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1 Ogenitor Cells (sprocs) play a vital role in hepatic regeneration.
2 before liver resection suffered from delayed hepatic regeneration.
3 is required for gammadeltaT cells to promote hepatic regeneration.
4 with regenerative growth factors to promote hepatic regeneration.
5 a specific inhibitor of Zn-HDAC activity) on hepatic regeneration.
6 jury, limit vascular inflammation, and boost hepatic regeneration.
7 ony-stimulating factor (G-CSF) could promote hepatic regeneration.
8 tosis resulted in significant suppression of hepatic regeneration.
9 systemic milieu that does not support normal hepatic regeneration.
10 inophen-induced injury, which contributes to hepatic regeneration.
11 mia/reperfusion but is also mitogenic during hepatic regeneration.
12 uld impair hepatocellular function and limit hepatic regeneration.
13 70), after two-thirds hepatectomy to prevent hepatic regeneration.
14 cumulation would be associated with impaired hepatic regeneration.
15 ide-1/glucagon receptor agonist, on NASH and hepatic regeneration.
16 MIP-2 doses after hepatic injury accelerate hepatic regeneration.
17 f CREB by phosphorylation that occurs during hepatic regeneration.
18 gave rise to donor-derived hematopoietic and hepatic regeneration.
19 liver injuries alter metabolism and initiate hepatic regeneration.
20 n in the rat has been useful in the study of hepatic regeneration.
21 ion of glucuronidation during the process of hepatic regeneration.
22 enol glucuronide formation was unaffected by hepatic regeneration 1, 2, and 5 days posthepatectomy wh
23 not the cellular alterations associated with hepatic regeneration affect the efficacy and toxicity of
24 ss the effects of undifferentiated UCMSCs in hepatic regeneration after an acute liver injury, we tra
30 nisms prevail: (1) cirrhosis associated with hepatic regeneration after tissue damage caused by hepat
31 f CD39 activity is associated with decreased hepatic regeneration and failure of vascular reconstitut
32 ighlights the role of activated HPCs in both hepatic regeneration and fibrosis during liver injury.
33 te that IL-6 can function acutely to improve hepatic regeneration and repair, but that more chronic e
36 ects of the signaling mechanisms involved in hepatic regeneration are under active investigation.
42 treated mice exhibited significantly reduced hepatic regeneration compared with controls, as assessed
43 vestigated the time course of changes during hepatic regeneration for Na, K-ATPase activity, lipid co
46 ent may be useful in assessing the degree of hepatic regeneration in patients with clinical liver dis
47 M-HSCs) have been shown to act as source for hepatic regeneration in rodent models; however, their ab
49 emic endotoxemia, which normally accompanies hepatic regeneration induced by PH, also decreased clear
52 ectomy rat model to elucidate the effects of hepatic regeneration on the various components of the mi
54 partial hepatectomy (PH) of 70% to stimulate hepatic regeneration showed decreased asialoglycoprotein
55 nvestigated the role of gammadeltaT cells in hepatic regeneration using mice with disruptions in Tcrd
58 to ischemia and hepatectomy showed impaired hepatic regeneration when compared with wild-type mice s
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