ライフサイエンスコーパス: herpes

1 role for IFI16 in protection against genital herpes.

2 transmission, through prevention of neonatal herpes.

3 ompared with control patients without ocular herpes.

4 ted with protection against recurrent ocular herpes.

5 ichomoniasis (1 study; n = 779), and genital herpes (1 study; n = 779).

6 In a cohort of patients with genital herpes and healthy controls, the minor G allele of the I

7 rus 1 (HSV-1) causes cold sores and neonatal herpes and is a leading cause of blindness.

8 l estimates of the annual number of neonatal herpes cases during 2010-15.

9 We estimated that the most neonatal herpes cases occurred in Africa, due to high maternal HS

10 ula, gastroenteritis, viral gastroenteritis, herpes dermatitis, multiple fractures, respiratory failu

11 considered antiretrovirals, such as the anti-herpes drugs Aciclovir and Ganciclovir and the anti-canc

12 cted with HSV-1 KOS, which induces transient herpes epithelial keratitis without herpes stromal kerat

13 INTERPRETATION: These neonatal herpes estimates mark the first attempt to quantify the

14 Herpes simplex virus type 2 (HSV-2; herpes) exacerbates human immunodeficiency virus type 1

15 Some infections by viruses in the herpes family are more common in the earlier childbearin

16 transgenic mice and reduced recurrent ocular herpes following UV-B-induced reactivation.

17 tance in CD8(+) T cell immunity to recurrent herpes has been poorly elucidated.

18 eutic strategies to treat blinding recurrent herpes infection and disease.

19 Using a mouse model of genital herpes infection, we demonstrate that both antibodies an

20 Neonatal herpes infection, while uncommon, can result in substant

21 upper respiratory tract infections, and oral herpes infections were more frequent with ocrelizumab th

22 against primary and secondary female genital herpes infections.

23 n global estimates of the number of neonatal herpes infections.

24 Importance: Genital herpes is a prevalent sexually transmitted infection in

25 Neonatal herpes is a rare but potentially devastating condition w

26 Caused by the herpes simplex virus (HSV), herpes is a viral infection that is one of the most wide

27 d Relevance: Serologic screening for genital herpes is associated with a high rate of false-positive

28 Herpes is associated with phenotypic changes in CD4+ T-c

29 tter collection of primary data for neonatal herpes is crucially needed to reduce uncertainty and ref

30 es extract for the prophylactic treatment of herpes labialis and suggest possible activity of Ca-SP a

31 Many viruses, including herpes simplex (HSV), are recruited to their host cells

32 Herpes simplex encephalitis (HSE) is the most common for

33 cent reviews; these include the relevance of herpes simplex encephalitis and of epilepsy to AD, the a

34 netic association between TRIF mutations and herpes simplex encephalitis in patients.

35 l intervention is crucial to the survival of herpes simplex encephalitis patients; however, many surv

36 including necrotizing stromal keratitis and herpes simplex encephalitis.

37 ), polyoma (BK) viremia (8.2% vs 11.1%), and herpes simplex infections (5.5% vs 4.0%).

38 ree patients with a unilateral and relapsing herpes simplex keratitis (HSK group) that was quiescent

39 ity of life (QoL) in patients with quiescent herpes simplex keratitis compared with control patients

40 al patients were diagnosed sequentially with herpes simplex keratitis, then Acanthamoeba keratitis be

41 for dendritiform keratopathy, such as prior herpes simplex keratitis, varicella-zoster viral keratit

42 ated with antiviral medications for presumed herpes simplex keratitis; 4 patients underwent diagnosti

43 petic anterior uveitis (AU), owing to either herpes simplex or varicella zoster virus, by using the S

44 lopment of a safe and effective asymptomatic herpes simplex vaccine that is selectively based on CD8(

45 lopment of a safe and effective T-cell-based herpes simplex vaccine.

