ライフサイエンスコーパス: hepatic stellate cell

1 ibrogenesis is limited compared with that of hepatic stellate cells.

2 ocytes activates fibrosis-related markers in hepatic stellate cells.

3 l, the L4F nanoparticles were incubated with hepatic stellate cells.

4 patocytes, sinusoidal endothelial cells, and hepatic stellate cells.

5 ctivation of neutrophils, Kupffer cells, and hepatic stellate cells.

6 secretion and inhibit collagen deposition in hepatic stellate cells.

7 ta (TGF-beta) signaling pathway in activated hepatic stellate cells.

8 mune response induced by B. abortus-infected hepatic stellate cells.

9 stern diet, indicating reduced activation of hepatic stellate cells.

10 both preneoplastic hepatocytes and activated hepatic stellate cells.

11 collagen I and alpha-smooth muscle actin, by hepatic stellate cells.

12 C), as well as primary human hepatocytes and hepatic stellate cells.

13 fibrosis marker activation in primary human hepatic stellate cells.

14 d in the liver, their expansion supported by hepatic stellate cells.

15 in embryonic stem cells, gastric tumors, and hepatic stellate cells.

16 e lineage tracing to follow transplanted rat hepatic stellate cells, a resident liver mesenchymal cel

17 Hepatic stellate cells activate during chronic liver inj

18 lic fatty liver disease activity scores, and hepatic stellate cell activation (alpha-smooth muscle ac

19 th hepatic alphaSMA expression, a marker for hepatic stellate cell activation (r = -0.31, P < 0.02).

20 hat coincided with alterations in markers of hepatic stellate cell activation and extracellular matri

21 dently benefitted liver fibrosis via altered hepatic stellate cell activation and extracellular matri

22 the mechanism by which adiponectin modulates hepatic stellate cell activation and fibrogenesis.

23 recedes fibrosis, but is also permissive for hepatic stellate cell activation and fibrosis.

24 evance, C1QTNF2 expression is reduced during hepatic stellate cell activation in culture and in a mou

25 Finally, MSDC-0602 directly reduced hepatic stellate cell activation in vitro, and MSDC-0602

26 on, L4F nanoparticles were found to suppress hepatic stellate cell activation in vitro.

27 FR reduced hepatic stellate cell activation markers (transforming g

28 LOX inhibition attenuated hepatic stellate cell activation markers and promoted F4

29 idence demonstrate that the hepatic fibrosis/hepatic stellate cell activation may be an important gen

30 Hepatic stellate cell activation represents a critical e

31 suppression of infiltrating macrophages and hepatic stellate cell activation through modulation of i

32 Hepatic stellate cell activation was detected by immunof

33 olic zonation, sinusoid capillarization, and hepatic stellate cell activation were assessed by anti-c

34 enesis partly mediated through inhibition of hepatic stellate cell activation, and significant decrea

35 ction induced liver fibrosis associated with hepatic stellate cell activation, hepatitis, and liver i

36 ary proliferation, senescence, fibrosis, and hepatic stellate cell activation, which were reduced in

37 HO-1 induction significantly suppressed hepatic stellate cell activation.

38 chanisms for increased hepatocyte growth and hepatic stellate cell activation.

39 phage activation, prothrombin activation and hepatic stellate cell activation.

40 culture models of monocyte/macrophage and/or hepatic stellate cell activation.

41 HCV-activated monocytes/macrophages promoted hepatic stellate cell activation.

42 However, the molecular mechanisms for hepatic stellate-cell activation by HCV-infected hepatoc

43 Addition of hepatic stellate cells allowed generation of myeloid-der

44 In vitro experiments were performed in rat hepatic stellate cells and hepatocytes.

45 tumors; and to detect pathways from profiled hepatic stellate cells and hippocampal neurons.

46 We identify potent MMIC activity in hepatic stellate cells and liver sinusoidal endothelial

47 expression and/or activity in primary human hepatic stellate cells and LX-2 cells.

