ライフサイエンスコーパス: hepatic microsome

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1 esters as determined in incubations with rat hepatic microsomes.

2 d 6-hydroxy-2-nitropyrene from 2-NP in human hepatic microsomes.

3 mers were more metabolically stable in human hepatic microsomes.

4 Incubation of 17AAG with human hepatic microsomes and cyclohexene oxide, a known inhibi

5 ssentially similar to those found with human hepatic microsomes and in human cells.

6 and vandetanib) were characterized by using hepatic microsomes and recombinant proteins.

7 y the same metabolites as produced by murine hepatic microsomes, but the 2-min metabolite was the maj

8 L1 preadipocyte cells, TBMEHP inhibited rat hepatic microsome deiodinase activity and was an agonist

9 effect on CCl4-induced lipid peroxidation of hepatic microsomes, exceeds that produced by alpha-tocop

10 The recovery of sEH inhibitors from rat hepatic microsomes, fortified at levels of 50, 100, and

11 reaction for CYP3A4) was reduced by >50% in hepatic microsomes from CYP3A4-HBN mice compared with co

12 tro glucuronidation of SN-38 was screened in hepatic microsomes from normal rats (n = 4), normal huma

13 le epoxide hydrolase (sEH) inhibitors in rat hepatic microsomes is described.

14 e formation of the HMM bands was observed in hepatic microsomes isolated from rats treated 1 week or

15 With rat hepatic microsomes or purified CPR, the presence of NADP

16 Metabolism studies using hepatic microsomes suggest that this procedure would be

17 ected in rat, hamster, dog, monkey, or human hepatic microsomes, suggesting the lack of oral toxicity

18 of retinol metabolism have been described in hepatic microsomes that involve, in part, cytochrome P45

19 shown to be specific in human plasma and rat hepatic microsomes, was further combined with the SRM tr

20 Human hepatic microsomes were competent in metabolizing all th

21 on and butanol extraction, we found that all hepatic microsomes were competent to activate 3-NBA.

22 Hepatic microsomes were loaded with 45Ca2+, and superfus

23 Hepatic microsomes were prepared by standard sucrose den

24 12 was rapidly metabolized by hepatic microsomes, which supports its topical use.

25 sized sEH inhibitors were extracted from rat hepatic microsomes with ethyl acetate and were determine

26 tification of residual sEH inhibitors in rat hepatic microsomes without interference.

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