ライフサイエンスコーパス: herpes simplex virus 1

1 protein that is typified by ICP27 encoded by Herpes Simplex Virus 1.

2 sneuronal viral tract tracer, H129 strain of herpes simplex virus-1.

3 Adults with available results for herpes simplex virus 1/2 (HSV-1/2), varicella-zoster vir

4 V] 6, 18%; HHV8, 6%; Epstein-Barr virus, 3%; herpes simplex virus 1, 3%; varicella zoster virus, 3%;

5 ion of genetically predisposed subjects with herpes simplex virus 1, although there is substantial he

6 on library generated using the high-capacity herpes simplex virus-1 amplicon technology to deliver ba

7 However, infection with herpes simplex virus 1, an N-glycoprotein-bearing pathog

8 's cerebrospinal fluid was positive for both herpes simplex virus 1 and 2 (HSV-1 and HSV-2).

9 Glycoprotein G (gG) from herpes simplex virus 1 and 2 (HSV-1 and HSV-2, important

10 al antiviral agents against diseases such as herpes simplex virus 1 and 2 (HSV-1) (HSV-2), human immu

11 Herpes simplex virus 1 and 2 cause genital herpes, blind

12 unoglobulin M/immunoglobulin G antibodies to herpes simplex virus 1 and 2, cytomegalovirus, Epstein-B

13 us agalactiae, cytomegalovirus, enterovirus, herpes simplex virus 1 and 2, human herpesvirus 6, human

14 not been studied as extensively as those of herpes simplex virus 1 and human cytomegalovirus (HCMV).

15 ibition decreases replication of adenovirus, herpes simplex virus 1 and influenza A in cultured prima

16 ability to be infected by the herpesviruses herpes simplex virus 1 and murine herpesvirus 68 and the

17 was stable in four cell lines infected with herpes simplex virus 1 and that it was actively stabiliz

18 l tracing system based on the H129 strain of herpes simplex virus 1 and used this system in rats alon

19 lear and cytosolic DNA during infection with herpes simplex virus-1 and HIV.

20 Human herpes simplex viruses 1 and 2 (HSV-1 and HSV-2) are lar

21 Human herpes simplex viruses 1 and 2 (HSV-1 and HSV-2) are lar

22 Herpes simplex viruses 1 and 2 (HSV-1 and HSV-2) infect

23 rus, mumps virus, measles virus, lyssavirus, herpes simplex viruses 1 and 2, Epstein-Barr virus, ente

24 mmunoglobulin G seropositivity for CMV, EBV, herpes-simplex virus 1, and varicella-zoster virus were

25 Ocular infection with herpes simplex virus 1 can result in a chronic immunoinf

26 ere independent of varicella-zoster virus or herpes-simplex virus 1 coinfection.

27 The herpes simplex virus 1 deletion mutant HSV1716 is a cond

28 The catalytic subunit of herpes simplex virus 1 DNA polymerase (HSV-1 Pol) has be

29 lex, we individually mutated residues in the herpes simplex virus 1 gB coil-arm complex to alanine an

30 Here, we have characterized the full-length herpes simplex virus 1 gB in a native membrane by displa

31 pted this method to rapidly deliver a 152 kb herpes simplex virus 1 genome cloned in yeast into mamma

32 e CD99, PILR-associating neural protein, and Herpes simplex virus-1 glycoprotein B.

33 nt productive infection of cultured cells by herpes simplex virus 1 has made it a paradigm for this m

34 n of foreign DNA-sensing pathways.IMPORTANCE Herpes simplex virus 1 (HSV-1) afflicts 80% of the popul

35 Herpes simplex virus 1 (HSV-1) and 2 (HSV-2) cause a var

36 acacine herpesvirus 1) is closely related to herpes simplex virus 1 (HSV-1) and encodes gD, which sha

37 Herpes simplex virus 1 (HSV-1) and HSV-2 are large, doub

38 Herpes simplex virus 1 (HSV-1) and HSV-2 are medically s

39 qualitative detection and differentiation of herpes simplex virus 1 (HSV-1) and HSV-2 DNA in 1,351 cu

40 Herpes simplex virus 1 (HSV-1) and HSV-2 establish laten

41 Here, we report that human herpes simplex virus 1 (HSV-1) and HSV-2 had opposite im

42 Herpes simplex virus 1 (HSV-1) and HSV-2 infect many hum

43 aken to compare the host immune responses to herpes simplex virus 1 (HSV-1) and HSV-2 infection by th

44 Herpes simplex virus 1 (HSV-1) and HSV-2, two closely re

45 ecombinant glycoprotein G (gG) antigens from herpes simplex virus 1 (HSV-1) and HSV-2.

