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1 FF >/= 5% after exclusion of other causes of hepatic steatosis).
2 hat cause lipid accumulation in hepatocytes (hepatic steatosis).
3 asatinib and quercetin (D+Q) reduces overall hepatic steatosis.
4 ein B (apoB) lipoproteins but can also cause hepatic steatosis.
5 temic glucose homeostasis, inflammation, and hepatic steatosis.
6 downregulation may contribute to HCV-induced hepatic steatosis.
7 hepatic VLDL secretion that protects against hepatic steatosis.
8 xert anti-inflammatory effects and alleviate hepatic steatosis.
9 rdiac structure, energetics, and cardiac and hepatic steatosis.
10 pression of ER chaperone proteins alleviates hepatic steatosis.
11 re protected against HFD-induced obesity and hepatic steatosis.
12 es improved glucose tolerance and attenuated hepatic steatosis.
13 PPARgamma antagonists may be useful to treat hepatic steatosis.
14  obesity-associated inflammation and improve hepatic steatosis.
15 glucose intolerance, insulin resistance, and hepatic steatosis.
16 , metabolic profile, insulin resistance, and hepatic steatosis.
17 athway reversed the "Gli-code" and mitigated hepatic steatosis.
18  tissue, decreased gluconeogenesis, and less hepatic steatosis.
19 de therapeutic strategies in obesity-induced hepatic steatosis.
20 elopment of obesity, insulin resistance, and hepatic steatosis.
21 ulting in hyperglycemia, hyperlipidemia, and hepatic steatosis.
22 ulin resistance, diabetes, dyslipidemia, and hepatic steatosis.
23 or signatures of coronary artery disease and hepatic steatosis.
24                         HIV patients develop hepatic steatosis.
25 d oxidation and lipolysis, and thus promoted hepatic steatosis.
26  with protection from insulin resistance and hepatic steatosis.
27 o antibiotic-treated mice fed a HFD reversed hepatic steatosis.
28  brown adipose mass; and were protected from hepatic steatosis.
29 FLD compared to BMI-matched subjects without hepatic steatosis.
30 uracy of MRI-PDFF as an imaging biomarker of hepatic steatosis.
31 by the HFD and protected against HFD-induced hepatic steatosis.
32 stologic steatosis grading for assessment of hepatic steatosis.
33 P/iDTR) mice and prevented the resolution of hepatic steatosis.
34 ic and environmental interactions underlying hepatic steatosis.
35 associated with lipodystrophy, diabetes, and hepatic steatosis.
36 mately 40-50% of obese adults do not develop hepatic steatosis.
37 relative to biopsy for the quantification of hepatic steatosis.
38 hole body fuel metabolism and a reduction in hepatic steatosis.
39 creased, and the mice were protected against hepatic steatosis.
40 een identified as a novel genetic marker for hepatic steatosis.
41 ularly among alcohol drinkers and those with hepatic steatosis.
42 2-/- mice are also resistant to diet-induced hepatic steatosis.
43  increased insulin sensitivity and decreased hepatic steatosis.
44  represents a novel mechanism of HCV-induced hepatic steatosis.
45  0.82; 95% CI: 0.68, 0.98; P = 0.02) but not hepatic steatosis.
46 ssion was also independently associated with hepatic steatosis.
47 pression of adiponectin, but did not prevent hepatic steatosis.
48 ed impairment of mitochondrial oxidation and hepatic steatosis.
49 abolic disorders, including dyslipidemia and hepatic steatosis.
50 iated with blood levels of liver enzymes and hepatic steatosis.
51 o completed liver ultrasound examination for hepatic steatosis.
52 sity, insulin resistance, hyperlipidemia and hepatic steatosis.
53 intestinal hypoxia-inducible factor (HIF) in hepatic steatosis.
54 nd fibrosis that are thought to emanate from hepatic steatosis.
55 her analyzed to evaluate their relation with hepatic steatosis.
