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1 tis B, as therapy in 5 patients with chronic hepatitis D.
2 ant women, in children, and in patients with hepatitis D and C virus coinfection.
3 y was shown to be effective in patients with hepatitis D and ribavirin provides no additional benefit
4 hepatitis B e antigen [HBeAg] negative, anti-hepatitis D antigen [HDAg] positive, and HDV RNA positiv
5 s of diversifying selective pressures on the hepatitis D antigen gene (HDAg).
6 stigate the efficacy of entecavir in chronic hepatitis D (CHD).
7 gests that acute HCV infection, unrecognized hepatitis D infection, and hepatitis E may all represent
8 ring acute flares of disease, and with acute hepatitis D superinfection.
9                                          The hepatitis D virion is composed of a coat of HBV envelope
10                                              Hepatitis D virus (also known as hepatitis delta virus)
11  activity of vanitaracin A was observed with hepatitis D virus (HDV) but not hepatitis C virus.
12 ron alfa-2a in patients with chronic HBV and hepatitis D virus (HDV) co-infection.
13 s with the human hepatitis B virus (HBV) and hepatitis D virus (HDV) depend on species-specific host
14                                              Hepatitis D virus (HDV) infection affects 15-20 million
15  appears to be an autonomous, or "isolated," hepatitis D virus (HDV) infection following the transpla
16                             The emergence of hepatitis D virus (HDV) infection in the era of widespre
17      The role of hepatitis B virus (HBV) and hepatitis D virus (HDV) infection was investigated as th
18 and treatment of hepatitis B virus (HBV) and hepatitis D virus (HDV) infections, we still do not comp
19                          Superinfection with hepatitis D virus (HDV) may increase the risk for hepati
20                                              Hepatitis D virus (HDV) particles pseudotyped with HBV a
21                                              Hepatitis D virus (HDV) requires hepatitis B surface ant
22             As previously shown by using the hepatitis D virus (HDV) system, recombinant HBV-HDV part
23 ch inhibited the process of entry of HBV and hepatitis D virus (HDV).
24      We aimed to establish the prevalence of hepatitis D virus among HBsAg-positive populations in su
25 n binding and in vitro infection assays with hepatitis D virus as a surrogate for HBV, we established
26 tions if HBV recurrence occurs (i.e., HIV or hepatitis D virus coinfection, preexisting drug resistan
27                      The odds ratio for anti-hepatitis D virus detection among HBsAg-positive patient
28 TION: Findings suggest localised clusters of hepatitis D virus endemicity across sub-Saharan Africa.
29 a, where HBsAg prevalence is higher than 8%, hepatitis D virus might represent an important additive
30                                              Hepatitis D virus particles pseudotyped with surface pro
31        We systematically reviewed studies of hepatitis D virus prevalence among HBsAg-positive popula
32 y, only eight of which included detection of hepatitis D virus RNA among anti-hepatitis D virus serop
33 etection of hepatitis D virus RNA among anti-hepatitis D virus seropositive participants.
34 ects model to calculate a pooled estimate of hepatitis D virus seroprevalence.
35 dies should aim to define the reliability of hepatitis D virus testing methods, identify risk factors
36 In west Africa, the pooled seroprevalence of hepatitis D virus was 7.33% (95% CI 3.55-12.20) in gener
37 ) positive, human immunodeficiency virus and hepatitis D virus-negative patients with pretransplant H
38 , the countries therein, and permutations of hepatitis D virus.
39     The receptor that allows hepatitis B and hepatitis D viruses to enter human liver cells has been

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