ライフサイエンスコーパス: hepatic enzyme

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1 rmore, GBR diet can also reduce the level of hepatic enzymes.

2 epatocellular damage and elevation of plasma hepatic enzymes.

3 llular damage as revealed by serum levels of hepatic enzymes.

4 erlying sex differences in the expression of hepatic enzymes active in the metabolism of drugs, stero

5 ed by comparing results from measurements of hepatic enzyme activities in mice fed either deficient o

6 astric for 4 days), whereas the elevation of hepatic enzyme activities was less pronounced.

7 prague Dawley rats inhibited the increase in hepatic enzyme activity and mRNA levels of CYP1A1, 1A2,

8 disruption has pleiotropic implications for hepatic enzyme activity, lipid processing, nuclear recep

9 resent study was to examine whether elevated hepatic enzymes (alanine aminotransferase [ALT], asparta

10 of lipid peroxidation levels and activity of hepatic enzymes (alanine transferase, ALT and aspartate

11 lbutyrate increases the proportion of active hepatic enzyme and unphosphorylated form over the inacti

12 ic grade 3 to 4 toxicity, including elevated hepatic enzymes and hyperbilirubinemia, was less common.

13 In patients on each diet, levels of hepatic enzymes and markers of inflammation decreased, i

14 rbital, and primidone are potent inducers of hepatic enzymes, and decrease the plasma concentration o

15 of atherosclerosis, although the CE from the hepatic enzyme appeared to promote more atherosclerosis

16 ta remains controversial, although screening hepatic enzymes are recommended in the evaluation of non

17 ncy of PDA on ARG2 rather than the principal hepatic enzyme ARG1 opens a therapeutic window for obesi

18 asting total cholesterol, uric acid, and the hepatic enzymes aspartate transaminase and alanine trans

19 r plasma concentrations of the intracellular hepatic enzyme, aspartate aminotransferase.

20 es, plasma proteins, transporters, and other hepatic enzymes at levels equal to adult liver tissue.

21 lirubinemia resulting from deficiency of the hepatic enzyme bilirubin-UDP-glucuronosyltransferase.

22 erload disease, weakness, fatigue, increased hepatic enzyme concentrations, right upper quadrant pain

23 The NH2-terminal peptide of hepatic enzyme contained 18 amino acids not found in enz

24 PV are both substrates and inhibitors of the hepatic enzyme, cytochrome P450 3A, and the drug transpo

25 tein kinase C (aPKC) in liver and muscle and hepatic enzyme expression in mice consuming a moderate h

26 ostane receptor (hCAR) and its regulation of hepatic enzyme expression.

27 The hepatic enzyme glycine N-methyltransferase (GNMT) plays

28 DLTs comprised elevated hepatic enzymes, hypophosphatemia, and thrombocytopenia.

29 terin (BH4) is an important cofactor for two hepatic enzymes, inducible nitric oxide synthase (iNOS)

30 As has been observed previously, hepatic enzyme induction with PB or betaNF shifted the l

31 genetic polymorphisms encoding CYP2C19, the hepatic enzyme involved in biotransformation of clopidog

32 These data suggest that UGT2B10 is the major hepatic enzyme involved in nicotine/cotinine glucuronida

33 ochrome P450 1A2 (CYP1A2) is a predominantly hepatic enzyme known to be important in the metabolism o

34 dverse events were anemia (65%) and elevated hepatic enzyme levels (49%).

35 During the initial 24 weeks, elevated hepatic enzyme levels (P<.001), gastrointestinal tract c

36 en suspected in patients with abnormal serum hepatic enzyme levels and obstruction of the biliary sys

37 most pronounced in individuals with reduced hepatic enzyme levels, as occurs in approximately 10% of

38 Recipient survival rates, serum hepatic enzyme levels, coagulation, and metabolic parame

39 d pressure, hepatic artery blood flow, serum hepatic enzyme levels, or in weight compared with contro

40 t from BMI in predicting increased levels of hepatic enzymes, likely as a result of unrecognized fatt

41 e encoding MAT(alpha)1, the subunit of major hepatic enzymes MAT I ([alpha1]4) and III([alpha1]2).

42 this condition, but replacement of deficient hepatic enzymes might restore excretion of porphyrin pre

43 clerosis of the aorta, and upregulation of a hepatic enzyme of lipoprotein secretion, stearoyl coenzy

44 he identity, function, and regulation of the hepatic enzymes potentially involved in catalyzing the h

45 ion combined to control some, but not other, hepatic enzyme programs.

46 r weight gain and increases in the levels of hepatic enzymes, prolactin, fasting glucose, fasting tot

47 rosmolar rats were observed to have elevated hepatic enzyme release and decreased bile production com

48 tophan 2,3-dioxygenase (TDO) is an unrelated hepatic enzyme that also degrades tryptophan along the k

49 I iodothyronine 5'-deiodinase (5' D-I), the hepatic enzyme that converts thyroxine to T3 and that is

50 hosphate synthetase I (CPSI) is an essential hepatic enzyme that initiates the urea cycle.

51 fluxes and changes in the gene expression of hepatic enzymes that closely resemble those of fasting.

52 iciency on the expressions and activities of hepatic enzymes that regulate transmethylation reactions

53 ctor aryl hydrocarbon receptor (AhR) and the hepatic enzyme tryptophan 2,3-dioxygenase, which provide

54 A nephropathy was determined by blocking the hepatic enzyme uricase with oxonic acid (CSA-OA).

55 hat regulation by environmental compounds of hepatic enzymes via CAR and PXR may have impact on the m

56 Twelve patients demonstrated an increase in hepatic enzymes without jaundice during the pregnancy.

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