ライフサイエンスコーパス: herpes simplex virus

1 r, and parvovirus B19), cytomegalovirus, and herpes simplex virus.

2 e fusion also proved to be well tolerated in herpes simplex virus.

3 n of foreign DNA-sensing pathways.IMPORTANCE Herpes simplex virus 1 (HSV-1) afflicts 80% of the popul

4 Herpes simplex virus 1 (HSV-1) and HSV-2 are large, doub

5 qualitative detection and differentiation of herpes simplex virus 1 (HSV-1) and HSV-2 DNA in 1,351 cu

6 Herpes simplex virus 1 (HSV-1) and HSV-2 infect many hum

7 f UL37 homologs from two alphaherpesviruses, herpes simplex virus 1 (HSV-1) and pseudorabies virus (P

8 6p (VP1/2) is the largest protein encoded by herpes simplex virus 1 (HSV-1) and resides in the innerm

9 hat gB homologs from two alphaherpesviruses, herpes simplex virus 1 (HSV-1) and saimiriine herpesviru

10 te aspects of intrinsic immunity to restrict herpes simplex virus 1 (HSV-1) as well as other viruses.

11 The herpes simplex virus 1 (HSV-1) capsid is a huge assembly

12 ar disease in virus-infected mice.IMPORTANCE Herpes simplex virus 1 (HSV-1) causes cold sores and neo

13 Herpes simplex virus 1 (HSV-1) encodes the multifunction

14 Herpes simplex virus 1 (HSV-1) enters mice via olfactory

15 ponent of cell membranes and is required for herpes simplex virus 1 (HSV-1) entry (1-3).

16 The dissection of the herpes simplex virus 1 (HSV-1) entry mechanism is compli

17 ined whether Cbl mediates the removal of the herpes simplex virus 1 (HSV-1) entry receptor Nectin-1 f

18 Herpes simplex virus 1 (HSV-1) establishes latency withi

19 Herpes simplex virus 1 (HSV-1) establishes lifelong infe

20 ultaneous presence of a helper virus such as herpes simplex virus 1 (HSV-1) for productive replicatio

21 ave costimulatory properties, is targeted by herpes simplex virus 1 (HSV-1) for viral immune escape.

22 plication greatly enhances expression of the herpes simplex virus 1 (HSV-1) gamma2 late genes by stil

23 Herpes simplex virus 1 (HSV-1) glycoprotein B (gB)-speci

24 gp120) construct fused to a small portion of herpes simplex virus 1 (HSV-1) glycoprotein D (gD) so th

25 The herpes simplex virus 1 (HSV-1) ICP0 protein is an E3 ubi

26 mia nuclear bodies (PML NBs), is a target of herpes simplex virus 1 (HSV-1) ICP0-mediated degradation

27 For example, herpes simplex virus 1 (HSV-1) immediate early (IE) prot

28 As HCF1 is best known for its function in herpes simplex virus 1 (HSV-1) immediate early gene tran

29 teins have some functional similarities with herpes simplex virus 1 (HSV-1) immediate early protein I

30 tinocytes represent a primary entry site for herpes simplex virus 1 (HSV-1) in vivo.

31 Uncontrolled herpes simplex virus 1 (HSV-1) infection can advance to

32 Herpes simplex virus 1 (HSV-1) infection is an incurable

33 Herpes simplex virus 1 (HSV-1) infection is widespread a

34 f the intrinsic antiviral immune response to herpes simplex virus 1 (HSV-1) infection.

35 efects in IFN responses can result in lethal herpes simplex virus 1 (HSV-1) infections, usually from

36 Herpes simplex virus 1 (HSV-1) infects humans through st

37 Herpes simplex virus 1 (HSV-1) infects most people and c

38 The immediate early protein ICP0 of herpes simplex virus 1 (HSV-1) interacts with CIN85, an

39 We report here that UL37 of herpes simplex virus 1 (HSV-1) is a protein deamidase th

40 Human herpes simplex virus 1 (HSV-1) is a widespread pathogen,

41 Herpes simplex virus 1 (HSV-1) is among the most common

42 Infected cell protein 0 (ICP0) of herpes simplex virus 1 (HSV-1) is an alpha gene product

43 One aspect of intrinsic resistance to herpes simplex virus 1 (HSV-1) is conferred by component

44 The UL16 tegument protein of herpes simplex virus 1 (HSV-1) is conserved among all he

45 Herpes simplex virus 1 (HSV-1) is one of the eight herpe

46 viral DNA from NPC-bound capsids.IMPORTANCE Herpes simplex virus 1 (HSV-1) is the causative agent of