46 We generated recombinant herpes simplex virus 1 (HSV-1) and human cytomegalovirus

47 Here, we generated recombinant herpes simplex virus 1 (HSV-1) and human cytomegalovirus

48 f UL37 homologs from two alphaherpesviruses, herpes simplex virus 1 (HSV-1) and pseudorabies virus (P

49 6p (VP1/2) is the largest protein encoded by herpes simplex virus 1 (HSV-1) and resides in the innerm

50 hat gB homologs from two alphaherpesviruses, herpes simplex virus 1 (HSV-1) and saimiriine herpesviru

51 with demonstrated antiviral activity against herpes simplex virus 1 (HSV-1) and several other herpesv

52 te aspects of intrinsic immunity to restrict herpes simplex virus 1 (HSV-1) as well as other viruses.

53 Herpes simplex virus 1 (HSV-1) can establish life-long l

54 Herpes simplex virus 1 (HSV-1) can undergo a productive

55 The herpes simplex virus 1 (HSV-1) capsid is a huge assembly

56 Herpes simplex virus 1 (HSV-1) causes a chronic, lifelon

57 Herpes simplex virus 1 (HSV-1) causes a spectrum of dise

58 ar disease in virus-infected mice.IMPORTANCE Herpes simplex virus 1 (HSV-1) causes cold sores and neo

59 Consistent with this, herpes simplex virus 1 (HSV-1) causes invariable and rap

60 Herpes simplex virus 1 (HSV-1) causes one of the most pr

61 Herpes simplex virus 1 (HSV-1) causes recurrent mucocuta

62 f different strains of mice with recombinant herpes simplex virus 1 (HSV-1) constitutively expressing

63 On entry into the nucleus, herpes simplex virus 1 (HSV-1) DNA localizes to nuclear

64 Herpes simplex virus 1 (HSV-1) effectively counteracts a

65 Herpes simplex virus 1 (HSV-1) encodes an endoribonuclea

66 Herpes simplex virus 1 (HSV-1) encodes the multifunction

67 Herpes simplex virus 1 (HSV-1) enters mice via olfactory

68 ponent of cell membranes and is required for herpes simplex virus 1 (HSV-1) entry (1-3).

69 The dissection of the herpes simplex virus 1 (HSV-1) entry mechanism is compli

70 ined whether Cbl mediates the removal of the herpes simplex virus 1 (HSV-1) entry receptor Nectin-1 f

71 Binding of herpes simplex virus 1 (HSV-1) envelope glycoprotein D (

72 Herpes simplex virus 1 (HSV-1) establishes a lifelong la

73 Herpes simplex virus 1 (HSV-1) establishes latency withi

74 Herpes simplex virus 1 (HSV-1) establishes lifelong infe

75 Herpes simplex virus 1 (HSV-1) establishes lifelong late

76 Herpes simplex virus 1 (HSV-1) facilitates virus entry i

77 ultaneous presence of a helper virus such as herpes simplex virus 1 (HSV-1) for productive replicatio

78 ave costimulatory properties, is targeted by herpes simplex virus 1 (HSV-1) for viral immune escape.

79 plication greatly enhances expression of the herpes simplex virus 1 (HSV-1) gamma2 late genes by stil

80 Herpes simplex virus 1 (HSV-1) genomes are associated wi

81 Previous methods of herpes simplex virus 1 (HSV-1) genotype analysis have la

82 ns within the cytoplasmic tail (cytotail) of herpes simplex virus 1 (HSV-1) gH were previously observ

83 estigate the role of the signal sequences of herpes simplex virus 1 (HSV-1) gK on virus replication a

84 Herpes simplex virus 1 (HSV-1) glycoprotein B (gB)-speci

85 gp120) construct fused to a small portion of herpes simplex virus 1 (HSV-1) glycoprotein D (gD) so th

86 Herpes simplex virus 1 (HSV-1) glycoprotein E (gE) media

87 Herpes simplex virus 1 (HSV-1) glycoprotein K (gK) is ex

88 The herpes simplex virus 1 (HSV-1) glycoprotein K (gK)/UL20

89 on similar to that of a previously described herpes simplex virus 1 (HSV-1) homolog, pUL6.

