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1                                              Herpes simplex encephalitis (HSE) is the most common for
2                                          The herpes simplex encephalitis (HSE) model was used for ind
3 cent reviews; these include the relevance of herpes simplex encephalitis and of epilepsy to AD, the a
4                                     Although herpes simplex encephalitis has been extensively studied
5 netic association between TRIF mutations and herpes simplex encephalitis in patients.
6 l intervention is crucial to the survival of herpes simplex encephalitis patients; however, many surv
7 al keratitis, highly debilitating and lethal herpes simplex encephalitis, and generalized infections
8  including necrotizing stromal keratitis and herpes simplex encephalitis.
9                      Many viruses, including herpes simplex (HSV), are recruited to their host cells
10 ), polyoma (BK) viremia (8.2% vs 11.1%), and herpes simplex infections (5.5% vs 4.0%).
11                                      Genital herpes simplex is one of the most prevalent sexually tra
12 ree patients with a unilateral and relapsing herpes simplex keratitis (HSK group) that was quiescent
13 ity of life (QoL) in patients with quiescent herpes simplex keratitis compared with control patients
14  group initially were misdiagnosed as having herpes simplex keratitis versus 59 (41.8%) patients who
15 al patients were diagnosed sequentially with herpes simplex keratitis, then Acanthamoeba keratitis be
16  for dendritiform keratopathy, such as prior herpes simplex keratitis, varicella-zoster viral keratit
17 ated with antiviral medications for presumed herpes simplex keratitis; 4 patients underwent diagnosti
18 petic anterior uveitis (AU), owing to either herpes simplex or varicella zoster virus, by using the S
19 lopment of a safe and effective asymptomatic herpes simplex vaccine that is selectively based on CD8(
20 lopment of a safe and effective T-cell-based herpes simplex vaccine.
21 polymerase chain reaction (PCR) analysis for Herpes simplex, varicella zoster, cytomegalovirus, Epste
22 this assessment include adenovirus 8 and 19, herpes simplex virus (HSV) 1 and 2, human immunodeficien
23                                              Herpes simplex virus (HSV) 1 stimulates type I IFN expre
24 dy of neurotropic viruses in vitroIMPORTANCE Herpes simplex virus (HSV) affects millions of people wo
25 tions that US9 is neuron specific.IMPORTANCE Herpes simplex virus (HSV) and other alphaherpesviruses,
26                                              Herpes simplex virus (HSV) anterograde transport in neur
27  a broad antimicrobial factor that restricts herpes simplex virus (HSV) by activating type I interfer
28                                          The herpes simplex virus (HSV) capsid is released into the c
29 his study employed a novel strategy in which herpes simplex virus (HSV) carrying a small interfering
30                                              Herpes simplex virus (HSV) causes acute and relapsing sy
31 fluid (CSF) commonly predicts the absence of herpes simplex virus (HSV) central nervous system (CNS)
32                After entry into the nucleus, herpes simplex virus (HSV) DNA is coated with repressive
33                      During lytic infection, herpes simplex virus (HSV) DNA is replicated by a mechan
34                                              Herpes simplex virus (HSV) entry into a subset of cells
35 erpesvirus entry mediator (HVEM) facilitates herpes simplex virus (HSV) entry through interactions wi
36                                              Herpes simplex virus (HSV) establishes a latent reservoi
37                                              Herpes simplex virus (HSV) establishes latency and react
38 xpression of 'pore dead' GluA1 subunits (via herpes simplex virus (HSV) GluA1-Q582E) in the lateral c
39         Pritelivir is a well-tolerated novel herpes simplex virus (HSV) helicase-primase inhibitor th
40 is known as a transcriptional coactivator of herpes simplex virus (HSV) immediate early (IE) transcri
41                                          The herpes simplex virus (HSV) infected cell culture polypep
42  Neuron-virus interactions that occur during herpes simplex virus (HSV) infection are not fully under
43                          Importance: Genital herpes simplex virus (HSV) infection is a prevalent sexu
44                                              Herpes simplex virus (HSV) infection is restricted to ep
45                                              Herpes simplex virus (HSV) infection is widespread in th
46 for antithrombin III and plays a key role in herpes simplex virus (HSV) infection.