56 sponse to feeding, exacerbating diet-induced hepatic steatosis.
57 ronger among alcohol drinkers and those with hepatic steatosis.
58 lop glucose intolerance and high fat-induced hepatic steatosis.
59 CI: 25, 49) were higher in participants with hepatic steatosis.
60 glucose homeostasis and prevents obesity and hepatic steatosis.
61 lipolysis and prevents high-fat diet-induced hepatic steatosis.
62 ein had significant shared gene effects with hepatic steatosis.
63 esses adipose tissue lipolysis, ameliorating hepatic steatosis.
64        Adult cnr2 mutants are susceptible to hepatic steatosis.
65  obesity-associated abnormalities, including hepatic steatosis.
66 tor finasteride induced hyperinsulinemia and hepatic steatosis (10.6 +/- 1.2 vs. 7.0 +/- 1.0 mumol g(
67 ights, HCLD feeding: (1) induced more severe hepatic steatosis; (2) up-regulated hepatic expression o
68 epatic steatosis than in adolescents without hepatic steatosis (41% compared with 18%; P = 0.04).
69  mice were protected against fasting-induced hepatic steatosis (a model of enhanced exogenous FA deli
70                                              Hepatic steatosis, a common finding in living liver dono
71 echanisms of inhalation exposure to PM2.5 on hepatic steatosis, a precursor or manifestation of metab
72 passes a range of conditions associated with hepatic steatosis, aberrant accumulation of fat in hepat
73 , HFD-fed AdSod2 KO mice were protected from hepatic steatosis, adipose tissue inflammation, and gluc
74 m the glucose intolerance, hyperinsulinemia, hepatic steatosis, adiposity, hypertension, myopathy, an
75  high-fat diet, as demonstrated by decreased hepatic steatosis, adiposity, plasmatic and hepatic trig
76 e proportion of patients without significant hepatic steatosis after 48 weeks was greater for those w
77 sociations between ambient air pollution and hepatic steatosis among 2,513 participants from the Fram
78 hing efavirenz (EFV) to raltegravir (RAL) on hepatic steatosis among HIV-infected patients with nonal
79                      Dietary determinants of hepatic steatosis, an important precursor for nonalcohol
80 e Hedgehog pathway play an important role in hepatic steatosis and beyond.
81 esterol metabolism, triglyceride production, hepatic steatosis and biliary cholestasis.
82 roton density fat fraction (PDFF) in grading hepatic steatosis and change in hepatic steatosis in adu
83 ific 11beta-HSD1 KO mice were protected from hepatic steatosis and circulating fatty acid excess.
84 tions in obesity and type 2 diabetes include hepatic steatosis and disruption of glucose-glycogen hom
85 demonstrates that cellular senescence drives hepatic steatosis and elimination of senescent cells may
86      These improvements also lead to reduced hepatic steatosis and enhanced hepatic insulin sensitivi
87 udy examined the shared gene effects between hepatic steatosis and fibrosis and their associations wi
88                      Studies have shown that hepatic steatosis and fibrosis are heritable, but whethe
89                                              Hepatic steatosis and fibrosis had a highly significant
90 bing exogenous hepatic FA use modulates both hepatic steatosis and fibrosis in the setting of hepatic
91  had transient elastography (TE) to evaluate hepatic steatosis and fibrosis.
92 d Fgf21 expression and reduced lipid-induced hepatic steatosis and glucose intolerance, but these eff
93      A study of twins provides evidence that hepatic steatosis and hepatic fibrosis are heritable tra
94 BH lysine 294 mutation in the liver leads to hepatic steatosis and hyperlipidemia in animals under pr
95 ced WAT lipolysis, glucocorticoid- initiated hepatic steatosis and hypertriglyceridemia were improved
96 de novo lipogenesis and prevent the onset of hepatic steatosis and hypertriglyceridemia.
