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1 as identified recently as a receptor for the hepatitis A virus.
2 ths were Salmonella Typhi, Taenia solium and hepatitis A virus.
3 fectively prevented with vaccination against hepatitis A virus.
4 rent infection as the community reservoir of hepatitis A virus.
5 e genome is conserved among strains, such as hepatitis A virus.
6 solate, respectively, of the HM175 strain of hepatitis A virus.
7 the parameters for the ubiquitination of the hepatitis A virus 3C protease are K(m) = 20 +/- 5 microm
8 we amplified virtually the entire genomes of hepatitis A virus (a member of the Picornaviridae family
9 andomization, 1414 (31%) were susceptible to hepatitis A virus and 1090 were eligible for the per-pro
11 multiple human maladies, yet currently, only hepatitis A virus and poliovirus can be controlled with
12 ad through the oral-fecal route (salmonella, hepatitis A virus), and organisms spread through direct
13 , Chlamydia pneumoniae, Helicobacter pylori, hepatitis A virus, and herpes simplex virus-1 were measu
15 f passively transferred maternal antibody to hepatitis A virus (anti-HAV) on the duration of seroposi
17 es of the encephalomyocarditis virus and the hepatitis A virus are both type III substrates for the m
18 rhinovirus, encephalomyocarditis virus, and hepatitis A virus) are morphologically similar, comprisi
19 enteric viruses, such as Human Norovirus and Hepatitis A Virus, are readily transmitted via the fecal
20 cally linked to asthma and is a receptor for hepatitis A virus, but the endogenous ligand of TIM-1 is
21 an KIM-1 exhibits homology to a monkey gene, hepatitis A virus cell receptor 1 (HAVcr-1), which was i
24 e toxin binds to protein receptors including hepatitis A virus cellular receptor 1 (HAVCR1), but the
26 expression of the inhibitory receptors PD-1, hepatitis A virus cellular receptor 2 (TIM3), lymphocyte
27 in-containing protein 3 (TIM3, also known as hepatitis A virus cellular receptor 2), and their respec
28 munized sequentially with tetanus toxoid and hepatitis A virus failed to develop antibody to either a
32 dministered to persons after exposure to the hepatitis A virus has not been compared directly with im
34 Vaccination provides long-term immunity to hepatitis A virus (HAV) among the general population, bu
35 lages in rural Alaska between 2 epidemics of hepatitis A virus (HAV) and after the second epidemic (1
36 ess the protective 190/4 epitope, they bound hepatitis A virus (HAV) and gained limited susceptibilit
38 irus B3, human rhinovirus 14, mengovirus, or hepatitis A virus (HAV) capsid proteins were supplied in
46 r highly specific and sensitive detection of hepatitis A virus (HAV) in food and water are of particu
52 on has dramatically reduced the incidence of hepatitis A virus (HAV) infection, but new infections co
53 n of HEV infection has broad similarities to hepatitis A virus (HAV) infection, with most cases being
63 the high-risk groups for vaccination against hepatitis A virus (HAV) is unlikely to have a significan
75 lonal antibody (MAb) 190/4 blocks binding of hepatitis A virus (HAV) to the HAV cellular receptor 1 (
76 or Disease Control and Prevention recommends hepatitis A virus (HAV) vaccination for all children at
79 h men (MSM) receiving two and three doses of hepatitis A virus (HAV) vaccine and HIV-uninfected MSM r
80 the safety and immunogenicity of 2 doses of hepatitis A virus (HAV) vaccine followed by a booster do
83 ' nontranslated region in the replication of hepatitis A virus (HAV) was studied by analyzing the tra
84 uman (HM-175) and simian (AGM-27) strains of hepatitis A virus (HAV) were constructed to evaluate the
85 tream of the internal ribosome entry site of Hepatitis A virus (HAV) were studied, a 35mer (HAV-35),
88 of positive-strand RNA viruses that includes hepatitis A virus (HAV), an ancient human pathogen that
90 s known about the mechanism of cell entry of hepatitis A virus (HAV), and the identification of cellu
91 on, all participants were vaccinated against hepatitis A virus (HAV), and the increase of antibody ti
92 IgG antibodies to cytomegalovirus (CMV), hepatitis A virus (HAV), herpes simplex virus type 1 (HS
93 'NTR) of an attenuated, cell culture-adapted hepatitis A virus (HAV), HM175/P16, enhance growth in cu
96 y a stem-loop structure with cre function in hepatitis A virus (HAV), the type species of this genus,
104 udies show that some picornaviruses, notably hepatitis A virus (HAV; genus Hepatovirus) and some memb
105 respond to a recall antigen and neoantigen (hepatitis A virus [HAV] vaccine) after 3 vaccinations.
107 chimpanzee sera; sera from chimpanzees with hepatitis A virus, hepatitis B virus, or hepatitis C vir
110 Mutations which positively affect growth of hepatitis A virus in cell culture may negatively affect
111 uring serial passage of the HM-175 strain of hepatitis A virus in MRC-5 cell cultures in order to det
113 itis B virus now joins hepatitis C virus and hepatitis A virus in targeting the same innate immune re
114 der had a significantly higher prevalence of hepatitis A virus infection (37%) than children living i
115 ica, DRB1*1301 is associated with protracted hepatitis A virus infection which may enhance exposure t
117 passively transferred maternal antibodies to hepatitis A virus (maternal anti-HAV) may lower the infa
118 l bacteria in the gastrointestinal tract and hepatitis A virus, may normally induce the development o
119 erved as controls, with infections by either hepatitis A virus or hepatitis B virus (HBV), or a nonin
120 tion from transcripts containing the IRES of hepatitis A virus or hepatitis C virus in BS-C-1 cells a
121 that the heterogeneous mixture of infectious hepatitis A virus particles (virions and provirions) typ
124 ing information, and did genetic analysis of hepatitis A virus recovered from patients' serum and sto
126 ad elevated titers of antibody to H. pylori, hepatitis A virus, rotavirus, and malaria at the outset,
127 unoglobulins A (IgA), G (IgG), and M (IgM)], hepatitis A virus, rotavirus, tetanus toxoid (IgG), and
128 patitis A (HepA) vaccine was assessed in 133 hepatitis A virus-seronegative, human immunodeficiency v
130 idence interval: 0.8, 2.9) of H. pylori with hepatitis A virus seroprevalence that weakened after adj
131 tion since proteins of hepatitis C virus and hepatitis A virus similarly bind IPS-1 and target it for
133 lace, and postexposure prophylaxis with both hepatitis A virus vaccine and immunoglobulin was provide
134 to 18 years of age) to receive MenB-FHbp or hepatitis A virus vaccine and saline and assigned 3304 y
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