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1 glia) infected with a common viral pathogen (herpes simplex virus type 1).
2 RNA viruses and some DNA viruses, including herpes simplex virus type 1.
3 chnique for mapping O-glycosylation sites on herpes simplex virus type 1.
4 globulin G antibodies to cytomegalovirus and herpes simplex virus type 1.
5 required for the cytoplasmic envelopment of herpes simplex virus type 1.
6 have reduced susceptibility to infection by herpes simplex virus type 1.
7 mmunogenic situations such as infection with herpes simplex virus type 1.
10 sing purified replication factors encoded by herpes simplex virus type 1 and a 70-base minicircle tem
12 ling nucleic acid recognition, is usurped by herpes simplex virus type 1 and functions together with
13 ith the corresponding sequences of UL36 from herpes simplex virus type 1 and M48 from murine cytomega
14 o exhibited increased susceptibility to both herpes simplex virus type 1 and murine gammaherpesvirus
15 virus (PRV), an alphaherpesvirus related to herpes simplex virus type 1 and varicella-zoster virus,
16 ediate-early genes of the alphaherpesviruses herpes simplex virus type 1 and varicella-zoster virus.
17 on renders Ksrp(-)(/)(-) cells refractory to herpes simplex virus type 1 and vesicular stomatitis vir
18 vities and underwent serological testing for herpes simplex virus types 1 and 2 (HSV-1 and -2) and co
22 e = 11.1 weeks) to Epstein Barr virus (EBV), herpes simplex virus types 1 and 2 (HSV-1 and HSV-2), an
23 Trichomonas vaginalis and viral pathogens (herpes simplex virus types 1 and 2 and adenovirus) can c
25 Plus method for the qualitative detection of herpes simplex virus types 1 and 2 in cerebrospinal flui
27 ein Barr virus (EBV), cytomegalovirus (CMV), herpes simplex virus types 1 and 2, human herpesvirus 8)
28 licate in alpha1,3GT-positive cells, whereas herpes simplex viruses type 1 and type 2 (HSV-1 and HSV-
30 on between education and cytomegalovirus and herpes simplex virus type 1 antibody levels remained str
34 in UL25 attaches to the external vertices of herpes simplex virus type 1 capsids and is required for
37 l structure of a herpesvirus polymerase, the Herpes Simplex Virus type 1 DNA polymerase, at 2.7 A res
38 to activated arrays in cells expressing the herpes simplex virus type 1 E3 ubiquitin ligase, ICP0, w
46 med at engineering the prefusion form of the herpes simplex virus type 1 gB ectodomain, including mod
49 We found wide distribution of O-glycans on herpes simplex virus type 1 glycoproteins and demonstrat
50 VZV ORFs transcribed during latency to their herpes simplex virus type 1 homologues reveals that the
51 the critical gamma(1)34.5 virulence genes of herpes simplex virus type 1 (HSV) under translation cont
52 the central 970 residues of this protein for herpes simplex virus type 1 (HSV-1 UL36, 3164 residues).
55 transport is essential for the transport of herpes simplex virus type 1 (HSV-1) along axons, yet lit
59 region of alphaherpesviruses, termed US3 in herpes simplex virus type 1 (HSV-1) and pseudorabies vir
60 n (VP1/2cbd) into assemblons is conserved in herpes simplex virus type 1 (HSV-1) and that this recrui
61 by exploiting the high transgene capacity of herpes simplex virus type 1 (HSV-1) and the episomal ret
62 bution of the neurotropic alphaherpesviruses-herpes simplex virus type 1 (HSV-1) and type 2 (HSV-2) a
64 the availability of antiviral chemotherapy, herpes simplex virus type 1 (HSV-1) and type 2 (HSV-2) i
66 e-early (IE) genes of the alphaherpesviruses herpes simplex virus type 1 (HSV-1) and varicella-zoster
67 haherpesvirus related to the human pathogens herpes simplex virus type 1 (HSV-1) and varicella-zoster
68 Both cell lines supported the replication of herpes simplex virus type 1 (HSV-1) and vesicular stomat
69 etter define the roles of gE, gI, and Us9 in herpes simplex virus type 1 (HSV-1) anterograde spread u
70 We examine whether APOE determines CMV and herpes simplex virus type 1 (HSV-1) antibody levels and
71 e same subjects, there was no association of herpes simplex virus type 1 (HSV-1) antibody levels with
72 In this report we use lytic infection by herpes simplex virus type 1 (HSV-1) as a model to examin
75 y in infection of primary human fibroblasts, herpes simplex virus type 1 (HSV-1) disrupts the centros
76 age and packaging of replicated concatemeric herpes simplex virus type 1 (HSV-1) DNA corresponds to a
77 o determine the presence and copy numbers of herpes simplex virus type 1 (HSV-1) DNA in human trigemi
78 though the seven viral proteins required for herpes simplex virus type 1 (HSV-1) DNA replication have
80 estigate possible immune mechanisms in fatal herpes simplex virus type 1 (HSV-1) encephalitis (HSE) a
84 tations in the thymidine kinase gene (tk) of herpes simplex virus type 1 (HSV-1) explain most cases o
85 at, unlike host FcgammaRs but similar to the herpes simplex virus type 1 (HSV-1) Fc receptor gE-gI, g
88 lutamic acid, or the analogous residues from herpes simplex virus type 1 (HSV-1) gB (HR and RVEA).