47 We have shown previously that herpes simplex virus 1 (HSV-1) lacking expression of the

48 nal (NLS) sequences previously identified in herpes simplex virus 1 (HSV-1) large terminase and human

49 Herpes simplex virus 1 (HSV-1) latency entails the repre

50 s from a variant to target the mRNA encoding herpes simplex virus 1 (HSV-1) major transcription regul

51 Herpes simplex virus 1 (HSV-1) most commonly causes recr

52 ve previously shown that the live-attenuated herpes simplex virus 1 (HSV-1) mutant lacking the nuclea

53 During DNA encapsidation, herpes simplex virus 1 (HSV-1) procapsids are converted

54 Herpes simplex virus 1 (HSV-1) remodels nuclear membrane

55 ivating protein, p35, is important for acute herpes simplex virus 1 (HSV-1) replication in mice.

56 was to reexamine the requirement of UL21 for herpes simplex virus 1 (HSV-1) replication.

57 Infection of mature HD10.6 neurons by herpes simplex virus 1 (HSV-1) results in a delayed but

58 Ocular infection with herpes simplex virus 1 (HSV-1) sets off an inflammatory

59 sRNA, encephalomyocarditis virus (EMCV), and herpes simplex virus 1 (HSV-1) show impaired production

60 nd ICP34.5 are among the proteins encoded by herpes simplex virus 1 (HSV-1) that modulate type I IFN

61 Humans occasionally transmit herpes simplex virus 1 (HSV-1) to captive primates, who

62 e ability of the prototypic alphaherpesvirus herpes simplex virus 1 (HSV-1) to enter neurons via axon

63 Herpes simplex virus 1 (HSV-1) typically causes recurren

64 Herpes simplex virus 1 (HSV-1) UL20 plays a crucial role

65 The herpes simplex virus 1 (HSV-1) UL37 protein functions in

66 ogic protection requisite for an efficacious herpes simplex virus 1 (HSV-1) vaccine remain unclear wi

67 Herein, we report that the live-attenuated herpes simplex virus 1 (HSV-1) VC2 vaccine strain, which

68 Following infection of epithelial tissues, herpes simplex virus 1 (HSV-1) virions travel via axonal

69 Herpes simplex virus 1 (HSV-1), a leading cause of genit

70 lected TRIM proteins in autophagy induced by herpes simplex virus 1 (HSV-1), encephalomyocarditis vir

71 Persistent pathogens, such as herpes simplex virus 1 (HSV-1), have evolved a variety o

72 roinvasive alphaherpesviruses, such as human herpes simplex virus 1 (HSV-1), HSV-2, and veterinarian

73 trast, of the more than 80 mRNAs harbored by herpes simplex virus 1 (HSV-1), only 5 are spliced.

74 Following infection with herpes simplex virus 1 (HSV-1), PIAS1 is recruited to nu

75 Here we focus on two tegument proteins from herpes simplex virus 1 (HSV-1), pUL7 and pUL51, which ha

76 Although many herpesviruses, including herpes simplex virus 1 (HSV-1), stimulate mTORC1, how HS

77 zuelan equine encephalitis virus (VEEV), and herpes simplex virus 1 (HSV-1), suggesting that LIMK inh

78 as the replication of a related herpesvirus, herpes simplex virus 1 (HSV-1), was not impacted.

79 For herpes simplex virus 1 (HSV-1), we and others have previ

80 Herpes simplex virus 1 (HSV-1), which causes a variety o

81 Although most herpes simplex virus 1 (HSV-1)-infected individuals shed

82 esponse to restrict viral pathogens, such as herpes simplex virus 1 (HSV-1).

83 ganglia (TG) of mice latently infected with herpes simplex virus 1 (HSV-1).

84 of ICP27, an immediate-early (IE) protein of herpes simplex virus 1 (HSV-1).