47                                   Studies of herpes simplex virus (HSV) infections of humans are limi
48                                              Herpes simplex virus (HSV) infections of the central ner
49                                 During lytic herpes simplex virus (HSV) infections, the virion host s
50                             The terminase of herpes simplex virus (HSV) is composed of three subunits
51                                              Herpes simplex virus (HSV) is investigated not only as a
52 pediatric penetrating keratoplasty (PPK) for herpes simplex virus (HSV) keratitis.
53 ctivator HCF-1 is required for initiation of herpes simplex virus (HSV) lytic infection and for react
54 f aqueous or vitreous humor was positive for herpes simplex virus (HSV) or varicella zoster virus (VZ
55 od to visualize pseudorabies virus (PRV) and herpes simplex virus (HSV) particles in living cells.
56                             Despite frequent herpes simplex virus (HSV) reactivation, peripheral nerv
57                             The rate of oral herpes simplex virus (HSV) shedding was lower overall, a
58                      Chronic infections with herpes simplex virus (HSV) type 1 are highly prevalent i
59                                          The herpes simplex virus (HSV) type I alkaline nuclease, UL1
60                      Samples were tested for herpes simplex virus (HSV) types 1 and 2, Epstein-Barr v
61  synthesis during multiround transmission of herpes simplex virus (HSV) using pulse-labeling with eth
62 e by microinjecting before training a single herpes simplex virus (HSV) vector expressing either CRIS
63 tential role of a TLR4/MCP-1 signal, we used Herpes Simplex Virus (HSV) vectors (amplicons) that reta
64                    Presence of CMV, EBV, and herpes simplex virus (HSV) were independent predictors o
65                                Caused by the herpes simplex virus (HSV), herpes is a viral infection
66     Infection is caused by 2 subtypes of the herpes simplex virus (HSV), HSV-1 and HSV-2.
67 itro nanomolar irreversible activity against herpes simplex virus (HSV), human papilloma virus, respi
68  cell entry and subsequent lateral spread of herpes simplex virus (HSV), requires the four envelope g
69                                              Herpes simplex virus (HSV), which triggers the STING sig
70                                              Herpes simplex virus (HSV)-1 and HSV-2 are significant h
71 cally significant species of simplexviruses, herpes simplex virus (HSV)-1 and HSV-2, with estimated d
72 ACASI), before test HIV counselling, HIV and herpes simplex virus (HSV)-2 testing, and post-test coun
73                                           In herpes simplex virus (HSV)-infected cells, ND10 bodies a
74 creased) particle-to-PFU ratio in vitro than herpes simplex virus (HSV).
75 enchymal brain inflammation, commonly due to herpes simplex virus (HSV).
76 interferon (IFN) is important for control of herpes simplex virus (HSV-1) in the central nervous syst
77 e delivered to the same sensory neurons that herpes simplex virus (HSV-1) infects when applied periph
78  alone mediates recognition and clearance of herpes simplex virus (HSV1)-infected cells.
79  ablation on tga20/CD11b-thymidine kinase of Herpes simplex virus (HSVTK) cerebellar organotypic cult
80                                    Oncolytic herpes simplex virus (oHSV) selectively replicates in ca
81 nd tested the fate and efficacy of oncolytic herpes simplex virus (oHSV)-armed mesenchymal stem cells
82 n of foreign DNA-sensing pathways.IMPORTANCE Herpes simplex virus 1 (HSV-1) afflicts 80% of the popul
83                                              Herpes simplex virus 1 (HSV-1) and HSV-2 are large, doub
84 qualitative detection and differentiation of herpes simplex virus 1 (HSV-1) and HSV-2 DNA in 1,351 cu
85                                              Herpes simplex virus 1 (HSV-1) and HSV-2 infect many hum
86 f UL37 homologs from two alphaherpesviruses, herpes simplex virus 1 (HSV-1) and pseudorabies virus (P
87 hat gB homologs from two alphaherpesviruses, herpes simplex virus 1 (HSV-1) and saimiriine herpesviru
88 te aspects of intrinsic immunity to restrict herpes simplex virus 1 (HSV-1) as well as other viruses.