48 triggering cellular senescence in activated hepatic stellate cells and portal fibroblasts by engagin

49 e system to study differentiation of primary hepatic stellate cells and portal fibroblasts from rats

50 EIIIA is dispensable for differentiation of hepatic stellate cells and portal fibroblasts to myofibr

51 he MIF receptor, CD74, that is released from hepatic stellate cells and that binds MIF, neutralizing

52 rate fibrosis in a yin-yang interaction with hepatic stellate cells, and are a key component of tumor

53 , dendritic cells, biliary epithelial cells, hepatic stellate cells, and hepatocytes.

54 promotes fibrogenic activation of attenuated hepatic stellate cells, and limits fibrosis reversal.

55 ent were also assessed in human hepatocytes, hepatic stellate cells, and LX-2 cells.

56 with eosinophils, neutrophils, macrophages, hepatic stellate cells, and lymphocytes all identified a

57 Endothelin-induced vasoconstriction by hepatic stellate cells, and not platelet accumulation or

58 sion of a subset of TGF-beta target genes in hepatic stellate cells, and the cooperation between the

59 Pericytes such as hepatic stellate cells are an important cell within the

60 Hepatic stellate cells are liver-specific mesenchymal ce

61 Activated hepatic stellate cells are the main source of excessive

62 were studied using K-562, P815, and primary hepatic stellate cells as targets.

63 milieu in the liver, including activation of hepatic stellate cells as well as expression of type 1 c

64 ding of the formation and characteristics of hepatic stellate cells, as well as their function in liv

65 poptosis in CAF and in myofibroblastic human hepatic stellate cells but lacked similar effects in qui

66 ver, the major cellular source of HGF is the hepatic stellate cell, but after liver injury, HGF expre

67 s was accompanied by increased activation of hepatic stellate cells, but hepatic mediators of inflamm

68 mide-induced fibrosis, which requires motile hepatic stellate cells, but not from bile duct ligation-

69 In vitro, CO increased HGF expression in hepatic stellate cells, but not HC, and when cocultured

70 rin induces Bmp6 mRNA expression in isolated hepatic stellate cells, but not in hepatocytes.

71 o, AGE exposure decreased Sirt1 and Timp3 in hepatic stellate cells by a NOX2-dependent pathway, and

72 tly induced production of collagen type I in hepatic stellate cells by activating the signal transduc

73 large numbers of hepatocytes, Kupffer cells, hepatic stellate cells, CD4-positive and CD8-positive ly

74 of an in situ liver perfusion system and on hepatic stellate cell contraction in vitro.

75 is, hepatic Rho-kinase activity (a marker of hepatic stellate cell contraction), and the endothelial

76 Instead, hepatic stellate cells cultured on EIIIA-containing cell

77 inhibition of miR-200b in cholangiocytes and hepatic stellate cells decreased the expression of miR-2

78 issue inflammation accompanied with elevated hepatic stellate cell-derived TnC and Toll-like receptor

79 Activated hepatic stellate cells drive fibrogenesis, changing the

80 Activation of the autophagic pathway in hepatic stellate cells during Brucella infection could h

81 nd activities of hepatocytes, Kupffer cells, hepatic stellate cells, endothelial cells, and circulati

82 We analyzed hepatic stellate cells from C57BL/6 wild-type, Atg7(F/F)

83 ysiological relevance of this interaction in hepatic stellate cells from ethanol-administered rats in

84 MDSC were generated by addition of hepatic stellate cells from various stain mice into dend

85 Mice devoid of Stat3 signaling in hepatic stellate cells (GFAPStat3(-/-) mice) were less s

86 It is becoming increasingly clear that hepatic stellate cells have a profound impact on the dif

87 ors to apoptosis resistance in primary human hepatic stellate cells (hHSC).

