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1  the correct schedule and before exposure to hepatitis B virus.
2  A3s against retroviruses, parvoviruses, and hepatitis B virus.
3 gle-stranded DNA (ssDNA) parvovirus, but not hepatitis B virus.
4                                 The study of hepatitis B virus and development of curative antivirals
5 es in the longer term through the control of hepatitis B virus and hepatitis C virus infections by va
6 ecrosis factors [screening tests looking for hepatitis B virus and hepatitis C virus status and laten
7 ruses (Africa for HIV and southeast Asia for hepatitis B virus and hepatitis C virus).
8                       We analyzed changes in hepatitis B virus and hepatitis delta virus (HDV) viral
9          Sharing the same envelope proteins, hepatitis B virus and hepatitis delta virus use the sodi
10 eptor mediating hepatocyte-specific entry of hepatitis B virus and hepatitis delta virus.
11               We also confirm suppression of hepatitis B virus and poliovirus by ARB.
12 on-Pfizer monkey virus), two hepadnaviruses (hepatitis B virus and woodchuck hepatitis virus), and an
13 sponsible for significant disease in humans (hepatitis B virus) and have been reported from a diverse
14 pentavalent (diphtheria, tetanus, pertussis, hepatitis B virus, and Haemophilus influenzae type b), y
15 d DARE for studies of the prevalence of HIV, hepatitis B virus, and hepatitis C virus in people with
16  hepatitis C virus with and without alcohol, hepatitis B virus, and hepatocellular carcinoma (group I
17 tomegalovirus, human immunodeficiency virus, hepatitis B virus, and neonatal herpes simplex virus, fr
18 Bx, restores replication of an HBx-deficient hepatitis B virus, and rescues wild-type hepatitis B vir
19 s SERS assay was applied to the detection of Hepatitis B virus antigen (HBsAg) in human blood plasma.
20                  The low detection limit for Hepatitis B virus antigen was estimated to be 0.01IU/mL.
21 s pollen allergens and the PreS protein from hepatitis B virus as a carrier were expressed in Escheri
22 ses, including influenza A virus (FLUAV) and hepatitis B virus but not retroviruses.
23 e show that cell-culture and patient-derived hepatitis B virus can establish persistent infection for
24                                          The hepatitis B virus capsid (core) can be composed of eithe
25                                      Chronic hepatitis B virus carriers are at risk of developing fib
26                                              Hepatitis B virus causes chronic infections in 250 milli
27 rcumcision, home birth, tribal scarring, and hepatitis B virus coinfection.
28  model of hepatitis B: infectious transgenic hepatitis B virus composed of a complete virus plus a fo
29 pid, high yield and economical production of Hepatitis B Virus core (HBc) particles.
30 s associated with a similar modest change in hepatitis B virus core antigen polypeptide (HBcAg/p21) s
31 ) and VP2 (aa141-155) epitopes of EV71 using hepatitis B virus core protein (HBc) as a carrier, desig
32                                              Hepatitis B virus core protein has 183 amino acids divid
33                                              Hepatitis B virus core protein has multiple roles in the
34  for novel biomarkers toward better defining hepatitis B virus cure should occur in parallel with dev
35  will likely be needed to achieve functional hepatitis B virus cure.
36                                          The hepatitis B virus deploys the hepatitis B virus X protei
37                              The Pol of duck hepatitis B virus (DHBV) also localized to the mitochond
38 (FISH)-based assay for the detection of duck hepatitis B virus (DHBV) cccDNA and HBV nuclear DNA in e
39              For the avian hepadnavirus duck hepatitis B virus (DHBV), CTD is dephosphorylated subseq
40 antly differ according to the VR definition (hepatitis B virus DNA <200, < 2000, < 20,000 IU/mL) or d
41                           The peak levels of hepatitis B virus DNA and hepatitis B core-related antig
42                                   Rebound of hepatitis B virus DNA and hepatitis B core-related antig
43 toring of CHB patients with detectable serum hepatitis B virus DNA in European tertiary referral cent
44 =1 alanine aminotransferase and 37% had >/=1 hepatitis B virus DNA level assessed annually.