85 pe dynamics as well as the nuclear egress of herpes simplex virus 1 (HSV-1).

86 SP141), bound to RIG-I during infection with herpes simplex virus 1 (HSV-1).

87 between AAV2 and one of its helper viruses, herpes simplex virus 1 (HSV-1).

88 f mice that have been ocularly infected with herpes simplex virus 1 (HSV-1).

89 choriomeningitis virus (LCMV) infection, or herpes simplex virus 1 (HSV1) infection was profoundly d

90 svirus [KSHV], Epstein-Barr virus [EBV], and herpes simplex virus 1 [HSV-1]) genomes and induces the

91 unoglobulin M/immunoglobulin G antibodies to herpes simplex virus 1 and 2, cytomegalovirus, Epstein-B

92 us agalactiae, cytomegalovirus, enterovirus, herpes simplex virus 1 and 2, human herpesvirus 6, human

93 ibition decreases replication of adenovirus, herpes simplex virus 1 and influenza A in cultured prima

94 ability to be infected by the herpesviruses herpes simplex virus 1 and murine herpesvirus 68 and the

95 Here, we have characterized the full-length herpes simplex virus 1 gB in a native membrane by displa

96 pted this method to rapidly deliver a 152 kb herpes simplex virus 1 genome cloned in yeast into mamma

97 nt productive infection of cultured cells by herpes simplex virus 1 has made it a paradigm for this m

98 e the molecular organization of chromatin in herpes simplex virus 1 infection and its effect on the t

99 er describes the extremely rapid response to herpes simplex virus 1 infection of cellular protein IFI

100 155 expression was up-regulated after ocular herpes simplex virus 1 infection, with the increased Mir

101 promised survival during influenza virus and herpes simplex virus 1 infections.

102 The conserved UL51 gene of herpes simplex virus 1 plays important roles in cell-to-

103 er replication of BKV, whereas influenza and herpes simplex virus 1 replication were clearly reduced.

104 study focuses on two tegument proteins from herpes simplex virus 1 that are conserved in all herpesv

105 tein domains, as well as a truncated form of herpes simplex virus 1 thymidine kinase (HSV-TK).

106 ture of its conserved N-terminal domain from herpes simplex virus 1 to 2.0-A resolution, which reveal

107 wed evidence of acute herpesvirus infection; herpes simplex virus 1 was found most often.

108 tudy, the structural, material properties of herpes simplex virus 1 were investigated.

109 V] 6, 18%; HHV8, 6%; Epstein-Barr virus, 3%; herpes simplex virus 1, 3%; varicella zoster virus, 3%;

110 e, whereas infection with influenza A virus, herpes simplex virus 1, or cytomegalovirus induced a str

111 provision of antagomir-155 nanoparticles to herpes simplex virus 1-infected mice led to diminished S

112 Random walk modelling of herpes simplex virus 1-sized particles in a three-dimens

113 Human herpes simplex viruses 1 and 2 (HSV-1 and HSV-2) are lar

114 Herpes simplex viruses 1 and 2 (HSV-1 and HSV-2) infect

115 rus, mumps virus, measles virus, lyssavirus, herpes simplex viruses 1 and 2, Epstein-Barr virus, ente

116 ere independent of varicella-zoster virus or herpes-simplex virus 1 coinfection.

117 mmunoglobulin G seropositivity for CMV, EBV, herpes-simplex virus 1, and varicella-zoster virus were

118 Herpes simplex virus-1 (HSV-1) causes life-long morbidit

119 Herpes simplex virus-1 (HSV-1) is a double-stranded DNA

120 Herpes Simplex Virus-1 (HSV-1) is a ubiquitous human pat

121 Herein we demonstrate that oncolytic herpes simplex virus-1 (HSV-1) potently activates human

122 ed interferon-alpha secretion in response to herpes simplex virus-1 (HSV-1), whereas granzyme-B induc

123 76-9 (MHC I low) tumors respond to oncolytic herpes simplex virus-1 (oHSV-1) and PD-1 blockade combin

124 on library generated using the high-capacity herpes simplex virus-1 amplicon technology to deliver ba

125 ers in the lung, and are resistant to lethal herpes simplex virus-1 infection due to enhanced product

126 Here, we address this deficit, focusing on a herpes simplex virus-1 protein, ICP27.

127 sneuronal viral tract tracer, H129 strain of herpes simplex virus-1.