88 We have reported that hepatic stellate cells (HpSCs), the stromal cells in the

89 n the transcriptome of rat PMFs, compared to hepatic stellate cell HSC-derived myofibroblasts in cult

90 ceptor (A2AR) activation is known to enhance hepatic stellate cell (HSC) activation and A2AR deficien

91 ing on the role of inflammatory mediators in hepatic stellate cell (HSC) activation and HSC survival

92 Liver X receptors (LXRs) are determinants of hepatic stellate cell (HSC) activation and liver fibrosi

93 such as dendritic cells (DCs) contribute to hepatic stellate cell (HSC) activation and liver fibrosi

94 ceptor tyrosine kinases, which contribute to hepatic stellate cell (HSC) activation and liver fibrosi

95 Hepatic stellate cell (HSC) activation and transforming

96 Hepatic stellate cell (HSC) activation is a pivotal even

97 Hepatic stellate cell (HSC) activation is an essential e

98 Hepatic stellate cell (HSC) activation on liver injury f

99 ive polymerase chain reaction for markers of hepatic stellate cell (HSC) activation.

100 ation of extracellular matrix components and hepatic stellate cell (HSC) activation.

101 I), which enhances TGF-beta signaling during hepatic stellate cell (HSC) activation.

102 is induced as an immediate-early gene during hepatic stellate cell (HSC) activation.

103 nds against oxidative stress, which promotes hepatic stellate cell (HSC) activation.

104 bition could partly account for reduction of hepatic stellate cell (HSC) activation.

105 R (HuR), during cholestatic liver injury and hepatic stellate cell (HSC) activation.

106 e, little is known about its contribution to hepatic stellate cell (HSC) activation.

107 ming growth factor (TGF)-beta and subsequent hepatic stellate cell (HSC) activation.

108 cantly increased after pIVCL concurrent with hepatic stellate cell (HSC) activation.

109 M-CSF-Mphi and IL-34-Mphi also express the hepatic stellate cell (HSC) activators, platelet-derived

110 on by exploring the impact of the AzI SNP on hepatic stellate cell (HSC) activity.

111 e role and the molecular mediators of EMT in hepatic stellate cell (HSC) and human liver cancer cells

112 study was to identify the role of Nogo-B in hepatic stellate cell (HSC) apoptosis in cirrhotic liver

113 liferation, LPC biliary differentiation, and hepatic stellate cell (HSC) chemotaxis.

114 tor type 4 (CXCR4) pathway directly promotes hepatic stellate cell (HSC) differentiation and activati

115 thymosin beta-4 (TB4) involved in regulating hepatic stellate cell (HSC) functions remain unclear.

116 PDGF-dependent hepatic stellate cell (HSC) recruitment is an essential

117 Hepatic stellate cell (HSC) transdifferentiation from a

118 sition of extracellular matrix components by hepatic stellate cell (HSC)-derived myofibroblasts.

119 reaction (qRT-PCR) and western blotting and hepatic stellate cell (HSC)-MF contractility by gel cont

120 eceptor type 2 (CCR2) is expressed by active hepatic stellate cells (HSC) and is a key monocyte recru

121 Erlotinib reduced EGFR phosphorylation in hepatic stellate cells (HSC) and reduced the total numbe

122 ion would normalize crosstalk with activated hepatic stellate cells (HSC) and thereby promote quiesce

123 Hepatic stellate cells (HSC) are a major source of the i

124 (CCl(4)) model of liver fibrosis, quiescent hepatic stellate cells (HSC) are activated to become myo

125 Hepatic stellate cells (HSC) are believed to be the majo

126 Hepatocyte apoptosis and activation of hepatic stellate cells (HSC) are critical events in fibr

127 To investigate whether the RA producing hepatic stellate cells (HSC) are part of the liver toler

128 Hepatic stellate cells (HSC) are the main effectors of l

129 Hepatic stellate cells (HSC) are the major source of ext

130 ibrosis is mediated by the transformation of hepatic stellate cells (HSC) from a quiescent to an acti

131 Recently, human hepatic stellate cells (HSC) have been reported to induc

132 tro by co-culturing control hepatocytes with hepatic stellate cells (HSC) isolated from ethanol-fed m

133 Activated hepatic stellate cells (HSC) play a critical role in fib

134 n (AR) involvement in liver fibrogenesis and hepatic stellate cells (HSC) regulation is under study.

