ライフサイエンスコーパス: herd immunity

1 Population-scale immunity is often termed 'herd immunity'.

2 mission into a human population with limited herd immunity.

3 can hardly be achieved due to the effects of herd immunity.

4 previous virus exposure, and monitoring sow herd immunity.

5 ecause of the decreasing marginal returns of herd immunity.

6 ly mechanisms contributing to the evasion of herd immunity.

7 associated disease, and provided evidence of herd immunity.

8 ory loss, vaccination protects polymicrobial herd immunity.

9 of cost-effectiveness, both with and without herd immunity.

10 llow escape from antibody neutralization and herd immunity.

11 be rescued by management approaches based on herd immunity.

12 ly limited in their ability to raise lasting herd immunity.

13 eral population because of high coverage and herd immunity.

14 s of vaccination coverage required to attain herd immunity.

15 epitopes, and likely evolves in response to herd immunity.

16 ation levels that are sufficient to maintain herd immunity.

17 lution of insecticide resistance and loss of herd immunity.

18 ulation immunity (90%-92%) needed to achieve herd immunity.

19 ion and for the protection of others through herd immunity.

20 g antigenic variation, likely in response to herd immunity.

21 persistence in the face of human population herd immunity.

22 of group C meningococci was consistent with herd immunity.

23 t-term vaccine effectiveness and substantial herd immunity.

24 actors such as socio-economic conditions and herd immunity.

25 achieve the high seroprevalence required for herd immunity.

26 decline in 2016 and fade-out in 2017 due to herd-immunity.

27 Transmission has the benefit of increasing herd immunity above that achieved by direct vaccination

28 Heterotypic genotypes did not contribute to herd immunity against GII.4 NoVs based on their inabilit

29 hildren were vaccinated, it is possible that herd immunity against influenza was achieved in Japan.

30 Therefore, to induce herd immunity against Streptococcus pneumoniae it will b

31 d in the wider population, presumably due to herd immunity against their seasonal H1 antigen.

32 juvenile fish through recruitment may reduce herd immunity, allow VHSV to persist, and drive superann

33 kely allowed GII.4-2012 to escape from human herd immunity and emerge as the new predominant strain.

34 chieve levels of coverage adequate to confer herd immunity and interrupt the diphtheria epidemic.

35 for at least partial escape from protective herd immunity and provide epidemiological support for th

36 luded in the conjugate vaccine may be due to herd immunity and serotype replacement effects in the ge

37 hanging pneumococcal population dynamics via herd immunity and serotype replacement.

38 yngeal carriage of H. influenzae and provide herd immunity and suggest that this effect is mediated t

39 vaccination campaigns to maintain long-term herd immunity and, hence, indirect protection of the unv

40 The balance between the loss of herd-immunity and viral re-importation will dictate futu

41 n maintains GII.4 persistence in the face of herd immunity, as the emergence of new pandemic strains

42 New research has examined the effect of 'herd immunity' associated with immunizing all school-age

43 In time, we predict that herd immunity benefits will diffuse from the larger whit

44 ch the overall attack rates and the level of herd immunity cannot be accurately estimated.

45 etween the direct effects of vaccination and herd immunity could lead to unintuitive consequences for

46 g children; and because of the potential for herd immunity decreasing morbidity and mortality among a

47 e new host population that wreak havoc until herd immunity develops.

48 V-related cancer by ethnicity, partly due to herd immunity disproportionately benefiting subgroups wi

49 s, which we suggest resulted from changes in herd immunity due to an increase in vaccination coverage

50 poultry populations where there was elevated herd immunity due to maternal and active immunity.

51 lations is consistent with a vaccine-induced herd immunity effect.

52 unt for all vaccine type-related cancers and herd immunity effects from vaccinating girls and boys.

53 er studies will be needed to assess possible herd immunity effects with meningococcal serogroup B vac

54 ing that the decline was likely secondary to herd-immunity effects.

55 eveloped by a Delphi expert panel; indirect (herd immunity) effects resulting from childhood PCV13 va

56 roportion of the population becomes immune, "herd immunity" emerges.

57 ealth interventions, because little residual herd immunity exists now that vaccination has ceased.

58 Herd immunity for unvaccinated children may occur in sch

59 isolates that are potentially able to escape herd immunity from earlier isolates.

60 easonal influenza, compromise the buildup of herd immunity from natural infection or deployment of cu

61 bstantially reduce the residual burden until herd immunity from pediatric PCV13 is fully established.

62 Both PPV23 and herd immunity from pediatric PCV7 were associated with r

63 emphatic in support of measurable impact of herd immunity given the uncertainty associated with pert

64 among other possible factors, GII.4-specific herd immunity had little role in the emergence of the ne

65 ts consistently point to profound direct and herd immunity impacts of the rotavirus vaccine program i

66 high-activity group will result in improved herd immunity in both the high-risk group and the popula

67 cination safety further contribute to waning herd immunity in developed countries, resulting in recen

68 asymptomatic infection contributes little to herd immunity in Ebola, and even if infectious, would ac

69 2 IAVs, suggesting that there may be minimal herd immunity in humans.

70 nd PsaA may be useful for the elicitation of herd immunity in humans.

71 he crucial role of monotypic and heterotypic herd immunity in shaping dengue epidemic behavior.

72 uently boosted by reexposure, so maintaining herd immunity in the face of potentially eroding individ

73 These results support the hypothesis that herd immunity is a driving force for GII.4 evolution in

74 e the observed epidemic cycles suggests that herd immunity is driving the epidemic dynamics caused by

75 with a mature PCV programme and established herd immunity is likely to maintain population control o

76 We postulate a herd-immunity model of C difficile diarrhoea, and examin

77 ether serogroup replacement or protection by herd immunity occurred.

78 ssusception and do not account for potential herd immunity or non-fatal outcomes.

79 l examined pointing to a substantial rise in herd immunity over the past 30 y.

80 ndings by documenting signatures of changing herd immunity over the study period.

81 needed, our results suggest that increasing herd immunity, perhaps with a combination of preemptive

82 t allows the virus to escape from protective herd immunity, resulting in new epidemic strains.

83 odel suggested that indirect protection, or "herd immunity," resulting from vaccination of school-age

84 ly lowering transmission rates and improving herd immunity--significantly moderate both influenza pan

85 However, part of the effect was caused by herd immunity, since vaccinated infants were more likely

86 mple by broadening the effective duration of herd immunity that can be achieved with currently used i

87 d containing epidemics if there was no prior herd immunity (that is, no prior immunologic protection

88 wentieth century populations with negligible herd immunity, the numbers of cases initially rose expon

89 ffs depend nonlinearly on whether or not the herd immunity threshold is reached.

90 In this paper, taking into consideration the herd immunity threshold, we present an evolutionary N-pe

91 For example, herd immunity to multiple pathogens has been observed at

92 f a new primary strain every 2 to 4 years as herd immunity to the previously circulating strain is ov

93 ay suggest that a sufficiently high level of herd immunity was reached during the first wave.

94 take into account the protective effects of herd immunity, we developed a new approach that combines

95 to result in a population level signature of herd immunity; we detail this and also discuss other pop

96 infants also benefited from household-level herd immunity when antenatal vaccination for every pregn

97 oth direct protection to those immunized and herd immunity, which is indirect protection of those who

98 ecticide resistance, which, with the loss of herd immunity, will increase the magnitude of future den

99 fluenza B virus are the result of changes in herd immunity, with reassortment continuously generating