ライフサイエンスコーパス: heterodimerization

1 eve high transporter stability and efficient heterodimerization.

2 ithin the TM domain is necessary for p45-p75 heterodimerization.

3 by bi-specific ligands that induce receptor heterodimerization.

4 the N terminus of MGAT2, is required for the heterodimerization.

5 e acetylation state of PAF49 does not affect heterodimerization.

6 R) domains of the receptors undergo homo- or heterodimerization.

7 retained in the cytosol upon wild-type DJ-1 heterodimerization.

8 ate cell survival and death through homo- or heterodimerization.

9 oint mutations in FGFR3 (A391E and G380R) on heterodimerization.

10 In Caco-2 cells, CCK enhanced CCK1R/CCK2R heterodimerization.

11 oGEFs as well as their co-recruitment due to heterodimerization.

12 the amount of ghrelin and state of receptor heterodimerization.

13 ing a Rac-1 construct via rapamycin-mediated heterodimerization.

14 ains that mediate either homodimerization or heterodimerization.

15 arrangements associated with the c-Fos/c-Jun heterodimerization.

16 s that are likely involved in their homo- or heterodimerization.

17 AK3 and PAK1 signaling may be coordinated by heterodimerization.

18 F49 were sufficient to provide the basis for heterodimerization.

19 lex and that they may serve to stabilize I-D heterodimerization.

20 and that blade I is required for CD44 MMP-14 heterodimerization.

21 cross-activation of GRPR signaling by MOR1D heterodimerization.

22 inactivating proteins with rapamycin-induced heterodimerization.

23 and PEG coimmobilized through leucine zipper heterodimerization.

24 clear localization of RARgamma through their heterodimerization.

25 lated HDAC1 can trans-regulate HDAC2 through heterodimerization.

26 of constitutive or monovalent ligand-induced heterodimerization.

27 gnaling by blocking ligand-induced HER2/HER3 heterodimerization.

28 f estrogenic ligands to promote ER homo- vs. heterodimerization.

29 gs appear to proceed through a non-templated heterodimerization.

30 ny biological processes mainly via homo- and heterodimerization.

31 oes not identify interactions that may favor heterodimerization.

32 e origin of stereoselectivity in this ketene heterodimerization.

33 s of the interactions inducing CH3 interface heterodimerization.

34 d, due to analytical challenges in measuring heterodimerization.

35 Toll-like receptor-1/2 (TLR-1/2) and TLR-2/6 heterodimerization.

36 mical properties, many times related to poor heterodimerization.

37 c was weakened, attenuating IL-2Rbeta-gammac heterodimerization.

38 tant domain Calpha is dependent on alphabeta heterodimerization.

39 deficiency, has on ZnT1, ZnT3, and ZnT4 upon heterodimerization.

40 D is a dimerization domain for PHYB homo and heterodimerization.

41 an interfacial cavity formed through subunit heterodimerization.

42 GIT1 to activate Ras and promote B-Raf/c-Raf heterodimerization.

43 responding to a segment that is required for heterodimerization.

44 elective small molecule inhibitor of MYC-MAX heterodimerization, 10058-F4, on myeloma cell lines as w

45 This disruption of heterodimerization allows the resulting monomers of p85

46 ted by these findings, we here uncovered the heterodimerization, altered subcellular localization, an

47 transformation and correlated with enhanced heterodimerization and activation of C-Raf.

48 ferences on the C-lobe surface influence the heterodimerization and activation of ErbB kinase domains

49 is of (-)-communesin F based on a late-stage heterodimerization and aminal exchange is described.

50 The functional significance of rod PDE6 heterodimerization and conserved differences between PDE

51 ary zone electrophoresis to measure rates of heterodimerization and DeltaGHet for seven ALS-variant a

52 ed TLR2-lipopeptide complex, it prevents TLR heterodimerization and downstream signaling.

53 an epidermal growth factor receptor 2 (HER2) heterodimerization and has single-agent activity in recu

54 f to CBFbeta is mutually exclusive with RUNX heterodimerization and impacts the expression of genes w

55 usion, sumoylation of Nrf2 and MafG enhances heterodimerization and increases GCLC expression, which

56 occur at multiple levels, including receptor heterodimerization and intracellular signaling.

57 /L as was nearly complete inhibition of HER3 heterodimerization and phosphorylation, thereby preventi

58 e-linked construct to induce IL-36R.IL-1RAcP heterodimerization and predicted the binding affinity du

59 ed to the strength of homodimerization, Sox2 heterodimerization and self-renewal activity.