128 viously established that cells infected with herpes simplex virus 2 (HSV-2) are disrupted in their ab

129 Subunit vaccines based on the herpes simplex virus 2 (HSV-2) glycoprotein D (gD-2) hav

130 nital herpes prophylactic vaccine containing herpes simplex virus 2 (HSV-2) glycoproteins C (gC2) and

131 Several prophylactic vaccines targeting herpes simplex virus 2 (HSV-2) have failed in the clinic

132 Herpes simplex virus 2 (HSV-2) is a causative agent of g

133 Herpes simplex virus 2 (HSV-2) is a major global pathoge

134 ified virion host shutoff protein (vhs) as a herpes simplex virus 2 (HSV-2) protein capable of disrup

135 llions of people worldwide are infected with herpes simplex virus 2 (HSV-2), and to date, an efficaci

136 %; varicella zoster virus, 3%; HHV7, 2%; and herpes simplex virus 2, 1%.

137 ted with human immunodeficiency virus (HIV), herpes simplex virus 2, and CMV starting antiretroviral

138 1 and other sexually transmitted infections (herpes simplex virus-2, bacteria, fungi), which reprogra

139 Interestingly, the replication of herpes simplex virus, a human herpesvirus that is closel

140 Alpha herpesviruses, such as herpes simplex virus and pseudorabies virus (PRV), are n

141 5 on the structural stability of icosahedral herpes simplex virus capsids.

142 Herpes simplex viruses cause oral and genital sores as w

143 ital specimens each containing >/=105 copies herpes simplex virus DNA/ml collected a median of 5 mont

144 The herpes simplex virus for example evades immune surveilla

145 iency virus, hepatitis B virus, and neonatal herpes simplex virus, from which lessons for the evaluat

146 The transcription factor ICP4 from herpes simplex virus has a central role in regulating th

147 this assessment include adenovirus 8 and 19, herpes simplex virus (HSV) 1 and 2, human immunodeficien

148 Herpes simplex virus (HSV) 1 stimulates type I IFN expre

149 dy of neurotropic viruses in vitroIMPORTANCE Herpes simplex virus (HSV) affects millions of people wo

150 tions that US9 is neuron specific.IMPORTANCE Herpes simplex virus (HSV) and other alphaherpesviruses,

151 Herpes simplex virus (HSV) anterograde transport in neur

152 a broad antimicrobial factor that restricts herpes simplex virus (HSV) by activating type I interfer

153 The herpes simplex virus (HSV) capsid is released into the c

154 his study employed a novel strategy in which herpes simplex virus (HSV) carrying a small interfering

155 Herpes simplex virus (HSV) causes acute and relapsing sy

156 fluid (CSF) commonly predicts the absence of herpes simplex virus (HSV) central nervous system (CNS)

157 After entry into the nucleus, herpes simplex virus (HSV) DNA is coated with repressive

158 During lytic infection, herpes simplex virus (HSV) DNA is replicated by a mechan

159 Herpes simplex virus (HSV) entry into a subset of cells

160 erpesvirus entry mediator (HVEM) facilitates herpes simplex virus (HSV) entry through interactions wi

161 Herpes simplex virus (HSV) establishes a latent reservoi

162 Herpes simplex virus (HSV) establishes latency and react

163 xpression of 'pore dead' GluA1 subunits (via herpes simplex virus (HSV) GluA1-Q582E) in the lateral c

164 Pritelivir is a well-tolerated novel herpes simplex virus (HSV) helicase-primase inhibitor th

165 is known as a transcriptional coactivator of herpes simplex virus (HSV) immediate early (IE) transcri

166 The herpes simplex virus (HSV) infected cell culture polypep

167 Neuron-virus interactions that occur during herpes simplex virus (HSV) infection are not fully under

168 Importance: Genital herpes simplex virus (HSV) infection is a prevalent sexu

169 Herpes simplex virus (HSV) infection is restricted to ep

170 Herpes simplex virus (HSV) infection is widespread in th

171 for antithrombin III and plays a key role in herpes simplex virus (HSV) infection.

