ライフサイエンスコーパス: hepatitis virus

1 urotropic coronavirus (rJ2.2 strain of mouse hepatitis virus).

2 BV and of woodchucks infected with woodchuck hepatitis virus.

3 that was chronically infected with woodchuck hepatitis virus.

4 A synthesis in a related coronavirus, murine hepatitis virus.

5 fection of p85beta-deficient mice with mouse hepatitis virus.

6 ction by the neurotropic JHM strain of mouse hepatitis virus.

7 human Hepatitis B virus (HBV) and Woodchuck hepatitis virus.

8 ction by the neurotropic JHM strain of mouse hepatitis virus.

9 irus chikungunya virus and coronavirus mouse hepatitis virus.

10 capsid protein of a model coronavirus, mouse hepatitis virus.

11 gh-risk etiological challenges, most notably hepatitis virus.

12 ol acute infection with the cytopathic mouse hepatitis virus.

13 ion units of blood are not tested for HIV or hepatitis viruses.

14 nodeficiency virus (HIV) and four species of hepatitis viruses.

15 CV vaccine and future research needs for the hepatitis viruses.

16 They are avian encephalomyelitis virus, duck hepatitis virus 1, duck picornavirus, porcine teschoviru

17 Intranasal mouse hepatitis virus-1 (MHV-1) infection of susceptible mouse

18 sses the recent advances in our knowledge of hepatitis viruses A through G, focusing on the literatur

19 n-A, non-B hepatitis cases were unrelated to hepatitis viruses A,B,C, and G, suggesting another unide

20 Mouse hepatitis virus, a beta-CoV in group A, uses the galecti

21 Intracerebral infection of mice with mouse hepatitis virus, a member of the Coronaviridae family, r

22 the role of MDA5 during infection with mouse hepatitis virus, a murine coronavirus used to model vira

23 ity was examined in mice infected with mouse hepatitis virus, a well-described model of virus-induced

24 Lpro inhibitors in mice infected with murine hepatitis virus A59, a hepatotropic coronavirus, resulte

25 as helminths, mycobacteria, Plasmodium, and hepatitis viruses affect more than a third of the human

26 urprisingly, two beta-CoVs in group A, mouse hepatitis virus and HKU1, have evolved to use different

27 odchucks chronically infected with woodchuck hepatitis virus and vaccinated with woodchuck hepatitis

28 e estimated risk for transfusion transmitted hepatitis viruses and retroviruses is now vanishingly sm

29 adnaviruses (hepatitis B virus and woodchuck hepatitis virus), and an intron-retaining transcript enc

30 ic enzyme from two CoVs, SARS-CoV and murine hepatitis virus, and its monomeric homologue, XendoU fro

31 n the characterization of several fastidious hepatitis viruses, and we investigated the feasibility o

32 tis aplastic anemia, antibodies to the known hepatitis viruses are absent; the unknown infectious age

33 methylator phenotype and the contribution of hepatitis viruses are poorly understood.

34 's murine encephalomyelitis virus and murine hepatitis virus, are used to induce infectious models of

35 splant recipients commonly are infected with hepatitis viruses, are immunosuppressed, and have other

36 sed from the replicase polyprotein of murine hepatitis virus as fusions with nonstructural protein 2

37 s within the replicase polyprotein of murine hepatitis virus as fusions with nonstructural proteins c

38 d HIV1 and HIV2, as well as of two different hepatitis viruses, attaining results of approximately 87

39 ucks chronically infected with the woodchuck hepatitis virus before and during 30 weeks of therapy wi

40 temperature-sensitive (ts) mutant of Murine hepatitis virus, Bristol ts31 (MHV-Brts31), that defines

41 al nervous system (CNS) by neurotropic mouse hepatitis virus but do not suffice to achieve sterile im

42 ntiviral protein IFN-alpha2 is used to treat hepatitis viruses but has proven rather ineffective agai

43 Five human hepatitis viruses cause most of the acute and chronic li

44 Analysis of several HBV/woodchuck hepatitis virus chimeras corroborated the findings from

45 Human immunodeficiency virus (HIV) and hepatitis virus coinfection amplify and accelerate hepat

46 We followed 22 910 participants without hepatitis virus coinfection for 114 478 person-years.

47 a monax) chronically infected with woodchuck hepatitis virus contained at least 100,000 clones of >1,

48 n development of HCV sequence databases, the Hepatitis Virus Database (Japan), euHCVdb (France), and

49 ts for lung cancer, and immunodeficiency and hepatitis virus effects for liver cancer.

