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3 cent reviews; these include the relevance of herpes simplex encephalitis and of epilepsy to AD, the a
6 l intervention is crucial to the survival of herpes simplex encephalitis patients; however, many surv
7 al keratitis, highly debilitating and lethal herpes simplex encephalitis, and generalized infections
12 ree patients with a unilateral and relapsing herpes simplex keratitis (HSK group) that was quiescent
13 ity of life (QoL) in patients with quiescent herpes simplex keratitis compared with control patients
14 group initially were misdiagnosed as having herpes simplex keratitis versus 59 (41.8%) patients who
15 al patients were diagnosed sequentially with herpes simplex keratitis, then Acanthamoeba keratitis be
16 for dendritiform keratopathy, such as prior herpes simplex keratitis, varicella-zoster viral keratit
17 ated with antiviral medications for presumed herpes simplex keratitis; 4 patients underwent diagnosti
18 petic anterior uveitis (AU), owing to either herpes simplex or varicella zoster virus, by using the S
19 lopment of a safe and effective asymptomatic herpes simplex vaccine that is selectively based on CD8(
21 polymerase chain reaction (PCR) analysis for Herpes simplex, varicella zoster, cytomegalovirus, Epste
22 this assessment include adenovirus 8 and 19, herpes simplex virus (HSV) 1 and 2, human immunodeficien
24 dy of neurotropic viruses in vitroIMPORTANCE Herpes simplex virus (HSV) affects millions of people wo
25 tions that US9 is neuron specific.IMPORTANCE Herpes simplex virus (HSV) and other alphaherpesviruses,
27 a broad antimicrobial factor that restricts herpes simplex virus (HSV) by activating type I interfer
29 his study employed a novel strategy in which herpes simplex virus (HSV) carrying a small interfering
31 fluid (CSF) commonly predicts the absence of herpes simplex virus (HSV) central nervous system (CNS)
35 erpesvirus entry mediator (HVEM) facilitates herpes simplex virus (HSV) entry through interactions wi
38 xpression of 'pore dead' GluA1 subunits (via herpes simplex virus (HSV) GluA1-Q582E) in the lateral c
40 is known as a transcriptional coactivator of herpes simplex virus (HSV) immediate early (IE) transcri
42 Neuron-virus interactions that occur during herpes simplex virus (HSV) infection are not fully under
53 ctivator HCF-1 is required for initiation of herpes simplex virus (HSV) lytic infection and for react
54 f aqueous or vitreous humor was positive for herpes simplex virus (HSV) or varicella zoster virus (VZ
55 od to visualize pseudorabies virus (PRV) and herpes simplex virus (HSV) particles in living cells.
61 synthesis during multiround transmission of herpes simplex virus (HSV) using pulse-labeling with eth
62 e by microinjecting before training a single herpes simplex virus (HSV) vector expressing either CRIS
63 tential role of a TLR4/MCP-1 signal, we used Herpes Simplex Virus (HSV) vectors (amplicons) that reta
67 itro nanomolar irreversible activity against herpes simplex virus (HSV), human papilloma virus, respi
68 cell entry and subsequent lateral spread of herpes simplex virus (HSV), requires the four envelope g
71 cally significant species of simplexviruses, herpes simplex virus (HSV)-1 and HSV-2, with estimated d
72 ACASI), before test HIV counselling, HIV and herpes simplex virus (HSV)-2 testing, and post-test coun
76 interferon (IFN) is important for control of herpes simplex virus (HSV-1) in the central nervous syst
77 e delivered to the same sensory neurons that herpes simplex virus (HSV-1) infects when applied periph
79 ablation on tga20/CD11b-thymidine kinase of Herpes simplex virus (HSVTK) cerebellar organotypic cult
81 nd tested the fate and efficacy of oncolytic herpes simplex virus (oHSV)-armed mesenchymal stem cells
82 n of foreign DNA-sensing pathways.IMPORTANCE Herpes simplex virus 1 (HSV-1) afflicts 80% of the popul
84 qualitative detection and differentiation of herpes simplex virus 1 (HSV-1) and HSV-2 DNA in 1,351 cu
86 f UL37 homologs from two alphaherpesviruses, herpes simplex virus 1 (HSV-1) and pseudorabies virus (P
87 hat gB homologs from two alphaherpesviruses, herpes simplex virus 1 (HSV-1) and saimiriine herpesviru
88 te aspects of intrinsic immunity to restrict herpes simplex virus 1 (HSV-1) as well as other viruses.