172 Studies of herpes simplex virus (HSV) infections of humans are limi

173 Herpes simplex virus (HSV) infections of the central ner

174 During lytic herpes simplex virus (HSV) infections, the virion host s

175 The terminase of herpes simplex virus (HSV) is composed of three subunits

176 Herpes simplex virus (HSV) is investigated not only as a

177 pediatric penetrating keratoplasty (PPK) for herpes simplex virus (HSV) keratitis.

178 ctivator HCF-1 is required for initiation of herpes simplex virus (HSV) lytic infection and for react

179 f aqueous or vitreous humor was positive for herpes simplex virus (HSV) or varicella zoster virus (VZ

180 od to visualize pseudorabies virus (PRV) and herpes simplex virus (HSV) particles in living cells.

181 Despite frequent herpes simplex virus (HSV) reactivation, peripheral nerv

182 The rate of oral herpes simplex virus (HSV) shedding was lower overall, a

183 Chronic infections with herpes simplex virus (HSV) type 1 are highly prevalent i

184 The herpes simplex virus (HSV) type I alkaline nuclease, UL1

185 Samples were tested for herpes simplex virus (HSV) types 1 and 2, Epstein-Barr v

186 synthesis during multiround transmission of herpes simplex virus (HSV) using pulse-labeling with eth

187 e by microinjecting before training a single herpes simplex virus (HSV) vector expressing either CRIS

188 tential role of a TLR4/MCP-1 signal, we used Herpes Simplex Virus (HSV) vectors (amplicons) that reta

189 Presence of CMV, EBV, and herpes simplex virus (HSV) were independent predictors o

190 Caused by the herpes simplex virus (HSV), herpes is a viral infection

191 Infection is caused by 2 subtypes of the herpes simplex virus (HSV), HSV-1 and HSV-2.

192 itro nanomolar irreversible activity against herpes simplex virus (HSV), human papilloma virus, respi

193 cell entry and subsequent lateral spread of herpes simplex virus (HSV), requires the four envelope g

194 Herpes simplex virus (HSV), which triggers the STING sig

195 Herpes simplex virus (HSV)-1 and HSV-2 are significant h

196 cally significant species of simplexviruses, herpes simplex virus (HSV)-1 and HSV-2, with estimated d

197 ACASI), before test HIV counselling, HIV and herpes simplex virus (HSV)-2 testing, and post-test coun

198 oposed to detect herpesviral DNA directly in herpes simplex virus (HSV)-infected cells and initiate i

199 In herpes simplex virus (HSV)-infected cells, ND10 bodies a

200 creased) particle-to-PFU ratio in vitro than herpes simplex virus (HSV).

201 enchymal brain inflammation, commonly due to herpes simplex virus (HSV).

202 interferon (IFN) is important for control of herpes simplex virus (HSV-1) in the central nervous syst

203 e delivered to the same sensory neurons that herpes simplex virus (HSV-1) infects when applied periph

204 Herpes simplex viruses (HSV) are human pathogens that sw

205 significant effect.IMPORTANCE Infections by herpes simplex viruses (HSV) cause painful cold sores or

206 l B virus isolates to use entry receptors of herpes simplex viruses (HSV).

207 alone mediates recognition and clearance of herpes simplex virus (HSV1)-infected cells.

208 Herpes simplex viruses (HSVs) are human pathogens that c

209 Herpes simplex viruses (HSVs) are unusual in that unlike

210 A key property of herpes simplex viruses (HSVs) is their ability to establ

211 ablation on tga20/CD11b-thymidine kinase of Herpes simplex virus (HSVTK) cerebellar organotypic cult

212 ing of mucosal immunity and then compare the herpes simplex virus, human immunodeficiency virus, and

213 , cytomegalovirus, Epstein-Barr virus (EBV), herpes simplex virus I and II, and vitreous cultures for

214 bitor ICP47, a small protein produced by the herpes simplex virus I.

215 and generalized infections that can lead to herpes simplex virus-induced acute liver failure.

216 h inhibits HIV-1, HIV-2, Influenza virus and herpes simplex virus infection, and enhances the potenti

217 ons, rubella, cytomegalovirus infection, and herpes simplex virus infections (TORCH), Apgar score <5

218 icase-primase inhibitor for the treatment of herpes simplex virus infections, was prepared.