50 CCL3(-/-) mice infected with mouse hepatitis virus exhibited a significant reduction of vir

51 r results support the hypothesis that murine hepatitis virus ExoN activity is required for resistance

52 he 3' untranslated region (UTR) of the mouse hepatitis virus genome contains two essential and overla

53 (-/-) mice infected with a neurotropic mouse hepatitis virus had an impaired ability to control viral

54 end-stage liver disease (ESLD) secondary to hepatitis viruses has evolved rapidly during the last tw

55 of the SARS-associated coronavirus and mouse hepatitis virus have evolved to promote optimal frameshi

56 e livers chronically infected with woodchuck hepatitis virus, (i) hepadnavirus superinfection and cel

57 g RNA element (WPRE), derived from woodchuck hepatitis virus in combination with an antibody-cytokine

58 ncoded by ORF6, enhanced the growth of mouse hepatitis virus in tissue culture cells and in mice.

59 against CCL5 to mice with established mouse hepatitis virus-induced demyelination and impaired motor

60 opathogenesis, is essential for the study of hepatitis virus-induced liver disease and for therapeuti

61 ined with significant receptor expression in hepatitis virus-infected livers, suggests that IL-29 may

62 To determine functional significance, mouse hepatitis virus-infected mice were treated with anti-Mig

63 us (OR = 9.4; 95% CI = 2.7-32.7) and chronic hepatitis virus infection (OR = 31.2; 95% CI = 6.3-153.2

64 etween heavy alcohol consumption and chronic hepatitis virus infection (OR, 53.9; 95% CI, 7.0-415.7)

65 from cancer worldwide and is associated with hepatitis virus infection in 80% of cases.

66 The role of hepatitis virus infection in glucose homeostasis is unce

67 Mouse hepatitis virus infection of mice provides a useful tool

68 In human hepatitis virus infection, ADAR1 has been shown to targe

69 d models were used to estimate the effect of hepatitis virus infection, and adjusted for potential co

70 t synergy between heavy alcohol consumption, hepatitis virus infection, and diabetes mellitus may sug

71 ividuals that is not attributable to chronic hepatitis virus infection.

72 as similarly observed during acute woodchuck hepatitis virus infection.

73 synergistic interaction between obesity and hepatitis virus infection.

74 r stomatitis virus infection, but not murine hepatitis virus infection.

75 roduction in all viral hepatitis infections: Hepatitis virus infections did not alter NK cell differe

76 d its role in sensing and protecting against hepatitis virus infections is uncertain.

77 A related element from woodchuck hepatitis virus is known as the woodchuck posttranscript

78 V-JHM strain of the murine coronavirus mouse hepatitis virus is much more neurovirulent than the MHV-

79 Mouse hepatitis virus is used as well studied examplar to re-e

80 Coinfection with hepatitis viruses is common in individuals infected with

81 ase, but the intrahepatic immune response to hepatitis viruses is poorly understood because of a lack

82 ample, C57BL/6 (B6) mice infected with mouse hepatitis virus (JHM strain, JHMV) develop severe enceph

83 ontaining CNS cells were infected with mouse hepatitis virus-JHM, which causes fatal encephalitis in

84 omyelitis induced by the JHM strain of mouse hepatitis virus (JHMV) and sustained during viral persis

85 ted with the neurotropic JHM strain of mouse hepatitis virus (JHMV) develop acute and chronic demyeli

86 (CNS) by the neurotropic JHM strain of mouse hepatitis virus (JHMV) induces an acute encephalomyeliti

87 ion with the neurotropic JHM strain of mouse hepatitis virus (JHMV) into the central nervous system (

88 Control of neurotropic mouse hepatitis virus (JHMV) requires the collaboration of CD4

89 ion with the neurotropic JHM strain of mouse hepatitis virus (JHMV) resulted in an acute encephalomye

90 CNS with the neurotropic JHM strain of mouse hepatitis virus (JHMV) results in an immune-mediated dem

91 CNS with the neurotropic JHM strain of mouse hepatitis virus (JHMV) was examined.

92 ion with the neurotropic JHM strain of mouse hepatitis virus (JHMV).