90 The herpes simplex virus 1 (HSV-1) ICP0 protein is an E3 ubi

91 mia nuclear bodies (PML NBs), is a target of herpes simplex virus 1 (HSV-1) ICP0-mediated degradation

92 Herpes simplex virus 1 (HSV-1) ICP27 is a shuttling prot

93 The herpes simplex virus 1 (HSV-1) ICP34.5 protein strongly

94 Herpes simplex virus 1 (HSV-1) ICP4 that lacks a TAD ver

95 herpesvirus (KSHV) ORF6 is homologous to the herpes simplex virus 1 (HSV-1) ICP8 and Epstein-Barr vir

96 Herpes simplex virus 1 (HSV-1) ICP8 is a single-stranded

97 For example, herpes simplex virus 1 (HSV-1) immediate early (IE) prot

98 As HCF1 is best known for its function in herpes simplex virus 1 (HSV-1) immediate early gene tran

99 teins have some functional similarities with herpes simplex virus 1 (HSV-1) immediate early protein I

100 The herpes simplex virus 1 (HSV-1) immediate early protein I

101 Herpes simplex virus 1 (HSV-1) immediate-early protein I

102 Herpes simplex virus 1 (HSV-1) immediate-early protein I

103 The herpes simplex virus 1 (HSV-1) immediate-early protein,

104 man foreskin fibroblasts (HFF) infected with herpes simplex virus 1 (HSV-1) in the absence of viral g

105 tinocytes represent a primary entry site for herpes simplex virus 1 (HSV-1) in vivo.

106 The herpes simplex virus 1 (HSV-1) infected cell protein 0 (

107 Similar tests were done with persons with herpes simplex virus 1 (HSV-1) infection as a model chro

108 factor for human cytomegalovirus (HCMV) and herpes simplex virus 1 (HSV-1) infection by inhibiting b

109 Uncontrolled herpes simplex virus 1 (HSV-1) infection can advance to

110 For example, the requirement for ICP0 during herpes simplex virus 1 (HSV-1) infection can be largely

111 Ocular herpes simplex virus 1 (HSV-1) infection can lead to mul

112 Thus, we have studied herpes simplex virus 1 (HSV-1) infection in the tree shr

113 Herpes simplex virus 1 (HSV-1) infection is an incurable

114 Herpes simplex virus 1 (HSV-1) infection is widespread a

115 We investigated the effect of herpes simplex virus 1 (HSV-1) infection on transcriptio

116 Herpes simplex virus 1 (HSV-1) infection triggers specif

117 During herpes simplex virus 1 (HSV-1) infection, empty procapsi

118 esponse to an in vivo, TLR2-dependent model, herpes simplex virus 1 (HSV-1) infection.

119 f the intrinsic antiviral immune response to herpes simplex virus 1 (HSV-1) infection.

120 Herpes simplex virus 1 (HSV-1) infections are common and

121 efects in IFN responses can result in lethal herpes simplex virus 1 (HSV-1) infections, usually from