90 res of the ectodomains of two FL1 mutants of herpes simplex virus type 1 (HSV-1) gB to clarify whethe
91 entry of the viral genome into the nucleus, herpes simplex virus type 1 (HSV-1) gene expression is r
93 the chromatin structures associated with the herpes simplex virus type 1 (HSV-1) genome during latenc
95 e latency-associated transcript (LAT) of the herpes simplex virus type 1 (HSV-1) genome is transcribe
102 have shown previously that immunization with herpes simplex virus type 1 (HSV-1) glycoprotein K (gK)
106 ed to result in partial complementation of a herpes simplex virus type 1 (HSV-1) ICP0 null (ICP0(-))
113 igeminal ganglia (TG) latently infected with herpes simplex virus type 1 (HSV-1) indicate the presenc
117 innate responses are involved in controlling herpes simplex virus type 1 (HSV-1) infection and that t
118 n important role in the optimal clearance of herpes simplex virus type 1 (HSV-1) infection in mice.
119 sor protein CoREST is involved in repressing herpes simplex virus type 1 (HSV-1) infection in the abs
120 ect of glycoprotein K (gK) overexpression on herpes simplex virus type 1 (HSV-1) infection in two dif
136 toplasmic domain of glycoprotein B (gB) from herpes simplex virus type 1 (HSV-1) is an important regu
138 nine (S/T) kinase encoded by the US3 gene of herpes simplex virus type 1 (HSV-1) is conserved in vari
144 riched in acetyl histone H3 (K9, K14) during herpes simplex virus type 1 (HSV-1) latency, whereas all
148 munoglobulin G antibody responses to CMV and herpes simplex virus type 1 (HSV-1) measured in archived
155 rophylaxis, recommended to prevent recurrent herpes simplex virus type 1 (HSV-1) ocular disorders, ma
157 te both adaptive and innate immunity against herpes simplex virus type 1 (HSV-1) ocular infection.
158 interferon signaling gene were infected with herpes simplex virus type 1 (HSV-1) or an attenuated rec
159 Women with primary or secondary syphilis, herpes simplex virus type 1 (HSV-1) or HSV-2 infection,
160 bits latently infected with either wild-type herpes simplex virus type 1 (HSV-1) or the latency-assoc
161 lly occurs during delivery from mothers with herpes simplex virus type 1 (HSV-1) or type 2 (HSV-2) ge
162 Two in-frame, C-terminal isoforms of the herpes simplex virus type 1 (HSV-1) origin binding prote
163 rt the replication of plasmid DNA containing herpes simplex virus type 1 (HSV-1) origin sequences in
169 Viral genes sufficient and required for herpes simplex virus type 1 (HSV-1) reactivation were id
172 n modulating the acquired immune response to herpes simplex virus type 1 (HSV-1) remains ill defined;
173 ological stress.IMPORTANCE Like all viruses, herpes simplex virus type 1 (HSV-1) reproduction relies
174 initial infection and cell-to-cell spread by herpes simplex virus type 1 (HSV-1) require the interact
179 lycoprotein E (gE) is required for efficient herpes simplex virus type 1 (HSV-1) spread from the inoc
180 vitro serial passage of a Deltagamma(1)34.5 herpes simplex virus type 1 (HSV-1) strain in SK-N-SH ne
182 ortant phase in the development of an ocular herpes simplex virus type 1 (HSV-1) subunit vaccine is t
193 we reported a new series of highly defective herpes simplex virus type 1 (HSV-1) vectors that were fu
195 glycoprotein C (gC), a major constituent of herpes simplex virus type 1 (HSV-1) virions, by hijackin
198 erent strains of mice infected ocularly with herpes simplex virus type 1 (HSV-1) was investigated.
