ライフサイエンスコーパス: hepatic vein

1 ory hepatic vein, and one had a double right hepatic vein.

2 an intrahepatic portal vein and an adjacent hepatic vein.

3 percutaneous access route through the fetal hepatic vein.

4 ) via a quadripolar catheter positioned in a hepatic vein.

5 tions of ammonia, glutamine, and urea in the hepatic vein.

6 ar catheter positioned in a subdiaphragmatic hepatic vein.

7 ol subjects by direct catheterization of the hepatic vein.

8 and sampled from femoral artery and vein and hepatic vein.

9 not adequately drained by a remaining major hepatic vein.

10 treatment for BCS with diffuse occlusion of hepatic veins.

11 %) patients had involvement of the portal or hepatic veins.

12 he short, nongrafted portions of the outflow hepatic veins.

13 inserted in the superior mesenteric and left hepatic veins.

14 6 nmol/min), and cortisol appearance in the hepatic vein after oral cortisone was unchanged.

15 ostprocessing depicted arterial, portal, and hepatic vein anatomy traversing the anticipated surgical

16 min with blood sampling from catheters in a hepatic vein and a radial artery (concentrations of (18)

17 Samples were obtained from the hepatic vein and an arterialized hand vein at steady sta

18 egmentectomy with reconstruction of the left hepatic vein and one patient died at 3 months after rese

19 effective for controlling bleeding from the hepatic vein and safer than increasing pneumoperitoneum

20 hepatectomies extended to include the middle hepatic vein and the caudate lobe but preserving the maj

21 SHAPE data were collected from a portal and hepatic vein and were compared with invasive measurement

22 Conclusion During microwave ablation of HCC, hepatic veins and arteries were resistant to vessel occl

23 ns in three, portal tracts in five, and both hepatic veins and portal tracts in two patients.

24 asurements were performed after clamping the hepatic veins and recipient hepatectomy.

25 the lungs, caused by altered diameter of the hepatic veins and sinusoids in Bmpr2(+/-) mice.

26 8 years) with fatty infiltration surrounding hepatic veins and/or portal tracts were retrospectively

27 ar predisposition to fat accumulation around hepatic veins and/or portal tracts.

28 Dogs had sampling (artery, portal vein, and hepatic vein) and infusion (vena cava and portal vein) c

29 simultaneous sampling from the portal vein, hepatic vein, and an arterialized peripheral vein.

30 ed (ie., gastroduodenal artery, portal vein, hepatic vein, and femoral artery), perfusion rates in he

31 xteen had at least one significant accessory hepatic vein, and one had a double right hepatic vein.

32 obtained (i.e., portal vein, carotid artery, hepatic vein, and pulmonary artery), perfusion rates wer

33 Complete occlusion of the portal veins, hepatic veins, and hepatic arteries within and directly

34 ein, superior mesenteric vein, splenic vein, hepatic veins, and inferior vena cava (IVC) were evaluat

35 myography-guided PN monitoring using a novel hepatic vein approach for prevention of PNP.

36 uinaud's segment, in which the corresponding hepatic veins are exposed on the raw surface.

37 The pattern of amino acids appearing in hepatic vein blood was affected by changes in amino acid

38 he first 10 did not include the right middle hepatic vein branches in the graft.

39 ine medially to incorporate the right middle hepatic vein branches into the donor graft.

40 were scored for visualization of portal and hepatic vein branches, liver edge sharpness, cardiac pul

41 monitoring using a catheter positioned in a hepatic vein can aid in preventing phrenic nerve palsy (

42 Liver resection with reconstruction of the hepatic veins can be performed in selected cases.

43 pneumoperitoneum pressure, bleeding from the hepatic vein cannot be controlled under high airway pres

44 ght-matched control subjects by means of the hepatic vein catheterization (HVC) technique.

45 e obtained in 12 control subjects undergoing hepatic vein catheterization and infusion of identical t

46 We used isotope dilution/hepatic vein catheterization techniques to examine wheth

47 on, directly measured using the arterial and hepatic vein catheters, did not differ (67 +/- 3 vs. 71

48 Hepatic vein-cava stenoses occurred after a mean of 37.2

49 s occurred in 32 LRD, 3 RSS, and 3 FS, while hepatic vein-cava stenoses occurred in 2 LRD, 8 RSS, and

50 tified in 49 patients (38 portal vein and 12 hepatic vein-cava stenoses).

51 significant reduction in the number of large hepatic veins compared to embryonic wild-type (WT) liver

52 n inflammatory cells surrounding the central hepatic vein, compared with WT.