45                                              Hepatitis B virus DNA targets in human serum are simulta
46 nts had alanine aminotransferase and 44% had hepatitis B virus DNA testing; hepatitis B e antigen and
47                                              Hepatitis B virus encodes the regulatory HBx protein who
48 f viruses, including HIV, hepatitis C virus, hepatitis B virus, enterovirus 71, influenza virus, resp
49          Myrcludex B, a peptide inhibitor of hepatitis B virus entry, is assumed to specifically targ
50                            New inhibitors of hepatitis B virus entry, replication, assembly, or secre
51 sue of Immunity, Ou and colleagues show that hepatitis B virus exploits these naturally occurring imm
52       The management for occupational HIV or hepatitis B virus exposures includes postexposure prophy
53  relieves the inhibition to allow productive hepatitis B virus gene expression.
54 ates with extrachromosomal reporters and the hepatitis B virus genome, suggesting a direct mechanism
55                         Chronic infection by hepatitis B virus (HBV) afflicts hundreds of millions wo
56 valence of HIV, hepatitis C virus (HCV), and hepatitis B virus (HBV) among PWID.
57                                              Hepatitis B virus (HBV) and hepatitis C virus (HCV) are
58                                              Hepatitis B virus (HBV) and hepatitis C virus (HCV) caus
59                      Advances in therapy for hepatitis B virus (HBV) and hepatitis C virus (HCV) have
60                              Whether chronic hepatitis B virus (HBV) and hepatitis C virus (HCV) infe
61 uss common challenges to the burden posed by hepatitis B virus (HBV) and hepatitis C virus (HCV), to
62                    Infections with the human hepatitis B virus (HBV) and hepatitis D virus (HDV) depe
63 tions and displays excellent potency against hepatitis B virus (HBV) and varicella-zoster virus (VZV)
64                              We investigated hepatitis B virus (HBV) antibodies and galactomannan enz
65           Outcomes of chronic infection with hepatitis B virus (HBV) are varied, with increased morbi
66                                  Assembly of hepatitis B virus (HBV) begins with packaging of the pre
67    In contrast to horizontal transmission of hepatitis B virus (HBV) between adults, which often lead
68 transferase 1 (PRMT1) only modestly increase hepatitis B virus (HBV) biosynthesis.
69                    Cell culture (cc)-derived hepatitis B virus (HBV) can infect differentiated HepaRG
70 been used to track the assembly of the T = 4 hepatitis B virus (HBV) capsid in real time.
71                                              Hepatitis B virus (HBV) capsid proteins (Cps) assemble a
72 ices to analyze in real time the assembly of Hepatitis B Virus (HBV) capsids below the pseudocritical
73                                          The hepatitis B virus (HBV) causes acute and chronic liver i
74                                              Hepatitis B virus (HBV) chronic infection affects up to
75                                              Hepatitis B virus (HBV) chronically infects 250 million
76 re (ACLF) were developed in patients with no Hepatitis B virus (HBV) cirrhosis (CANONIC study).
77 rus (HIV)-infected patients with and without hepatitis B virus (HBV) coinfection on antiretroviral th
78                               HDV requires a hepatitis B virus (HBV) coinfection to provide HDV with
79 se associated with persistent infection with hepatitis B virus (HBV) continues to be a major health p
80                    Although an isolated anti-hepatitis B virus (HBV) core antibody (anti-HBc) serolog
81 tion state of the C-terminal domain (CTD) of hepatitis B virus (HBV) core or capsid protein is highly
82                             Mutations in the hepatitis B virus (HBV) core promoter (CP) have been sho
83                     Multiple subunits of the hepatitis B virus (HBV) core protein (HBc) assemble into
84                                   Though the hepatitis B virus (HBV) core protein is an important par
85                                          The hepatitis B virus (HBV) core protein is essential for HB
86                                              Hepatitis B virus (HBV) covalently closed circular (CCC)
87 tion and the factors involved.IMPORTANCE The hepatitis B virus (HBV) covalently closed circular (CCC)
88            Currently in vitro infection with hepatitis B virus (HBV) depends on cell culture-derived
89                              Serum levels of hepatitis B virus (HBV) DNA (</=2000 IU/mL) and hepatiti
90 ified APOBEC deaminases as enzymes targeting hepatitis B virus (HBV) DNA in the nucleus thus affectin
91 ith nucleos(t)ide analogues (NAs) suppresses hepatitis B virus (HBV) DNA production but does not affe
92 r chronic hepatitis B involve suppression of hepatitis B virus (HBV) DNA with the use of nucleoside a
93 nd 95%, 99%, 100%, and 100% had undetectable hepatitis B virus (HBV) DNA, respectively.
