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1 e gold sensor surface and post modified with hepatitis B surface antigen.
2 in HCC deaths from screening in carriers of hepatitis B surface antigen.
3 vitro were observed in response to RTS,S or hepatitis B surface antigen.
4 s, syphilis, human T lymphocyte virus I, and hepatitis B surface antigen.
5 he TDF+placebo group; no patient had loss of hepatitis B surface antigen.
6 y 1 patient (0.7%) in the FTC/TDF group lost hepatitis B surface antigen.
7 33 patients and staff who were positive for hepatitis B surface antigen, 30 (91%) showed complete nu
8 tis B e antigen (94% vs. 40%; P < 0.001) and hepatitis B surface antigen (71% vs 8.3%; P < 0.001).
9 ble HBV DNA in plasma and loss of detectable hepatitis B surface antigen after cessation of therapy.
10 eover, HepG2/NTCP cells secreted very little hepatitis B surface antigen after infection with cell cu
11 was defined by presence of serum antibody to hepatitis B surface antigen among children aged 2 years.
12 f a binary vector containing a gene encoding hepatitis B surface antigen and a gene encoding the kana
13 tor high-risk children at 1 year of age with hepatitis B surface antigen and anti-hepatitis B to iden
14 onal hepatocyte expansion, and expression of hepatitis B surface antigen and core antigen in liver ti
16 nations and biochemical studies were normal, hepatitis B surface antigen and hepatitis B core antigen
17 all patients were taking 3TC or FTC and were hepatitis B surface antigen and hepatitis B e antigen po
18 ected on pregnant women testing positive for hepatitis B surface antigen and on their infants, childr
19 r hepatitis B viral infection by testing for hepatitis B surface antigen and the antibody to hepatiti
21 e identified 21,419 veterans with a positive hepatitis B surface antigen, and 97% of patients had ala
22 tested for antibodies to hepatitis C virus, hepatitis B surface antigen, and IgM antibodies to hepat
23 ducation, tobacco smoking, alcohol drinking, hepatitis B surface antigen, and/or anti-hepatitis C vir
24 >6 months of age were tested for antibody to hepatitis B surface antigen (anti-HBs) 22 years later.
25 d subjects seropositive for both antibody to hepatitis B surface antigen (anti-HBs) and anti-HBc (13
26 B vaccine, and 94% demonstrated antibody to hepatitis B surface antigen (anti-HBs) at levels > or =
27 ollected in 2001 were tested for antibody to hepatitis B surface antigen (anti-HBs) by AUSAB enzyme-l
28 article vaccine) in inducing protective anti-hepatitis B surface antigen (anti-HBs) concentrations in
29 s B core antigen (anti-HBc), and antibody to hepatitis B surface antigen (anti-HBs) in 1214 serum sam
30 emic areas have reported loss of antibody to hepatitis B surface antigen (anti-HBs) in a high proport
31 e the direct detection of antibodies against hepatitis B surface antigen (anti-HBs) in clinical serum
32 ude and duration of the antibody response to hepatitis B surface antigen (anti-HBs) induced by the HB
34 icting studies about whether the antibody to hepatitis B surface antigen (anti-HBs) protects against
