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1 the correct schedule and before exposure to hepatitis B virus.
2 A3s against retroviruses, parvoviruses, and hepatitis B virus.
3 gle-stranded DNA (ssDNA) parvovirus, but not hepatitis B virus.
5 es in the longer term through the control of hepatitis B virus and hepatitis C virus infections by va
6 ecrosis factors [screening tests looking for hepatitis B virus and hepatitis C virus status and laten
12 on-Pfizer monkey virus), two hepadnaviruses (hepatitis B virus and woodchuck hepatitis virus), and an
13 sponsible for significant disease in humans (hepatitis B virus) and have been reported from a diverse
14 pentavalent (diphtheria, tetanus, pertussis, hepatitis B virus, and Haemophilus influenzae type b), y
15 d DARE for studies of the prevalence of HIV, hepatitis B virus, and hepatitis C virus in people with
16 hepatitis C virus with and without alcohol, hepatitis B virus, and hepatocellular carcinoma (group I
17 tomegalovirus, human immunodeficiency virus, hepatitis B virus, and neonatal herpes simplex virus, fr
18 Bx, restores replication of an HBx-deficient hepatitis B virus, and rescues wild-type hepatitis B vir
19 s SERS assay was applied to the detection of Hepatitis B virus antigen (HBsAg) in human blood plasma.
21 s pollen allergens and the PreS protein from hepatitis B virus as a carrier were expressed in Escheri
23 e show that cell-culture and patient-derived hepatitis B virus can establish persistent infection for
28 model of hepatitis B: infectious transgenic hepatitis B virus composed of a complete virus plus a fo
30 s associated with a similar modest change in hepatitis B virus core antigen polypeptide (HBcAg/p21) s
31 ) and VP2 (aa141-155) epitopes of EV71 using hepatitis B virus core protein (HBc) as a carrier, desig
34 for novel biomarkers toward better defining hepatitis B virus cure should occur in parallel with dev
38 (FISH)-based assay for the detection of duck hepatitis B virus (DHBV) cccDNA and HBV nuclear DNA in e
40 antly differ according to the VR definition (hepatitis B virus DNA <200, < 2000, < 20,000 IU/mL) or d
43 toring of CHB patients with detectable serum hepatitis B virus DNA in European tertiary referral cent
46 nts had alanine aminotransferase and 44% had hepatitis B virus DNA testing; hepatitis B e antigen and
48 f viruses, including HIV, hepatitis C virus, hepatitis B virus, enterovirus 71, influenza virus, resp
51 sue of Immunity, Ou and colleagues show that hepatitis B virus exploits these naturally occurring imm
54 ates with extrachromosomal reporters and the hepatitis B virus genome, suggesting a direct mechanism
61 uss common challenges to the burden posed by hepatitis B virus (HBV) and hepatitis C virus (HCV), to
63 tions and displays excellent potency against hepatitis B virus (HBV) and varicella-zoster virus (VZV)
67 In contrast to horizontal transmission of hepatitis B virus (HBV) between adults, which often lead
72 ices to analyze in real time the assembly of Hepatitis B Virus (HBV) capsids below the pseudocritical
77 rus (HIV)-infected patients with and without hepatitis B virus (HBV) coinfection on antiretroviral th
79 se associated with persistent infection with hepatitis B virus (HBV) continues to be a major health p
81 tion state of the C-terminal domain (CTD) of hepatitis B virus (HBV) core or capsid protein is highly
87 tion and the factors involved.IMPORTANCE The hepatitis B virus (HBV) covalently closed circular (CCC)
90 ified APOBEC deaminases as enzymes targeting hepatitis B virus (HBV) DNA in the nucleus thus affectin
91 ith nucleos(t)ide analogues (NAs) suppresses hepatitis B virus (HBV) DNA production but does not affe
92 r chronic hepatitis B involve suppression of hepatitis B virus (HBV) DNA with the use of nucleoside a
94 plantation, 39 (72%) patients had detectable hepatitis B virus (HBV) DNA, with a median of 4.5 log co
98 the human immunodeficiency virus (HIV) gene, hepatitis B virus (HBV) gene and human T-lymphotropic vi
106 tion, and HBV-driven tumor growth.IMPORTANCE Hepatitis B virus (HBV) HBx protein plays a critical rol