53 even consecutive patients with tumors at the hepatic vein confluence were prospectively evaluated wit

54 pects of the procedure, including the middle hepatic vein controversy and the "small for size syndrom

55 other hand, hepatic artery, portal vein, and hepatic vein cortisol concentrations did not differ (0.3

56 quires no retrocaval, hepatic vein, or short hepatic vein dissection, and the inferior vena cava can

57 we present a liver graft with abnormal left hepatic vein draining directly to the right atrium of th

58 mental system used for this study mimics the hepatic vein draining into the inferior vena cava and al

59 often employed to control bleeding from the hepatic vein during pure laparoscopic hepatectomy; howev

60 d metabolite concentrations over time in the hepatic vein during toxin-induced liver damage and regen

61 ation, portal vein endothelial inflammation, hepatic vein endothelial inflammation, and centrilobular

62 bnormalities, including aberrant subcapsular hepatic veins, enlarged glomeruli, intestinal polyps con

63 al mitral annular e' velocity, and prominent hepatic vein expiratory diastolic flow reversals are ind

64 ombination with either medial e'>/=9 cm/s or hepatic vein expiratory diastolic reversal ratio >/=0.79

65 ial mitral annular e' to lateral e', and (5) hepatic vein expiratory diastolic reversal ratio.

66 septal shift, (2) medial mitral e', and (3) hepatic vein expiratory diastolic reversal ratio.

67 stal trocars to expose the root of the right hepatic vein for segmentectomy VII and VIII.

68 inuous spectral doppler ultrasonography of a hepatic vein from 20 s before to 3 min after a periphera

69 ari syndrome (BCS) with diffuse occlusion of hepatic veins has a high mortality rate and remains chal

70 artery (RA) catheter, portal vein (PV), and hepatic vein (HV) during the dissection phase and was re

71 nt via percutaneous access through the fetal hepatic vein in a sheep model.

72 ary artery, carotid artery, portal vein, and hepatic vein in swine infused with PGE1 (range, 0.67-4.9

73 sed to the confluence of the left and middle hepatic veins in all cases.

74 ) in level 1-2 buds and becomes CD34(-) near hepatic veins in level 3-4 buds.

75 t hepatic vein (RHV) confluence of accessory hepatic veins in the surgical plane.

76 Fatty infiltration surrounded hepatic veins in three, portal tracts in five, and both

77 ying the galactose infusion point (simulated hepatic vein) in a 15-cm conduit was 1.7 to 2.8 mm, or 1

78 1) but did not differ in the portal vein and hepatic vein, indicating net uptake across the viscera b

79 Hepatic vein involvement by hepatic malignancy does not

80 provide comparable results for assessment of hepatic vein involvement by tumor.

81 ameters were measured 15-20 mm caudal to the hepatic vein junction and recorded by bidimensional imag

82 terial blood supply, leucine export into the hepatic vein, leucine oxidation and transamination, and

83 Anatomy of the left hepatic vein (LHV) was studied in a series of 53 consecu

84 del that PH in combination with right median hepatic vein ligation (RMHV-L) caused confluent parenchy

85 hepatectomy plane to the right of the middle hepatic vein (MHV) and separating the right and left lob

86 the individualized management of the middle hepatic vein (MHV).

87 ws adequate venous outflow through the right hepatic vein more than 1 cm, which is demonstrated by th

88 oportal vein (n = 24), vena cava (n = 3), or hepatic vein (n = 3).

89 Sixteen hepatic veins (nine right, four middle, three left) were

90 rly and transient increase in posttransplant hepatic vein nitrate levels (pretransplant, 90 microM; 2

91 Monocrotaline caused hepatic vein NO to decrease by 30% at 24 hours and by 70

92 Hepatic vein NOx was significantly higher than circulati

93 Cellular overgrowth from the hepatic vein occluded the end of one graft at 3 months,

94 SSPCS in BCS with hepatic vein occlusion alone results in reversal of live

95 In the 32 patients with BCS resulting from hepatic vein occlusion alone, SSPCS had a surgical death

96 Hepatic vein occlusion was significantly correlated with

97 The right hepatic vein of the donor graft was anastomosed to the c

98 and have a patent main portal vein and major hepatic veins on duplex ultrasonography.

99 vein drainage to the inferior vena cava and hepatic vein or of the inferior phrenic vein (n = 7).

100 two veins closely merging toward the median hepatic vein, or (C) a double outflow.