46 this assessment include adenovirus 8 and 19, herpes simplex virus (HSV) 1 and 2, human immunodeficien

47 Herpes simplex virus (HSV) 1 stimulates type I IFN expre

48 dy of neurotropic viruses in vitroIMPORTANCE Herpes simplex virus (HSV) affects millions of people wo

49 tions that US9 is neuron specific.IMPORTANCE Herpes simplex virus (HSV) and other alphaherpesviruses,

50 Herpes simplex virus (HSV) anterograde transport in neur

51 a broad antimicrobial factor that restricts herpes simplex virus (HSV) by activating type I interfer

52 The herpes simplex virus (HSV) capsid is released into the c

53 his study employed a novel strategy in which herpes simplex virus (HSV) carrying a small interfering

54 Herpes simplex virus (HSV) causes acute and relapsing sy

55 fluid (CSF) commonly predicts the absence of herpes simplex virus (HSV) central nervous system (CNS)

56 During lytic infection, herpes simplex virus (HSV) DNA is replicated by a mechan

57 Herpes simplex virus (HSV) entry into a subset of cells

58 erpesvirus entry mediator (HVEM) facilitates herpes simplex virus (HSV) entry through interactions wi

59 Herpes simplex virus (HSV) establishes a latent reservoi

60 Herpes simplex virus (HSV) establishes latency and react

61 is known as a transcriptional coactivator of herpes simplex virus (HSV) immediate early (IE) transcri

62 The herpes simplex virus (HSV) infected cell culture polypep

63 Neuron-virus interactions that occur during herpes simplex virus (HSV) infection are not fully under

64 Herpes simplex virus (HSV) infection is restricted to ep

65 Herpes simplex virus (HSV) infection is widespread in th

66 Herpes simplex virus (HSV) infections of the central ner

67 The terminase of herpes simplex virus (HSV) is composed of three subunits

68 pediatric penetrating keratoplasty (PPK) for herpes simplex virus (HSV) keratitis.

69 ctivator HCF-1 is required for initiation of herpes simplex virus (HSV) lytic infection and for react

70 f aqueous or vitreous humor was positive for herpes simplex virus (HSV) or varicella zoster virus (VZ

71 od to visualize pseudorabies virus (PRV) and herpes simplex virus (HSV) particles in living cells.

72 Despite frequent herpes simplex virus (HSV) reactivation, peripheral nerv

73 The rate of oral herpes simplex virus (HSV) shedding was lower overall, a

74 Chronic infections with herpes simplex virus (HSV) type 1 are highly prevalent i

75 The herpes simplex virus (HSV) type I alkaline nuclease, UL1

76 e by microinjecting before training a single herpes simplex virus (HSV) vector expressing either CRIS

77 Presence of CMV, EBV, and herpes simplex virus (HSV) were independent predictors o

78 Caused by the herpes simplex virus (HSV), herpes is a viral infection

79 Infection is caused by 2 subtypes of the herpes simplex virus (HSV), HSV-1 and HSV-2.

80 itro nanomolar irreversible activity against herpes simplex virus (HSV), human papilloma virus, respi

81 cell entry and subsequent lateral spread of herpes simplex virus (HSV), requires the four envelope g

82 Herpes simplex virus (HSV), which triggers the STING sig

83 cally significant species of simplexviruses, herpes simplex virus (HSV)-1 and HSV-2, with estimated d

84 In herpes simplex virus (HSV)-infected cells, ND10 bodies a

85 creased) particle-to-PFU ratio in vitro than herpes simplex virus (HSV).

86 enchymal brain inflammation, commonly due to herpes simplex virus (HSV).

87 interferon (IFN) is important for control of herpes simplex virus (HSV-1) in the central nervous syst

88 alone mediates recognition and clearance of herpes simplex virus (HSV1)-infected cells.

