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1                          In the absence of a heterooligomeric adaptor complex composed of FCHO, Eps15
2   6-Phosphofructokinases (Pfk) are homo- and heterooligomeric, allosteric enzymes that catalyze one o
3 re illustrated by separating components from heterooligomeric assemblies formed between tetrameric tr
4 that cofactors and Torsins associate to form heterooligomeric assemblies with a defined Torsin-activa
5  initial signals for both homooligomeric and heterooligomeric assembly into receptors with GABAC prop
6 g Kvbeta subunits, our findings suggest that heterooligomeric assembly of these subunits occurs to in
7 he presence of an intermediate population of heterooligomeric channels consisting of Kvalpha1.2 with
8 related, mechanism(s); namely, "locking" the heterooligomeric channels in their closed state.
9 ant role(s) in modulation of the kinetics of heterooligomeric channels.
10 l point interact with BamA, a component of a heterooligomeric complex (Bam complex) that catalyzes OM
11 chemical analyses revealed that the PTP is a heterooligomeric complex composed of VDAC, SPG7, and Cyp
12                              We analyzed the heterooligomeric complex of LmClpP1 and LmClpP2 via coex
13 al domain can function normally within the P heterooligomeric complex to carry out transcription and
14 light the functional importance of Mfn1-Mfn2 heterooligomeric complexes and the close interplay betwe
15                           NMDA receptors are heterooligomeric complexes comprised of both NR1 and NR2
16 )beta subunits form multimolecular homo- and heterooligomeric complexes in human vascular smooth musc
17 fn1, but the functional significance of such heterooligomeric complexes is unknown.
18 with this hypothesis, Pop1p and Pop2p formed heterooligomeric complexes when overexpressed, and bindi
19 becomes the dominant HSF and is able to form heterooligomeric complexes with HSFA1.
20         SEP3 has been shown to form homo and heterooligomeric complexes with other MADS domain transc
21 lements mutant Mfn2 through the formation of heterooligomeric complexes, including complexes that for
22 proteins that assemble into symmetric linear heterooligomeric complexes, which in turn are able to po
23 on in vivo, notwithstanding the formation of heterooligomeric complexes.
24 nnels, kinetic properties of single homo and heterooligomeric ENaCs formed by the subunits with indiv
25  kinetic behavior of both homooligomeric and heterooligomeric ENaCs, although the carboxy-terminal do
26 sitizing component, even faster than that of heterooligomeric GABA(A) receptors, in striking contrast
27 hether the same region contained signals for heterooligomeric interaction with rho2 subunits.
28 netic resonance (EPR) to probe the homo- and heterooligomeric interactions of reconstituted sarcoplas
29                          GABAA receptors are heterooligomeric ligand-gated anion channels that exhibi
30 americ module, eight of which form a single, heterooligomeric midplane ring, which is flexible in a d
31 vity in a heterogeneous protein mixture or a heterooligomeric protein (20), or determining if multipl
32  isolation, but together form a thermostable heterooligomeric Rca that can use both alpha-proteobacte
33  be regulated by factor-binding-induced homo/heterooligomeric restructuring, paving the way to cell m
34                           The flagellum is a heterooligomeric structure that protrudes from the surfa
35 tive phenotype results from the formation of heterooligomeric VacA complexes with defective functiona

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