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1 hat the accessory factor binding region A is hepatocyte nuclear factor-1.
3 olamine dehydratase/dimerization cofactor of hepatocyte nuclear factor 1 alpha), a gene that was rece
4 odenal homeobox-1, BETA2/NeuroD, Nkx6.1, and hepatocyte nuclear factor 1 alpha), beta-cell metabolic
6 zygous mutations in the transcription factor hepatocyte nuclear factor-1 alpha (HNF1A or TCF1 gene) r
7 mon polymorphisms of the HNF1A gene encoding hepatocyte nuclear factor-1 alpha and plasma C-reactive
8 ent genome-wide association study identified hepatocyte nuclear factor 1-alpha (HNF1A) as a key regul
9 48; 95% CI, 2.83-10.61; P = 4.4 x 10(-7)) in hepatocyte nuclear factor 1-alpha (HNF1A), the gene resp
10 ent, the liver-enriched transcription factor hepatocyte nuclear factor 1, and an unknown liver-enrich
11 hat the accessory factor binding region A is hepatocyte nuclear factor-1, and we show here that the f
12 mobility shift assays provided evidence for hepatocyte nuclear factor 1 binding within footprint A a
13 egulatory elements, including an Sp-1 and an hepatocyte nuclear factor-1 binding site and a CAAT box.
14 lamine dehydratase/dimerization cofactor for hepatocyte nuclear factor 1 (DCoH) has been elucidated b
15 ding a chloroplast dimerization co-factor of hepatocyte nuclear factor 1 (DCoH)/pterin-4alpha-carbino
16 es for known trans-acting factors, including hepatocyte nuclear factor 1, Forkhead box A1 and CCCTC-b
17 GFBP-1 promoter activation was via an intact hepatocyte nuclear factor 1 (HNF-1) site and was depende
19 the binding site of the transcription factor hepatocyte nuclear factor-1 (HNF-1) and specifically sup
20 , in LLC-PK cells, but not in HepG2 cells, a hepatocyte nuclear factor-1 (HNF-1) binding site was cri
22 arkedly decreases mRNA and protein levels of hepatocyte nuclear factor-1 (HNF-1), a transcription fac
24 so identical to the dimerization cofactor of hepatocyte nuclear factor 1 (HNF1) (DCoH) that is able t
25 cells, we have identified a highly conserved hepatocyte nuclear factor 1 (HNF1) binding site residing
26 hree regions contained binding sites for the hepatocyte nuclear factor 1 (HNF1), a transcriptional re
28 experimental process was defined by studying hepatocyte nuclear factor 1 (HNF1), which binds DNA as a
29 horetic mobility shift analyses revealed two hepatocyte nuclear factor-1 (HNF1), three CCAAT/enhancer
31 loss of liver-enriched transcription factors hepatocyte nuclear factor 1 (HNF1alpha) and hepatocyte n
32 ntly down-regulated pathways associated with hepatocyte nuclear factor 1 homeobox A (Hnf1a) and hepat
36 ODY3 and 5, rare forms of diabetes caused by hepatocyte nuclear factor-1 mutations, is discussed.
37 lpha1-antitrypsin messenger RNA, albumin and hepatocyte nuclear factor 1 protein were detected in the
40 tissue-specific transcription factor HNF-1 (hepatocyte nuclear factor-1) through binding the coactiv
41 ing and for the transcription factor variant hepatocyte nuclear factor 1 (vhnf1) in specification of
42 th factor (Fgf) signals from r4; and variant hepatocyte nuclear factor 1 (vhnf1, also known as tcf2),
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