89 ablation on tga20/CD11b-thymidine kinase of Herpes simplex virus (HSVTK) cerebellar organotypic cult

90 Oncolytic herpes simplex virus (oHSV) selectively replicates in ca

91 nd tested the fate and efficacy of oncolytic herpes simplex virus (oHSV)-armed mesenchymal stem cells

92 n of foreign DNA-sensing pathways.IMPORTANCE Herpes simplex virus 1 (HSV-1) afflicts 80% of the popul

93 Herpes simplex virus 1 (HSV-1) and HSV-2 are large, doub

94 f UL37 homologs from two alphaherpesviruses, herpes simplex virus 1 (HSV-1) and pseudorabies virus (P

95 ar disease in virus-infected mice.IMPORTANCE Herpes simplex virus 1 (HSV-1) causes cold sores and neo

96 Herpes simplex virus 1 (HSV-1) encodes the multifunction

97 ponent of cell membranes and is required for herpes simplex virus 1 (HSV-1) entry (1-3).

98 The dissection of the herpes simplex virus 1 (HSV-1) entry mechanism is compli

99 ined whether Cbl mediates the removal of the herpes simplex virus 1 (HSV-1) entry receptor Nectin-1 f

100 Herpes simplex virus 1 (HSV-1) establishes latency withi

101 Herpes simplex virus 1 (HSV-1) establishes lifelong infe

102 ultaneous presence of a helper virus such as herpes simplex virus 1 (HSV-1) for productive replicatio

103 plication greatly enhances expression of the herpes simplex virus 1 (HSV-1) gamma2 late genes by stil

104 gp120) construct fused to a small portion of herpes simplex virus 1 (HSV-1) glycoprotein D (gD) so th

105 The herpes simplex virus 1 (HSV-1) ICP0 protein is an E3 ubi

106 mia nuclear bodies (PML NBs), is a target of herpes simplex virus 1 (HSV-1) ICP0-mediated degradation

107 Herpes simplex virus 1 (HSV-1) infection is an incurable

108 Herpes simplex virus 1 (HSV-1) infection is widespread a

109 efects in IFN responses can result in lethal herpes simplex virus 1 (HSV-1) infections, usually from

110 Herpes simplex virus 1 (HSV-1) infects most people and c

111 The immediate early protein ICP0 of herpes simplex virus 1 (HSV-1) interacts with CIN85, an

112 We report here that UL37 of herpes simplex virus 1 (HSV-1) is a protein deamidase th

113 Human herpes simplex virus 1 (HSV-1) is a widespread pathogen,

114 The UL16 tegument protein of herpes simplex virus 1 (HSV-1) is conserved among all he

115 Herpes simplex virus 1 (HSV-1) is one of the eight herpe

116 viral DNA from NPC-bound capsids.IMPORTANCE Herpes simplex virus 1 (HSV-1) is the causative agent of

117 nal (NLS) sequences previously identified in herpes simplex virus 1 (HSV-1) large terminase and human

118 Herpes simplex virus 1 (HSV-1) latency entails the repre

119 s from a variant to target the mRNA encoding herpes simplex virus 1 (HSV-1) major transcription regul

120 Herpes simplex virus 1 (HSV-1) most commonly causes recr

121 ve previously shown that the live-attenuated herpes simplex virus 1 (HSV-1) mutant lacking the nuclea

122 Herpes simplex virus 1 (HSV-1) remodels nuclear membrane

123 was to reexamine the requirement of UL21 for herpes simplex virus 1 (HSV-1) replication.

124 Infection of mature HD10.6 neurons by herpes simplex virus 1 (HSV-1) results in a delayed but

125 Ocular infection with herpes simplex virus 1 (HSV-1) sets off an inflammatory

126 sRNA, encephalomyocarditis virus (EMCV), and herpes simplex virus 1 (HSV-1) show impaired production

127 nd ICP34.5 are among the proteins encoded by herpes simplex virus 1 (HSV-1) that modulate type I IFN

128 e ability of the prototypic alphaherpesvirus herpes simplex virus 1 (HSV-1) to enter neurons via axon

129 Herpes simplex virus 1 (HSV-1) UL20 plays a crucial role

130 The herpes simplex virus 1 (HSV-1) UL37 protein functions in

131 Herein, we report that the live-attenuated herpes simplex virus 1 (HSV-1) VC2 vaccine strain, which

132 Following infection of epithelial tissues, herpes simplex virus 1 (HSV-1) virions travel via axonal

133 lected TRIM proteins in autophagy induced by herpes simplex virus 1 (HSV-1), encephalomyocarditis vir

134 Persistent pathogens, such as herpes simplex virus 1 (HSV-1), have evolved a variety o

135 roinvasive alphaherpesviruses, such as human herpes simplex virus 1 (HSV-1), HSV-2, and veterinarian

136 trast, of the more than 80 mRNAs harbored by herpes simplex virus 1 (HSV-1), only 5 are spliced.