60 ing to the increased potential for EGFR/HER2 heterodimerization and signaling.

61 We saw increased JAK2 heterodimerization and sustained JAK2 activation in cell

62 triggers hPer2 degradation, thus controlling heterodimerization and ultimately gene transcription.

63 hen a PKA-activated Ras promotes Raf-1/B-Raf heterodimerization, and are inhibited by interfering wit

64 embrane skeleton, suppression of C-RAF/B-RAF heterodimerization, and inhibition of C-RAF kinase activ

65 PtrSND1s, making them nonproductive through heterodimerization, and thereby modulating the SND1 tran

66 Par-6 and aPKC interact via PB1 domain heterodimerization, and this interaction activates aPKC

67 uR1 and mGluR5 in a manner inconsistent with heterodimerization, and thus suggest an interaction betw

68 istic insights on Ang2 antagonism, Tie1/Tie2 heterodimerization, and Tie2 clustering.

69 o-hole interface, the molecular mechanism of heterodimerization, and to engineer Fc domains that coul

70 domain 4 homodimerization and UvrB-domain 4 heterodimerization are allowed.

71 and functional roles of homodimerization and heterodimerization are still unclear.

72 imers of isolated subunits are inhibitory to heterodimerization as illustrated by a covalently linked

73 ependent MAPK signalling by antagonizing RAF heterodimerization as well as the conformational changes

74 was important to support efficient TonB-ExbD heterodimerization at these specific regions.

75 Heterodimerization between A and B allows coimmobilizati

76 Heterodimerization between ACS isoforms from distinct su

77 reactivation of JAK-STAT signalling and with heterodimerization between activated JAK2 and JAK1 or TY

78 te from a dominant-negative effect caused by heterodimerization between AQP4 and AQP4-Delta4, which w

79 Intermolecular FRET studies confirmed heterodimerization between PAR(2) and PAR(4).

80 Protein-protein interaction assays reveal heterodimerization between PpSMF1 and PpSCRM1, which, to

81 -terminal domain (NTD) of the GluN1 promotes heterodimerization between the NTDs of GluN1 and GluN2,

82 entiates the activation process by enhancing heterodimerization between the two proteins and vastly i

83 Our data argue against 2C4 as a universal heterodimerization blocker for p185(her2/neu), but indic

84 n of PDGFRA by EGFR and EGF-induced receptor heterodimerization, both of which are abolished by EGFR

85 uggests a possible structural basis for Her2 heterodimerization, but all available structures for dim

86 olecules designed to inhibit HIF-2alpha-ARNT heterodimerization by binding an internal cavity of the

87 s that allows the quantification of receptor heterodimerization by dual-color fluorescence cross-corr

88 main with an alpha-helical cap that mediates heterodimerization by forming an intermolecular helix bu

89 that the temporal regulation of Neurog2-E47 heterodimerization by GSK3 is a central component of the

90 ns are similar and compatible with Tie2/Tie1 heterodimerization by the same mechanism.

91 oreover, we find that events influencing Raf heterodimerization can alter the transforming potential

92 By heterodimerization, CCRL2 could regulate membrane expres

93 Heterodimerization creates V-shaped molecules with a hin

94 ion, pertuzumab-mediated inhibition of ErbB2 heterodimerization decreased AKT phosphorylation, cell g

95 a Nab3 self-association defect, a Nab3-Nrd1 heterodimerization defect, a Nrd1-polymerase II binding

96 The rate, mechanism, and DeltaG of heterodimerization (DeltaGHet) all remain undetermined,

97 Rates of heterodimerization did not correlate with survival time

98 Heterodimerization domain (HD) mutations in NOTCH1 induc

99 us substitutions affecting the extracellular heterodimerization domain (HD).