89                                          The herpes simplex virus 1 (HSV-1) capsid is a huge assembly
90 ar disease in virus-infected mice.IMPORTANCE Herpes simplex virus 1 (HSV-1) causes cold sores and neo
91                                              Herpes simplex virus 1 (HSV-1) encodes the multifunction
92                                              Herpes simplex virus 1 (HSV-1) enters mice via olfactory
93 ponent of cell membranes and is required for herpes simplex virus 1 (HSV-1) entry (1-3).
94                        The dissection of the herpes simplex virus 1 (HSV-1) entry mechanism is compli
95 ined whether Cbl mediates the removal of the herpes simplex virus 1 (HSV-1) entry receptor Nectin-1 f
96                                              Herpes simplex virus 1 (HSV-1) establishes latency withi
97                                              Herpes simplex virus 1 (HSV-1) establishes lifelong infe
98 ultaneous presence of a helper virus such as herpes simplex virus 1 (HSV-1) for productive replicatio
99 plication greatly enhances expression of the herpes simplex virus 1 (HSV-1) gamma2 late genes by stil
100                                              Herpes simplex virus 1 (HSV-1) glycoprotein B (gB)-speci
101 gp120) construct fused to a small portion of herpes simplex virus 1 (HSV-1) glycoprotein D (gD) so th
102                                          The herpes simplex virus 1 (HSV-1) ICP0 protein is an E3 ubi
103 mia nuclear bodies (PML NBs), is a target of herpes simplex virus 1 (HSV-1) ICP0-mediated degradation
104                                 For example, herpes simplex virus 1 (HSV-1) immediate early (IE) prot
105    As HCF1 is best known for its function in herpes simplex virus 1 (HSV-1) immediate early gene tran
106 teins have some functional similarities with herpes simplex virus 1 (HSV-1) immediate early protein I
107 tinocytes represent a primary entry site for herpes simplex virus 1 (HSV-1) in vivo.
108                                 Uncontrolled herpes simplex virus 1 (HSV-1) infection can advance to
109                                              Herpes simplex virus 1 (HSV-1) infection is an incurable
110                                              Herpes simplex virus 1 (HSV-1) infection is widespread a
111 f the intrinsic antiviral immune response to herpes simplex virus 1 (HSV-1) infection.
112 efects in IFN responses can result in lethal herpes simplex virus 1 (HSV-1) infections, usually from
113                                              Herpes simplex virus 1 (HSV-1) infects humans through st
114                                              Herpes simplex virus 1 (HSV-1) infects most people and c
115          The immediate early protein ICP0 of herpes simplex virus 1 (HSV-1) interacts with CIN85, an
116                  We report here that UL37 of herpes simplex virus 1 (HSV-1) is a protein deamidase th
117                                        Human herpes simplex virus 1 (HSV-1) is a widespread pathogen,
118                                              Herpes simplex virus 1 (HSV-1) is among the most common
119            Infected cell protein 0 (ICP0) of herpes simplex virus 1 (HSV-1) is an alpha gene product
120                 The UL16 tegument protein of herpes simplex virus 1 (HSV-1) is conserved among all he
121                                              Herpes simplex virus 1 (HSV-1) is one of the eight herpe
122  viral DNA from NPC-bound capsids.IMPORTANCE Herpes simplex virus 1 (HSV-1) is the causative agent of
123                We have shown previously that herpes simplex virus 1 (HSV-1) lacking expression of the
124 nal (NLS) sequences previously identified in herpes simplex virus 1 (HSV-1) large terminase and human
125                                              Herpes simplex virus 1 (HSV-1) latency entails the repre
126 s from a variant to target the mRNA encoding herpes simplex virus 1 (HSV-1) major transcription regul
127                                              Herpes simplex virus 1 (HSV-1) most commonly causes recr
128 ve previously shown that the live-attenuated herpes simplex virus 1 (HSV-1) mutant lacking the nuclea
129                    During DNA encapsidation, herpes simplex virus 1 (HSV-1) procapsids are converted
130                                              Herpes simplex virus 1 (HSV-1) remodels nuclear membrane
131 ivating protein, p35, is important for acute herpes simplex virus 1 (HSV-1) replication in mice.
132 was to reexamine the requirement of UL21 for herpes simplex virus 1 (HSV-1) replication.