137 Following infection with herpes simplex virus 1 (HSV-1), PIAS1 is recruited to nu

138 Here we focus on two tegument proteins from herpes simplex virus 1 (HSV-1), pUL7 and pUL51, which ha

139 Although many herpesviruses, including herpes simplex virus 1 (HSV-1), stimulate mTORC1, how HS

140 zuelan equine encephalitis virus (VEEV), and herpes simplex virus 1 (HSV-1), suggesting that LIMK inh

141 For herpes simplex virus 1 (HSV-1), we and others have previ

142 Herpes simplex virus 1 (HSV-1), which causes a variety o

143 Although most herpes simplex virus 1 (HSV-1)-infected individuals shed

144 esponse to restrict viral pathogens, such as herpes simplex virus 1 (HSV-1).

145 SP141), bound to RIG-I during infection with herpes simplex virus 1 (HSV-1).

146 between AAV2 and one of its helper viruses, herpes simplex virus 1 (HSV-1).

147 f mice that have been ocularly infected with herpes simplex virus 1 (HSV-1).

148 choriomeningitis virus (LCMV) infection, or herpes simplex virus 1 (HSV1) infection was profoundly d

149 us agalactiae, cytomegalovirus, enterovirus, herpes simplex virus 1 and 2, human herpesvirus 6, human

150 ability to be infected by the herpesviruses herpes simplex virus 1 and murine herpesvirus 68 and the

151 Here, we have characterized the full-length herpes simplex virus 1 gB in a native membrane by displa

152 pted this method to rapidly deliver a 152 kb herpes simplex virus 1 genome cloned in yeast into mamma

153 e the molecular organization of chromatin in herpes simplex virus 1 infection and its effect on the t

154 promised survival during influenza virus and herpes simplex virus 1 infections.

155 er replication of BKV, whereas influenza and herpes simplex virus 1 replication were clearly reduced.

156 study focuses on two tegument proteins from herpes simplex virus 1 that are conserved in all herpesv

157 tein domains, as well as a truncated form of herpes simplex virus 1 thymidine kinase (HSV-TK).

158 wed evidence of acute herpesvirus infection; herpes simplex virus 1 was found most often.

159 tudy, the structural, material properties of herpes simplex virus 1 were investigated.

160 e, whereas infection with influenza A virus, herpes simplex virus 1, or cytomegalovirus induced a str

161 Random walk modelling of herpes simplex virus 1-sized particles in a three-dimens

162 viously established that cells infected with herpes simplex virus 2 (HSV-2) are disrupted in their ab

163 Several prophylactic vaccines targeting herpes simplex virus 2 (HSV-2) have failed in the clinic

164 llions of people worldwide are infected with herpes simplex virus 2 (HSV-2), and to date, an efficaci

165 Alpha herpesviruses, such as herpes simplex virus and pseudorabies virus (PRV), are n

166 5 on the structural stability of icosahedral herpes simplex virus capsids.

167 ital specimens each containing >/=105 copies herpes simplex virus DNA/ml collected a median of 5 mont

168 The transcription factor ICP4 from herpes simplex virus has a central role in regulating th

169 h inhibits HIV-1, HIV-2, Influenza virus and herpes simplex virus infection, and enhances the potenti

170 icase-primase inhibitor for the treatment of herpes simplex virus infections, was prepared.

171 r development of antiviral agents.IMPORTANCE Herpes simplex virus is a major pathogen, and although n

172 The capsid of herpes simplex virus is built up of a variety of protein

173 ovir decreased the recurrence of any type of herpes simplex virus keratitis by approximately half.

