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1 Ogenitor Cells (sprocs) play a vital role in hepatic regeneration.
2 before liver resection suffered from delayed hepatic regeneration.
3 is required for gammadeltaT cells to promote hepatic regeneration.
4  with regenerative growth factors to promote hepatic regeneration.
5 a specific inhibitor of Zn-HDAC activity) on hepatic regeneration.
6 jury, limit vascular inflammation, and boost hepatic regeneration.
7 ony-stimulating factor (G-CSF) could promote hepatic regeneration.
8 tosis resulted in significant suppression of hepatic regeneration.
9 systemic milieu that does not support normal hepatic regeneration.
10 inophen-induced injury, which contributes to hepatic regeneration.
11 mia/reperfusion but is also mitogenic during hepatic regeneration.
12 uld impair hepatocellular function and limit hepatic regeneration.
13 70), after two-thirds hepatectomy to prevent hepatic regeneration.
14 cumulation would be associated with impaired hepatic regeneration.
15 ide-1/glucagon receptor agonist, on NASH and hepatic regeneration.
16  MIP-2 doses after hepatic injury accelerate hepatic regeneration.
17 f CREB by phosphorylation that occurs during hepatic regeneration.
18 gave rise to donor-derived hematopoietic and hepatic regeneration.
19 liver injuries alter metabolism and initiate hepatic regeneration.
20 n in the rat has been useful in the study of hepatic regeneration.
21 ion of glucuronidation during the process of hepatic regeneration.
22 enol glucuronide formation was unaffected by hepatic regeneration 1, 2, and 5 days posthepatectomy wh
23 not the cellular alterations associated with hepatic regeneration affect the efficacy and toxicity of
24 ss the effects of undifferentiated UCMSCs in hepatic regeneration after an acute liver injury, we tra
25 F) and epidermal growth factor (EGF) mediate hepatic regeneration after liver injury.
26  systemic inflammatory reactions may trigger hepatic regeneration after major tissue loss.
27 lar matrix remodeling events associated with hepatic regeneration after partial hepatectomy.
28           In conclusion, the early stages of hepatic regeneration after resection provide an environm
29 g protein (ENA-78), a CXC chemokine, retards hepatic regeneration after resection.
30 nisms prevail: (1) cirrhosis associated with hepatic regeneration after tissue damage caused by hepat
31 f CD39 activity is associated with decreased hepatic regeneration and failure of vascular reconstitut
32 ighlights the role of activated HPCs in both hepatic regeneration and fibrosis during liver injury.
33 te that IL-6 can function acutely to improve hepatic regeneration and repair, but that more chronic e
34                  Analysis of liver function, hepatic regeneration, and comprehensive genomic and meta
35 th elevated portal venous pressure, impaired hepatic regeneration, and postoperative morbidity.
36 ects of the signaling mechanisms involved in hepatic regeneration are under active investigation.
37                        Therefore, we studied hepatic regeneration at day 1, day 2, and day 4 in a mod
38                             bFGF accelerated hepatic regeneration by 42%, 19%, 16%, and 16% on postop
39                            TNP-470 inhibited hepatic regeneration by 46%, 74%, 67%, and 64% on postop
40         We have shown that G-CSF facilitates hepatic regeneration by increasing the migration of BM-d
41                   gammadeltaT cells regulate hepatic regeneration by producing IL-22 and IL-17, which
42 treated mice exhibited significantly reduced hepatic regeneration compared with controls, as assessed
43 vestigated the time course of changes during hepatic regeneration for Na, K-ATPase activity, lipid co
44 c liver failure, and erythropoietin promoted hepatic regeneration in animal studies.
45  Interleukin 6 (IL-6) is a potent inducer of hepatic regeneration in normal and ischemic livers.
46 ent may be useful in assessing the degree of hepatic regeneration in patients with clinical liver dis
47 M-HSCs) have been shown to act as source for hepatic regeneration in rodent models; however, their ab
48   These results indicate major impairment of hepatic regeneration in steatotic livers.
49 emic endotoxemia, which normally accompanies hepatic regeneration induced by PH, also decreased clear
50                                              Hepatic regeneration is a heterogeneous phenomenon invol
51                                       During hepatic regeneration, MMP-9 was detected in the urine at
52 ectomy rat model to elucidate the effects of hepatic regeneration on the various components of the mi
53  and enhanced the proliferative capacity and hepatic regeneration ratio in mice on the MCD diet.
54 partial hepatectomy (PH) of 70% to stimulate hepatic regeneration showed decreased asialoglycoprotein
55 nvestigated the role of gammadeltaT cells in hepatic regeneration using mice with disruptions in Tcrd
56                                     Although hepatic regeneration was associated with a reduced chole
57                                              Hepatic regeneration was determined by liver weight/tota
58  to ischemia and hepatectomy showed impaired hepatic regeneration when compared with wild-type mice s
59 lts provide a therapeutic roadmap to achieve hepatic regeneration without provoking fibrosis.

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