200 svirus 8 (HHV-8), cytomegalovirus (CMV), and herpes simplex virus type 1 (HSV-1) were detected in 90%
202 pathogens, varicella-zoster virus (VZV) and herpes simplex virus type 1 (HSV-1), as well as two vete
203 Here, we use affinity assays to show that herpes simplex virus type 1 (HSV-1), but not HSV-2, bind
205 infection of human monocyte-derived DCs with herpes simplex virus type 1 (HSV-1), CYTIP is rapidly de
206 ed States population is infected with either herpes simplex virus type 1 (HSV-1), herpes simplex viru
211 with viral encephalitic agents: enterovirus, herpes simplex virus type 1 (HSV-1), varicella-zoster vi
212 ormational approach to genome engineering of herpes simplex virus type 1 (HSV-1), which has a large D
213 tion in bacterial lipopolysaccharide (LPS)-, herpes simplex virus type 1 (HSV-1)-, or adenovirus (ser
215 stress-inducing agents, protein synthesis in herpes simplex virus type 1 (HSV-1)-infected cells is no
218 estigated their suppressive capacities using herpes simplex virus type 1 (HSV-1)-specific effector T
228 After replicating in surface epithelia, herpes simplex virus type-1 (HSV-1) enters the axonal te
230 an sulfate (3-OS HS), contribute strongly to herpes simplex virus type-1 (HSV-1) infection in vitro.
233 rr virus (EBV), human herpesvirus 6 (HHV-6), herpes simplex virus types 1 (HSV-1) and 2 (HSV-2), and
234 to mimic genital herpes infections caused by herpes simplex virus types 1 (HSV-1) and 2 (HSV-2).
235 ript mapping to open reading frame (ORF) 61 (herpes simplex virus type 1 [HSV-1] ICP0 homolog) was co
237 tegument proteins VP13/14, VP22, and VP16 of herpes simplex virus type 1 (HSV1) are components of pri
239 e types of viruses, adenovirus type 5 (AV5), herpes simplex virus type 1 (HSV1), simian virus 40 (SV4
241 e association of 4 human herpesviruses (CMV, herpes simplex virus type 1, human herpesvirus type 6, a
242 previously that the plating efficiency of a herpes simplex virus type 1 ICP0-null mutant was enhance
250 P22 is required for efficient replication of herpes simplex virus type 1 in some cell types (permissi
251 ransneuronal transport of the H129 strain of herpes simplex virus type 1 in the Cebus monkey to label
253 pathogens were detected by mNGS (4 cases of herpes simplex virus type 1, including 1 case of coinfec
254 e factor 15, Slug, and SPDEF, stimulated the herpes simplex virus type 1-infected cell protein 0 (ICP
260 neration Ad (Ad) vector, which expresses the herpes simplex virus type 1 mutant thymidine kinase (HSV
264 n is influenced by sequence context and that herpes simplex virus type 1 Pol may dissociate more freq
269 crystal structure of the gB ectodomain from herpes simplex virus type 1 reveals a multidomain trimer
270 meric red fluorescent protein, and truncated herpes simplex virus type 1 sr39 thymidine kinase) by us
271 as used for PET imaging of the reporter gene herpes simplex virus type-1 sr39 thymidine kinase (HSV1-
272 ation of the representative alphaherpesvirus herpes simplex virus type 1, the representative gammaher
273 ed and tested as reporter probes for imaging herpes simplex virus type 1 thymidine kinase (HSV1- tk)
274 nic HC-Ad driving constitutive expression of herpes simplex virus type 1 thymidine kinase (HSV1-TK) a
276 gene encoding the prodrug-converting enzyme herpes simplex virus type 1 thymidine kinase (HSV1-tk) i
278 -MDHT), which can be used for imaging of the herpes simplex virus type 1 thymidine kinase (HSV1-tk) r
279 ydroxymethyl]butyl)guanine ((18)F-FHBG), the herpes simplex virus type 1 thymidine kinase (HSV1-tk) v
280 n (hCEA) and with a fusion reporter gene for herpes simplex virus type 1 thymidine kinase (HSV1tk) an
281 ycytidine kinase double mutant (hdCKDM), and herpes simplex virus type 1 thymidine kinase (hsvTK) rep
283 egalovirus (CMV) promoter driving the mutant herpes simplex virus type 1 thymidine kinase reporter ge
284 ssion tomography (PET)-based split reporter (herpes simplex virus type 1 thymidine kinase), cleaved b
285 stably transfected with a mutant version of herpes simplex virus type 1 thymidine kinase, HSV1-sr39t
287 ceuticals ((18)F-FEAU) comparable to that of herpes simplex virus type-1 thymidine kinase (HSV1-tk)-t
288 gue as a probe for imaging the expression of herpes simplex virus type-1 thymidine kinase (HSV1-TK).
289 mography (PET) is used for the assessment of herpes simplex virus type-1 thymidine kinase gene expres
290 essing a PET reporter gene, the mutant viral herpes simplex virus type 1-thymidine kinase (HSV1-sr39t
291 Ads) carrying the conditional cytotoxic gene herpes simplex virus type 1-thymidine kinase (TK) induce
297 identification of charge cluster mutants of herpes simplex virus type 1 UL34 that localize properly
300 odds ratio for being in a higher tertile of herpes simplex virus type 1 was 1.63 (95% confidence int
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