93 ion with the neurotropic JHM strain of mouse hepatitis virus (JHMV).

94 The 5' 140 nucleotides of the mouse hepatitis virus (MHV) 5' untranslated region (5'UTR) are

95 We used mouse hepatitis virus (MHV) A59 as a model to gain insight int

96 The endoribonuclease activity of the mouse hepatitis virus (MHV) A59 Nsp15 was also increased by pR

97 he outcome of viral encephalomyelitis [mouse hepatitis virus (MHV) A59, Theiler's encephalomyelitis v

98 Infection with the murine coronavirus mouse hepatitis virus (MHV) activates the pattern recognition

99 uses within the genus Betacoronavirus, mouse hepatitis virus (MHV) and MERS-CoV, encode 2',5'-phospho

100 5' untranslated regions (5'UTRs) from mouse hepatitis virus (MHV) and severe acute respiratory syndr

101 Murine hepatitis virus (MHV) and severe acute respiratory syndr

102 acute respiratory syndrome (SARS)-CoV, mouse hepatitis virus (MHV) and the human CoV OC43 S2 subunits

103 The p28 and p65 proteins of mouse hepatitis virus (MHV) are the most amino-terminal protei

104 oprotein (S) of the murine coronavirus mouse hepatitis virus (MHV) binds to viral murine CEACAM recep

105 Some strains of mouse hepatitis virus (MHV) can induce chronic inflammatory de

106 The neurotropic JHM strain of mouse hepatitis virus (MHV) causes acute encephalitis and chro

107 Murine coronavirus mouse hepatitis virus (MHV) causes demyelination of the centra

108 Murine coronavirus mouse hepatitis virus (MHV) causes persistent infection of the

109 ) of the 5'-untranslated region of the mouse hepatitis virus (MHV) contains a highly conserved pental

110 Mouse hepatitis virus (MHV) does not induce interferon (IFN) p

111 or clearance of the murine coronavirus mouse hepatitis virus (MHV) during acute infection.

112 h the recombinant JHM (RJHM) strain of mouse hepatitis virus (MHV) elicits a weak CD8(+) T-cell respo

113 Various strains of mouse hepatitis virus (MHV) exhibit different pathogenic pheno

114 Overall, our data suggest murine hepatitis virus (MHV) ExoN activity is required for resi

115 Most strains of murine coronavirus mouse hepatitis virus (MHV) express a cleavable spike glycopro

116 ing the recombinant murine coronavirus mouse hepatitis virus (MHV) expressing the T cell-chemoattract

117 Murine coronavirus mouse hepatitis virus (MHV) gene 1 encodes several nonstructur

118 The mouse hepatitis virus (MHV) genome's 3' untranslated region co

119 Murine hepatitis virus (MHV) has long served as a model system

120 us work with the prototype coronavirus mouse hepatitis virus (MHV) has shown that a major determinant

121 Many strains of the murine coronavirus mouse hepatitis virus (MHV) have distinct, S-dependent organ a

122 revious studies have demonstrated that mouse hepatitis virus (MHV) hepatotropism is determined largel

123 We have used several strains of murine hepatitis virus (MHV) in cell culture and in vivo in mou

124 ucted mutants of the model coronavirus mouse hepatitis virus (MHV) in which all or part of the M prot

125 nts of a severely defective mutant of murine hepatitis virus (MHV) in which the N gene was replaced w

126 The murine coronavirus mouse hepatitis virus (MHV) induced the expression of type I i

127 The coronavirus mouse hepatitis virus (MHV) induces a minimal type I interfero

128 determine the requirement of nsp4 for murine hepatitis virus (MHV) infection in culture, engineered d

129 Murine hepatitis virus (MHV) infection provides a model system

130 Thus, during mouse hepatitis virus (MHV) infection, hepatitis, which damage

131 function in the context of a related murine hepatitis virus (MHV) infection.