108 dvice 1: Clinicians should vaccinate against hepatitis B virus (HBV) in all unvaccinated adults (incl
110 Despite the introduction of immunisation for hepatitis B virus (HBV) in the 1990s, HBV-related morbid
113 iviral treatment-naive patients with chronic hepatitis B virus (HBV) infection but not in treatment-e
114 optosis proteins (cIAPs) impair clearance of hepatitis B virus (HBV) infection by preventing TNF-medi
116 (RNAi)-based therapeutic ARC-520 for chronic hepatitis B virus (HBV) infection consists of a melittin
117 tive prevention and treatment interventions, hepatitis B virus (HBV) infection continues to cause nea
119 rugs in patients with HBeAg-negative chronic hepatitis B virus (HBV) infection in a non-inferiority s
120 rugs in patients with HBeAg-positive chronic hepatitis B virus (HBV) infection in a non-inferiority s
121 iral compounds.The lack of models that mimic hepatitis B virus (HBV) infection in a physiologically r
123 iated with diabetes in patients with chronic hepatitis B virus (HBV) infection in North America are u
126 regarding the prevalence and distribution of hepatitis B virus (HBV) infection in U.S. Hispanics/Lati
143 on or cirrhosis due to other causes, such as hepatitis B virus (HBV) infection or alcohol, remains un
144 he heterogeneous clinical courses of chronic hepatitis B virus (HBV) infection reflect the complex ho
150 ions have among the highest rates of chronic hepatitis B virus (HBV) infection worldwide, but little
151 hepatitis C virus (HCV) infection, 17.9% had hepatitis B virus (HBV) infection, and 2.2% had both.
152 achieving better treatment and prevention of hepatitis B virus (HBV) infection, anti-HBV agents targe
153 govern distinct clinical phases of a chronic hepatitis B virus (HBV) infection-immune tolerant (IT),
164 patitis B surface antigen (HBsAg) in chronic hepatitis B virus (HBV) infections of China remains uncl
170 plication-competent viral capsids.IMPORTANCE Hepatitis B virus (HBV) is a major human pathogen, and n
175 hronic hepatitis B (CHB), failure to control hepatitis B virus (HBV) is associated with T cell dysfun
177 antiviral regimens with no activity against hepatitis B virus (HBV) may increase the risk for HBV re
180 ), most cases of which are related to either hepatitis B virus (HBV) or hepatitis C virus (HCV).
182 ron gamma and interleukin 10) to overlapping hepatitis B virus (HBV) peptides (preS, S, preC, core, a
184 racterizes and defines the clinical value of hepatitis B virus (HBV) quasispecies with reverse transc
189 udies report conflicting data on the risk of hepatitis B virus (HBV) reactivation in rheumatologic pa
190 tumor chemotherapy regimens pose a risk for hepatitis B virus (HBV) reactivation, but screening and
192 ate cotransporting polypeptide (NTCP) as the hepatitis B virus (HBV) receptor enabled researchers to
193 noglobulin (HBIg) is effective in preventing hepatitis B virus (HBV) recurrence after liver transplan
194 an integral component of prophylaxis against hepatitis B virus (HBV) recurrence in liver transplantat
195 al or mother-to-child transmission (MTCT) of hepatitis B virus (HBV) remains the major risk factor fo
196 eraction mechanisms for achieving an optimal hepatitis B virus (HBV) replication have been largely un
198 es, such as entecavir or tenofovir, suppress hepatitis B virus (HBV) replication, improve liver funct
201 Persistent or chronic infection with the hepatitis B virus (HBV) represents one of the most commo
203 ation, there is still evidence of suboptimal hepatitis B virus (HBV) screening among patients at high
205 B vaccine, despite loss of antibody against hepatitis B virus (HBV) surface antigen (anti-HBs), is u
206 natural killer (NK) cells in the process of hepatitis B virus (HBV) surface antigen (HBsAg) clearanc
212 ctiveness of strategies to prevent perinatal hepatitis B virus (HBV) transmission in the United State
213 fumarate (TDF) in reducing mother-to-infant hepatitis B virus (HBV) transmissions is not clearly und
217 tide sequence characteristic of DNA from the hepatitis B virus (HBV) with a detection limit of 85 pM.