35 HBsAg) clearance with or without antibody to hepatitis B surface antigen (anti-HBs) seroconversion.
36 years, cumulative persistence of antibody to hepatitis B surface antigen (anti-HBs) was 85%, and cumu
38 rom OBI carriers with or without antibody to hepatitis B surface antigen (anti-HBs) were expressed in
39 children (50%) had undetectable antibody to hepatitis B surface antigen (anti-HBs), and only 8 (13%)
40 chronic (current) infection; and antibody to hepatitis B surface antigen (anti-HBs), indicative of im
42 fter the chimpanzees developed antibodies to hepatitis B surface antigen (anti-HBs), they were challe
44 presence of any of HBV serological markers (hepatitis B surface antigen, anti-hepatitis B core antib
45 oking, serum alanine aminotransferase, serum hepatitis B surface antigen, anti-hepatitis C virus anti
46 ng, elevated alanine aminotransferase, serum hepatitis B surface antigen, anti-hepatitis C virus posi
47 ent, and immunoprecipitation with monoclonal hepatitis B surface antigen antibodies (anti-HBs), indic
48 negative, over half (52.9%) had antibody to hepatitis B surface antigen antibody levels of <10 mIU/m
49 Practice Advice 2: Clinicians should screen (hepatitis B surface antigen, antibody to hepatitis B cor
50 og10 decrease in levels of serum HBV DNA and hepatitis B surface antigen, as well as liver HBV RNA, w
52 t the N terminus of the pre-S1 region of the hepatitis B surface antigen between residues 17 and 21,
53 or both human immunodeficiency virus 1/2 and hepatitis B surface antigen by Western blot and neutrali
56 roconversion (RR = 2.1, 95% CI 1.4-3.3), and hepatitis B surface antigen clearance (RR = 5.8, 95% CI
57 ersion), along with HBeAg seroconversion and hepatitis B surface antigen clearance during long-term f
58 ppression (RR = 1.4, 95% CI 1.1-1.8) but not hepatitis B surface antigen clearance or seroconversion.
59 of seroconversion (hepatitis B e antigen or hepatitis B surface antigen) compared to single drug the
60 HBs titers necessary to minimize the risk of hepatitis B surface antigen detectability were >500 IU/L
61 se of the absence of a documented history of hepatitis B surface antigen for at least six months.
62 hepatitis B infection (negative results for hepatitis B surface antigen) for findings associated wit
63 to hepatitis B core antigen, and antibody to hepatitis B surface antigen) for HBV in high-risk person
65 add-on led to significantly more decline in hepatitis B surface antigen, HBeAg, and HBV DNA (all P <
66 atitis B virus (HBV) DNA (</=2000 IU/mL) and hepatitis B surface antigen (HBsAg) (<1000 IU/mL) have b
67 odds ratio for prevalent diabetes comparing hepatitis B surface antigen (HBsAg) (+) to HBsAg (-) par
69 chronic HBV infection was assessed by serum hepatitis B surface antigen (HBsAg) and AFB1 exposure by
70 By using this strategy, we investigated the hepatitis B surface antigen (HBsAg) and alpha-fetoprotei
72 o were born after July 1987 were assayed for hepatitis B surface antigen (HBsAg) and antibodies to HB
73 studies had to report seroprevalence of both hepatitis B surface antigen (HBsAg) and antibodies to HC
74 -1-positive subjects who tested negative for hepatitis B surface antigen (HBsAg) and antibody to HBsA
75 n patients with cancer who test positive for hepatitis B surface antigen (HBsAg) and are undergoing c
77 in blood donors is achieved by screening for hepatitis B surface antigen (HBsAg) and for antibodies a
79 n be predicted by on-treatment monitoring of hepatitis B surface antigen (HBsAg) and hepatitis B viru
80 selected samples for quantitative levels of hepatitis B surface antigen (HBsAg) and hepatitis delta
81 describe the binding to MIF of two peptides, hepatitis B surface antigen (HBsAg) and insulin B (InsB)
82 tains full-length HDV RNA and an envelope of hepatitis B surface antigen (HBsAg) and is not different
88 cipients of livers from donors without serum hepatitis B surface antigen (HBsAg) but with antibody to
89 protein design to improve expression of the hepatitis B surface antigen (HBsAg) by attempting to mim
90 Mutations may also impact the detection of hepatitis B surface antigen (HBsAg) by commercial assays
91 hepatitis B e antigen (HBeAg)-seropositive, hepatitis B surface antigen (HBsAg) carrier children, wh
93 described instances of reactivation occur in hepatitis B surface antigen (HBsAg) carriers with inacti
94 t common risk factors were cohabitation with hepatitis B surface antigen (HBsAg) carriers, intravenou
97 A post-treatment response was defined as hepatitis B surface antigen (HBsAg) clearance with or wi
100 Among 2334 RA patients who had available hepatitis B surface antigen (HBsAg) data, 123 patients p
103 cent (12 of 60) of the AHBV-ALF group had no hepatitis B surface antigen (HBsAg) detectable on admiss
105 kinetics of hepatitis delta virus (HDV) and hepatitis B surface antigen (HBsAg) during interferon-al
106 cohort of women after they were screened for hepatitis B surface antigen (HBsAg) during pregnancy.