132 e replacements were engineered into a murine hepatitis virus (MHV) infectious clone in place of conse

133 The prototype JHM strain of murine hepatitis virus (MHV) is an enveloped, RNA-containing co

134 The M protein of mouse hepatitis virus (MHV) is an integral membrane protein wi

135 synthesis by the prototype coronavirus mouse hepatitis virus (MHV) is carried out by a replicase-tran

136 Mouse hepatitis virus (MHV) isolates JHM.WU and JHM.SD promote

137 ive roles of specific ISGs against the mouse hepatitis virus (MHV) members of the coronaviruses are l

138 Mouse hepatitis virus (MHV) neurotropism varies by strain: MHV

139 monstrated that the murine coronavirus mouse hepatitis virus (MHV) nonstructural protein 2 (ns2) is a

140 We modeled the CoV murine hepatitis virus (MHV) nsp12-RdRp structure and superimpo

141 Here, we engineered mutations in murine hepatitis virus (MHV) nsp14 N7-MTase at residues D330 an

142 ween the endoribonuclease activity of murine hepatitis virus (MHV) Nsp15 (mNsp15) and its role in vir

143 magnetic resonance (NMR) structure of mouse hepatitis virus (MHV) nsp3a and show, using isothermal t

144 en shown previously that mutations in murine hepatitis virus (MHV) nsp4 loop 1 that alter nsp4 glycos

145 everse genetic mutagenesis of the CoV murine hepatitis virus (MHV) nsp5, we identified a new temperat

146 The coronavirus mouse hepatitis virus (MHV) performs RNA replication on double

147 Murine hepatitis virus (MHV) PLP2 and orthologs recognize and c

148 Infection of the CNS with the mouse hepatitis virus (MHV) provides a unique model situation

149 Mouse hepatitis virus (MHV) replicase products nsp3, nsp4, and

150 Mouse hepatitis virus (MHV) replication in actively growing DB

151 proteins and their processing during murine hepatitis virus (MHV) replication.

152 cultured cells with murine coronavirus mouse hepatitis virus (MHV) resulted in activation of the mito

153 t infection of rat oligodendrocytes by mouse hepatitis virus (MHV) resulted in apoptosis, which is ca

154 cranial infection of C57BL/6 mice with mouse hepatitis virus (MHV) results in an acute encephalomyeli

155 Intracerebral infection of mice with mouse hepatitis virus (MHV) results in an acute encephalomyeli

156 ) A1 has previously been shown to bind mouse hepatitis virus (MHV) RNA at the 3' end of both plus and

157 to function as regulatory factors for mouse hepatitis virus (MHV) RNA synthesis as a result of their

158 Mouse hepatitis virus (MHV) RNA synthesis is mediated by a vir

159 The 3' cis-acting element for mouse hepatitis virus (MHV) RNA synthesis resides entirely wit

160 The Murine hepatitis virus (MHV) strain A59 ns2 protein is a 30-kDa

161 important for clearance of neurotropic mouse hepatitis virus (MHV) strain A59, although their possibl

162 The murine coronavirus, mouse hepatitis virus (MHV) strain A59, causes acute encephali

163 temperature-sensitive (TS) mutants of murine hepatitis virus (MHV) strain A59, proposes that an inter

164 C57BL/6 (B6) mice infected with mouse hepatitis virus (MHV) strain JHM develop a clinically ev

165 We observed that the nonfusogenic mouse hepatitis virus (MHV) strain MHV-2 reached a titer of ap

166 We demonstrate that MHV-A59, a mouse hepatitis virus (MHV) strain that causes brain and spina

167 Sequencing and reversion analysis of murine hepatitis virus (MHV) temperature-sensitive (ts) viruses

168 previously generated E gene mutants of mouse hepatitis virus (MHV) that had marked defects in viral g

169 urrent studies, neurotropic strains of mouse hepatitis virus (MHV) that induce meningitis, encephalit

170 usly generated E gene point mutants of mouse hepatitis virus (MHV) that were defective in growth and

171 previously generated E gene mutants of mouse hepatitis virus (MHV) that were defective in viral growt

172 at infection by the murine coronavirus mouse hepatitis virus (MHV) triggers the proximal UPR transduc

