1  have increased levels of Cdc25A and develop hepatic cysts).

2  intragenic, somatic mutations are common in hepatic cysts.

3 ominant polycystic kidney disease, developed hepatic cysts.

4 95%, with internal septations present in the hepatic cysts.

5 recognized and treated differently than most hepatic cysts.

6 at somatic loss of Pkd1 results in renal and hepatic cysts.

7                                              Hepatic cysts and bile duct proliferation, characteristi

8  Additionally, DEN(N2ICD) mice develop large hepatic cysts, dysplasia of the biliary epithelium, and

9                     More prominent in women, hepatic cysts emerge after the onset of puberty and dram

10 garding mechanism, the epithelium that lines hepatic cysts exhibited markedly higher levels of phosph

11 ue of somatostatin known to inhibit cAMP) in hepatic cyst growth using an in vitro model of cystogene

12  would be positioned to modulate the rate of hepatic cyst growth.

13                      Volumes of total liver, hepatic cysts, hepatic parenchyma, and total kidney were

14 stic kidney disease (ADPKD) increases liver, hepatic cyst, or kidney volume.

15                                              Hepatic cysts were lined by single or multiple layers of

16 otopic liver transplantation for symptomatic hepatic cysts with excellent long-term results and minim