174 nfections of the superficial cornea, such as herpes simplex virus keratitis or Acanthamoeba keratitis

175 ) relocation of nonintegrin receptors (e.g., herpes simplex virus nectin1 and Kaposi's sarcoma-associ

176 h clinically diagnosed ARN, PCR-positive for herpes simplex virus or varicella zoster virus and evalu

177 iral reactivation, exhibiting parallels with herpes simplex virus reactivation.

178 of the lipid raft-dwelling US9 protein from Herpes Simplex Virus strikingly overlaps with that of th

179 transgenic mice expressing the suicide gene, herpes simplex virus thymidine kinase (HSVtk), driven by

180 g AAVP particles served to deliver the human Herpes simplex virus thymidine kinase type-1 (HSVtk) gen

181 Infectious titers of EBOV or herpes simplex virus type 1 (HSV-1) in detergents-treate

182 Lytic infection with herpes simplex virus type 1 (HSV-1) induces profound mod

183 Herpes simplex virus type 1 (HSV-1) is a leading cause o

184 Glycoprotein D (gD) of herpes simplex virus type 1 (HSV-1) is one of four glyco

185 lly occurs during delivery from mothers with herpes simplex virus type 1 (HSV-1) or type 2 (HSV-2) ge

186 ological stress.IMPORTANCE Like all viruses, herpes simplex virus type 1 (HSV-1) reproduction relies

187 we reported a new series of highly defective herpes simplex virus type 1 (HSV-1) vectors that were fu

188 ormational approach to genome engineering of herpes simplex virus type 1 (HSV-1), which has a large D

189 Knockout of TRIM14 impairs herpes simplex virus type 1 (HSV-1)-triggered antiviral

190 Support for the concept that herpes simplex virus type 1 (HSV1), when present in the

191 Herpes simplex virus type 1 and Alzheimer's disease: pos

192 athway are associated with susceptibility to herpes simplex virus type 1 encephalitis (HSE).

193 We found wide distribution of O-glycans on herpes simplex virus type 1 glycoproteins and demonstrat

194 gene encoding the prodrug-converting enzyme herpes simplex virus type 1 thymidine kinase (HSV1-tk) i

195 CMV, herpes simplex virus type 1, and human herpesvirus 6 inf

196 e association of 4 human herpesviruses (CMV, herpes simplex virus type 1, human herpesvirus type 6, a

197 pathogens were detected by mNGS (4 cases of herpes simplex virus type 1, including 1 case of coinfec

198 fumarate (TDF) has in vitro activity against herpes simplex virus type 2 (HSV-2) and reduced HSV-2 ac

199 Despite the high prevalence of herpes simplex virus type 2 (HSV-2) in sub-Saharan Afric

200 Genital herpes simplex virus type 2 (HSV-2) infection causes rec

201 mmune system's protective effect against one herpes simplex virus type 2 (HSV-2) infection protects a

202 or NK cell function in response to a mucosal herpes simplex virus type 2 (HSV-2) infection.

203 HIV and herpes simplex virus type 2 (HSV-2) infections cause a s

204 ART) has been incompletely characterized for herpes simplex virus type 2 (HSV-2).

205 Herpes simplex virus type 2 (HSV-2; herpes) exacerbates

206 against cervical cancer, cervical dysplasia, herpes simplex virus type 2, chlamydia, and syphilis.

207 f herpetic neuralgia using a murine model of Herpes Simplex Virus Type-1 (HSV-1) infection.

208 CB) women, including bacterial vaginosis and herpes simplex virus type-2 (HSV-2) infection.

209 he impact of antiretroviral therapy (ART) on herpes simplex virus type-2 (HSV-2) replication is uncle

210 ded controls for sociodemographic variables, herpes simplex virus type-2 status, and recreational dru

211 rr virus (EBV), human herpesvirus 6 (HHV-6), herpes simplex virus types 1 (HSV-1) and 2 (HSV-2), and

212 ein Barr virus (EBV), cytomegalovirus (CMV), herpes simplex virus types 1 and 2, human herpesvirus 8)

213 iency virus, hepatitis B virus, and neonatal herpes simplex virus, from which lessons for the evaluat