133        Infection of mature HD10.6 neurons by herpes simplex virus 1 (HSV-1) results in a delayed but
134                        Ocular infection with herpes simplex virus 1 (HSV-1) sets off an inflammatory
135 sRNA, encephalomyocarditis virus (EMCV), and herpes simplex virus 1 (HSV-1) show impaired production
136 nd ICP34.5 are among the proteins encoded by herpes simplex virus 1 (HSV-1) that modulate type I IFN
137                 Humans occasionally transmit herpes simplex virus 1 (HSV-1) to captive primates, who
138 e ability of the prototypic alphaherpesvirus herpes simplex virus 1 (HSV-1) to enter neurons via axon
139                                              Herpes simplex virus 1 (HSV-1) typically causes recurren
140                                              Herpes simplex virus 1 (HSV-1) UL20 plays a crucial role
141                                          The herpes simplex virus 1 (HSV-1) UL37 protein functions in
142 ogic protection requisite for an efficacious herpes simplex virus 1 (HSV-1) vaccine remain unclear wi
143   Herein, we report that the live-attenuated herpes simplex virus 1 (HSV-1) VC2 vaccine strain, which
144   Following infection of epithelial tissues, herpes simplex virus 1 (HSV-1) virions travel via axonal
145                                              Herpes simplex virus 1 (HSV-1), a leading cause of genit
146 lected TRIM proteins in autophagy induced by herpes simplex virus 1 (HSV-1), encephalomyocarditis vir
147                Persistent pathogens, such as herpes simplex virus 1 (HSV-1), have evolved a variety o
148 roinvasive alphaherpesviruses, such as human herpes simplex virus 1 (HSV-1), HSV-2, and veterinarian
149 trast, of the more than 80 mRNAs harbored by herpes simplex virus 1 (HSV-1), only 5 are spliced.
150                     Following infection with herpes simplex virus 1 (HSV-1), PIAS1 is recruited to nu
151  Here we focus on two tegument proteins from herpes simplex virus 1 (HSV-1), pUL7 and pUL51, which ha
152       Although many herpesviruses, including herpes simplex virus 1 (HSV-1), stimulate mTORC1, how HS
153 zuelan equine encephalitis virus (VEEV), and herpes simplex virus 1 (HSV-1), suggesting that LIMK inh
154 as the replication of a related herpesvirus, herpes simplex virus 1 (HSV-1), was not impacted.
155                                          For herpes simplex virus 1 (HSV-1), we and others have previ
156                                              Herpes simplex virus 1 (HSV-1), which causes a variety o
157                                Although most herpes simplex virus 1 (HSV-1)-infected individuals shed
158 esponse to restrict viral pathogens, such as herpes simplex virus 1 (HSV-1).
159  ganglia (TG) of mice latently infected with herpes simplex virus 1 (HSV-1).
160 SP141), bound to RIG-I during infection with herpes simplex virus 1 (HSV-1).
161  between AAV2 and one of its helper viruses, herpes simplex virus 1 (HSV-1).
162 f mice that have been ocularly infected with herpes simplex virus 1 (HSV-1).
163  choriomeningitis virus (LCMV) infection, or herpes simplex virus 1 (HSV1) infection was profoundly d
164 svirus [KSHV], Epstein-Barr virus [EBV], and herpes simplex virus 1 [HSV-1]) genomes and induces the
165 unoglobulin M/immunoglobulin G antibodies to herpes simplex virus 1 and 2, cytomegalovirus, Epstein-B
166 us agalactiae, cytomegalovirus, enterovirus, herpes simplex virus 1 and 2, human herpesvirus 6, human
167 ibition decreases replication of adenovirus, herpes simplex virus 1 and influenza A in cultured prima
168  ability to be infected by the herpesviruses herpes simplex virus 1 and murine herpesvirus 68 and the
169  Here, we have characterized the full-length herpes simplex virus 1 gB in a native membrane by displa
170 pted this method to rapidly deliver a 152 kb herpes simplex virus 1 genome cloned in yeast into mamma
171 nt productive infection of cultured cells by herpes simplex virus 1 has made it a paradigm for this m
172 e the molecular organization of chromatin in herpes simplex virus 1 infection and its effect on the t
173 155 expression was up-regulated after ocular herpes simplex virus 1 infection, with the increased Mir
174 promised survival during influenza virus and herpes simplex virus 1 infections.