94 plantation, 39 (72%) patients had detectable hepatitis B virus (HBV) DNA, with a median of 4.5 log co
95                                         Anti-hepatitis B virus (HBV) drugs are currently limited to n
96                            The management of hepatitis B virus (HBV) e antigen-positive viremic patie
97                                              Hepatitis B virus (HBV) encodes a multifunction reverse
98 the human immunodeficiency virus (HIV) gene, hepatitis B virus (HBV) gene and human T-lymphotropic vi
99                              To evaluate how hepatitis B virus (HBV) genetic variation affected progr
100                                              Hepatitis B virus (HBV) genotype and its role in disease
101                                              Hepatitis B virus (HBV) genotype C causes prolonged chro
102                                              Hepatitis B virus (HBV) genotypes can influence treatmen
103                                              Hepatitis B virus (HBV) has a detrimental effect on huma
104                                          The hepatitis B virus (HBV) has been described as stealth vi
105                              Reactivation of hepatitis B virus (HBV) has been reported in hepatitis C
106 tion, and HBV-driven tumor growth.IMPORTANCE Hepatitis B virus (HBV) HBx protein plays a critical rol
107                                              Hepatitis B virus (HBV) immunization has been effectivel
108 dvice 1: Clinicians should vaccinate against hepatitis B virus (HBV) in all unvaccinated adults (incl
109             The basis for the persistence of hepatitis B virus (HBV) in hepatocytes, even in the pres
110 Despite the introduction of immunisation for hepatitis B virus (HBV) in the 1990s, HBV-related morbid
111                                      Chronic hepatitis B virus (HBV) infection affects 240 million pe
112                                              Hepatitis B virus (HBV) infection afflicts millions worl
113 iviral treatment-naive patients with chronic hepatitis B virus (HBV) infection but not in treatment-e
114 optosis proteins (cIAPs) impair clearance of hepatitis B virus (HBV) infection by preventing TNF-medi
115                                              Hepatitis B virus (HBV) infection can result in a spectr
116 (RNAi)-based therapeutic ARC-520 for chronic hepatitis B virus (HBV) infection consists of a melittin
117 tive prevention and treatment interventions, hepatitis B virus (HBV) infection continues to cause nea
118        What are the screening guidelines for hepatitis B virus (HBV) infection for asymptomatic, nonp
119 rugs in patients with HBeAg-negative chronic hepatitis B virus (HBV) infection in a non-inferiority s
120 rugs in patients with HBeAg-positive chronic hepatitis B virus (HBV) infection in a non-inferiority s
121 iral compounds.The lack of models that mimic hepatitis B virus (HBV) infection in a physiologically r
122                          However, studies of hepatitis B virus (HBV) infection in chimpanzees have in
123 iated with diabetes in patients with chronic hepatitis B virus (HBV) infection in North America are u
124                   Despite the high burden of hepatitis B virus (HBV) infection in sub-Saharan Africa,
125           The number of persons with chronic hepatitis B virus (HBV) infection in the United States i
126 regarding the prevalence and distribution of hepatitis B virus (HBV) infection in U.S. Hispanics/Lati
127                                      Chronic hepatitis B virus (HBV) infection is a common cause of t
128                                      Chronic hepatitis B virus (HBV) infection is a global public hea
129                                      Chronic hepatitis B virus (HBV) infection is a global public hea
130                                      Chronic hepatitis B virus (HBV) infection is a major factor in h
131                                      Chronic hepatitis B virus (HBV) infection is a major global heal
132                                      Chronic hepatitis B virus (HBV) infection is a major risk factor
133                                      Chronic hepatitis B virus (HBV) infection is a major risk factor
134                                              Hepatitis B virus (HBV) infection is a serious public he
135                                   Persistent hepatitis B virus (HBV) infection is established by the