97 FGF21 administration reduced alcohol-induced hepatic steatosis and inflammation in WT mice.
98  diet), CX3CR1 protected mice from excessive hepatic steatosis and inflammation, as well as systemic
99  improved insulin sensitivity, and decreased hepatic steatosis and inflammation.
100 rom peripheral lipolysis, showed exacerbated hepatic steatosis and inflammation.
101 ween diet, bile acids, sex, and dysbiosis in hepatic steatosis and inflammation.
102 ric HCLD feeding induced greater severity in hepatic steatosis and inflammatory response than HFLD fe
103                                              Hepatic steatosis and insulin resistance are among the m
104 t absorption and are protected from obesity, hepatic steatosis and insulin resistance associated with
105 es suggest that DPP-4 inhibition ameliorates hepatic steatosis and insulin resistance by suppressing
106             On high-fat diet, JAK2L mice had hepatic steatosis and insulin resistance despite protect
107                           The development of hepatic steatosis and insulin resistance in CBA/J mice o
108  also efficacious in preventing and treating hepatic steatosis and insulin resistance induced by a hi
109 s an attractive target for the management of hepatic steatosis and insulin resistance.
110 0-mediated adipose tissue lipolysis promotes hepatic steatosis and insulin resistance.
111  or feeding them a high-fat diet, results in hepatic steatosis and insulin resistance.
112 in green coffee beans, prevents diet-induced hepatic steatosis and insulin resistance.
113  are protected against high-fat diet-induced hepatic steatosis and insulin resistance.
114 of SYK activation diminished alcohol-induced hepatic steatosis and interferon regulatory factor 3-med
115 ining 3 (PNPLA3) is robustly associated with hepatic steatosis and its progression to steatohepatitis
116 patic fatty acid esterification, ameliorates hepatic steatosis and lipid-induced insulin resistance.
117       Mice with high-fat diet-induced simple hepatic steatosis and lipid-loaded Huh7 cells had reduce
118  FGF21 depletion exacerbated alcohol-induced hepatic steatosis and liver injury, which was associated
119 terms of differences in insulin sensitivity, hepatic steatosis and metabolic outcomes in participants
120 BR may be a new therapeutic strategy against hepatic steatosis and non-alcoholic steatohepatitis.
121 nsumption have been implicated as a cause of hepatic steatosis and obesity but the effect of meal pat
122 ion of Hif2a, in which high-fat-diet-induced hepatic steatosis and obesity were substantially lower a
123 e Western diet, independently contributes to hepatic steatosis and obesity.
124    These changes were accompanied by reduced hepatic steatosis and oxidative stress in adipose tissue
125     The DPP-4 inhibitor prevented WD-induced hepatic steatosis and reduced hepatic insulin resistance
126 IO mice, which was linked to amelioration of hepatic steatosis and reduced inflammation in liver and
127 nd liver injury with significantly increased hepatic steatosis and serum AST level as well as hepatic
128                      On chow, JAK2L mice had hepatic steatosis and severe whole-body and hepatic insu
129 om MCD formulas developed varying degrees of hepatic steatosis and steatohepatitis, in the order star
130 tivation of FGF21 by AHR contributed to both hepatic steatosis and systemic insulin hypersensitivity,
131 ion protects mice from high-fat diet-induced hepatic steatosis and that mutation of the SLC13A5 ortho
132 et (HFD) and high-carbohydrate diet (HCD) on hepatic steatosis and the underlying mechanisms, especia
133                                              Hepatic steatosis and VAT associated with significant ch
134 s of sugar and fat intake as determinants of hepatic steatosis and visceral obesity in overweight ado
135 duced adipose tissue inflammation as well as hepatic steatosis and was more effective at correcting h
136               Long-term GC treatment induces hepatic steatosis and weight gain.
137 ed hypertriglyceridemia, insulin resistance, hepatic steatosis, and diabetes.
138 posity, weight gain, hormonal dysregulation, hepatic steatosis, and dyslipidemia was reduced or rever
139 on against cardiac hypertrophy and fibrosis, hepatic steatosis, and gastrointestinal barrier dysfunct
140 , protection against diet-induced adiposity, hepatic steatosis, and glucose intolerance.