175 er replication of BKV, whereas influenza and herpes simplex virus 1 replication were clearly reduced.
176  study focuses on two tegument proteins from herpes simplex virus 1 that are conserved in all herpesv
177 tein domains, as well as a truncated form of herpes simplex virus 1 thymidine kinase (HSV-TK).
178 wed evidence of acute herpesvirus infection; herpes simplex virus 1 was found most often.
179 tudy, the structural, material properties of herpes simplex virus 1 were investigated.
180 V] 6, 18%; HHV8, 6%; Epstein-Barr virus, 3%; herpes simplex virus 1, 3%; varicella zoster virus, 3%;
181 e, whereas infection with influenza A virus, herpes simplex virus 1, or cytomegalovirus induced a str
182  provision of antagomir-155 nanoparticles to herpes simplex virus 1-infected mice led to diminished S
183                     Random walk modelling of herpes simplex virus 1-sized particles in a three-dimens
184 viously established that cells infected with herpes simplex virus 2 (HSV-2) are disrupted in their ab
185                Subunit vaccines based on the herpes simplex virus 2 (HSV-2) glycoprotein D (gD-2) hav
186 nital herpes prophylactic vaccine containing herpes simplex virus 2 (HSV-2) glycoproteins C (gC2) and
187      Several prophylactic vaccines targeting herpes simplex virus 2 (HSV-2) have failed in the clinic
188                                              Herpes simplex virus 2 (HSV-2) is a causative agent of g
189                                              Herpes simplex virus 2 (HSV-2) is a major global pathoge
190 ified virion host shutoff protein (vhs) as a herpes simplex virus 2 (HSV-2) protein capable of disrup
191 llions of people worldwide are infected with herpes simplex virus 2 (HSV-2), and to date, an efficaci
192 %; varicella zoster virus, 3%; HHV7, 2%; and herpes simplex virus 2, 1%.
193 ted with human immunodeficiency virus (HIV), herpes simplex virus 2, and CMV starting antiretroviral
194                 Alpha herpesviruses, such as herpes simplex virus and pseudorabies virus (PRV), are n
195 5 on the structural stability of icosahedral herpes simplex virus capsids.
196 ital specimens each containing >/=105 copies herpes simplex virus DNA/ml collected a median of 5 mont
197                                          The herpes simplex virus for example evades immune surveilla
198           The transcription factor ICP4 from herpes simplex virus has a central role in regulating th
199 bitor ICP47, a small protein produced by the herpes simplex virus I.
200 h inhibits HIV-1, HIV-2, Influenza virus and herpes simplex virus infection, and enhances the potenti
201 ons, rubella, cytomegalovirus infection, and herpes simplex virus infections (TORCH), Apgar score <5
202 icase-primase inhibitor for the treatment of herpes simplex virus infections, was prepared.
203 r development of antiviral agents.IMPORTANCE Herpes simplex virus is a major pathogen, and although n
204                                The capsid of herpes simplex virus is built up of a variety of protein
205 ens from entering the endoplasmic reticulum, herpes simplex virus is hidden from cytotoxic T lymphocy
206 ovir decreased the recurrence of any type of herpes simplex virus keratitis by approximately half.
207 nfections of the superficial cornea, such as herpes simplex virus keratitis or Acanthamoeba keratitis
208 us keratitis is significantly different from herpes simplex virus keratitis, and further studies usin
209              Further, in the murine model of herpes simplex virus keratitis, corneal pathology and ly
210 itis is significantly different from that of herpes simplex virus keratitis.
211  of autophagy/xenophagy results with mutated herpes simplex virus lacking its ICP34.5 neurovirulence
212 ) relocation of nonintegrin receptors (e.g., herpes simplex virus nectin1 and Kaposi's sarcoma-associ
213 h clinically diagnosed ARN, PCR-positive for herpes simplex virus or varicella zoster virus and evalu
214 iral reactivation, exhibiting parallels with herpes simplex virus reactivation.