122 Herpes simplex virus 1 (HSV-1) infects humans through st

123 Herpes simplex virus 1 (HSV-1) infects most people and c

124 The immediate early protein ICP0 of herpes simplex virus 1 (HSV-1) interacts with CIN85, an

125 Herpes simplex virus 1 (HSV-1) is a common human pathoge

126 Herpes simplex virus 1 (HSV-1) is a double-stranded DNA

127 Herpes simplex virus 1 (HSV-1) is a double-stranded DNA

128 Infected cell protein 0 (ICP0) of herpes simplex virus 1 (HSV-1) is a key regulator in bot

129 We report here that UL37 of herpes simplex virus 1 (HSV-1) is a protein deamidase th

130 The infected cell polypeptide 4 (ICP4) of herpes simplex virus 1 (HSV-1) is a regulator of viral t

131 Herpes simplex virus 1 (HSV-1) is a ubiquitous and impor

132 Herpes simplex virus 1 (HSV-1) is a ubiquitous virus tha

133 Human herpes simplex virus 1 (HSV-1) is a widespread pathogen,

134 Herpes simplex virus 1 (HSV-1) is among the most common

135 Infected cell protein 0 (ICP0) of herpes simplex virus 1 (HSV-1) is an alpha gene product

136 Herpes simplex virus 1 (HSV-1) is an important human pat

137 One aspect of intrinsic resistance to herpes simplex virus 1 (HSV-1) is conferred by component

138 The UL16 tegument protein of herpes simplex virus 1 (HSV-1) is conserved among all he

139 Herpes simplex virus 1 (HSV-1) is one of the eight herpe

140 In this study, we demonstrate that herpes simplex virus 1 (HSV-1) is targeted by tetherin.

141 viral DNA from NPC-bound capsids.IMPORTANCE Herpes simplex virus 1 (HSV-1) is the causative agent of

142 We have shown previously that herpes simplex virus 1 (HSV-1) lacking expression of the

143 nal (NLS) sequences previously identified in herpes simplex virus 1 (HSV-1) large terminase and human

144 Herpes simplex virus 1 (HSV-1) latency entails the repre

145 In the final stages of the herpes simplex virus 1 (HSV-1) life cycle, a viral nucle

146 ermeasure, infected cell protein 0 (ICP0) of herpes simplex virus 1 (HSV-1) localizes to ND10s, degra

147 ICP0 is required for efficient initiation of herpes simplex virus 1 (HSV-1) lytic infection and produ

148 s from a variant to target the mRNA encoding herpes simplex virus 1 (HSV-1) major transcription regul

149 Herpes simplex virus 1 (HSV-1) most commonly causes recr

150 ies to protect against ocular infection with herpes simplex virus 1 (HSV-1) must address the issue of

151 ve previously shown that the live-attenuated herpes simplex virus 1 (HSV-1) mutant lacking the nuclea

152 Herpes simplex virus 1 (HSV-1) mutants that lack the gam

153 Assembly of herpes simplex virus 1 (HSV-1) occurs in the cytoplasm,

154 istar rats were intranasally inoculated with herpes simplex virus 1 (HSV-1) or phosphate-buffered sal

155 ide that was sufficient to interact with the herpes simplex virus 1 (HSV-1) portal protein and was ne

156 During DNA encapsidation, herpes simplex virus 1 (HSV-1) procapsids are converted

157 Herpes simplex virus 1 (HSV-1) protein ICP27 enables vir

158 The herpes simplex virus 1 (HSV-1) protein ICP34.5, encoded

159 Herpes simplex virus 1 (HSV-1) regulatory protein ICP0 s

160 Herpes simplex virus 1 (HSV-1) remodels nuclear membrane

161 ivating protein, p35, is important for acute herpes simplex virus 1 (HSV-1) replication in mice.

162 Previous work has shown that LMB blocks herpes simplex virus 1 (HSV-1) replication in Vero cells

163 was to reexamine the requirement of UL21 for herpes simplex virus 1 (HSV-1) replication.

164 Herpes simplex virus 1 (HSV-1) required cholesterol or d

165 Ocular infection with herpes simplex virus 1 (HSV-1) results in a chronic immu

166 Infection of mature HD10.6 neurons by herpes simplex virus 1 (HSV-1) results in a delayed but

167 determined whether cytomegalovirus (CMV) and herpes simplex virus 1 (HSV-1) seropositivity were assoc

168 Ocular infection with herpes simplex virus 1 (HSV-1) sets off an inflammatory

169 sRNA, encephalomyocarditis virus (EMCV), and herpes simplex virus 1 (HSV-1) show impaired production

170 The herpes simplex virus 1 (HSV-1) strain McKrae is highly v

171 The herpes simplex virus 1 (HSV-1) tegument is known to cont

172 nd ICP34.5 are among the proteins encoded by herpes simplex virus 1 (HSV-1) that modulate type I IFN

173 Humans occasionally transmit herpes simplex virus 1 (HSV-1) to captive primates, who

174 e ability of the prototypic alphaherpesvirus herpes simplex virus 1 (HSV-1) to enter neurons via axon