141 gen XVIII, developed reduced bulk-adiposity, hepatic steatosis, and hypertriglyceridemia.
142 FD-induced adipose and hepatic inflammation, hepatic steatosis, and insulin resistance.
143 yme, is increased in the livers of mice with hepatic steatosis, and knocking down Mogat1 improves glu
144 epr(db/db) HF mice developed marked obesity, hepatic steatosis, and more than 50% progressed to NASH
145 r's disease (AD), dermatological conditions, hepatic steatosis, and oncology.
146 and alanine aminotransferase concentrations; hepatic steatosis; and hepatic expression of IL-1alpha,
147  1 unit increase), diabetes (aOR: 1.56), and hepatic steatosis (aOR: 1.78) at the time of initial bio
148 sh that RBP4 expressed in adipocytes induces hepatic steatosis arising from primary effects occurring
149 ce were protected from high-fat diet-induced hepatic steatosis as well as from insulin resistance.
150 om insulin resistance, but are more prone to hepatic steatosis, as compared with their HFD-fed Ptpn6(
151 uctose-treated fish prevented development of hepatic steatosis, as did treatment of tunicamycin- or v
152 EFV to RAL showed decreases in the degree of hepatic steatosis, as measured by CAP, compared with tho
153 tly regulated and excessive lipolysis causes hepatic steatosis, as NEFA released from adipose tissue
154                 BBR treatment also decreased hepatic steatosis, as well as the expression of acetyl-C
155 sults in reductions in circulating IGF-1 and hepatic steatosis, associated with systemic insulin resi
156                                   Changes in hepatic steatosis at 48 weeks of follow-up over baseline
157 between magnesium intake and alcohol use and hepatic steatosis at baseline were not significant (P >
158                         NAFLD was defined as hepatic steatosis at nonenhanced CT (liver minus spleen
159 age, sex, and ethnicity, the heritability of hepatic steatosis (based on MRI-PDFF) was 0.52 (95% conf
160 ol (beta = -0.020; P = 7 x 10(-37)), greater hepatic steatosis (beta = 0.021; P = 3 x 10(-4)), higher
161 R of db/db mice reverted insulin resistance, hepatic steatosis, body weight and adiposity to those of
162 t promise for the quantitative assessment of hepatic steatosis but has not been validated in children
163 een reported to attenuate the development of hepatic steatosis, but the potential mechanisms remain p
164 ion of MED13 in mice conferred resistance to hepatic steatosis by activating a metabolic gene program
165  conclude that chronic inflammation precedes hepatic steatosis by disrupting the balance between fatt
166 report here that liver BVRA protects against hepatic steatosis by inhibiting glycogen synthase kinase
167 idly induces hepatic autophagy and abrogates hepatic steatosis by inhibiting hexose transport via the
168        Nrg4 gene transfer curbed HFD-induced hepatic steatosis by inhibiting lipogenesis and PPARgamm
169 activation play key roles in the response to hepatic steatosis by up-regulating apoA-IV and promoting
170  food allergies, type 1 and type 2 diabetes, hepatic steatosis, cardiovascular disease, and inflammat
171  & AIMS: Effective treatments are needed for hepatic steatosis characterized by accumulation of trigl
172 e had higher serum EtOH levels and developed hepatic steatosis characterized by micro- and macrovesic
173 density fat fraction (PDFF), a biomarker for hepatic steatosis, compared to histologic steatosis grad
174 e developed a multi-component classifier for hepatic steatosis comprised of phenotypic, genomic, and
175 agon-like peptide-1 (GLP-1) analogues reduce hepatic steatosis, concentrations of liver enzymes, and
176 R1 deficiency induces insulin resistance and hepatic steatosis, consistent with the intrahepatic accu
177 bidities (for example, diabetes mellitus and hepatic steatosis) contribute to approximately 2.5 milli
178                              The presence of hepatic steatosis correlated between monozygotic twins (
179 mination of liver samples from patients with hepatic steatosis demonstrated activation of Torc1 signa
180 mprovements in weight, glucose tolerance and hepatic steatosis despite differential effects on hepati
181  with chylomicron retention disorder develop hepatic steatosis, despite impaired intestinal fat malab
182 od why some, but not other, individuals with hepatic steatosis develop NASH.