136                                      Chronic hepatitis B virus (HBV) infection is estimated to affect
137                                      Chronic Hepatitis B Virus (HBV) infection is generally not curab
138                                              Hepatitis B virus (HBV) infection is more common in Afri
139                                      Chronic hepatitis B virus (HBV) infection is partly responsible
140                                      Chronic hepatitis B virus (HBV) infection is prevalent, deadly,
141                        A hallmark of chronic hepatitis B virus (HBV) infection is the functional impa
142                                      Chronic hepatitis B virus (HBV) infection often develop into cir
143 on or cirrhosis due to other causes, such as hepatitis B virus (HBV) infection or alcohol, remains un
144 he heterogeneous clinical courses of chronic hepatitis B virus (HBV) infection reflect the complex ho
145                                      Chronic hepatitis B virus (HBV) infection remains the most commo
146                                              Hepatitis B virus (HBV) infection represents a significa
147                 Vaccine failure with chronic hepatitis B virus (HBV) infection still develops in chil
148                       Patients with resolved hepatitis B virus (HBV) infection who are treated for he
149         It can occur with active or resolved hepatitis B virus (HBV) infection with a clinical spectr
150 ions have among the highest rates of chronic hepatitis B virus (HBV) infection worldwide, but little
151 hepatitis C virus (HCV) infection, 17.9% had hepatitis B virus (HBV) infection, and 2.2% had both.
152 achieving better treatment and prevention of hepatitis B virus (HBV) infection, anti-HBV agents targe
153 govern distinct clinical phases of a chronic hepatitis B virus (HBV) infection-immune tolerant (IT),
154 s and are particularly promising for chronic hepatitis B virus (HBV) infection.
155 n patients with current or prior exposure to hepatitis B virus (HBV) infection.
156  the liver diseases that result from chronic hepatitis B virus (HBV) infection.
157 atory effect are rarely addressed in chronic hepatitis B virus (HBV) infection.
158 surface Ag (HBsAg) seroconversion in chronic hepatitis B virus (HBV) infection.
159  Greenland remains a highly endemic area for hepatitis B virus (HBV) infection.
160 th metabolic risk factors, for patients with hepatitis B virus (HBV) infection.
161 rcinoma (HCC), often associated with chronic hepatitis B virus (HBV) infection.
162 age to care can reduce the burden of chronic hepatitis B virus (HBV) infection.
163                      An estimated 20 000 new hepatitis B virus (HBV) infections occur each year in th
164 patitis B surface antigen (HBsAg) in chronic hepatitis B virus (HBV) infections of China remains uncl
165 presents an endpoint of treatment of chronic hepatitis B virus (HBV) infections.
166                           BACKGROUND & AIMS: Hepatitis B virus (HBV) infects hepatocytes, but the mec
167                                              Hepatitis B virus (HBV) infects hundreds of millions of
168                                              Hepatitis B virus (HBV) is a major global health concern
169                                              Hepatitis B virus (HBV) is a major human pathogen, and a
170 plication-competent viral capsids.IMPORTANCE Hepatitis B virus (HBV) is a major human pathogen, and n
171       Mother-to-child transmission (MTCT) of hepatitis B virus (HBV) is a major route of HBV transmis
172                       Chronic infection with hepatitis B virus (HBV) is a risk factor for developing
173                       Chronic infection with hepatitis B virus (HBV) is a risk factor for developing
174                                              Hepatitis B virus (HBV) is an important global human pat
175 hronic hepatitis B (CHB), failure to control hepatitis B virus (HBV) is associated with T cell dysfun
176                                              Hepatitis B virus (HBV) is endemic in sub-Saharan Africa
177  antiviral regimens with no activity against hepatitis B virus (HBV) may increase the risk for HBV re
178                                              Hepatitis B virus (HBV) modulates microRNA (miRNA) expre
179         We examined the associations between hepatitis B virus (HBV) or hepatitis C virus (HCV) infec
180 ), most cases of which are related to either hepatitis B virus (HBV) or hepatitis C virus (HCV).