175 ts as a restriction factor against ICP0-null herpes simplex virus 1 (HSV-1) to limit viral replicatio

176 A) genomic stability pathway is exploited by herpes simplex virus 1 (HSV-1) to promote viral DNA synt

177 Lytic infection by herpes simplex virus 1 (HSV-1) triggers a change in many

178 Herpes simplex virus 1 (HSV-1) typically causes recurren

179 The herpes simplex virus 1 (HSV-1) U(L)21 gene encodes a 62-

180 cellular protein IFI16 colocalizes with the herpes simplex virus 1 (HSV-1) ubiquitin ligase ICP0 at

181 Herpes simplex virus 1 (HSV-1) UL20 plays a crucial role

182 The herpes simplex virus 1 (HSV-1) UL25 gene product is a mi

183 The herpes simplex virus 1 (HSV-1) UL37 protein functions in

184 The herpes simplex virus 1 (HSV-1) UL37 protein has also bee

185 Herpes simplex virus 1 (HSV-1) UL5/8/52 helicase-primase

186 The herpes simplex virus 1 (HSV-1) UL51 gene encodes a 244-a

187 The herpes simplex virus 1 (HSV-1) UL6 portal protein forms

188 ogic protection requisite for an efficacious herpes simplex virus 1 (HSV-1) vaccine remain unclear wi

189 Herein, we report that the live-attenuated herpes simplex virus 1 (HSV-1) VC2 vaccine strain, which

190 Herpes simplex virus 1 (HSV-1) viral glycoproteins gD (c

191 The herpes simplex virus 1 (HSV-1) virion DNA contains nicks

192 incorporation of tegument proteins into the herpes simplex virus 1 (HSV-1) virion during virion asse

193 Following infection of epithelial tissues, herpes simplex virus 1 (HSV-1) virions travel via axonal

194 Herpes simplex virus 1 (HSV-1) virions, like those of al

195 Herpes simplex virus 1 (HSV-1) virus is the leading caus

196 Herpes simplex virus 1 (HSV-1) was shown to contain cata

197 Herpes simplex virus 1 (HSV-1), a leading cause of genit

198 Herpes simplex virus 1 (HSV-1), a ubiquitous human patho

199 Herpes simplex virus 1 (HSV-1), a ubiquitous virus that

200 e PCR detected varicella-zoster virus (VZV), herpes simplex virus 1 (HSV-1), and HSV-2 DNA in 117 fre

201 carrying latent mouse cytomegalovirus (CMV), herpes simplex virus 1 (HSV-1), and/or murine gammaherpe

202 In herpes simplex virus 1 (HSV-1), binding clusters enriche

203 at ICP0, a key regulatory protein encoded by herpes simplex virus 1 (HSV-1), binds ubiquitin-specific

204 The latency-associated transcript (LAT) of herpes simplex virus 1 (HSV-1), CD8alpha(+) dendritic ce

205 In the course of primary infection with herpes simplex virus 1 (HSV-1), children with inborn err

206 virus type 1 (HIV-1), cytomegalovirus (CMV), herpes simplex virus 1 (HSV-1), Ebola virus, vaccinia vi

207 lected TRIM proteins in autophagy induced by herpes simplex virus 1 (HSV-1), encephalomyocarditis vir

208 rus, human immunodeficiency virus type 1, or herpes simplex virus 1 (HSV-1), failed to reveal any enh

209 Persistent pathogens, such as herpes simplex virus 1 (HSV-1), have evolved a variety o

210 say for the detection and differentiation of herpes simplex virus 1 (HSV-1), HSV-2, and varicella-zos

211 sitioned laboratory-developed PCR assays for herpes simplex virus 1 (HSV-1), HSV-2, and varicella-zos

212 roinvasive alphaherpesviruses, such as human herpes simplex virus 1 (HSV-1), HSV-2, and veterinarian

213 replication of four different herpesviruses, herpes simplex virus 1 (HSV-1), HSV-2, HCMV, and mouse c

214 ecifically, FIT-039 inhibited replication of herpes simplex virus 1 (HSV-1), HSV-2, human adenovirus,

215 During infection by herpes simplex virus 1 (HSV-1), macrophages acquire enha

216 trast, of the more than 80 mRNAs harbored by herpes simplex virus 1 (HSV-1), only 5 are spliced.