215  of the lipid raft-dwelling US9 protein from Herpes Simplex Virus strikingly overlaps with that of th
216 transgenic mice expressing the suicide gene, herpes simplex virus thymidine kinase (HSVtk), driven by
217 g AAVP particles served to deliver the human Herpes simplex virus thymidine kinase type-1 (HSVtk) gen
218                 Infectious titers of EBOV or herpes simplex virus type 1 (HSV-1) in detergents-treate
219                         Lytic infection with herpes simplex virus type 1 (HSV-1) induces profound mod
220 mount of virus reactivation following ocular herpes simplex virus type 1 (HSV-1) infection.
221                                              Herpes simplex virus type 1 (HSV-1) is a leading cause o
222                                              Herpes simplex virus type 1 (HSV-1) is a ubiquitous viru
223                       Glycoprotein D (gD) of herpes simplex virus type 1 (HSV-1) is one of four glyco
224 lly occurs during delivery from mothers with herpes simplex virus type 1 (HSV-1) or type 2 (HSV-2) ge
225 ological stress.IMPORTANCE Like all viruses, herpes simplex virus type 1 (HSV-1) reproduction relies
226 we reported a new series of highly defective herpes simplex virus type 1 (HSV-1) vectors that were fu
227 ormational approach to genome engineering of herpes simplex virus type 1 (HSV-1), which has a large D
228                   Knockout of TRIM14 impairs herpes simplex virus type 1 (HSV-1)-triggered antiviral
229                 Support for the concept that herpes simplex virus type 1 (HSV1), when present in the
230                                              Herpes simplex virus type 1 and Alzheimer's disease: pos
231 athway are associated with susceptibility to herpes simplex virus type 1 encephalitis (HSE).
232   We found wide distribution of O-glycans on herpes simplex virus type 1 glycoproteins and demonstrat
233  gene encoding the prodrug-converting enzyme herpes simplex virus type 1 thymidine kinase (HSV1-tk) i
234                                         CMV, herpes simplex virus type 1, and human herpesvirus 6 inf
235 e association of 4 human herpesviruses (CMV, herpes simplex virus type 1, human herpesvirus type 6, a
236  pathogens were detected by mNGS (4 cases of herpes simplex virus type 1, including 1 case of coinfec
237        Talimogene laherparepvec (T-VEC) is a herpes simplex virus type 1-derived oncolytic immunother
238 chnique for mapping O-glycosylation sites on herpes simplex virus type 1.
239 fumarate (TDF) has in vitro activity against herpes simplex virus type 2 (HSV-2) and reduced HSV-2 ac
240                                We focused on herpes simplex virus type 2 (HSV-2) because prior publis
241 ndoms for protection against transmission of herpes simplex virus type 2 (HSV-2) has been examined in
242               Despite the high prevalence of herpes simplex virus type 2 (HSV-2) in sub-Saharan Afric
243                                      Genital herpes simplex virus type 2 (HSV-2) infection causes rec
244                                    Globally, herpes simplex virus type 2 (HSV-2) infection is the mos
245 mmune system's protective effect against one herpes simplex virus type 2 (HSV-2) infection protects a
246 or NK cell function in response to a mucosal herpes simplex virus type 2 (HSV-2) infection.
247                                      HIV and herpes simplex virus type 2 (HSV-2) infections cause a s
248 tor in clinical development for treatment of herpes simplex virus type 2 (HSV-2) infections.
249                                              Herpes simplex virus type 2 (HSV-2) reactivation is acco
250 ART) has been incompletely characterized for herpes simplex virus type 2 (HSV-2).
251                                              Herpes simplex virus type 2 (HSV-2; herpes) exacerbates
252 against cervical cancer, cervical dysplasia, herpes simplex virus type 2, chlamydia, and syphilis.
253 l among 3408 persons coinfected with HIV and herpes simplex virus type 2.
254 nd spinal cord in response to infection with herpes simplex virus type 2.
255 mydia trachomatis, Neisseria gonorrhoeae, or herpes simplex virus type 2.
256 lar functions affected by lytic infection of Herpes Simplex Virus type I in Human primary fibroblasts
257 f herpetic neuralgia using a murine model of Herpes Simplex Virus Type-1 (HSV-1) infection.
