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1 nts with idiopathic scleritis and those with herpes infection.
2 ccurately diagnose anogenital lesions due to herpes infection.
3 significance of HS and 3-OS HS during ocular herpes infection.
4 D) contribute to protective immunity against herpes infection.
5 en (36 percent) had symptoms consistent with herpes infection.
6 al morbidity or with any cases of congenital herpes infection.
7 e seropositive reported a history of genital herpes infection.
8 n global estimates of the number of neonatal herpes infections.
9 against primary and secondary female genital herpes infections.
10 vaccine formulation that could combat ocular herpes infection and disease in humans.
11 eutic strategies to treat blinding recurrent herpes infection and disease.
12 dependent protective immunity against ocular herpes infection and disease.
13  epitope (gB498-505), protect against ocular herpes infection and disease.
14 ective immunity against sexually transmitted herpes infection and disease.
15 epitope-based vaccine against primary ocular herpes infection and disease.
16 onses, associated with a reduction in ocular herpes infection and disease.
17 D T-cell epitopes protected mice from ocular herpes infection and disease.
18 dependent protective immunity against ocular herpes infection and disease.
19 B)-specific CD8(+) T cells protect mice from herpes infection and disease.
20  a strong protective immunity against ocular herpes infection and disease.
21 mate the prevalence and incidence of genital herpes infection and to assess the relation between HSV-
22                                              Herpes infections are among the most common sexually tra
23                                              Herpes infections are among the most common sexually tra
24 rvix and ectocervix/vagina) to mimic genital herpes infections caused by herpes simplex virus types 1
25 ncreased risk of PTD associated with genital herpes infection if left untreated and a potential benef
26 effective for blocking the spread of topical herpes infection in an animal model.
27 ive for the suppression of recurrent genital herpes infection in HIV-infected individuals.
28 can provide passive immunity against genital herpes infections in mice; orally administered polymeric
29 utinely used for topical treatment of ocular herpes infections in the United States.
30 caused by HSV-2, which suggests that genital herpes infection likely increases the efficiency of the
31 ications in mitigating the effect of genital herpes infection on the risk of PTD.
32 ma, iron or nutrient starvation, concomitant herpes infection, or maturation of the host cell into it
33 tric infectious diseases concerning neonatal herpes infections, poliovirus immunization schedule, and
34  (16%) patients treated with alemtuzumab had herpes infections (predominantly cutaneous) compared wit
35               Importance: Current therapy of herpes infections relies on nucleoside analogues.
36  has been shown to protect mice from genital herpes infection, the mechanism by which these agents pr
37       In diseases such as diabetes, HIV, and herpes infections, the resultant neuropathic pain is oft
38              Interestingly, during an ocular herpes infection, there was a selective increase in the
39 he proportion of HSV-1 among initial genital herpes infections was higher among men who had sex with
40               Using a mouse model of genital herpes infection, we demonstrate that both antibodies an
41 upper respiratory tract infections, and oral herpes infections were more frequent with ocrelizumab th
42                                     Neonatal herpes infection, while uncommon, can result in substant
43              A vaccine that prevents genital herpes infection will have major public health benefits.

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