107 inical trial evaluated the immunogenicity of hepatitis B surface antigen (HBsAg) expressed in potatoe
109 clearance of hepatitis B virus (HBV) DNA and hepatitis B surface antigen (HBsAg) from serum is less o
111 , R21 particles are formed from a single CSP-hepatitis B surface antigen (HBsAg) fusion protein, and
113 liver lesions of transgenic mice bearing the hepatitis B surface antigen (HBsAg) gene of hepatitis B
116 Portal vein infusion of an rSV40 expressing hepatitis B surface antigen (HBsAg) in a naive Gunn rat
117 The age-specific seroclearance pattern of hepatitis B surface antigen (HBsAg) in chronic hepatitis
118 a showing oral immunogenicity of recombinant hepatitis B surface antigen (HBsAg) in preclinical anima
120 amuscular injection, a TAP fragment encoding hepatitis B surface antigen (HBsAg) induced anti-HBsAg a
121 which the circumsporozoite protein fused to hepatitis B surface antigen (HBsAg) is expressed togethe
125 elevated alanine aminotransferase, although hepatitis B surface antigen (HBsAg) kinetics may help pr
126 This study investigated the role of serum hepatitis B surface antigen (HBsAg) levels and establish
127 S/S gene variability has any impact on serum hepatitis B surface antigen (HBsAg) levels and to analyz
129 inetics of hepatitis B e antigen (HBeAg) and hepatitis B surface antigen (HBsAg) levels during flares
130 e profile and clinical significance of serum hepatitis B surface antigen (HBsAg) levels during long-t
131 In patients with low viral loads, higher hepatitis B surface antigen (HBsAg) levels have been sho
134 e use of organs from marginal donors who are hepatitis B surface antigen (HBsAg) negative and hepatit
135 B e antigen (HBeAg) and antibody (anti-HBe), hepatitis B surface antigen (HBsAg) or antibody (anti-HB
136 hepatitis B core antibody (anti-HBc) without hepatitis B surface antigen (HBsAg) or hepatitis B surfa
137 h acute HBV infection until the clearance of hepatitis B surface antigen (HBsAg) or the development o
138 asured in cells, extracellular vesicles, and hepatitis B surface antigen (HBsAg) particles of hepatom
139 e kidneys into recipients who are themselves hepatitis B surface antigen (HbsAg) positive with approp
143 cted persons, persons born in countries with hepatitis B surface antigen (HBsAg) prevalence of more t
144 isporivir and NIM811) on HBV replication and hepatitis B surface antigen (HBsAg) production in cell l
146 g of adult community-based screening using a hepatitis B surface antigen (HBsAg) rapid test and subse
149 utic ETV at the time of HBV reactivation and hepatitis B surface antigen (HBsAg) reverse seroconversi
150 d communities in western Gambia were offered hepatitis B surface antigen (HBsAg) screening via a poin
152 morphisms (SNPs) at HLA-DP and IL28B loci on hepatitis B surface antigen (HBsAg) seroclearance in chr
153 ed with HCV-monoinfected patients and a high hepatitis B surface antigen (HBsAg) seroclearance rate.