135 Effects of NKT cell-derived factors on hepatic stellate cells (HSC) were examined and fibrogene

136 Murine FHL2(-/-) and hepatic stellate cells (HSC) were isolated and investiga

137 s ectopically expressed during activation of hepatic stellate cells (HSC) when it is responsible for

138 cotransplantation with liver stromal cells, hepatic stellate cells (HSC), achieves long-term surviva

139 -22 ameliorates liver fibrosis by inhibiting hepatic stellate cells (HSC), and loss of miR-200a is as

140 in response (UPR) both promote activation of hepatic stellate cells (HSC), however the link between t

141 e influence on viability and mitochondria of hepatic stellate cells (HSC), the toxicity of the codrug

142 growth factor (CCN2) drives fibrogenesis in hepatic stellate cells (HSC).

143 are hallmarks of the transdifferentiation of hepatic stellate cells (HSC).

144 r are principally regulated by activation of hepatic stellate cells (HSC).

145 ole of secreted bioactive TGF-beta1 in human hepatic stellate cells (HSCs) activation and invasion.

146 on-kinse (FAK) plays a key role in promoting hepatic stellate cells (HSCs) activation in vitro and li

147 (CTGF) is constantly expressed in activated hepatic stellate cells (HSCs) and acts downstream of TGF

148 was principally expressed in quiescent mouse hepatic stellate cells (HSCs) and directly suppressed pr

149 OPN biological actions were studied in human hepatic stellate cells (HSCs) and in precision-cut liver

150 Paracrine signaling between hepatic stellate cells (HSCs) and liver endothelial cell

151 Hepatic stellate cells (HSCs) and portal fibroblasts (PF

152 n induce liver fibrosis remission by killing hepatic stellate cells (HSCs) and producing interferon (

153 Liver repair involves phenotypic changes in hepatic stellate cells (HSCs) and reactivation of morpho

154 at MAT2A expression is enhanced in activated hepatic stellate cells (HSCs) and that silencing this ge

155 IL-20 activated quiescent rat hepatic stellate cells (HSCs) and up-regulated TGF-beta1

156 that accumulate during tissue fibrosis, and hepatic stellate cells (HSCs) are believed to be the maj

157 Hepatic stellate cells (HSCs) are critical for hepatic w

158 Hepatic stellate cells (HSCs) are key players in the dev

159 Hepatic stellate cells (HSCs) are liver-specific mesench

160 ownstream of TLRs on Kupffer cells (KCs) and hepatic stellate cells (HSCs) are poorly defined.

161 Activated hepatic stellate cells (HSCs) are the major source for a

162 During liver fibrosis, activated hepatic stellate cells (HSCs) are the major source of th

163 In liver fibrogenesis, hepatic stellate cells (HSCs) are thought to transdiffer

164 e factor-1alpha (HIF-1alpha) is activated in hepatic stellate cells (HSCs) by hypoxia and regulates g

165 fibrillary acidic protein positive (GFAP(+)) hepatic stellate cells (HSCs) can express epithelial mar

166 tyrosine kinase receptor, is up-regulated in hepatic stellate cells (HSCs) during chronic liver injur

167 ) generates reactive oxygen species (ROS) in hepatic stellate cells (HSCs) during liver fibrosis.

168 Similarly, normal/quiescent hepatic stellate cells (HSCs) express high levels of MMP

169 e the expression and processing of CtsB/D in hepatic stellate cells (HSCs) from ASMase-null mice and

170 Effects of LPS on fibrogenic hepatic stellate cells (HSCs) from WT and TLR4-KO mice w

171 Hepatic stellate cells (HSCs) have been identified as th

172 Retinoid-storing hepatic stellate cells (HSCs) have recently been describ

173 ve shown potent immunoregulatory activity of hepatic stellate cells (HSCs) in mice.