90 ar disease in virus-infected mice.IMPORTANCE Herpes simplex virus 1 (HSV-1) causes cold sores and neo
95 ined whether Cbl mediates the removal of the herpes simplex virus 1 (HSV-1) entry receptor Nectin-1 f
98 ultaneous presence of a helper virus such as herpes simplex virus 1 (HSV-1) for productive replicatio
99 plication greatly enhances expression of the herpes simplex virus 1 (HSV-1) gamma2 late genes by stil
101 gp120) construct fused to a small portion of herpes simplex virus 1 (HSV-1) glycoprotein D (gD) so th
103 mia nuclear bodies (PML NBs), is a target of herpes simplex virus 1 (HSV-1) ICP0-mediated degradation
105 As HCF1 is best known for its function in herpes simplex virus 1 (HSV-1) immediate early gene tran
106 teins have some functional similarities with herpes simplex virus 1 (HSV-1) immediate early protein I
112 efects in IFN responses can result in lethal herpes simplex virus 1 (HSV-1) infections, usually from
122 viral DNA from NPC-bound capsids.IMPORTANCE Herpes simplex virus 1 (HSV-1) is the causative agent of
124 nal (NLS) sequences previously identified in herpes simplex virus 1 (HSV-1) large terminase and human
126 s from a variant to target the mRNA encoding herpes simplex virus 1 (HSV-1) major transcription regul
128 ve previously shown that the live-attenuated herpes simplex virus 1 (HSV-1) mutant lacking the nuclea
131 ivating protein, p35, is important for acute herpes simplex virus 1 (HSV-1) replication in mice.
135 sRNA, encephalomyocarditis virus (EMCV), and herpes simplex virus 1 (HSV-1) show impaired production
136 nd ICP34.5 are among the proteins encoded by herpes simplex virus 1 (HSV-1) that modulate type I IFN
138 e ability of the prototypic alphaherpesvirus herpes simplex virus 1 (HSV-1) to enter neurons via axon
142 ogic protection requisite for an efficacious herpes simplex virus 1 (HSV-1) vaccine remain unclear wi
143 Herein, we report that the live-attenuated herpes simplex virus 1 (HSV-1) VC2 vaccine strain, which
144 Following infection of epithelial tissues, herpes simplex virus 1 (HSV-1) virions travel via axonal
146 lected TRIM proteins in autophagy induced by herpes simplex virus 1 (HSV-1), encephalomyocarditis vir
148 roinvasive alphaherpesviruses, such as human herpes simplex virus 1 (HSV-1), HSV-2, and veterinarian
149 trast, of the more than 80 mRNAs harbored by herpes simplex virus 1 (HSV-1), only 5 are spliced.
151 Here we focus on two tegument proteins from herpes simplex virus 1 (HSV-1), pUL7 and pUL51, which ha
153 zuelan equine encephalitis virus (VEEV), and herpes simplex virus 1 (HSV-1), suggesting that LIMK inh
163 choriomeningitis virus (LCMV) infection, or herpes simplex virus 1 (HSV1) infection was profoundly d
164 svirus [KSHV], Epstein-Barr virus [EBV], and herpes simplex virus 1 [HSV-1]) genomes and induces the
165 unoglobulin M/immunoglobulin G antibodies to herpes simplex virus 1 and 2, cytomegalovirus, Epstein-B
166 us agalactiae, cytomegalovirus, enterovirus, herpes simplex virus 1 and 2, human herpesvirus 6, human
167 ibition decreases replication of adenovirus, herpes simplex virus 1 and influenza A in cultured prima
168 ability to be infected by the herpesviruses herpes simplex virus 1 and murine herpesvirus 68 and the
169 Here, we have characterized the full-length herpes simplex virus 1 gB in a native membrane by displa
170 pted this method to rapidly deliver a 152 kb herpes simplex virus 1 genome cloned in yeast into mamma
171 nt productive infection of cultured cells by herpes simplex virus 1 has made it a paradigm for this m
172 e the molecular organization of chromatin in herpes simplex virus 1 infection and its effect on the t
173 155 expression was up-regulated after ocular herpes simplex virus 1 infection, with the increased Mir
175 er replication of BKV, whereas influenza and herpes simplex virus 1 replication were clearly reduced.
176 study focuses on two tegument proteins from herpes simplex virus 1 that are conserved in all herpesv
180 V] 6, 18%; HHV8, 6%; Epstein-Barr virus, 3%; herpes simplex virus 1, 3%; varicella zoster virus, 3%;
181 e, whereas infection with influenza A virus, herpes simplex virus 1, or cytomegalovirus induced a str
182 provision of antagomir-155 nanoparticles to herpes simplex virus 1-infected mice led to diminished S
184 viously established that cells infected with herpes simplex virus 2 (HSV-2) are disrupted in their ab
186 nital herpes prophylactic vaccine containing herpes simplex virus 2 (HSV-2) glycoproteins C (gC2) and
187 Several prophylactic vaccines targeting herpes simplex virus 2 (HSV-2) have failed in the clinic
190 ified virion host shutoff protein (vhs) as a herpes simplex virus 2 (HSV-2) protein capable of disrup
191 llions of people worldwide are infected with herpes simplex virus 2 (HSV-2), and to date, an efficaci
193 ted with human immunodeficiency virus (HIV), herpes simplex virus 2, and CMV starting antiretroviral
196 ital specimens each containing >/=105 copies herpes simplex virus DNA/ml collected a median of 5 mont
200 h inhibits HIV-1, HIV-2, Influenza virus and herpes simplex virus infection, and enhances the potenti
201 ons, rubella, cytomegalovirus infection, and herpes simplex virus infections (TORCH), Apgar score <5
203 r development of antiviral agents.IMPORTANCE Herpes simplex virus is a major pathogen, and although n
205 ens from entering the endoplasmic reticulum, herpes simplex virus is hidden from cytotoxic T lymphocy
206 ovir decreased the recurrence of any type of herpes simplex virus keratitis by approximately half.