214 ing of mucosal immunity and then compare the herpes simplex virus, human immunodeficiency virus, and

215 papilloma virus, hepatitis B and C viruses, herpes simplex virus, norovirus, rotavirus, parvovirus,

216 use substantial impairment: cytomegalovirus, herpes simplex virus, rubella virus, Toxoplasma gondii,

217 ded toxoplasmosis, rubella, cytomegalovirus, herpes simplex virus, syphilis, and human immunodeficien

218 97%; PPV range, 52%-95%; NPV range, 79%-80%; herpes simplex virus, vulvar ulcerations: sensitivity, 2

219 Herpes simplex virus-1 (HSV-1) causes life-long morbidit

220 Herpes Simplex Virus-1 (HSV-1) is a ubiquitous human pat

221 76-9 (MHC I low) tumors respond to oncolytic herpes simplex virus-1 (oHSV-1) and PD-1 blockade combin

222 ers in the lung, and are resistant to lethal herpes simplex virus-1 infection due to enhanced product

223 Here, we address this deficit, focusing on a herpes simplex virus-1 protein, ICP27.

224 Furthermore, an unrelated herpes simplex virus-thymidine kinase (HSV-TK) promoter

225 r, and parvovirus B19), cytomegalovirus, and herpes simplex virus.

226 e fusion also proved to be well tolerated in herpes simplex virus.

227 Herpes simplex viruses (HSV) are human pathogens that sw

228 significant effect.IMPORTANCE Infections by herpes simplex viruses (HSV) cause painful cold sores or

229 Herpes simplex viruses 1 and 2 (HSV-1 and HSV-2) infect

230 rus, mumps virus, measles virus, lyssavirus, herpes simplex viruses 1 and 2, Epstein-Barr virus, ente

231 domains from the Drosophila fushi tarazu and Herpes simplex VP16 was created.

232 polymerase chain reaction (PCR) analysis for Herpes simplex, varicella zoster, cytomegalovirus, Epste

233 ere independent of varicella-zoster virus or herpes-simplex virus 1 coinfection.

234 viruses: KSHV, Epstein-Barr virus (EBV), and herpes simplexvirus-2 (HSV-2).

235 ransient herpes epithelial keratitis without herpes stromal keratitis sequelae, possessed a significa

236 transient discomfort to the blinding disease herpes stromal keratitis, yet most patients experience a

237 ngs, a novel "prime/pull" therapeutic ocular herpes vaccine strategy is proposed and discussed.

238 This study shows that a herpes viral infection of FMs sensitizes the tissue to l

239 a-associated lymphoid tissue lymphoma, human herpes virus (HHV)-6, HHV-7, chlamydia, Epstein-Barr vir

240 Kaposi's sarcoma-associated herpes virus (KSHV) polyadenylated nuclear (PAN) RNA fac

241 Human herpes virus 8 (HHV-8), also known as Kaposi's sarcoma a

242 tivity; 14.9% (95% CI: 12.4-17.4%) for human herpes virus 8 infection.

243 Kaposi sarcoma-associated herpesvirus/human herpes virus 8 replication and uptake of the ORF45 tegum

244 Kaposi sarcoma-associated herpesvirus/human herpes virus 8.

245 ng respiratory virus infection, we show that herpes virus entry mediator (HVEM; TNFRSF14), a member o

246 Epstein-Barr virus (EBV) is a human herpes virus that infects >90% of individuals.

247 cancer), viral (e.g., human papillomavirus, herpes virus) and bacterial (e.g., Helicobacter pylori,

248 ovirus (HCMV) is the prototypical human beta-herpes virus.

249 We found that herpes viruses (e.g., subtypes HHV4, HHV5, and HHV6) tha

250 ivo human tonsillar tissue system) and human herpes viruses.

251 of activity that also covers hepatitis B and herpes viruses.