219 r development of antiviral agents.IMPORTANCE Herpes simplex virus is a major pathogen, and although n

220 The capsid of herpes simplex virus is built up of a variety of protein

221 ens from entering the endoplasmic reticulum, herpes simplex virus is hidden from cytotoxic T lymphocy

222 ovir decreased the recurrence of any type of herpes simplex virus keratitis by approximately half.

223 nfections of the superficial cornea, such as herpes simplex virus keratitis or Acanthamoeba keratitis

224 us keratitis is significantly different from herpes simplex virus keratitis, and further studies usin

225 Further, in the murine model of herpes simplex virus keratitis, corneal pathology and ly

226 itis is significantly different from that of herpes simplex virus keratitis.

227 of autophagy/xenophagy results with mutated herpes simplex virus lacking its ICP34.5 neurovirulence

228 ) relocation of nonintegrin receptors (e.g., herpes simplex virus nectin1 and Kaposi's sarcoma-associ

229 papilloma virus, hepatitis B and C viruses, herpes simplex virus, norovirus, rotavirus, parvovirus,

230 Oncolytic herpes simplex virus (oHSV) selectively replicates in ca

231 nd tested the fate and efficacy of oncolytic herpes simplex virus (oHSV)-armed mesenchymal stem cells

232 h clinically diagnosed ARN, PCR-positive for herpes simplex virus or varicella zoster virus and evalu

233 iral reactivation, exhibiting parallels with herpes simplex virus reactivation.

234 use substantial impairment: cytomegalovirus, herpes simplex virus, rubella virus, Toxoplasma gondii,

235 of the lipid raft-dwelling US9 protein from Herpes Simplex Virus strikingly overlaps with that of th

236 ded toxoplasmosis, rubella, cytomegalovirus, herpes simplex virus, syphilis, and human immunodeficien

237 Modified herpes simplex viruses that are unable to produce glycop

238 In herpes simplex virus, the interaction between the capsid

239 transgenic mice expressing the suicide gene, herpes simplex virus thymidine kinase (HSVtk), driven by

240 g AAVP particles served to deliver the human Herpes simplex virus thymidine kinase type-1 (HSVtk) gen

241 incorporation of pro-drug converting enzyme, herpes simplex virus thymidine kinase, into therapeutic

242 Furthermore, an unrelated herpes simplex virus-thymidine kinase (HSV-TK) promoter

243 e (CTL), Th1 cell, or Th17 cell responses to herpes simplex virus, Toxoplasma gondii, and Citrobacter

244 Infectious titers of EBOV or herpes simplex virus type 1 (HSV-1) in detergents-treate

245 Lytic infection with herpes simplex virus type 1 (HSV-1) induces profound mod

246 mount of virus reactivation following ocular herpes simplex virus type 1 (HSV-1) infection.

247 Herpes simplex virus type 1 (HSV-1) is a leading cause o

248 Herpes simplex virus type 1 (HSV-1) is a ubiquitous viru

249 Glycoprotein D (gD) of herpes simplex virus type 1 (HSV-1) is one of four glyco

250 munoglobulin G antibody responses to CMV and herpes simplex virus type 1 (HSV-1) measured in archived

251 lly occurs during delivery from mothers with herpes simplex virus type 1 (HSV-1) or type 2 (HSV-2) ge

252 ological stress.IMPORTANCE Like all viruses, herpes simplex virus type 1 (HSV-1) reproduction relies

253 we reported a new series of highly defective herpes simplex virus type 1 (HSV-1) vectors that were fu

254 ormational approach to genome engineering of herpes simplex virus type 1 (HSV-1), which has a large D

255 Knockout of TRIM14 impairs herpes simplex virus type 1 (HSV-1)-triggered antiviral

256 Support for the concept that herpes simplex virus type 1 (HSV1), when present in the

257 sing purified replication factors encoded by herpes simplex virus type 1 and a 70-base minicircle tem

258 Herpes simplex virus type 1 and Alzheimer's disease: pos

259 athway are associated with susceptibility to herpes simplex virus type 1 encephalitis (HSE).