174 t the potential role of progenitor cells and hepatic stellate cells (HSCs) in promoting the early eve

175 Activation of hepatic stellate cells (HSCs) in response to injury is a

176 Here we report that fibrogenic hepatic stellate cells (HSCs) in the liver are negative

177 hepatic gammadelta T cells colocalized with hepatic stellate cells (HSCs) in vivo and promoted apopt

178 Hepatic stellate cells (HSCs) induce hepatic inflammatio

179 Hepatic stellate cells (HSCs) inhibit T cells, a process

180 We answered the questions of whether hepatic stellate cells (HSCs) interact with CD4+ T cells

181 e activation and transformation of quiescent hepatic stellate cells (HSCs) into myofibroblast-like ce

182 the transition of quiescent hepatocytes and hepatic stellate cells (HSCs) into the cell cycle.

183 Activation of hepatic stellate cells (HSCs) is a critical step in the

184 Activation of hepatic stellate cells (HSCs) is a key event in the init

185 Activation of hepatic stellate cells (HSCs) is crucial to the developm

186 onic liver disease mediated by activation of hepatic stellate cells (HSCs) leads to liver fibrosis.

187 Hepatic stellate cells (HSCs) may play an important role

188 Proliferating cholangiocytes and activated hepatic stellate cells (HSCs) participate in the promoti

189 Hepatic stellate cells (HSCs) play a major role increasi

190 Hepatic stellate cells (HSCs) play critical roles in liv

191 ammatory and pro-fibrogenic environment with hepatic stellate cells (HSCs) remodeling the extracellul

192 (NKT) cells to the liver to remove activated hepatic stellate cells (HSCs) significantly and ameliora

193 We demonstrated previously that mouse hepatic stellate cells (HSCs) suppress T cells via progr

194 n of matrix proteins, such as collagen I, by hepatic stellate cells (HSCs) that culminates in cirrhos

195 developed a novel model for depleting mouse hepatic stellate cells (HSCs) that has allowed us to cla

196 Transdifferentiation of hepatic stellate cells (HSCs) to a myofibroblast-like ph

197 These secreted cytokines may activate hepatic stellate cells (HSCs) toward fibrosis.

198 Hepatic stellate cells (HSCs) undergo myofibroblastic ac

199 Hepatic stellate cells (HSCs) undergo myofibroblastic tr

200 Finally, the effect of leptin on hepatic stellate cells (HSCs) was evaluated.

201 assess direct effects on OPN expression, and hepatic stellate cells (HSCs) were cultured in medium wi

202 Hh-sensitive, hepatic stellate cells (HSCs) were identified as the sou

203 Kupffer cells and hepatic stellate cells (HSCs) were isolated from WT mice

204 Hepatic stellate cells (HSCs) were recently identified a

205 Hepatic stellate cells (HSCs) were recently postulated a

206 Hepatic stellate cells (HSCs) were treated with or witho

207 s increased during the culture activation of hepatic stellate cells (HSCs), a liver resident fibrobla

208 novel Cre-transgenic mouse that marks 99% of hepatic stellate cells (HSCs), a liver-specific pericyte

209 Hepatic stellate cells (HSCs), a population of liver non

210 ading to increased LPA levels, activation of hepatic stellate cells (HSCs), and amplification of prof

211 nd NOX2 in endogenous liver cells, including hepatic stellate cells (HSCs), and bone marrow (BM)-deri

212 ver sinusoidal endothelial cells (LSECs) and hepatic stellate cells (HSCs), are the first liver cells

213 ion of myofibroblasts derived from quiescent hepatic stellate cells (HSCs), but the mechanisms that c

214 acellular matrix (ECM) cytokine expressed in hepatic stellate cells (HSCs), could drive fibrogenesis

215 In cultured hepatic stellate cells (HSCs), disrupting Hedgehog signa

216 lls of the gastrointestinal tract, including hepatic stellate cells (HSCs), endothelial cells, and he

217 periments were performed with primary rodent hepatic stellate cells (HSCs), Kupffer cells (KCs), and