156 cipants, 603,585 had baseline data for serum hepatitis B surface antigen (HBsAg) status and were incl
160 le HBV viral load, 7 had positive results on hepatitis B surface antigen (HBsAg) testing and had an u
162 or Disease Control and Prevention recommends hepatitis B surface antigen (HBsAg) testing to identify
164 plasmid expression vectors for lacZ and the hepatitis B surface antigen (HBsAg) to intact skin induc
166 Intradermal immunization of mice against hepatitis B surface antigen (HBsAg) using a novel real-t
173 ies to hepatitis C virus antigen (anti-HCV), hepatitis B surface antigen (HBsAg), and antibodies to h
174 body to hepatitis B core antigen (anti-HBc), hepatitis B surface antigen (HBsAg), and antibody to hep
175 nts of serum alanine aminotransferase (ALT), hepatitis B surface antigen (HBsAg), and HBV DNA monthly
176 eponema pallidum, and hepatitis B, tests for hepatitis B surface antigen (HBsAg), and polymerase chai
178 Angeles County for the presence in serum of hepatitis B surface antigen (HBsAg), antibodies to HBsAg
179 and 72 age-matched controls were tested for hepatitis B surface antigen (HBsAg), antibodies to hepat
182 d HBV DNA from the serum; 1 went on to clear hepatitis B surface antigen (HBsAg), before transplantat
183 ultiplex cytokine measurements, quantitative hepatitis B surface antigen (HBsAg), HBeAg levels, HBV g
184 reactivation and hepatitis we identified all hepatitis B surface antigen (HBsAg), HBV DNA, and alanin
185 t of CHB based on age-specific prevalence of hepatitis B surface antigen (HBsAg), hepatitis B e antig
186 6 months thereafter, we tested for HBeAg and hepatitis B surface antigen (HBsAg), hepatitis B virus (
187 -HBc), indicative of previous HBV infection; hepatitis B surface antigen (HBsAg), indicative of chron
188 ose with human immunodeficiency virus (HIV), hepatitis B surface antigen (HBsAg), prevalent hepatic d
189 variant became detectable shortly after the hepatitis B surface antigen (HBsAg), suggesting a correl
190 119E mutation impaired detection of secreted hepatitis B surface antigen (HBsAg), suggesting immune e
192 , IL-12p70, CXCL10 and subsequent decline in hepatitis B surface antigen (HBsAg), with 20% HBsAg loss
193 spective, multicenter trial and enrolled 118 hepatitis B surface antigen (HBsAg)- and hepatitis B e a
194 l groups: HBIg-treated patients who remained hepatitis B surface antigen (HBsAg)-negative (n = 4) and
195 us (HBV) infection (OBI), serum samples from hepatitis B surface antigen (HBsAg)-negative subjects <1
196 Hepatitis B virus (HBV) reactivation in hepatitis B surface antigen (HBsAg)-negative, antibody t
199 spective, multicenter, cohort study of 2,562 hepatitis B surface antigen (HBsAg)-positive individuals
200 in (HBIg) reduces the risk of reinfection in hepatitis B surface antigen (HBsAg)-positive liver trans
201 rate of HBV infection among children born to hepatitis B surface antigen (HBsAg)-positive mothers and
202 ased on findings from a prospective study of hepatitis B surface antigen (HBsAg)-positive patients, w
203 as detected by TaqMan in 164 of 195 (84%) of hepatitis B surface antigen (HBsAg)-positive samples.
