ライフサイエンスコーパス: heterophil

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1 with the initial influx of chMMP-9-positive heterophils.

2 ripheral blood mononuclear cells but not for heterophils.

3 ronger, up-regulated gene expression of line heterophils A than line B, and these genes include sever

4 in vivo a further infiltration of endogenous heterophils and monocytes and dramatically rescued the i

5 properties; transferases cloned from chicken heterophils and red cells have signal peptides and may b

6 To dissect the interplay between heterophils and SE infection, we utilized large-scale ge

7 trated in vascular endothelium, macrophages, heterophils, and cardiac myocytes.

8 mplants of activated fibroblasts, monocytes, heterophils, and endothelial cells.

9 phil-like inflammatory cells, namely chicken heterophils, and the development of new blood vessels.

10 This early heterophil arrival was followed by the infiltration of m

11 us stents induced significantly more luminal heterophils/eosinophils and fibrin deposition than Cyphe

12 greater fibrin deposition, medial cell loss, heterophils/eosinophils, and late neointimal hyperplasia

13 Large numbers of heterophils, equivalent to neutrophils in mammals, and i

14 Moreover, purified heterophils incorporated into onplants lacking growth fa

15 The exogenously added heterophils induced in vivo a further infiltration of en

16 Previously, heterophils isolated from broilers with different geneti

17 In vivo, a decreased heterophil (neutrophil-like cell) migration was observed

18 d the MIL4 Ab, which detects both guinea pig heterophils (neutrophils) and eosinophils, to provide th

19 A direct addition of isolated heterophils or purified chMMP-9 into the HT-1080 onplant

20 se of the rabbit skeletal muscle and chicken heterophil, respectively.

21 Birds with microfilariae had elevated heterophil to lymphocyte ratios and were all co-infected

22 atinase B (chMMP-9) as a specific marker for heterophils, we now show that the onset and extent of an

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