100 long intra-molecular coiled-coil arms with a heterodimerization domain at one end and an ABC-like nuc

101 it releases the autoinhibitory clamp on the heterodimerization domain imposed by the Lin12/Notch rep

102 NOTCH1 heterodimerization domain mutations were associated with

103 fibroblasts with mutations in the alpha/beta heterodimerization domain seems to be associated with a

104 ng mutations outside the alpha/beta spectrin heterodimerization domain, four had normal brain imaging

105 3 Lin12/Notch repeats and the juxtamembrane heterodimerization domain, the region of Notch1 most fre

106 constituent LNR (Lin12-Notch repeat) and HD (heterodimerization) domains, at forces similar to those

107 nts can be physiologically important through heterodimerization, even when inactive alone, and can co

108 erbB3 homo- and heterodimerization events were captured in real time on

109 Using a similar approach, ALX/FPR1 heterodimerization evoked using the panagonist peptide A

110 Despite the significance of TF heterodimerization for gene regulation, a quantitative u

111 omains required for actin-binding, homo- and heterodimerization, high-order chromatin organization, a

112 The structural basis for Her2 heterodimerization, however, remains poorly understood.

113 onditions, whether heterodimers form, or how heterodimerization impacts enzyme activity.

114 Braf35-iBraf heterodimerization impairs Braf35 interaction with the L

115 d in an inverted orientation or capture only heterodimerization in a single assay.

116 Yet heterodimerization in achondroplasia has not been charac

117 While methods exist for studying receptor heterodimerization in cell membranes, they are limited t

118 However, a role for opioid receptor heterodimerization in neurons has yet to be identified.

119 The in vivo significance of Mdm2-Mdm4 heterodimerization in regulation of p53 function is unkn

120 This suggests a role for NHERF2/NHERF3 heterodimerization in the regulation of NHE3 activity.

121 he physiological relevance of 5-HT2 receptor heterodimerization in vivo Accordingly, exogenous expres

122 t 2.47 A resolution, revealed a mechanism of heterodimerization in which UL50 clamps onto helices of

123 of the temperature and salt dependencies of heterodimerization indicate a modest unfavorable enthalp

124 age RNA homodimerization or to encourage RNA heterodimerization, indicating that HIV-1 and HIV-2 RNA

125 In addition, we demonstrate that heterodimerization influences the stability of ACS prote

126 HER2 antibody pertuzumab, which blocks HER2 heterodimerization, inhibited growth induced by hereguli

127 trengths and weaknesses of all of the PA-PB1 heterodimerization inhibitors, we analyze their hypothes

128 cellular sites within CaV1.2-alpha1C permits heterodimerization-initiated channel inhibition with rap

129 to surface residues in the characterized M50 heterodimerization interface substantially decreased UL5

130 ined asymmetric architecture, with extensive heterodimerization interfaces and AHR interdomain intera

131 Mechanistically, Mdm2-Mdm4 heterodimerization is critical for inhibiting lethal p53

132 cificity in receptor homodimerization versus heterodimerization is essential in determining the role

133 CCK2 receptors to determine whether receptor heterodimerization is involved in such modulation.

134 NHERF2/NHERF3 heterodimerization is mediated by PDZ domains of NHERF2

135 C- and E-class protein heterodimerization is predicted by the floral quartet mo

136 ilar levels of internalization, the role for heterodimerization is probably at the level of receptor

137 Mst1/Mst2 heterodimerization is strongly promoted by oncogenic H-r

138 FSHR heterodimerization is unique to the ovary because in the

139 dividual K8, K18, or K19 but is limited upon heterodimerization (K8/K18 or K8/K19) in the absence of

140 te that although both partners contribute to heterodimerization, ligand-bound ERalpha plays a dominan

141 ucines abolished protein disulfide isomerase heterodimerization, lipid transfer, and apoB secretion,

142 N. crassa and indicate that LOV-LOV homo- or heterodimerization may be a mechanism for regulating lig

143 of common target promoters, suggesting that heterodimerization may be required for the full regulati

144 dorsal root ganglion neurons indicating that heterodimerization may provide greater diversity of leak

145 regulation of MST1/2 includes both homo- and heterodimerization, mediated by helical SARAH domains, t

146 ults demonstrate that Pi-regulated PiT1-PiT2 heterodimerization mediates Pi sensing independently of

147 This study suggests that NHERF2/NHERF3 heterodimerization mediates the formation of NHE3 macroc

148 at substrate dimerization (homo- or possibly heterodimerization) might represent a general principle

149 ether, our results provide evidence that the heterodimerization of 6TM-MOR with beta2-AR underlies a

150 ure models suggest that homodimerization and heterodimerization of 7-transmembrane receptors regulate

151 The unique heterodimerization of a functional Nox4/p22(phox) Y121H

152 AM1 and AM2 receptors formed by the obligate heterodimerization of a G protein-coupled receptor, the

153 protease also cleaves the iPQM relevant for heterodimerization of a subgroup of neurotoxins.