217 Following infection with herpes simplex virus 1 (HSV-1), PIAS1 is recruited to nu

218 Here we focus on two tegument proteins from herpes simplex virus 1 (HSV-1), pUL7 and pUL51, which ha

219 such complex regulation, viruses, including herpes simplex virus 1 (HSV-1), rely on multifunctional

220 Although many herpesviruses, including herpes simplex virus 1 (HSV-1), stimulate mTORC1, how HS

221 zuelan equine encephalitis virus (VEEV), and herpes simplex virus 1 (HSV-1), suggesting that LIMK inh

222 Herpes simplex virus 1 (HSV-1), the prototypic member of

223 During infection by herpes simplex virus 1 (HSV-1), the viral capsid is tran

224 as the replication of a related herpesvirus, herpes simplex virus 1 (HSV-1), was not impacted.

225 For herpes simplex virus 1 (HSV-1), we and others have previ

226 nfected cellular protein 0 (ICP0) from human herpes simplex virus 1 (HSV-1), which also disrupts PML-

227 Herpes simplex virus 1 (HSV-1), which causes a variety o

228 role in the anterograde axonal transport of herpes simplex virus 1 (HSV-1), yet the molecular mechan

229 Herpes simplex virus 1 (HSV-1)-infected cell protein 0 (

230 those from EBV-infected and, more recently, herpes simplex virus 1 (HSV-1)-infected cells, cement th

231 onent glycoprotein-tegument complex found in herpes simplex virus 1 (HSV-1)-infected cells.

232 Although most herpes simplex virus 1 (HSV-1)-infected individuals shed

233 In animals, herpes simplex virus 1 (HSV-1)-infected Nlrc3(-/-) mice

234 essed immunogenicity and vaccine efficacy in herpes simplex virus 1 (HSV-1)-seropositive guinea pigs

235 SP141), bound to RIG-I during infection with herpes simplex virus 1 (HSV-1).

236 between AAV2 and one of its helper viruses, herpes simplex virus 1 (HSV-1).

237 of ICP27, an immediate-early (IE) protein of herpes simplex virus 1 (HSV-1).

238 pe dynamics as well as the nuclear egress of herpes simplex virus 1 (HSV-1).

239 zation of a replication-competent controlled herpes simplex virus 1 (HSV-1).

240 ediator (HVEM) can both mediate the entry of herpes simplex virus 1 (HSV-1).

241 us type 2 is known to inhibit replication of herpes simplex virus 1 (HSV-1).

242 r group for human cytomegalovirus (HCMV) and herpes simplex virus 1 (HSV-1).

243 es, including the genomes of viruses such as herpes simplex virus 1 (HSV-1).

244 f mice that have been ocularly infected with herpes simplex virus 1 (HSV-1).

245 reported to exacerbate the pathogenicity of herpes simplex virus 1 (HSV-1).

246 sensitivity and possible countermeasures of herpes simplex virus 1 (HSV-1).

247 d protein kinases (MAPKs) are deregulated by herpes simplex virus 1 (HSV-1).

248 cient replication and reactivation of latent herpes simplex virus 1 (HSV-1).

249 esponse to restrict viral pathogens, such as herpes simplex virus 1 (HSV-1).

250 ganglia (TG) of mice latently infected with herpes simplex virus 1 (HSV-1).

251 tive diagnostic tool to confirm diagnosis of herpes simplex virus-1 (HSV) in corneal scrapings with h

252 one marrow-derived macrophage infection with herpes simplex virus-1 (HSV-1) and Salmonella typhimuriu

253 Herpesviruses exemplified by herpes simplex virus-1 (HSV-1) attach to cell surface he

254 Herpes simplex virus-1 (HSV-1) causes life-long morbidit

255 Childhood herpes simplex virus-1 (HSV-1) encephalitis (HSE) may re

256 f many human promoters, but is essential for herpes simplex virus-1 (HSV-1) gene transcription and re

257 ed that xiamycin A is a potent agent against herpes simplex virus-1 (HSV-1) in vitro.