181 -replicative (HBV DNA <20,000 IU/mL) chronic hepatitis B virus (HBV) patients.
182 ron gamma and interleukin 10) to overlapping hepatitis B virus (HBV) peptides (preS, S, preC, core, a
183                       Chronic infection with hepatitis B virus (HBV) progresses through different pha
184 racterizes and defines the clinical value of hepatitis B virus (HBV) quasispecies with reverse transc
185           Reports were published recently on hepatitis B virus (HBV) reactivation (HBV-R) in patients
186                                              Hepatitis B virus (HBV) reactivation during immunosuppre
187                                              Hepatitis B virus (HBV) reactivation has been reported i
188                                              Hepatitis B virus (HBV) reactivation in hepatitis B surf
189 udies report conflicting data on the risk of hepatitis B virus (HBV) reactivation in rheumatologic pa
190  tumor chemotherapy regimens pose a risk for hepatitis B virus (HBV) reactivation, but screening and
191                Immunosuppressants can induce hepatitis B virus (HBV) reactivation; however, informati
192 ate cotransporting polypeptide (NTCP) as the hepatitis B virus (HBV) receptor enabled researchers to
193 noglobulin (HBIg) is effective in preventing hepatitis B virus (HBV) recurrence after liver transplan
194 an integral component of prophylaxis against hepatitis B virus (HBV) recurrence in liver transplantat
195 al or mother-to-child transmission (MTCT) of hepatitis B virus (HBV) remains the major risk factor fo
196 eraction mechanisms for achieving an optimal hepatitis B virus (HBV) replication have been largely un
197                                              Hepatitis B Virus (HBV) replication in hepatocytes is re
198 es, such as entecavir or tenofovir, suppress hepatitis B virus (HBV) replication, improve liver funct
199           To understand subcellular sites of hepatitis B virus (HBV) replication, we visualized core
200 osphates (dNTPs) are essential for efficient hepatitis B virus (HBV) replication.
201     Persistent or chronic infection with the hepatitis B virus (HBV) represents one of the most commo
202                               Infection with hepatitis B virus (HBV) results in disparate degrees of
203 ation, there is still evidence of suboptimal hepatitis B virus (HBV) screening among patients at high
204 pies according to hepatitis C virus (HCV) or hepatitis B virus (HBV) status.
205  B vaccine, despite loss of antibody against hepatitis B virus (HBV) surface antigen (anti-HBs), is u
206  natural killer (NK) cells in the process of hepatitis B virus (HBV) surface antigen (HBsAg) clearanc
207                       Humans vaccinated with hepatitis B virus (HBV) surface antigen (HBsAg) sometime
208                  REP 2139 clears circulating hepatitis B virus (HBV) surface antigen (HBsAg), enhanci
209       Here, we investigated the CTD in empty hepatitis B virus (HBV) T=4 capsids.
210                                          New hepatitis B virus (HBV) therapies are expected to have b
211                   Despite immunoprophylaxis, hepatitis B virus (HBV) transmission in highly viremic m
212 ctiveness of strategies to prevent perinatal hepatitis B virus (HBV) transmission in the United State
213  fumarate (TDF) in reducing mother-to-infant hepatitis B virus (HBV) transmissions is not clearly und
214                                              Hepatitis B virus (HBV) transmitted from infected mother
215                 Here, the CTD from the human hepatitis B virus (HBV) was found to be dephosphorylated
216                               Sequences from hepatitis B virus (HBV) were detected in 7 individuals w
217 tide sequence characteristic of DNA from the hepatitis B virus (HBV) with a detection limit of 85 pM.