219 to identify NAFLD, hepatitis C virus (HCV), hepatitis B virus (HBV), alcoholic liver disease (ALD),
220 Previous estimates of the burden of HIV, hepatitis B virus (HBV), and hepatitis C virus (HCV) amo
221 cquiring human immunodeficiency virus (HIV), hepatitis B virus (HBV), and hepatitis C virus (HCV).
222 f 47,591 adults wait-listed for LT from HCV, hepatitis B virus (HBV), and nonalcoholic steatohepatiti
223 pidemiology of HIV, hepatitis C virus (HCV), hepatitis B virus (HBV), and tuberculosis in prisoners.
225 role of the transcriptional template of the hepatitis B virus (HBV), covalently closed circular DNA
226 atitis delta virus (HDV), a satellite of the hepatitis B virus (HBV), increases viral liver disease s
227 is in patients with hepatitis C virus (HCV), hepatitis B virus (HBV), NAFLD, and alcoholic liver dise
228 th infections by either hepatitis A virus or hepatitis B virus (HBV), or a noninfectious cause for th
230 of the reverse transcriptase protein (RT) of hepatitis B virus (HBV), sampled from patients with rapi
232 eral common loci associated with the risk of hepatitis B virus (HBV)- or hepatitis C virus (HCV)-rela
233 The incidences of chronic hepatitis B (CHB), Hepatitis B virus (HBV)-associated cirrhosis and HBV-ass
235 pha elicits a pleiotropic antiviral state in hepatitis B virus (HBV)-infected hepatocytes, but whethe
237 disturbance of Mg(2+) homeostasis on chronic hepatitis B virus (HBV)-infected natural killer (NK) and
240 n conducted on the genetic susceptibility of hepatitis B virus (HBV)-related hepatocellular carcinoma
247 compatible with acute, resolved, and chronic hepatitis B virus (HBV)infection but might also signify
249 d with hepatitis C virus [HCV] and five with hepatitis B virus [HBV]) of 600 HIV-infected patients wh
255 re found in the sera from patients with AIH, hepatitis B virus, hepatitis C virus, and nonalcoholic s
258 of the pregenomic RNA encapsidation in human Hepatitis B virus in vivo using a coarse-grained molecul
260 drug-induced liver injury DILI (22%), acute hepatitis B virus infection (12%), autoimmune hepatitis
261 C virus infection (41%), alcohol (39%), and hepatitis B virus infection (22%) the commonest etiologi
262 an guidelines recommend treatment of chronic hepatitis B virus infection (CHB) with the nucleos(t)ide
263 A cross-sectional analysis of prevalence of hepatitis B virus infection (HBV) among rural couples wa
265 f LT, center, and hepatitis C virus (HCV) or hepatitis B virus infection) with non-HIV-infected contr
268 and obesity; 12.5% (95% CI: 10.6-14.3%) for hepatitis B virus infection; 29.1% (95% CI: 23.6-34.5%)
269 copies per mL); HLA-B*5701-negative; had no hepatitis B virus infection; screening genotypes showing
275 9 HDV-infected patients, 25 individuals with hepatitis B virus monoinfection and 18 healthy controls.
280 e (PR = 2.57; 95% CI: 2.33-2.84; P = 0.001), hepatitis B virus (PR = 1.32; 95% CI: 1.09-1.59; P = 0.0
281 lly regulates several direct target genes of hepatitis B virus protein X (HBx), a viral co-factor.
285 transfer of T cells engineered to express a hepatitis B virus-specific (HBV-specific) T cell recepto
287 axis, mother-to-child transmission (MTCT) of Hepatitis B Virus still occurs in approximately 2-5% of
289 for malignant disease, testing positive for hepatitis B virus surface antigen, pregnancy, creatinine
293 ment of hepatitis C virus and suppression of hepatitis B virus, treatment and management principles f
296 in Egypt (1054 [84%] of 1251 patients), and hepatitis B virus was the leading cause in the other Afr
297 of this algorithm is demonstrated using the Hepatitis B virus X (HBx) protein, a protein of unknown
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