154 hat controls ACS protein stability through a heterodimerization of ACS isoforms.

155 Recent development suggests that homo- and heterodimerization of APP and APP-like proteins (APLPs),

156 NPD1 also enhanced the heterodimerization of Bcl-x(L) with its counterpart, pro

157 Heterodimerization of both proteins with the NXT (NTF2-r

158 The Ca(2+)-dependent antiparallel heterodimerization of con-G and con-T(K7 gamma), two pep

159 Finally, we explored a possible heterodimerization of Cul3 and Cul5 and indeed discovere

160 The heterodimerization of EGFR with HER2 induces a more pote

161 These data demonstrate that heterodimerization of erbB2/erbB3 is a prerequisite for

162 d decrease sGCbeta1 H-NOXA homodimerization, heterodimerization of full-length heterodimeric sGC was

163 Heterodimerization of G protein-coupled receptors has an

164 al and can be applied to study the homo- and heterodimerization of GPCRs and other transmembrane prot

165 get protein is accomplished through specific heterodimerization of GR1 and GR2 adapters, followed by

166 Heterodimerization of human epidermal growth factor rece

167 hat mutations within either interface weaken heterodimerization of isolated half hinges in vitro but

168 Catalytic heterodimerization of ketenes can lead to important four

169 scribe an unprecedented catalytic asymmetric heterodimerization of ketenes of wide substrate scope.

170 xtensive studies of the catalytic asymmetric heterodimerization of ketenes to give ketene heterodimer

171 been shown to be extremely important for the heterodimerization of many TFIID subunits.

172 M2 and MDMX by inhibitor-driven homo- and/or heterodimerization of MDM2 and MDMX proteins.

173 Our results suggest a model by which heterodimerization of monoubiquitinated FANCD2 and FANCI

174 Heterodimerization of MOP and CCK2 receptors therefore i

175 Here we show that heterodimerization of muOR with delta opioid receptors (

176 he Max-Max homodimer and interferes with the heterodimerization of Myc and Max in a dose-dependent ma

177 iently varying oleate concentration; and 3), heterodimerization of Oaf1p and Pip2p does not appear to

178 of unsymmetrical cyclobutanes by controlled heterodimerization of olefins remains a substantial chal

179 trogen receptor and also did not affect homo/heterodimerization of p68/p72.

180 Consistent with the obligate heterodimerization of phyC and phyE, phyC is made unstab

181 Heterodimerization of RcsB with various auxiliary regula

182 pendent TGF-beta signaling by disrupting the heterodimerization of TbetaRI and TbetaRII receptors.

183 cubane 4Fe-4S cluster, which is crucial for heterodimerization of TFEalpha/beta and its engagement w

184 Heterodimerization of the alpha4 integrin with endogenou

185 ltiple Fc variant pairs that drive efficient heterodimerization of the antibody heavy chains.

186 restricted to the receptor itself, allowing heterodimerization of the four EGFR family members witho

187 ligand-mediated co-internalization following heterodimerization of the GHS-R1a receptor with the dopa

188 t the first demonstration of agonist-induced heterodimerization of the H1R and H2R.

189 tes lymphocyte function by signaling through heterodimerization of the IL-2Rbeta and gammac receptor

190 Binding of CHOP prevented heterodimerization of the receptor subunits GABAB1 and G

191 To evaluate potential heterodimerization of the receptors, sensitized emission

192 The interaction is also required for proper heterodimerization of the transporter.

193 of enhancing procaspase-8 activation through heterodimerization of their respective protease domains.

194 of the NC, ND and NE sequences and directed heterodimerization of these photosensory regions with th

195 ltaneous measurement of homodimerization and heterodimerization of type I receptor domains in their n

196 To study the consequences of homo- and heterodimerization of wild-type and mutant PTEN in vivo,

197 This likely resulted from the heterodimerization of wild-type and mutant TRESK subunit

198 Efficient [2+2] heterodimerizations of dissimilar acyclic enones can be

199 Although homo- and heterodimerizations of G protein-coupled receptors (GPCR

200 gth among all possible homodimerizations and heterodimerizations of these three NHERF proteins by pul

201 identify the unique determinants that favor heterodimerization over procaspase-8 homodimerization, a

202 AHR and its heterodimerization partner aryl hydrocarbon receptor nuc

203 )) and the marked-box domains of E2F and its heterodimerization partner DP.