260 We found wide distribution of O-glycans on herpes simplex virus type 1 glycoproteins and demonstrat

261 gene encoding the prodrug-converting enzyme herpes simplex virus type 1 thymidine kinase (HSV1-tk) i

262 CMV, herpes simplex virus type 1, and human herpesvirus 6 inf

263 e association of 4 human herpesviruses (CMV, herpes simplex virus type 1, human herpesvirus type 6, a

264 pathogens were detected by mNGS (4 cases of herpes simplex virus type 1, including 1 case of coinfec

265 Talimogene laherparepvec (T-VEC) is a herpes simplex virus type 1-derived oncolytic immunother

266 chnique for mapping O-glycosylation sites on herpes simplex virus type 1.

267 ncy virus (HIV) agent that decreased risk of herpes simplex virus type 2 (HSV-2) acquisition in HIV p

268 fumarate (TDF) has in vitro activity against herpes simplex virus type 2 (HSV-2) and reduced HSV-2 ac

269 We focused on herpes simplex virus type 2 (HSV-2) because prior publis

270 ndoms for protection against transmission of herpes simplex virus type 2 (HSV-2) has been examined in

271 Despite the high prevalence of herpes simplex virus type 2 (HSV-2) in sub-Saharan Afric

272 Genital herpes simplex virus type 2 (HSV-2) infection causes rec

273 Globally, herpes simplex virus type 2 (HSV-2) infection is the mos

274 mmune system's protective effect against one herpes simplex virus type 2 (HSV-2) infection protects a

275 or NK cell function in response to a mucosal herpes simplex virus type 2 (HSV-2) infection.

276 HIV and herpes simplex virus type 2 (HSV-2) infections cause a s

277 tor in clinical development for treatment of herpes simplex virus type 2 (HSV-2) infections.

278 Herpes simplex virus type 2 (HSV-2) reactivation is acco

279 ART) has been incompletely characterized for herpes simplex virus type 2 (HSV-2).

280 Herpes simplex virus type 2 (HSV-2; herpes) exacerbates

281 he association between DMPA use and incident herpes simplex virus type 2 (HSV2) infection in women.

282 load, viral set point, CD4(+) T-cell count, herpes simplex virus type 2 detection, and gonorrhea.

283 against cervical cancer, cervical dysplasia, herpes simplex virus type 2, chlamydia, and syphilis.

284 l among 3408 persons coinfected with HIV and herpes simplex virus type 2.

285 nd spinal cord in response to infection with herpes simplex virus type 2.

286 mydia trachomatis, Neisseria gonorrhoeae, or herpes simplex virus type 2.

287 lar functions affected by lytic infection of Herpes Simplex Virus type I in Human primary fibroblasts

288 f herpetic neuralgia using a murine model of Herpes Simplex Virus Type-1 (HSV-1) infection.

289 Herpes simplex virus type-1 (HSV-1) is one of the most w

290 CB) women, including bacterial vaginosis and herpes simplex virus type-2 (HSV-2) infection.

291 he impact of antiretroviral therapy (ART) on herpes simplex virus type-2 (HSV-2) replication is uncle

292 ded controls for sociodemographic variables, herpes simplex virus type-2 status, and recreational dru

293 rr virus (EBV), human herpesvirus 6 (HHV-6), herpes simplex virus types 1 (HSV-1) and 2 (HSV-2), and

294 Herpes simplex virus types 1 and 2 (HSV-1 and HSV-2) are

295 e = 11.1 weeks) to Epstein Barr virus (EBV), herpes simplex virus types 1 and 2 (HSV-1 and HSV-2), an

296 Trichomonas vaginalis and viral pathogens (herpes simplex virus types 1 and 2 and adenovirus) can c

297 Seroprevalence of EBV, CMV, herpes simplex virus types 1 and 2, and human herpesviru

298 ein Barr virus (EBV), cytomegalovirus (CMV), herpes simplex virus types 1 and 2, human herpesvirus 8)

299 e, HIV, rabies, Japanese encephalitis virus, herpes simplex virus, varicella zoster virus, cytomegalo

300 97%; PPV range, 52%-95%; NPV range, 79%-80%; herpes simplex virus, vulvar ulcerations: sensitivity, 2