204 on-years, respectively, among women who were hepatitis B surface antigen (HBsAg)-seronegative and HBs
213 isone) chemotherapy: screen all patients for hepatitis B surface antigen (HBsAg; Screen-All), screen
214 viewed our experience with liver grafts from hepatitis-B surface antigen (HBsAg)(-), antibody to core
215 homa patients with "resolved" HBV infection (hepatitis B surface antigen [HBsAg] negative and hepatit
216 nts either untreated (25) or NUC treated (36 hepatitis B surface antigen [HBsAg](+) and 10 HBsAg(-)/h
217 510A greatly reduced small envelope protein (hepatitis B surface antigen [HBsAg]) levels both inside
218 ating the request for a serological profile (hepatitis B surface antigen [HBsAg], anti-HBc, and anti-
219 mending screening for hepatitis B infection (hepatitis B surface antigen [HBsAg], antihepatitis B cor
220 fined HBV coinfection as a positive test for hepatitis B surface antigen, HBV DNA, or hepatitis B e a
221 spectively for human immunodeficiency virus, hepatitis B surface antigen, hepatitis C virus, and huma
222 tectable HBV DNA serum levels or antibody to hepatitis B surface antigen/hepatitis B surface antigen
225 dence of ongoing viral infection: persistent hepatitis B surface antigen in three (6%), HCV antibody
226 ts also responded to later vaccinations with hepatitis B surface antigen, indicating that no immunolo
230 for liver injury (i.e., negative HCV RNA and hepatitis B surface antigen, low alcohol consumption, no
232 cy virus (HIV), hepatitis C virus (HCV), and hepatitis B surface antigen nearly tripled between 1 wee
233 The patient is clinically normal, serum hepatitis B surface antigen negative at 10 months of fol
234 ivation in patients with resolved infection (hepatitis B surface antigen negative) receiving chemothe
237 vious or occult HBV infection in a cohort of hepatitis B surface antigen-negative patients with histo
238 Hepatitis B virus infection originating from hepatitis B surface antigen-negative, hepatitis B core a
239 han genotype 1b, if they tested positive for hepatitis B surface antigen or anti-HIV antibody at scre
240 ze (OR, 1.23; 95% CI: 1.09-1.40; P = 0.001), hepatitis B surface antigen, or anti-hepatitis C virus p
242 ERA plasmid DNA and a plasmid expressing the hepatitis B surface antigen (pCMV-s) by the i.m. route r
243 s part of an intradermal delivery system for hepatitis B surface antigen, PCPP demonstrates superior
244 developed de novo HBV: two recipients became hepatitis B surface antigen positive at 70 and 23 months
246 eated patients who were HBV DNA negative but hepatitis B surface antigen positive had lower levels of
247 tigen positive) or partial (HBV DNA negative/hepatitis B surface antigen positive) control of their i
248 o had complete (HBV DNA negative/antibody to hepatitis B surface antigen positive) or partial (HBV DN
249 ) were HCV-antibody positive, 85 (7.4%) were hepatitis B surface antigen positive, and 17 (1.5%) were
250 luded the following: illicit drug use, serum hepatitis B surface antigen positive, grade 1 encephalop
252 patitis B vaccine prophylaxis of newborns of hepatitis B surface antigen-positive mothers, were extra
253 ery during ART was significantly impaired in hepatitis B surface antigen-positive patients and in tho
254 tive EGC-HCC association became stronger for hepatitis B surface antigen-positive persons who also ha
257 holic drinks per week (IRR, 1.68, P < .001), hepatitis B surface antigen positivity (IRR, 1.68, P < .
258 ng for age at HCV infection, past or current hepatitis B surface antigen positivity, and history of a
259 th human immunodeficiency virus coinfection, hepatitis B surface antigen positivity, cirrhosis, and H
260 ing those with human immunodeficiency virus, hepatitis B surface antigen positivity, hepatocellular c
261 This 120 kDa fusion protein between the hepatitis B surface antigen preS2 sequence and beta-gala
262 ization of the baboon fetus with recombinant hepatitis B surface antigen produced a specific fetal Ig
263 n dipeptidyl peptidase IV or from transgenic hepatitis B surface antigen-producing G26 mice with hepa
264 apy, the estimated overall seroprevalence of hepatitis B surface antigen remains high at 6.1% (95% un
265 plete HBV genome and the gene coding for the hepatitis B surface antigen (S gene) identified sequence
272 l therapy reduced MTCT, as defined by infant hepatitis B surface antigen seropositivity (risk ratio =
273 ional adjustment for liver enzyme levels and hepatitis B surface antigen status (men: HR, 0.60; P tre
274 status, ascertained semiannually, and their hepatitis-B surface antigen status (HBsAg), which we asc
275 n wild-type mice, and in mice that carried a hepatitis B surface antigen transgene-this to model the
276 hepatitis B surface antigen mice, expressing hepatitis B surface antigen under control of the albumin
279 ositive patients lost hepatitis B e antigen; hepatitis B surface antigen was undetectable in 3 (6%);
280 onal cure characterized by sustained loss of hepatitis B surface antigen with or without hepatitis B
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