218                                              Hepatitis B virus (HBV), a small enveloped DNA virus, ch
219  to identify NAFLD, hepatitis C virus (HCV), hepatitis B virus (HBV), alcoholic liver disease (ALD),
220     Previous estimates of the burden of HIV, hepatitis B virus (HBV), and hepatitis C virus (HCV) amo
221 cquiring human immunodeficiency virus (HIV), hepatitis B virus (HBV), and hepatitis C virus (HCV).
222 f 47,591 adults wait-listed for LT from HCV, hepatitis B virus (HBV), and nonalcoholic steatohepatiti
223 pidemiology of HIV, hepatitis C virus (HCV), hepatitis B virus (HBV), and tuberculosis in prisoners.
224                                              Hepatitis B virus (HBV), belonging to Hepadnaviridae fam
225  role of the transcriptional template of the hepatitis B virus (HBV), covalently closed circular DNA
226 atitis delta virus (HDV), a satellite of the hepatitis B virus (HBV), increases viral liver disease s
227 is in patients with hepatitis C virus (HCV), hepatitis B virus (HBV), NAFLD, and alcoholic liver dise
228 th infections by either hepatitis A virus or hepatitis B virus (HBV), or a noninfectious cause for th
229 otype 1 hepatitis C virus (HCV) and 203 with hepatitis B virus (HBV), respectively.
230 of the reverse transcriptase protein (RT) of hepatitis B virus (HBV), sampled from patients with rapi
231           Here, using mutational analyses of hepatitis B virus (HBV), we found that Hsp90 stimulates
232 eral common loci associated with the risk of hepatitis B virus (HBV)- or hepatitis C virus (HCV)-rela
233 The incidences of chronic hepatitis B (CHB), Hepatitis B virus (HBV)-associated cirrhosis and HBV-ass
234                                              Hepatitis B virus (HBV)-encoded X protein (HBx) plays a
235 pha elicits a pleiotropic antiviral state in hepatitis B virus (HBV)-infected hepatocytes, but whethe
236                  We have sampled healthy and hepatitis B virus (HBV)-infected human livers to probe f
237 disturbance of Mg(2+) homeostasis on chronic hepatitis B virus (HBV)-infected natural killer (NK) and
238                    Elucidating mechanisms of hepatitis B virus (HBV)-mediated hepatocarcinogenesis is
239                                In this study hepatitis B virus (HBV)-naive older adults received thre
240 n conducted on the genetic susceptibility of hepatitis B virus (HBV)-related hepatocellular carcinoma
241                                Activation of hepatitis B virus (HBV)-specific CD8 T cells by therapeu
242                                Activated and hepatitis B virus (HBV)-specific T cells, particularly t
243 cy virus (HIV), hepatitis C virus (HCV), and hepatitis B virus (HBV).
244 is delta virus (HDV) is a satellite virus of hepatitis B virus (HBV).
245 on, Kupffer cells (KCs) likely interact with hepatitis B virus (HBV).
246 f death for patients coinfected with HIV and hepatitis B virus (HBV).
247 compatible with acute, resolved, and chronic hepatitis B virus (HBV)infection but might also signify
248  elevated in HCV+ liver biopsies compared to hepatitis B virus (HBV+) and uninfected samples.
249 d with hepatitis C virus [HCV] and five with hepatitis B virus [HBV]) of 600 HIV-infected patients wh
250 tors and centered on CTNNB1 (alcohol), TP53 (hepatitis B virus, HBV) and AXIN1.
251                                              Hepatitis B viruses (HBVs), which are enveloped viruses
252                              Hepadnaviruses (hepatitis B viruses [HBVs]) are the only animal viruses
253                                 Treatment of hepatitis B virus, hepatitis C virus, and acute T cell-m
254 a number of major human pathogens, including hepatitis B virus, hepatitis C virus, and malaria.
255 re found in the sera from patients with AIH, hepatitis B virus, hepatitis C virus, and nonalcoholic s
256                       The prevalence of HIV, hepatitis B virus, hepatitis C virus, and tuberculosis a
257 ent hepatitis B virus, and rescues wild-type hepatitis B virus in a DDB1-knockdown background.