258                                              Herpes simplex virus type-1 (HSV-1) is one of the most w
259 CB) women, including bacterial vaginosis and herpes simplex virus type-2 (HSV-2) infection.
260 he impact of antiretroviral therapy (ART) on herpes simplex virus type-2 (HSV-2) replication is uncle
261 ded controls for sociodemographic variables, herpes simplex virus type-2 status, and recreational dru
262 rr virus (EBV), human herpesvirus 6 (HHV-6), herpes simplex virus types 1 (HSV-1) and 2 (HSV-2), and
263                                              Herpes simplex virus types 1 and 2 (HSV-1 and HSV-2) are
264 e = 11.1 weeks) to Epstein Barr virus (EBV), herpes simplex virus types 1 and 2 (HSV-1 and HSV-2), an
265   Trichomonas vaginalis and viral pathogens (herpes simplex virus types 1 and 2 and adenovirus) can c
266                  Seroprevalence of EBV, CMV, herpes simplex virus types 1 and 2, and human herpesviru
267 ein Barr virus (EBV), cytomegalovirus (CMV), herpes simplex virus types 1 and 2, human herpesvirus 8)
268 iency virus, hepatitis B virus, and neonatal herpes simplex virus, from which lessons for the evaluat
269 ing of mucosal immunity and then compare the herpes simplex virus, human immunodeficiency virus, and
270  papilloma virus, hepatitis B and C viruses, herpes simplex virus, norovirus, rotavirus, parvovirus,
271 use substantial impairment: cytomegalovirus, herpes simplex virus, rubella virus, Toxoplasma gondii,
272 ded toxoplasmosis, rubella, cytomegalovirus, herpes simplex virus, syphilis, and human immunodeficien
273 e (CTL), Th1 cell, or Th17 cell responses to herpes simplex virus, Toxoplasma gondii, and Citrobacter
274 97%; PPV range, 52%-95%; NPV range, 79%-80%; herpes simplex virus, vulvar ulcerations: sensitivity, 2
275                                              Herpes simplex virus-1 (HSV-1) causes life-long morbidit
276                                              Herpes Simplex Virus-1 (HSV-1) is a ubiquitous human pat
277         Herein we demonstrate that oncolytic herpes simplex virus-1 (HSV-1) potently activates human
278 ed interferon-alpha secretion in response to herpes simplex virus-1 (HSV-1), whereas granzyme-B induc
279 76-9 (MHC I low) tumors respond to oncolytic herpes simplex virus-1 (oHSV-1) and PD-1 blockade combin
280 on library generated using the high-capacity herpes simplex virus-1 amplicon technology to deliver ba
281 ers in the lung, and are resistant to lethal herpes simplex virus-1 infection due to enhanced product
282 Here, we address this deficit, focusing on a herpes simplex virus-1 protein, ICP27.
283 sneuronal viral tract tracer, H129 strain of herpes simplex virus-1.
284  and generalized infections that can lead to herpes simplex virus-induced acute liver failure.
285                    Furthermore, an unrelated herpes simplex virus-thymidine kinase (HSV-TK) promoter
286 r, and parvovirus B19), cytomegalovirus, and herpes simplex virus.
287 e fusion also proved to be well tolerated in herpes simplex virus.
288 ere independent of varicella-zoster virus or herpes-simplex virus 1 coinfection.
289 mmunoglobulin G seropositivity for CMV, EBV, herpes-simplex virus 1, and varicella-zoster virus were
290                                              Herpes simplex viruses (HSV) are human pathogens that sw
291  significant effect.IMPORTANCE Infections by herpes simplex viruses (HSV) cause painful cold sores or
292 l B virus isolates to use entry receptors of herpes simplex viruses (HSV).
293                                              Herpes simplex viruses (HSVs) are human pathogens that c
294                                              Herpes simplex viruses (HSVs) are unusual in that unlike
295                            A key property of herpes simplex viruses (HSVs) is their ability to establ
296                                        Human herpes simplex viruses 1 and 2 (HSV-1 and HSV-2) are lar
297                                              Herpes simplex viruses 1 and 2 (HSV-1 and HSV-2) infect
298 rus, mumps virus, measles virus, lyssavirus, herpes simplex viruses 1 and 2, Epstein-Barr virus, ente
299                                     Modified herpes simplex viruses that are unable to produce glycop
300 domains from the Drosophila fushi tarazu and Herpes simplex VP16 was created.

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