173 The mouse hepatitis virus (MHV) variant MHV/BHK can infect hamster

174 We previously described mouse hepatitis virus (MHV) variant V51 derived from a persist

175 Antibodies directed against mouse hepatitis virus (MHV) virions and against the putative R

176 kaging signal (PS) for the coronavirus mouse hepatitis virus (MHV) was originally identified as an el

177 Mouse hepatitis virus (MHV) was used as a model to study the i

178 In previous work with the coronavirus mouse hepatitis virus (MHV), a highly defective M protein muta

179 murine CEACAM1 serve as receptors for mouse hepatitis virus (MHV), a murine coronavirus.

180 eta IFN (IFN-alpha/beta) receptor with mouse hepatitis virus (MHV), a murine coronavirus.

181 stion, we used the rJHM strain (rJ) of mouse hepatitis virus (MHV), a neurotropic coronavirus that ca

182 erminal domain (NTD) of N protein from mouse hepatitis virus (MHV), a virus most closely related to S

183 d by the prototypical Betacoronavirus, mouse hepatitis virus (MHV), and by Middle East respiratory sy

184 The murine coronavirus, mouse hepatitis virus (MHV), causes acute hepatitis in its nat

185 vely, including the murine coronavirus mouse hepatitis virus (MHV), Haemophilus influenzae, Neisseria

186 For the prototype coronavirus mouse hepatitis virus (MHV), it has previously been establishe

187 pe of a highly neurovirulent strain of mouse hepatitis virus (MHV), JHM, is thought to be essential f

188 In murine hepatitis virus (MHV), nsps 1, 2, and 3 are processed by

189 Infection of mice with mouse hepatitis virus (MHV), strain JHM, results in acute and

190 ed virus entry and cell-cell fusion of mouse hepatitis virus (MHV), suggesting the importance of lipi

191 For the coronavirus mouse hepatitis virus (MHV), the first proteolytic processing

192 In mouse hepatitis virus (MHV), the NTD binds the transcriptional

193 y, we exploited the model coronavirus, mouse hepatitis virus (MHV), to investigate the genotype and p

194 the N protein of the model coronavirus mouse hepatitis virus (MHV), we constructed mutants in which e

195 f N protein domains in the coronavirus mouse hepatitis virus (MHV), we replaced the MHV N gene with i

196 ation and expression is altered due to mouse hepatitis virus (MHV)-A59 infection both in vivo and in

197 activated protein kinases (MAPKs) in a mouse hepatitis virus (MHV)-infected macrophage-derived J774.1

198 eases replication fidelity of the CoV murine hepatitis virus (MHV).

199 sponse to intracerebral infection with mouse hepatitis virus (MHV).

200 t in the prototype group 2 coronavirus mouse hepatitis virus (MHV).

201 ry syndrome coronavirus (SARS-CoV) and mouse hepatitis virus (MHV).

202 nfection by the enveloped coronavirus murine hepatitis virus (MHV).

203 against a member of the Coronaviridae, Mouse hepatitis virus (MHV).

204 UTR) of the genome of the coronavirus mouse hepatitis virus (MHV).

205 tory syndrome CoV, and the murine CoV, mouse hepatitis virus (MHV).

206 hibitor in a coronavirus model system, mouse hepatitis virus (MHV).

207 f in vivo infection by the coronavirus mouse hepatitis virus (MHV).

208 titution mutations into the genome of murine hepatitis virus (MHV-A59) containing ExoN activity [ExoN

209 tralize the neurotropic JHM strain of murine hepatitis virus (MHV-JHM).