204 enic mutants, is critically dependent on its heterodimerization partner ErbB-3, and surprisingly, doe

205 ncur with RXRalpha functioning as obligatory heterodimerization partner for several nuclear receptors

206 determination in a manner beyond that of its heterodimerization partner MEIS2.

207 ntral MHC class III region, and its obligate heterodimerization partner Msh4 have a critical role in

208 on of Notch-1, ErbB4, pErbB4, and pEGFR, the heterodimerization partner of ErbB4, suggesting increase

209 targeted via bexarotene, a ligand of Nurr1's heterodimerization partner retinoid X receptor (RXR).

210 as >100-fold lower in vitro affinity for its heterodimerization partner, hypoxia-inducible factor 2al

211 sponse element (XRE) in association with the heterodimerization partner, the AhR nuclear translocator

212 the C. elegans homolog of DP, the human E2F-heterodimerization partner.

213 taining loosely organized domains, while its heterodimerization partners use a surface patch on their

214 Ligand binding, heterodimerization, phosphorylation of ERBB3, and AKT si

215 and tetrasubstituted cyclobutanes through a heterodimerization process involving two different alken

216 e map dimerization determinants and define a heterodimerization profile.

217 Thus, heterodimerization provides a means for diversifying fun

218 ny TALE either using effector molecules or a heterodimerization reaction.

219 complexes, the specificity and mechanism of heterodimerization remain unclear.

220 tatic interactions that facilitate efficient heterodimerization, resulting in bispecific antibodies w

221 Furthermore, KSR2-BRAF heterodimerization results in an increase of BRAF-induce

222 r receptor Nurr1 can be activated by RXR via heterodimerization (RXR-Nurr1) and is a promising target

223 engineered is by creating "knob" and "hole" heterodimerization sites in the CH3 domains of two antib

224 Structural determinants responsible for heterodimerization specificity of bZIP53 are poorly unde

225 tions that modulate the homodimerization and heterodimerization states to define additional roles of

226 d DAF-38 form heterodimers, and we show that heterodimerization strongly increases cAMP inhibition in

227 Here, we show that the protein heterodimerization switches FKBP-rapalog-FRB can be harn

228 We use a small molecule-regulated heterodimerization system to rapidly control the localiz

229 ted by recruitment of a 4-phosphatase with a heterodimerization system.

230 ibitor GRK2ct (GRK2ct-KERE) and the FRB/FKBP heterodimerization system.

231 or a variety of regulatory possibilities via heterodimerization that could impact song behavior in ze

232 f amino acid substitutions that restored (i) heterodimerization to a noninteracting FosLZ variant, an

233 of Mycobacterium tuberculosis induces TLR1/2 heterodimerization to elicit proinflammatory-type respon

234 successfully drive human IgG1 CH3 or IgM CH4 heterodimerization to levels similar to or above those o

235 inflammation and suggest a new model of TLR heterodimerization triggered by coreceptor signaling eve

236 the molecular basis of TLR1 and TLR6-driven heterodimerization upon LPA binding underlines the highl

237 Light-induced heterodimerization using the phytochrome system has prev

238 ated with an increased receptor binding, and heterodimerization was associated with a decreased recep

239 In vivo NHERF2/NHERF3 heterodimerization was confirmed by FRET and FRAP (fluor

240 ligation assays demonstrated that BRAF-CRAF heterodimerization was increased in fixed tumor samples

241 were substituted with alanine, the PiT1-PiT2 heterodimerization was no longer regulated by extracellu

242 pendent Pi transport activity, the PiT1-PiT2 heterodimerization was still regulated by extracellular

243 ent subunits were linked together to enforce heterodimerization), we characterized the biophysical an

244 growth factor receptor (EGFR)-p185(her2/neu) heterodimerization, we noted that 2C4 formed association

245 pical DR1 sequence favors PPARalpha/RXRalpha heterodimerization, whereas the switch from RXRalpha to

246 y interacts with VDR and RXR promoting their heterodimerization, which is critical for VDR:RXR target

247 the cytostatic activity of C/EBPbeta through heterodimerization, which prevents senescence and suppre

248 Although heterodimerization with 5-HT2C receptors does not alter

249 1 cytoplasmic tail and does not entail beta1 heterodimerization with an alpha-subunit or its localiza

250 tidylinositol 3-kinase/Akt signaling through heterodimerization with and activation by other ErbB rec

251 A dimerization leads us to propose a mode of heterodimerization with ARNT that is supported by both b

252 f Bcl-xL in mitochondria, increase in Bcl-xL heterodimerization with Bax in mitochondria, and reduced

253 lation of the alpha3 precursor prevented its heterodimerization with beta1, whereas CD151 association

254 to wild-type B-Raf kinase domain leading to heterodimerization with C-Raf causing a paradoxical hype

255 hibits binding to activated Ras and disrupts heterodimerization with C-Raf, which is dependent on the

256 eased C/EBPbeta homodimer formation, whereas heterodimerization with C/EBPgamma was relatively unaffe

257 stitutions into the Runt domain that disrupt heterodimerization with CBFbeta but not DNA binding.