183               Six weeks of WD feeding caused hepatic steatosis development as evidenced by the 2.25-f
184 , sub-chronic WD feeding appears to increase hepatic steatosis development over a 6-week period but o
185 C1 confers broad spectrum protection against hepatic steatosis development.
186 induced Zip14 KO mice show greater levels of hepatic steatosis due to higher expression of genes invo
187        CREBH-deficient mice developed severe hepatic steatosis due to increased adipose tissue lipoly
188 timulation contributes to the development of hepatic steatosis, dyslipidemia and hyperglycemia.
189 rodent diets with citrus flavonoids prevents hepatic steatosis, dyslipidemia, and insulin resistance
190 ubsequent metabolism have been implicated in hepatic steatosis, dyslipidemia, obesity, and insulin re
191 dividuals presented with a mild phenotype of hepatic steatosis, elevated aminotransferases and alkali
192 rogression of metabolic disorders, including hepatic steatosis, glucose intolerance, and inflammation
193 nction and ameliorates high-fat-diet-induced hepatic steatosis, glucose tolerance, and insulin resist
194 ex score >/=1.5, hepatic fibrosis >/=F3, and hepatic steatosis >/=S2 were independent factors associa
195 of hepatic fibrosis, and the heritability of hepatic steatosis has not been assessed systematically i
196 gnetic resonance imaging (MRI) biomarkers of hepatic steatosis have demonstrated tremendous promise f
197 aerobic capacity had no impact on WD-induced hepatic steatosis; however, rats bred for low aerobic ca
198                                              Hepatic steatosis (HS) is common in individuals with hep
199                          We examined whether hepatic steatosis (HS) on ultrasound and liver enzyme ac
200                       Primary endpoints were hepatic steatosis (HS; quantified by magnetic resonance
201 gain and associated comorbidities, including hepatic steatosis, hypercholesterolemia, and glucose int
202 uses obesity with visceral fat accumulation, hepatic steatosis, hyperleptinemia, hyperinsulinemia, an
203 ivation of Tm6sf2 in mice is associated with hepatic steatosis, hypocholesterolemia, and transaminiti
204 enuation parameter (CAP) in the diagnosis of hepatic steatosis in a series of overweight or obese chi
205 ) in grading hepatic steatosis and change in hepatic steatosis in adults with nonalcoholic steatohepa
206 de novo ceramide synthesis reduced PLIN2 and hepatic steatosis in alcohol-fed mice, but only de novo
207  improved lipid metabolism, and also reduced hepatic steatosis in diet-induced obese mice.
208 f obesity, insulin resistance, diabetes, and hepatic steatosis in diet-induced obese mice.
209 rately, forced PHLPP2 expression ameliorates hepatic steatosis in diet-induced obese mice.
210  Finally, E2f1 deletion completely abrogated hepatic steatosis in different murine models of nonalcoh
211             However, PM2.5 exposure relieved hepatic steatosis in high-fat diet-induced obese mice.
212 viral drugs with a lower potential to induce hepatic steatosis in human immunodeficiency virus (HIV)
213  We sought to determine the role of FGF21 in hepatic steatosis in mice exposed to chronic alcohol tre
214 NF6 in livers of adult C57Bl/6 mice leads to hepatic steatosis in mice fed normal laboratory chow.
215 ne, a natural compound that protects against hepatic steatosis in mice fed the high-fat diet, as a no
216  hyperinsulinemia, hypertriglyceridemia, and hepatic steatosis in mice.