218 78a-3p, directly targeting Gli3 in activated hepatic stellate cells (HSCs), reduces expression of Gli

219 yte-macrophage colony-stimulating factor and hepatic stellate cells (HSCs), resulting in the generati

220 Activated hepatic stellate cells (HSCs), the main fibrogenic cell

221 T cell MPs fuse with cell membranes of hepatic stellate cells (HSCs), the major effector cells

222 levels of IL-10R2 and IL-22R1 expression on hepatic stellate cells (HSCs), the predominant cell type

223 A transcriptome signature of activated hepatic stellate cells (HSCs), the primary collagen-secr

224 entry into cells, are expressed on activated hepatic stellate cells (HSCs), the principle fibrogenic

225 at TGF-beta1 recruited IQGAP1 to TbetaRII in hepatic stellate cells (HSCs), the resident liver pericy

226 mechanism of this resistance by focusing on hepatic stellate cells (HSCs), which are known to regula

227 In human cultured hepatic stellate cells (HSCs), which express high levels

228 ved exosomes could regulate the phenotype of hepatic stellate cells (HSCs).

229 growth factor-beta (PDGFB) is a mitogen for hepatic stellate cells (HSCs).

230 ions of Kupffer cells (KCs), neutrophils, or hepatic stellate cells (HSCs).

231 d by the deposition of collagen by activated hepatic stellate cells (HSCs).

232 e stored as retinyl esters in the retina and hepatic stellate cells (HSCs).

233 SEMA7A is highly expressed in hepatic stellate cells (HSCs).

234 sponse involving TGFbeta1/SMAD activation of hepatic stellate cells (HSCs).

235 is by regulating the fibrogenic phenotype of hepatic stellate cells (HSCs).

236 ires proliferation and activation of resting hepatic stellate cells (HSCs).

237 acrophages and noninflammatory cells such as hepatic stellate cells (HSCs).

238 system and is mediated by Kupffer cells and hepatic stellate cells (HSCs).

239 Hepatic fibrosis is marked by activation of hepatic stellate cells (HSCs).

240 ra-epithelial neoplasia, or PDAC, as well as hepatic stellate cells (HSCs).

241 eins including laminin (Ln)-332, produced by hepatic stellate cells (HSCs).

242 o with special focus on the STAT3 pathway in hepatic stellate cells (HSCs).

243 iew them as more than just an alternative to hepatic stellate cells in fibrosis.

244 of IL-20 was much higher in hepatocytes and hepatic stellate cells in liver biopsies from patients w

245 ivated B cells (NFkappaB) in hepatocytes and hepatic stellate cells in monoculture; however, they do

246 cant Timp3 gene transcription was induced by hepatic stellate cells in the inflamed liver.

247 inhibition of miR-200b in cholangiocytes and hepatic stellate cells in vitro, we evaluated angiogenes

248 ion of a p53-dependent senescence program in hepatic stellate cells increases liver fibrosis and cirr

249 on of tumor-derived JAG1 signaling activated hepatic stellate cells, increasing their recruitment to

250 We recently demonstrated that hepatic stellate cells induce the differentiation of mye

251 These results demonstrated that hepatic stellate cell-induced MDSCs concurrently suppres

252 Cells showed decreased fibrogenesis, hepatic stellate cell infiltration, Kupffer cells and in

253 r was miniaturized and integrated with human hepatic stellate cells inside microfluidic devices.

254 brosis, the activation of CB(1) receptors on hepatic stellate cells is fibrogenic, and CB(1) blockade

255 emene (ELE) on the activation of human liver hepatic stellate cell line LX-2 cells.

256 This mechanism was confirmed in a hepatic stellate cell line stably that endogenously expr

257 PA-treated PRHs was applied to cultured rat hepatic stellate cell line, HSC-T6, with or without Flu-

258 l death involving the TRAIL receptors in the hepatic stellate cell line, LX2.