252 Serologic screening for genital herpes was associated with psychosocial harms, including

253 Globally the overall rate of neonatal herpes was estimated to be about ten cases per 100 000 l

254 erial vaginosis, trichomoniasis, and genital herpes was performed in a high-prevalence population wit

255 Persons with genital herpes were randomized into 3 dose cohorts to receive 3

256 1.04; 95% confidence interval 1.00-1.08) or herpes zoster (adjusted HR 1.03; 95% confidence interval

257 Tens of millions of seniors are at risk of herpes zoster (HZ) and its complications.

258 on emergency department (ED) utilization for herpes zoster (HZ) has not been examined to date.

259 Temporal increases in the incidence of herpes zoster (HZ) have been reported but studies have e

260 The incidence and severity of herpes zoster (HZ) increases with age.

261 Protection against herpes zoster (HZ) induced by the live attenuated zoster

262 e in a healthy population, the protection of herpes zoster (HZ) vaccine in end-stage renal disease (E

263 on for the four catch-up cohorts was 33% for herpes zoster (incidence rate ratio 0.67 [0.61-0.74]) an

264 Herpes zoster (shingles) causes significant morbidity in

265 equivalent to about 17 000 fewer episodes of herpes zoster and 3300 fewer episodes of postherpetic ne

266 a vaccine effectiveness of about 62% against herpes zoster and 70-88% against postherpetic neuralgia.

267 o estimate vaccine coverage and incidence of herpes zoster and postherpetic neuralgia consultations.

268 of the vaccination programme on incidence of herpes zoster and postherpetic neuralgia in this populat

269 ultations with patients aged 60-89 years for herpes zoster and postherpetic neuralgia occurring betwe

270 cinations administered and consultations for herpes zoster and postherpetic neuralgia, and aggregated

271 ce-attenuated vaccine against chickenpox and herpes zoster and providing a new target for interventio

272 To examine whether herpes zoster antigen (also called varicella-zoster viru

273 Herpes zoster antigen was detected in 3 of 25 temporal a

274 ups of patients, false-positive staining for herpes zoster antigen was detected in the presence of ca

275 False-positive staining for herpes zoster antigen was detected on several temporal a

276 Among the GCA-negative group, herpes zoster antigen was not detected in any biopsy.

277 group, 3 patients had positive staining for herpes zoster antigen.

278 r virus is an important human pathogen, with herpes zoster being a major health issue in the aging an

279 is commonly thought to increase the risk of herpes zoster by causing immunosuppression.

280 c neuralgia is the most important symptom of herpes zoster disease and it is very difficult to treat.

281 c neuralgia is the most important symptom of herpes zoster disease, which is caused by Varicella zost

282 for the three routine cohorts, incidence of herpes zoster fell by 35% (incidence rate ratio 0.65 [95

283 ated varicella-zoster virus vaccine prevents herpes zoster in adults older than 50 years.

284 three serious infections, and four cases of herpes zoster in patients who received tofacitinib durin

285 oups, and the rates of overall infection and herpes zoster infection were higher with tofacitinib tha

286 s, there were four serious infections, three herpes zoster infections, one myocardial infarction, and

287 5175 person-years of follow-up, 26 cases of herpes zoster occurred among those assigned acyclovir, c

288 ical record can reliably be used to identify herpes zoster ophthalmicus cases.

289 s was noteworthy because the patient had had herpes zoster ophthalmicus diagnosed 3 weeks before the

290 e immunogenicity and safety of an adjuvanted herpes zoster subunit (HZ/su) vaccine when coadministere

291 INTERPRETATION: The herpes zoster vaccination programme in England has had a

292 In 2013, a herpes zoster vaccination programme was introduced in En

293 We identified individual data on herpes zoster vaccinations administered and consultation

294 Live attenuated herpes zoster vaccine (HZV) reduces that risk, although

295 l outcomes of myocardial infarction (MI) and herpes zoster were also studied.

296 ced transient local skin reactions: 1 (4.8%) herpes zoster, 3 (14.3%) transaminase elevation, and 1 (

297 eased incidences of cytomegalovirus disease, herpes zoster, BK virus, and nephropathy, which led to t

298 on for development of treatments for painful herpes zoster.

299 s a highly contagious agent of varicella and herpes zoster.

300 VZV) is the causative agent of varicella and herpes zoster.