258 Herpes simplex virus-1 (HSV-1) is a double-stranded DNA

259 Herpes simplex virus-1 (HSV-1) is a human pathogen that

260 Herpes Simplex Virus-1 (HSV-1) is a ubiquitous human pat

261 Herein we demonstrate that oncolytic herpes simplex virus-1 (HSV-1) potently activates human

262 ed interferon-alpha secretion in response to herpes simplex virus-1 (HSV-1), whereas granzyme-B induc

263 dV), coxsackievirus A24, enterovirus 70, and herpes simplex-virus-1 (HSV-1) was tested in standard in

264 svirus [KSHV], Epstein-Barr virus [EBV], and herpes simplex virus 1 [HSV-1]) genomes and induces the

265 choriomeningitis virus (LCMV) infection, or herpes simplex virus 1 (HSV1) infection was profoundly d

266 re more susceptible to lethal infection with herpes simplex virus 1 (HSV1) than wild-type mice.

267 Here, an ultrastructural study of herpes simplex virus 1 (HSV1)-infected cells revealed en

268 Latent herpes simplex virus-1 (HSV1) genomes in peripheral nerv

269 provision of antagomir-155 nanoparticles to herpes simplex virus 1-infected mice led to diminished S

270 e the molecular organization of chromatin in herpes simplex virus 1 infection and its effect on the t

271 er describes the extremely rapid response to herpes simplex virus 1 infection of cellular protein IFI

272 Herpes simplex virus 1 infection triggers multiple chang

273 155 expression was up-regulated after ocular herpes simplex virus 1 infection, with the increased Mir

274 ers in the lung, and are resistant to lethal herpes simplex virus-1 infection due to enhanced product

275 n double-stranded DNA (dsDNA) stimulation or herpes simplex virus-1 infection, but also enhances auto

276 Furthermore, in a murine model of herpes simplex virus-1 infection, mice lacking DC-specif

277 promised survival during influenza virus and herpes simplex virus 1 infections.

278 To facilitate studies of herpes simplex virus 1 latency, cell culture models of q

279 76-9 (MHC I low) tumors respond to oncolytic herpes simplex virus-1 (oHSV-1) and PD-1 blockade combin

280 e, whereas infection with influenza A virus, herpes simplex virus 1, or cytomegalovirus induced a str

281 The U(S)3 protein kinase of herpes simplex virus 1 plays a key role in blocking apop

282 The conserved UL51 gene of herpes simplex virus 1 plays important roles in cell-to-

283 Here, we address this deficit, focusing on a herpes simplex virus-1 protein, ICP27.

284 er replication of BKV, whereas influenza and herpes simplex virus 1 replication were clearly reduced.

285 EBNA-3B and ICP4 from Epstein-Barr virus and herpes simplex virus 1, respectively, both members of th

286 Random walk modelling of herpes simplex virus 1-sized particles in a three-dimens

287 study focuses on two tegument proteins from herpes simplex virus 1 that are conserved in all herpesv

288 estrict gene expression and pathogenicity of herpes simplex virus 1, thereby promoting long-term infe

289 modified HOS cells (HOS-313) expressing the herpes simplex virus 1 thymidine kinase (HSV-1 TK).

290 tein domains, as well as a truncated form of herpes simplex virus 1 thymidine kinase (HSV-TK).

291 with that of a widely used PRG based on the herpes simplex virus 1 thymidine kinase.

292 ed by HDACI-induced and p21-driven truncated herpes simplex virus-1 thymidine kinase sr39 mutant (ttk

293 e the design and use of an engineered cyclic herpes simplex virus 1-thymidine kinase (HSV1-TK) PET re

294 ture of its conserved N-terminal domain from herpes simplex virus 1 to 2.0-A resolution, which reveal

295 15% amino acid sequence identities with the herpes simplex virus 1 UL11 and cytomegalovirus UL99 teg

296 Infected cell protein 8 (ICP8) from herpes simplex virus 1 was first identified as a single-

297 wed evidence of acute herpesvirus infection; herpes simplex virus 1 was found most often.

298 tudy, the structural, material properties of herpes simplex virus 1 were investigated.

299 BNA-3B from Epstein-Barr virus and ICP4 from herpes simplex virus 1, which are genes important for la

300 It has been proposed that herpes simplex virus 1 with VP22 deleted requires second