207 nfections of the superficial cornea, such as herpes simplex virus keratitis or Acanthamoeba keratitis
208 us keratitis is significantly different from herpes simplex virus keratitis, and further studies usin
211 of autophagy/xenophagy results with mutated herpes simplex virus lacking its ICP34.5 neurovirulence
212 ) relocation of nonintegrin receptors (e.g., herpes simplex virus nectin1 and Kaposi's sarcoma-associ
213 h clinically diagnosed ARN, PCR-positive for herpes simplex virus or varicella zoster virus and evalu
215 of the lipid raft-dwelling US9 protein from Herpes Simplex Virus strikingly overlaps with that of th
216 transgenic mice expressing the suicide gene, herpes simplex virus thymidine kinase (HSVtk), driven by
217 g AAVP particles served to deliver the human Herpes simplex virus thymidine kinase type-1 (HSVtk) gen
224 lly occurs during delivery from mothers with herpes simplex virus type 1 (HSV-1) or type 2 (HSV-2) ge
225 ological stress.IMPORTANCE Like all viruses, herpes simplex virus type 1 (HSV-1) reproduction relies
226 we reported a new series of highly defective herpes simplex virus type 1 (HSV-1) vectors that were fu
227 ormational approach to genome engineering of herpes simplex virus type 1 (HSV-1), which has a large D
232 We found wide distribution of O-glycans on herpes simplex virus type 1 glycoproteins and demonstrat
233 gene encoding the prodrug-converting enzyme herpes simplex virus type 1 thymidine kinase (HSV1-tk) i
235 e association of 4 human herpesviruses (CMV, herpes simplex virus type 1, human herpesvirus type 6, a
236 pathogens were detected by mNGS (4 cases of herpes simplex virus type 1, including 1 case of coinfec
239 fumarate (TDF) has in vitro activity against herpes simplex virus type 2 (HSV-2) and reduced HSV-2 ac
241 ndoms for protection against transmission of herpes simplex virus type 2 (HSV-2) has been examined in
245 mmune system's protective effect against one herpes simplex virus type 2 (HSV-2) infection protects a
252 against cervical cancer, cervical dysplasia, herpes simplex virus type 2, chlamydia, and syphilis.
256 lar functions affected by lytic infection of Herpes Simplex Virus type I in Human primary fibroblasts
260 he impact of antiretroviral therapy (ART) on herpes simplex virus type-2 (HSV-2) replication is uncle
261 ded controls for sociodemographic variables, herpes simplex virus type-2 status, and recreational dru
262 rr virus (EBV), human herpesvirus 6 (HHV-6), herpes simplex virus types 1 (HSV-1) and 2 (HSV-2), and
264 e = 11.1 weeks) to Epstein Barr virus (EBV), herpes simplex virus types 1 and 2 (HSV-1 and HSV-2), an
265 Trichomonas vaginalis and viral pathogens (herpes simplex virus types 1 and 2 and adenovirus) can c
267 ein Barr virus (EBV), cytomegalovirus (CMV), herpes simplex virus types 1 and 2, human herpesvirus 8)
268 iency virus, hepatitis B virus, and neonatal herpes simplex virus, from which lessons for the evaluat
269 ing of mucosal immunity and then compare the herpes simplex virus, human immunodeficiency virus, and
270 papilloma virus, hepatitis B and C viruses, herpes simplex virus, norovirus, rotavirus, parvovirus,
271 use substantial impairment: cytomegalovirus, herpes simplex virus, rubella virus, Toxoplasma gondii,
272 ded toxoplasmosis, rubella, cytomegalovirus, herpes simplex virus, syphilis, and human immunodeficien
273 e (CTL), Th1 cell, or Th17 cell responses to herpes simplex virus, Toxoplasma gondii, and Citrobacter
274 97%; PPV range, 52%-95%; NPV range, 79%-80%; herpes simplex virus, vulvar ulcerations: sensitivity, 2
278 ed interferon-alpha secretion in response to herpes simplex virus-1 (HSV-1), whereas granzyme-B induc
279 76-9 (MHC I low) tumors respond to oncolytic herpes simplex virus-1 (oHSV-1) and PD-1 blockade combin
280 on library generated using the high-capacity herpes simplex virus-1 amplicon technology to deliver ba
281 ers in the lung, and are resistant to lethal herpes simplex virus-1 infection due to enhanced product
289 mmunoglobulin G seropositivity for CMV, EBV, herpes-simplex virus 1, and varicella-zoster virus were
291 significant effect.IMPORTANCE Infections by herpes simplex viruses (HSV) cause painful cold sores or
298 rus, mumps virus, measles virus, lyssavirus, herpes simplex viruses 1 and 2, Epstein-Barr virus, ente
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