ライフサイエンスコーパス: hepatic parenchyma

1 th in the portal triad that effaced adjacent hepatic parenchyma.

2 rom the biliary tree and develops within the hepatic parenchyma.

3 tructure, which shows similarities to normal hepatic parenchyma.

4 hal insult to tumors while preserving normal hepatic parenchyma.

5 of amastigotes from foci of infection in the hepatic parenchyma.

6 ns, angiogenesis, and regeneration of normal hepatic parenchyma.

7 was no collateral damage to the neighboring hepatic parenchyma.

8 oth in portal regions and extending into the hepatic parenchyma.

9 ccompanying enhancement abnormalities of the hepatic parenchyma.

10 er the risk of toxic effects of drugs on the hepatic parenchyma.

11 e requirement for the formation of an active hepatic parenchyma.

12 foci of increased vascularization within the hepatic parenchyma.

13 neum (11 sites), lymph nodes (10 sites), and hepatic parenchyma.

14 negligible inflammation/necrosis within the hepatic parenchyma.

15 us activated Kupffer cells were noted in the hepatic parenchyma.

16 enous inflow block with hypoperfusion of the hepatic parenchyma.

17 the number of neutrophils sequestered in the hepatic parenchyma.

18 te out of the vessels and integrate into the hepatic parenchyma.

19 ounding the bile ductules and throughout the hepatic parenchyma.

20 r cells, which are rarely observed in normal hepatic parenchyma.

21 inflammation and the pathogenic processes in hepatic parenchyma.

22 suggested the infiltration of HSCs into the hepatic parenchyma after stem cell mobilization.

23 der of the tumor nodule from the surrounding hepatic parenchyma) also was measured.

24 ultifocal accumulations of leukocytes in the hepatic parenchyma and around portal tracts and central

25 inflammation leads to fibrosis involving the hepatic parenchyma and biliary tree, which can lead to c

26 lation, with less neutrophils present in the hepatic parenchyma and far more neutrophils adherent to

27 xpressing monocytes and Kupffer cells in the hepatic parenchyma and perivascular sites and absence of

28 teract with HSCs on their migration into the hepatic parenchyma, and a depletion or deactivation of H

29 ivo recruitment of NK cells to pulmonary and hepatic parenchyma, and s.c. tumor after treatment of mi

30 Volumes of total liver, hepatic cysts, hepatic parenchyma, and total kidney were measured by a

31 se the numbers of ECFC persisting within the hepatic parenchyma are needed in order to enhance ECFC t

32 profiles are associated with changes in the hepatic parenchyma as reflected in increased scores for

33 chemokine induction was infiltration of the hepatic parenchyma by neutrophils.

34 c +/- 3 (P < .0001 and P < .01) and those in hepatic parenchyma in healthy volunteers (745 msec +/- 6

35 y liver functions and can integrate into the hepatic parenchyma in vivo.

36 ed hepatocytes is essential for a functional hepatic parenchyma, including genes encoding several apo

37 ed the extent of the biliary tree within the hepatic parenchyma, including identification of the cana

38 auses of oval cell proliferation in that the hepatic parenchyma is relatively unaffected.

39 ere clinically equivocal (myocardium, lungs, hepatic parenchyma, jejunum, and renal cortex/medulla) a

40 s characterized by cystic replacement of the hepatic parenchyma, leading to hepatic dysfunction, port

41 hepatocellular tumors was identified in the hepatic parenchyma of A/JCr mice.

42 zing lesion was progressive and involved the hepatic parenchyma, portal triads, and intralobular venu

43 ously shown that the microenvironment of the hepatic parenchyma regulates hepatic tumor formation fro

44 ematoma was punctured to relieve pressure on hepatic parenchyma, retrieving 300 mL of blood.

45 Autoimmune hepatitis is a disease of the hepatic parenchyma that can present in acute or chronic

46 pancreatic islet autotransplantation in the hepatic parenchyma (the intrahepatic [IH] group, n = 9)

47 Recovery/regeneration of hepatic parenchyma was assessed by histologic assessment

48 Hepatic parenchyma was normal, whereas apoptosis and cel

49 stage 2 metastases mimicked the surrounding hepatic parenchyma, whereas stage 3 metastases exhibited

50 uminous subcapsular hematoma compressing the hepatic parenchyma, which appeared ischemic with a flatt

51 The thickness of hepatic parenchyma with intense perilesional enhancement

52 The thickness of hepatic parenchyma with intense perilesional enhancement

53 The mean percentage of hepatic parenchyma with nontumorous perfusion defects wa

54 Uniform necrosis of hepatic parenchyma within the cryolesion was confirmed.