258 merization, whereas blade I was required for heterodimerization with CD44.

259 Parp9 undergoes heterodimerization with Dtx3L, a histone E3 ligase invol

260 or via a ligand-independent process through heterodimerization with ErbB2 overexpressed in breast tu

261 s the dynamics of ErbB3 homodimerization and heterodimerization with ErbB2.

262 activity of FHY3 and its homodimerization or heterodimerization with FAR1.

263 mediating homodimerization and required for heterodimerization with Fbxo7.Skp1.

264 een shown to acquire membrane association by heterodimerization with GAD65.

265 Although heterodimerization with Gdf1 did not increase binding of

266 hese interactions appeared to stabilize HER2 heterodimerization with HER3 and induced receptor activa

267 ickel-liposomes, and is increased further by heterodimerization with Her3 or Her4.

268 lls with NO increased the level of ITGalpha6 heterodimerization with ITGbeta1 but not with ITGbeta4.

269 tivates the receptor kinase BRI1 by inducing heterodimerization with its co-receptor kinase BAK1; how

270 riptional activity by competing with Myc for heterodimerization with its obligatory partner, Max.

271 inal domain of PRG, which is responsible for heterodimerization with LARG, strongly inhibited Ca(2+)-

272 MO-1, which promoted Nrf2 binding to ARE and heterodimerization with MafG.

273 n signals and the bHLHZ domain essential for heterodimerization with Max and DNA binding.

274 is responsible for ubiquitination of p53 and heterodimerization with MDM4.

275 d cell entry, nectin-4 homodimerization, and heterodimerization with nectin-1.

276 The NHERF3-4A mutant is defective in heterodimerization with NHERF2 and does not support the

277 the nuclear portion was inactive because of heterodimerization with NPM1.

278 "knob-into-hole" technology for heavy chain heterodimerization with one heavy chain consisting of a

279 y identified regions in Ostbeta required for heterodimerization with Ostalpha, trafficking of the Ost

280 ot bind EGF-like ligands, relying instead on heterodimerization with other (ligand-bound) EGFR-family

281 essential for protein stability and possible heterodimerization with other isoforms of RGP.

282 Transcriptional activation by CHOP involves heterodimerization with other members of the basic leuci

283 centrations, presumably by disruption of RXR heterodimerization with other nuclear receptors.

284 ons of CXCR4 dimerization and studying CXCR4 heterodimerization with other receptors.

285 at mediate their homodimerization as well as heterodimerization with other SARAH domain-containing pr

286 competent itself as it requires aPC-induced heterodimerization with PAR-2 (human podocytes) or PAR-1

287 is controlled by the activating protease and heterodimerization with PAR2 or PAR3.

288 Dtx3L heterodimerization with Parp9 enables NAD(+) and poly(AD

289 Heterodimerization with prozyme displaces this sequence

290 Heterodimerization with R2 reduced the rate of internali

291 hrough homodimerization of MMP-14 as well as heterodimerization with the cell surface adhesion molecu

292 L1 in both proteins mediates DNA binding and heterodimerization with the core binding factor beta (CB

293 llowed by SARAH domains, which mediate Rassf heterodimerization with the Mst1/2 protein kinases.

294 PIF1 is also degraded in the dark by direct heterodimerization with the positively acting factor HFR

295 nding to PPARgamma response elements and for heterodimerization with the retinoid X receptors.

296 LR expressed on human monocytes, by inducing heterodimerization with TLR1 in an NADPH oxidase-depende

297 of UL53 crucial for homodimerization and for heterodimerization with UL50, we constructed and express

298 Its location in the nucleus and heterodimerization with WC-2, together with the presence

299 Furthermore, upon heterodimerization with ZnT1, the zinc transporters ZnT2

300 ne phosphorylation and ErbB3 and p185(c-neu) heterodimerization, with subsequent activation of intrac