ライフサイエンスコーパス: hepatic damage

1 are potential markers for Geniposide-induced hepatic damage.

2 patients with HCC in NCL lacked any sign of hepatic damage.

3 indicators of ongoing viral replication and hepatic damage.

4 nocytes in triggering liver inflammation and hepatic damage.

5 e detrimental host inflammatory response and hepatic damage.

6 ng their potential as a sensitive measure of hepatic damage.

7 disease because of the nature of preexisting hepatic damage.

8 ion plays a critical role in immune-mediated hepatic damage.

9 ups, which correlated with a lower degree of hepatic damage.

10 n of CXCR3(+) lymphocytes into the liver and hepatic damage.

11 identify candidate genes that contribute to hepatic damage.

12 a cytokine environment conducive to limited hepatic damage.

13 mice against endotoxin- and ischemia-induced hepatic damage.

14 lalanine, or tyrosine) resulted in renal and hepatic damage.

15 ansaminase (ALT) levels at 6 hours confirmed hepatic damage.

16 contribution to PMN and platelet adhesion in hepatic damage.

17 of lethally injected mice, indicating severe hepatic damage.

18 less Lm burden in liver and spleen, and less hepatic damage 3 days postinfection.

19 ion, PAI-1 protein is a negative effector of hepatic damage after HS-R through its influence on class

20 r instance, older organisms showed extensive hepatic damage, along with increased morbidity and morta

21 s, could play a key role in the variation in hepatic damage and be the target of the modifiers.

22 Excess iron is associated with hepatic damage and diabetes in humans, although the deta

23 ction, pronounced weight loss and markers of hepatic damage and disease were observed exclusively in

24 e hepatoprotective feature of STAT3 prevents hepatic damage and fibrosis under the condition of persi

25 vels positively correlate with the degree of hepatic damage and serum TNF-alpha, MIP-1alpha, and MIP-

26 Moreover, AM/AMBP-1 significantly attenuated hepatic damage and the elevation of plasma lactate, and

27 over, administration of AM/AMBP-1 attenuated hepatic damage and the increase in plasma lactate and pr

28 he absence of IL-12p40 induction and serious hepatic damage are involved in the death of IRF-1-/- mic

29 Depletion of Kupffer cells attenuated hepatic damage as seen by decreases of 53% (p < 0.05) in

30 Overall hepatic damage, assessed as increased expression of live

31 polysaccharide (LPS)-induced endotoxemia and hepatic damage associated with decreased proinflammatory

32 a1-antitrypsin Z (ATZ) in hepatocytes causes hepatic damage by a gain-of-function, "proteotoxic" mech

33 distinct layers of defense to prevent overt hepatic damage by bile acids during cholestasis.

34 A thermal injury, however, causes hepatic damage by inducing hepatic edema, fatty infiltra

35 We examined endothelial and hepatic damage by serologic or tissue studies and assess

36 bility of IRF-1-/- and ICSBP-/- mice; and 3) hepatic damage caused by Brucella virulence contributes

37 expression by combining 2-AAF with selective hepatic damage caused by either carbon tetrachloride (CC

38 IL-10 is known to suppress the extent of hepatic damage caused by parasite ova during natural inf

39 ld be carefully evaluated for progression of hepatic damage during cold storage and transport.

40 Hepatic damage following ischemia-reperfusion injury inv

41 Immune-mediated hepatic damage has been demonstrated in the pathogenesis

42 However, most patients with chronic hepatic damage have cirrhosis and fibrosis, which limit

43 then pass through the liver, contributing to hepatic damage, impaired microbial clearance, and impair

44 Indeed, TNF-alpha induced similar hepatic damage in both mice with hepatocyte-specific JNK

45 miologic data suggest that coffee may reduce hepatic damage in chronic liver disease.

46 ave applied this approach for indications of hepatic damage in experimental toxicity studies.

47 esistance to lamivudine may result in severe hepatic damage in immunocompromised patients.

48 h1 response and conferred protection against hepatic damage induced by F. hepatica infection.

49 Hepatic damage, microcirculation, regeneration, and vasc

50 ed oxidative stress and end-organ (renal and hepatic) damage, not to refractory hypotension.

51 causes a range of toxic responses, including hepatic damage, steatohepatitis, and a lethal wasting sy

52 ing high levels of FasL on the initiation of hepatic damage through analysis of chemokine and chemoki

53 Intoxication exacerbates postburn hepatic damage through p38-dependent interleukin-6 produ

54 involved in promoting liver inflammation and hepatic damage through the induction of chemokines.

55 This mitigation of hepatic damage was associated with a 54% decrease (p < 0

56 Hepatic damage was induced by migrating newborn larvae.

57 Hepatic damage was most marked in patients with the Cys2

58 evaluate the degree of primary and secondary hepatic damage, we generated a transgenic mouse with liv

59 pool can be instilled by BS feeding, without hepatic damage, which makes Hrn mice an attractive model

60 evalence of exposure to factors potentiating hepatic damage with acute hepatitis B contributed to the