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1 nts with idiopathic scleritis and those with herpes infection.
2 ccurately diagnose anogenital lesions due to herpes infection.
3 significance of HS and 3-OS HS during ocular herpes infection.
4 D) contribute to protective immunity against herpes infection.
5 en (36 percent) had symptoms consistent with herpes infection.
6 al morbidity or with any cases of congenital herpes infection.
7 e seropositive reported a history of genital herpes infection.
8 n global estimates of the number of neonatal herpes infections.
9 against primary and secondary female genital herpes infections.
21 mate the prevalence and incidence of genital herpes infection and to assess the relation between HSV-
24 rvix and ectocervix/vagina) to mimic genital herpes infections caused by herpes simplex virus types 1
25 ncreased risk of PTD associated with genital herpes infection if left untreated and a potential benef
28 can provide passive immunity against genital herpes infections in mice; orally administered polymeric
30 caused by HSV-2, which suggests that genital herpes infection likely increases the efficiency of the
32 ma, iron or nutrient starvation, concomitant herpes infection, or maturation of the host cell into it
33 tric infectious diseases concerning neonatal herpes infections, poliovirus immunization schedule, and
34 (16%) patients treated with alemtuzumab had herpes infections (predominantly cutaneous) compared wit
36 has been shown to protect mice from genital herpes infection, the mechanism by which these agents pr
39 he proportion of HSV-1 among initial genital herpes infections was higher among men who had sex with
41 upper respiratory tract infections, and oral herpes infections were more frequent with ocrelizumab th
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