ライフサイエンスコーパス: hepatitis C virus RNA

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1 nd can be primed with other PAMPs, including hepatitis C virus RNA.

2 B, and C, respectively; all had undetectable hepatitis C virus RNA 48 weeks after the end of therapy.

3 The primary endpoint was absence of hepatitis C virus RNA 6 months posttreatment.

4 through/relapse patients (undetectable serum hepatitis C virus RNA after 24 weeks of peginterferon-ri

5 ielded highly reproducible quantification of hepatitis C virus RNA and displayed a nearly 600-fold dy

6 e hepatitis C infection, close monitoring of hepatitis C virus RNA and treatment of patients with per

7 serum aminotransferases, the level of serum hepatitis C virus RNA, and histologic necroinflammatory

8 ic response (undetectable [<125 IU/mL] serum hepatitis C virus RNA at 24-week follow-up).

9 response (defined as undetectable levels of hepatitis C virus RNA at 4 weeks) and were eligible to p

10 ogic response (2 log10 reduction in level of hepatitis C virus RNA at week 12; n=466), and undetectab

11 rus RNA at week 12; n=466), and undetectable hepatitis C virus RNA at weeks 20 (n=320), 48 (end of tr

12 ponders (SRs) with a less than 2-log drop in hepatitis C virus RNA by week 4.

13 ype 1 hepatitis C virus infection with serum hepatitis C virus RNA concentrations of at least 5 log10

14 by 2.5- to 10.6-fold even at relatively low hepatitis C virus RNA concentrations.

15 ro-2'-C-methylcytidine (1) was designed as a hepatitis C virus RNA-dependent RNA polymerase (HCV RdRp

16 as inhibitors (IC(50) = 0.08-3.8 microM) of hepatitis C virus RNA-dependent RNA polymerase (RdRp).

17 rce microscopy (AFM) was used to detect HCV (hepatitis C virus) RNA directly and to quantitatively an

18 among patients with a >2-log(10) decrease in hepatitis C virus RNA during prior PEG-IFN/RBV therapy:

19 T allowed subjects who achieved undetectable hepatitis C virus RNA from weeks 4 and 12, known as exte

20 tural protein 5A (NS5A) encoded by the human hepatitis C virus RNA genome is shown here to induce the

21 The data suggest that hepatitis C virus RNA helicase, and therefore viral repl

22 The hepatitis C virus RNA helicase, NS3, is an important mod

23 ast 7 months posttransplant, with detectable hepatitis C virus RNA in serum and features of hepatitis

24 s, Foster City, Calif.) for the detection of hepatitis C virus RNA in serum and plasma.

25 A dramatic reduction in hepatitis C virus RNA level was observed after 2 days of

26 lant recipients was evaluated and effects on hepatitis C virus RNA level, quasispecies evolution, and

27 -2a treated patients had significantly lower hepatitis C virus RNA levels and more favorable changes

28 Patients with baseline hepatitis C virus-RNA levels (bHCV-RNA)>6 log IU/mL or c

29 A practical, efficient synthesis of 1, a hepatitis C virus RNA replication inhibitor, is describe

30 report the isolation and characterization of hepatitis C virus RNA replicons resistant to a novel ket

31 Hepatitis C virus RNA was detected in Gammagard, and the

32 achievement of sustained clearance of serum hepatitis C virus RNA, which is influenced, in turn, by

33 atients (F0-F3), if a >2-log(10) decrease in hepatitis C virus RNA with previous PEG-IFN/RBV treatmen

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