258 of the pregenomic RNA encapsidation in human Hepatitis B virus in vivo using a coarse-grained molecul
259 FNalpha was confirmed in HEV gt1, but not in Hepatitis B Virus infected animals.
260  drug-induced liver injury DILI (22%), acute hepatitis B virus infection (12%), autoimmune hepatitis
261  C virus infection (41%), alcohol (39%), and hepatitis B virus infection (22%) the commonest etiologi
262 an guidelines recommend treatment of chronic hepatitis B virus infection (CHB) with the nucleos(t)ide
263  A cross-sectional analysis of prevalence of hepatitis B virus infection (HBV) among rural couples wa
264                                      Chronic hepatitis B virus infection is a leading cause of cirrho
265 f LT, center, and hepatitis C virus (HCV) or hepatitis B virus infection) with non-HIV-infected contr
266 B vaccine is an effective measure to prevent hepatitis B virus infection.
267 with HIV infection are at increased risk for hepatitis B virus infection.
268  and obesity; 12.5% (95% CI: 10.6-14.3%) for hepatitis B virus infection; 29.1% (95% CI: 23.6-34.5%)
269  copies per mL); HLA-B*5701-negative; had no hepatitis B virus infection; screening genotypes showing
270                                     However, hepatitis B virus inhibited the AIM2 inflammasome by red
271                         For the discovery of hepatitis B virus integration sites from probe capture d
272                                              Hepatitis B virus is one of the smallest human pathogens
273             HCV is an RNA virus that, unlike hepatitis B virus, is unable to integrate into the host
274  might perform distinct functions during the hepatitis B virus lifecycle.
275 9 HDV-infected patients, 25 individuals with hepatitis B virus monoinfection and 18 healthy controls.
276                             Formation of the hepatitis B virus nucleocapsid is an essential step in t
277                                      Chronic hepatitis B virus or hepatitis C co-infection was allowe
278                  Patients with co-infection (hepatitis B virus or HIV infection), evidence of decompe
279                       Other viruses, such as hepatitis B virus or human herpesvirus 8 (HHV-8), establ
280 e (PR = 2.57; 95% CI: 2.33-2.84; P = 0.001), hepatitis B virus (PR = 1.32; 95% CI: 1.09-1.59; P = 0.0
281 lly regulates several direct target genes of hepatitis B virus protein X (HBx), a viral co-factor.
282                                              Hepatitis B virus reactivation (HBVr) is an important co
283                                              Hepatitis B virus reactivation is a newly identified saf
284                                              Hepatitis B virus reactivation, defined as an abrupt inc
285  transfer of T cells engineered to express a hepatitis B virus-specific (HBV-specific) T cell recepto
286 were available on covariates, such as HIV or hepatitis B virus status, for subgroup analyses.
287 axis, mother-to-child transmission (MTCT) of Hepatitis B Virus still occurs in approximately 2-5% of
288 yme as signal amplifier for determination of hepatitis B virus surface antigen (HBsAg).
289  for malignant disease, testing positive for hepatitis B virus surface antigen, pregnancy, creatinine
290  genomes, such as those of papillomavirus or hepatitis B virus that induce specific cancers.
291                                           In hepatitis B virus, this is manifested as a virion where
292 th on extrachromosomal reporter genes and on hepatitis B virus transcription.
293 ment of hepatitis C virus and suppression of hepatitis B virus, treatment and management principles f
294 onses to protein-based vaccines, such as the Hepatitis B virus vaccine formulated in alum.
295 n of CpG-C, using an interval similar to the Hepatitis B virus vaccine.
296  in Egypt (1054 [84%] of 1251 patients), and hepatitis B virus was the leading cause in the other Afr
297  of this algorithm is demonstrated using the Hepatitis B virus X (HBx) protein, a protein of unknown
298            The hepatitis B virus deploys the hepatitis B virus X protein (HBx) as a suppressor of hos
299 mined in HCC that spontaneously developed in hepatitis B virus X protein (HBx) transgenic mice.
300                                The enigmatic hepatitis B virus X protein was used as a test case for

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