259 1 up-regulation; coculture of hepatocyte and hepatic stellate cell lines significantly increased expr

260 iven profibrogenic program in hepatocyte and hepatic stellate cell lines through ROS, NFkappaB, and T

261 rix metalloproteinase-1 secretion induced by hepatic stellate cells (LX-2).

262 e performed in vitro studies on immortalized hepatic stellate cells (LX-2).

263 Human hepatic stellate cells (LX2 cells) were used to assess a

264 ted for up to 21 days using human cell lines hepatic stellate cells (LX2), hepatocellular carcinoma (

265 interacts with adipocytes, pancreatic cells, hepatic stellate cells, macrophages, and muscle cells.

266 Hepatic accumulation of myofibroblastic hepatic stellate cells (MF-HSCs) is pivotal in the patho

267 of studies employing Tg technology to target hepatic stellate cells, myofibroblasts, liver sinusoidal

268 CCA cells with myofibroblastic human primary hepatic stellate cells or LX-2 cells significantly decre

269 was not required for differentiation of rat hepatic stellate cells or portal fibroblasts into fibrog

270 ed production of hepatocyte growth factor in hepatic stellate cells postinjury, which, in turn, resul

271 (1) angioblasts, (2) mature endothelia, (3) hepatic stellate cell precursors, (4) mature stellate ce

272 ed livers: transition of resident, quiescent hepatic stellate cells (Q-HSCs) to MF-HSCs and expansion

273 Trans-differentiation of quiescent hepatic stellate cells (Q-HSCs), which exhibit epithelia

274 patic fibrosis, attenuated the activation of hepatic stellate cells, reduced frequencies of Th9, Th17

275 ors, liver sinusoidal endothelial cells, and hepatic stellate cells respond to liver injury and contr

276 Primary hepatic stellate cell-seeded hydrogels stiffened in situ

277 muscle actin mRNA), whereas EX increased the hepatic stellate cell senescence marker CCN1 (P < 0.01 v

278 p47phox KO endogenous liver cells, including hepatic stellate cells, showed a approximately 60% reduc

279 throughput image-based screen using primary hepatic stellate cells that identified the antifungal dr

280 egression and reduced clearance of activated hepatic stellate cells, the key fibrogenic cell in the l

281 the activation of the autophagic pathway in hepatic stellate cells to create a microenvironment that

282 findings linking senescence and autophagy in hepatic stellate cells to HCC have also been discovered,

283 lls, liver sinusoidal endothelial cells, and hepatic stellate cells to liver homeostasis and repair a

284 The TRAIL pathway can mediate apoptosis of hepatic stellate cells to promote the resolution of live

285 er autophagy also promotes loss of lipids in hepatic stellate cells to provide energy for their activ

286 Hepatic stellate cell transdifferentiation is a key even

287 bsence of CYLD, gene transcription of HGF in hepatic stellate cells was repressed through binding of

288 sis and DNA damage, whereas proliferation of hepatic stellate cells was stimulated by KCa3.1 inhibiti

289 Fibrotic status and the activation of hepatic stellate cells were improved upon pterostilbene

290 hanisms of Nogo-B's involvement in fibrosis, hepatic stellate cells were isolated from WT and NGB KO

291 nation showed that macrophages and activated hepatic stellate cells were the main cell types expressi

292 CCN1 also induced Jag1 expression in hepatic stellate cells, whereupon they interacted with h

293 The inflammatory cells activate hepatic stellate cells, which are the major source of my

294 parent that the activation of perisinusoidal hepatic stellate cells, which is a key event mediating t

295 sis of liver fibrosis involves activation of hepatic stellate cells, which is associated with depleti

296 -catenin signaling may lead to activation of hepatic stellate cells, which is required for fibrosis.

297 Treatment of mouse hepatic stellate cells with ethanol significantly increa

298 ons in vitro, we treated rat hepatocytes and hepatic stellate cells with PLS, which caused proteolyti

299 Mechanistically, PTX3 mediated the hepatic stellate cell wound-healing response.

300 They are distinct from hepatic stellate cells, yet like stellate cells differen