217  and genetic obesity, insulin resistance and hepatic steatosis in mice.
218 estigated mechanisms by which SIRT1 controls hepatic steatosis in mice.
219  SLC7A11 was associated with reduced risk of hepatic steatosis in participants (odds ratio, 0.69; 95%
220 ht gain, liver damage and the development of hepatic steatosis in prediabetic mice while protecting a
221 weight, enhance glycemic control and reverse hepatic steatosis in relevant rodent models.
222 sition in livers of control mice, but severe hepatic steatosis in SIRT1 LKO mice.
223                    Both diets induced severe hepatic steatosis in the LTKO mice as compared to WT con
224  or diagnostic tests have been developed for hepatic steatosis in the setting of obesity.
225 ressed adipose tissue lipolysis and improved hepatic steatosis in these mice.
226                              We investigated hepatic steatosis in transgenic mice expressing the HIV-
227 ed for treating the increasing prevalence of hepatic steatosis in Western populations.
228 ly, Crtc1 deficient mice develop spontaneous hepatic steatosis in young age.
229  several high-confidence candidate genes for hepatic steatosis, including Gde1, a glycerophosphodiest
230 f lipid dysregulation that can contribute to hepatic steatosis, including reduced triglyceride secret
231 on of 18:2 species with complete reversal of hepatic steatosis, increased hepatic injury, and worsene
232 hermore, ApoE-/-ITCH-/- mice exhibit reduced hepatic steatosis, increased mitochondrial oxidative cap
233 CD36Tg mice showed unexpected attenuation of hepatic steatosis, increased very low-density lipoprotei
234 s indexes (BMI), fatty liver index (FLI) and hepatic steatosis index (HSI) was analyzed using the imp
235  reduced obesity development and ameliorated hepatic steatosis induced by both high-fat diet treatmen
236 ation of pNaKtide reduced obesity as well as hepatic steatosis, inflammation and fibrosis.
237 enchyma as reflected in increased scores for hepatic steatosis, inflammation, fibrosis and NASH.
238 atic Ppargamma, i.e. C3H/HeN, do not develop hepatic steatosis, insulin resistance, or dysglycemia on
239 lic disorder characterized by lipodystrophy, hepatic steatosis, insulin resistance, severe diabetes,
240  host of diseases including atherosclerosis, hepatic steatosis, insulin resistance, type 2 diabetes,
241 ssociated fatty liver and insulin resistance.Hepatic steatosis is a common disease closely associated
242                                              Hepatic steatosis is a frequent complication associated
243                                              Hepatic steatosis is associated with a greater intake of
244                                              Hepatic steatosis is caused by metabolic imbalances that
245                                              Hepatic steatosis is characterised by excessive triglyce
246                                              Hepatic steatosis is common in obesity and insulin resis
247                                              Hepatic steatosis is defined by the accumulation of lipi
248                          This indicates that hepatic steatosis is dissociated from insulin resistance
249                                              Hepatic steatosis is independent of androgens in rats.
250  of epigenetic changes in the development of hepatic steatosis is largely unknown.
251 bution of each hormone to the development of hepatic steatosis is unclear.
252 ssociated with markers of liver function and hepatic steatosis, laying the groundwork for future diag
253 9 +/- 103 vs. 313 +/- 66 ng mL(-1) min), and hepatic steatosis (liver triglycerides 136.1 +/- 17.0 vs
254 models and prevalence ratios for presence of hepatic steatosis (LPR </= 0.33) using generalized linea
255           Among the cohort, 20% (26/130) had hepatic steatosis (magnetic resonance imaging-proton den
256                                              Hepatic steatosis may be benign or progress to hepatocyt
257 n experimental animals and, while overcoming hepatic steatosis, may have significant gastrointestinal
258 nt rats including obesity, hyperinsulinemia, hepatic steatosis, nephropathy, and infertility.
259 t2 knockout mice are leaner and resistant to hepatic steatosis, obesity and insulin resistance under
260 eatment of dyslipidemia, insulin resistance, hepatic steatosis, obesity, and atherosclerosis.