101 al hemorrhage, or suffered from portal vein, hepatic vein, or hepatic artery thrombosis

102 cavaplasty technique requires no retrocaval, hepatic vein, or short hepatic vein dissection, and the

103 een proximal FHVP (obtained at 2 cm from the hepatic vein outlet) and IVC (measured at the level of t

104 Levels were also determined from hepatic vein, portal vein, and systemic arterial blood i

105 s were located immediately adjacent to major hepatic veins, portal veins, or both; thus, they were no

106 he difference between wedged (WHVP) and free hepatic vein pressure (FHVP), predicts survival in patie

107 reening inflammatory serum biomarkers of the hepatic vein pressure gradient (HVPG) is based on the fa

108 The hepatic vein pressure gradient (HVPG) is the standard us

109 underwent a left hepatectomy without middle hepatic vein procurement.

110 donor liver transplantation that necessitate hepatic vein reconstruction can be applied to hepatic re

111 ts who underwent hepatic resection requiring hepatic vein reconstruction from 1996-2001 were reviewed

112 Involvement of the hepatic veins requiring reconstruction has traditionally

113 ANOVA) from hepatic artery to portal vein to hepatic vein, respectively, indicating 8 +/- 3 and 28 +/

114 presence, size, and distance from the right hepatic vein (RHV) confluence of accessory hepatic veins

115 nferior vena cava, through drainage into the hepatic vein, right atrium or left atrium.

116 d laceration or contusion extending into the hepatic vein(s), inferior vena cava, porta hepatis, or g

117 he present study, arterial, portal vein, and hepatic vein sampling catheters were surgically placed i

118 monitoring using a catheter positioned in a hepatic vein seems feasible and effective to prevent PNP

119 a catheter positioned in a subdiaphragmatic hepatic vein seems feasible during cryoballoon ablation.

120 aped (H-type portal-caval)], 2 had portal-to-hepatic vein shunts (portohepatic), and 2 had a persiste

121 nd US yielded identical findings at 26 of 27 hepatic vein sites.

122 n 39.7% of portal veins (29 of 73), 15.0% of hepatic veins (six of 40), and 14.2% of hepatic arteries

123 rrent variceal bleeding or ascites than were hepatic vein stenoses (odds ratio, 3.6; P = .02).

124 ted after 30 days; and in 19 patients (16%), hepatic vein stenoses were detected after 30 days.

125 Although common, hepatic vein stenoses were rarely associated with recurr

126 erved that during the first 48 h, most major hepatic veins, such as the portal and umbilical veins, n

127 (1.33 +/- 0.11; P < 0.001) were lower in the hepatic vein than in the portal vein, indicating product

128 ficantly higher leptin concentrations in the hepatic vein than lean sham-operated, fa/fa BDL, or fa/f

129 PAI-1 play pivotal and antagonistic roles in hepatic vein thrombosis and that PAI-1 is a potential ta

130 ncidence of hepatic artery, portal vein, and hepatic vein thrombosis is 0%, 0%, and 0%, respectively.

131 ccessful deceased donor liver transplant for hepatic vein thrombosis.

132 re largely protected from the development of hepatic vein thrombosis.

133 ribution was similar between portal vein and hepatic vein to cava, as was the mean patient age.

134 shunts were created from the middle or left hepatic vein to the left portal vein, and none of the su

135 rease of 10% above baseline levels indicated hepatic vein transit time (HVTT).

136 ups: the first had occlusion confined to the hepatic veins treated by direct side-to-side portacaval

137 isease (M), macrovascular involvement of all hepatic veins (V) or portal bifurcation (P), contiguous

138 negative gradient of M30 from the portal to hepatic vein was demonstrated in patients with acetamino

139 Injury involving at least one major hepatic vein was found in 15 (88%) of 17 patients who re

140 ays of gestation (term, 147 days), the fetal hepatic vein was punctured percutaneously under ultrasou

141 x patients with HCC and cirrhosis, the right hepatic vein was reconstructed to provide venous outflow

142 tumors, hepatic artery, portal vein, and the hepatic veins was developed.

143 The SHAPE gradient between the portal and hepatic veins was in good overall agreement with the hep

144 truction of either the inferior vena cava or hepatic veins was performed in five patients.

145 of the inferior vena cava, portal vein, and hepatic veins, was successfully managed by aggressive ch

146 spectral Doppler signals of right and middle hepatic veins were analyzed.

147 Forty-four accessory hepatic veins were identified in 40 patients; seven drai

148 Significant accessory hepatic veins were preserved, brought together in a comm

149 (PV) injection of plasmid DNA in mice whose hepatic veins were transiently occluded.

150 ch in treating BCS with diffuse occlusion of hepatic veins, which should be performed in time.

151 confluence of a venous tributary (i.e., the hepatic vein) with a major vascular channel (i.e., the v