210 nto a heterologous murine coronavirus (mouse hepatitis virus [MHV]) but is not essential for optimal

211 with a recombinant murine coronavirus (mouse hepatitis virus [MHV]) expressing the T-cell chemoattrac

212 of RNase L during murine coronavirus (mouse hepatitis virus [MHV]) infection of myeloid cells correl

213 Murine coronavirus (mouse hepatitis virus [MHV]) nonstructural protein 2 (ns2) is

214 Restricted analyses among hepatitis virus-negative subjects, nondrinkers, nondiabe

215 In this report, we show that the murine hepatitis virus nsp2 sequence was tolerated in ORF1b wit

216 the interaction between coronaviruses (mouse hepatitis virus) of different neurovirulences with prima

217 during clearance of the JHM strain of mouse hepatitis virus, only few virus-specific Ab-secreting ce

218 zation is critical for maintaining JHM mouse hepatitis virus persistence within the central nervous s

219 ar immunity in controlling neurotropic mouse hepatitis virus persistence within the CNS were determin

220 neurotropic coronavirus JHM strain of mouse hepatitis virus persists in oligodendroglia despite the

221 us-mouse phenylalanine hydroxylase-woodchuck hepatitis virus post-transcriptional response element (r

222 The woodchuck hepatitis virus posttranscriptional regulatory element (

223 s sc counterpart, which lacked the woodchuck hepatitis virus posttranscriptional regulatory element (

224 NS) with the neurotropic JHM strain of mouse hepatitis virus produces acute and chronic demyelination

225 ction by the neurotropic JHM strain of mouse hepatitis virus produces an acute demyelinating encephal

226 Detection of the mouse hepatitis virus receptor within the central nervous syst

227 duced high titer viremia and acute resolving hepatitis; viruses recovered from both animals lacked th

228 und that proteasome inhibitors blocked mouse hepatitis virus replication at an early step in the vira

229 toma cells are not able to support woodchuck hepatitis virus replication.

230 trypsinogen activation in pancreatitis, and hepatitis virus replication.

231 CXCL10-expressing murine coronavirus (mouse hepatitis virus) resulted in protection from disease and

232 neurotropic strains of the coronavirus mouse hepatitis virus results in an immune response-mediated d

233 Although HIV and the hepatitis viruses share common modes of transmission, an

234 The identification of hepatitis virus specific signatures provides a foundatio

235 ivation gene 2(-/-) mice with only non-mouse hepatitis virus-specific T cells, we show that CD8 T cel

236 ligation of T cells in vitro with the murine hepatitis virus spike protein, a natural ligand for the

237 Combining risk-score tertile levels and hepatitis virus status to stratify participants was more

238 ped using accessible variables combined with hepatitis virus status, which allows selection of asympt

239 n.) infection of A/J mice with the CoV mouse hepatitis virus strain 1 (MHV-1) induces an acute respir

240 nvestigation into the genetic basis of mouse hepatitis virus strain 1 (MHV-1) pneumovirulence.

241 ne coronaviruses expressing wild-type murine hepatitis virus strain 4 (MHV-4) or MHV-A59 spike glycop

242 induced in primary mouse astrocytes by mouse hepatitis virus strain A59 (MHV-A59) and MHV-2.

243 The murine hepatitis virus strain A59 (MHV-A59) model produces foca

244 Previous work showed that mouse hepatitis virus strain A59 (MHV-A59) with a mutated cata

245 cently established for the coronavirus mouse hepatitis virus strain A59 (MHV-A59), in which cDNA frag

246 tious cDNA of the group II coronavirus mouse hepatitis virus strain A59 (MHV-A59).

247 Mouse hepatitis virus strain A59 infection of mice is a useful

248 tranasal infection with the gliatropic mouse hepatitis virus strain A59.

249 Replication of the neurotropic mouse hepatitis virus strain JHM (JHMV) is controlled primaril

250 Mouse hepatitis virus strain JHM (MHV-JHM) causes acute enceph

251 Mouse hepatitis virus strain JHM causes a chronic demyelinatin

252 Mice infected with mouse hepatitis virus strain JHM develop an inflammatory demye

253 The coronavirus, mouse hepatitis virus strain JHM, causes acute and chronic neu

254 ected with the neurotropic coronavirus mouse hepatitis virus strain JHM.

255 fections, including mice infected with mouse hepatitis virus strain JHM.