261  represent an effective therapy for managing hepatic steatosis, obesity, and diabetes.
262 ge, which may be due to the manifestation of hepatic steatosis observed in the mice.
263               In the U.S. population, severe hepatic steatosis on ultrasound and liver enzyme elevati
264 ha-encoding gene Pik3r1 rescued diabetes and hepatic steatosis phenotypes of Kbtbd2(-/-) mice.
265 d catabolism, leads to AT atrophy and severe hepatic steatosis, phenotypes rescued by macrophage-spec
266                           HFD also increased hepatic steatosis, plasma alanine aminotransferase activ
267 al: -0.006, -0.001) and higher prevalence of hepatic steatosis (prevalence ratio = 1.16, 95% confiden
268 nditions, n3-PUFA exert beneficial effect on hepatic steatosis, regeneration and inflammatory insults
269                                              Hepatic steatosis renders liver more vulnerable to ische
270 this study had a high prevalence of elevated hepatic steatosis scores.
271   Carriers of the mutation had dyslipidemia, hepatic steatosis, systemic insulin resistance, and diab
272 sumption was more common in adolescents with hepatic steatosis than in adolescents without hepatic st
273 f livers revealed that asparaginase provoked hepatic steatosis that coincided with activation of anot
274             IL-1-type cytokines can regulate hepatic steatosis; the NLR family pyrin domain containin
275 inal HIF-2alpha could be a viable target for hepatic steatosis therapy.
276 bile acid composition and in fasting-induced hepatic steatosis through a novel mechanism involving ac
277  multiple loci that confer susceptibility to hepatic steatosis through diverse metabolic mechanisms.
278 -expression of PNPLA8 dramatically decreases hepatic steatosis through increased autophagy in hepatoc
279  reveal a novel function of SRA in promoting hepatic steatosis through repression of ATGL expression.
280 lic fatty liver diseases (NAFLD) ranges from hepatic steatosis to steatohepatitis and fibrosis.
281 ggesting a possible specific contribution of hepatic steatosis to subclinical vascular impairment.
282 topathologic features, ranging from isolated hepatic steatosis, to steatohepatitis with evidence of h
283        Thus, PLD1 plays an important role in hepatic steatosis via the regulation of autophagy.
284         Similarly, SCFA-induced reduction of hepatic steatosis was absent in mice lacking hepatic PPA
285                                              Hepatic steatosis was assessed at baseline and after 72
286                                              Hepatic steatosis was assessed with magnetic resonance i
287                                              Hepatic steatosis was effectively reduced only in ob/ob
288  mice (Mttp-LKO, i.e., double knockout mice) hepatic steatosis was greatly diminished and fibrosis pr
289 t of PM2.5 exposure on high-fat diet-induced hepatic steatosis was mediated through PM2.5-induced hep
290                                              Hepatic steatosis was quantified noninvasively by MRI an
291 scular disease, patchy cardiac fibrosis, and hepatic steatosis, was noted in the other organs.
292 ory to weight gain, glucose intolerance, and hepatic steatosis when challenged with high-fat diet.
293  accurately classifies grades and changes in hepatic steatosis when histologic analysis of biopsies i
294 g and fasting blood glucose, weight gain and hepatic steatosis while protecting against diabetic neur
295 iated with gastrointestinal side effects and hepatic steatosis, whose long-term sequelae remain uncle
296  mass, fat content, glucose intolerance, and hepatic steatosis with advancing age.
297 cient mice fed a high fat diet had increased hepatic steatosis with significantly higher insulin resi
298 n patients with biopsy-proven NASH (n = 13), hepatic steatosis without NASH (n = 11), and healthy con
299   DNP improved glucose tolerance and reduced hepatic steatosis without observed toxicity.
300  triglyceride transfer protein (Mttp) causes hepatic steatosis, yet the risks for developing hepatic

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