256 e to high-fat diet-caused obesity and murine hepatitis virus strain-3 (MHV-3)-induced fulminant hepat

257 Mice infected with the coronavirus mouse hepatitis virus, strain JHM (JHM) develop a disease that

258 infected with the murine coronavirus, mouse hepatitis virus, strain JHM (MHV) develop an immune-medi

259 ce infected with attenuated strains of mouse hepatitis virus, strain JHM, develop a chronic infection

260 cognized in C57BL/6 mice infected with mouse hepatitis virus, strain JHM, or lymphocytic choriomening

261 epatitis virus and vaccinated with woodchuck hepatitis virus surface antigen could be induced to reco

262 ivo during infection with a strain of murine hepatitis virus that causes a chronic, inflammatory demy

263 s are likely to be related to differences in hepatitis viruses that are most prevalent in a populatio

264 readily infected by another strain of mouse hepatitis virus, the A59 strain (MHV-A59).

265 Intranasal mouse hepatitis virus type 1 (MHV-1) infection of mice induces

266 recently been shown that cell entry of mouse hepatitis virus type 2 (MHV-2) is mediated through endoc

267 From a public health perspective, hepatitis virus vaccination programs and efforts to both

268 y of PLPs from SARS-CoV, MERS-CoV, and mouse hepatitis virus was evaluated against seven ISG15s origi

269 importance of the CD (SWWSFNPETNNL) in mouse hepatitis virus was investigated with a panel of mutant

270 sublethal infections with neurotropic mouse hepatitis virus was investigated.

271 s mice infected with the JHM strain of mouse hepatitis virus, which exhibit CTL escape variants with

272 he cis-acting element found in the woodchuck hepatitis virus (WHV) (the WHV posttranscriptional regul

273 onically infected with HBV-related woodchuck hepatitis virus (WHV) and already developed HCCs were us

274 her animal hepadnaviruses, such as woodchuck hepatitis virus (WHV) and duck hepatitis B virus (DHBV).

275 ciated with chronic infection with woodchuck hepatitis virus (WHV) and serve as a model of hepatitis

276 ed that the X-deficient mutants of woodchuck hepatitis virus (WHV) are not completely replication def

277 odchucks chronically infected with woodchuck hepatitis virus (WHV) are superinfected with HDV, they p

278 Woodchucks infected at birth with woodchuck hepatitis virus (WHV) cleared viremia and developed anti

279 Integrations of woodchuck hepatitis virus (WHV) DNA and rearrangements of the N-my

280 ucks chronically infected with the woodchuck hepatitis virus (WHV) elicited differential T-cell respo

281 odchucks chronically infected with woodchuck hepatitis virus (WHV) induces a transient decline in vir

282 atal woodchucks with self-limiting woodchuck hepatitis virus (WHV) infection to those woodchucks prog

283 issue from woodchucks with chronic woodchuck hepatitis virus (WHV) infection was assayed for randomly

284 ed (resolved) and chronic neonatal woodchuck hepatitis virus (WHV) infection.

285 immunity (vCMI) following neonatal woodchuck hepatitis virus (WHV) infection.

286 over during clearance of transient woodchuck hepatitis virus (WHV) infections.

287 Woodchuck hepatitis virus (WHV) is prone to aberrant assembly in v

288 (Marmota monax) infected with the woodchuck hepatitis virus (WHV) represent the best animal model fo

289 en the immunogenicity of an analog woodchuck hepatitis virus (WHV) surface antigen (WHsAg) pDNA vacci

290 ting the ability of the virions of woodchuck hepatitis virus (WHV) to induce productive acute infecti

291 ucks chronically infected with the woodchuck hepatitis virus (WHV) were treated with the antiviral dr

292 is study, we generated a series of woodchuck hepatitis virus (WHV) X mutants, including mutants of th

293 Woodchuck hepatitis virus (WHV), a close relative of human hepatit

294 monax) is naturally infected with woodchuck hepatitis virus (WHV), a hepadnavirus closely related to

295 from X/c-myc bitransgenic mice and woodchuck hepatitis virus (WHV)-infected woodchucks.

296 emonstrated for HBV or HBV-related woodchuck hepatitis virus (WHV).

297 rmota monax ) harbors a DNA virus (Woodchuck hepatitis virus [WHV]) that is similar in structure and

298 e (M) protein of the model coronavirus mouse hepatitis virus with its counterpart from a heterologous

299 ation of the neurotropic JHM strain of mouse hepatitis virus within the central nervous system is con

300 tional regulatory element from the woodchuck hepatitis virus (WPRE).