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1 ess advantages of between-population mating (heterosis).
2 development and may be a major component of heterosis.
3 regions may contribute disproportionally to heterosis.
4 ion in the hybrid and the molecular basis of heterosis.
5 o both parental strains, suggesting positive heterosis.
6 o speculate on mechanisms that might lead to heterosis.
7 cal dominant or overdominant explanations of heterosis.
8 of protein metabolism plays a role in growth heterosis.
9 ons of this work toward our understanding of heterosis.
10 isms, including overdominance, contribute to heterosis.
11 traits including egg-to-adult viability via heterosis.
12 s in the hybrid, which may have an impact on heterosis.
13 rtant role in the generation of the observed heterosis.
14 us showed a significant decrease in apparent heterosis.
15 ight at flowering was the only trait to show heterosis.
16 uantitative trait loci and investigations of heterosis.
17 na, according to the dominance hypothesis of heterosis.
18 re that overdominance does not contribute to heterosis.
19 ly related species, and the genetic basis of heterosis.
20 etic changes are a cause or a consequence of heterosis.
21 us than their parents, a phenomenon known as heterosis.
22 n-level dominance plays an important role in heterosis.
23 ibution of dosage to quantitative traits and heterosis.
24 s line that could contribute to the observed heterosis.
25 ution of parental methylation differences to heterosis.
26 mal productivity through the manipulation of heterosis.
27 lism, thus modulating biomass and leading to heterosis.
28 ay contribute to its high degree of observed heterosis.
29 he sizes of the ears are diagnostic of yield heterosis.
30 ses, and, in plants, may also play a role in heterosis.
31 intensities enhanced both photosynthesis and heterosis.
32 in bioenergetic processes may contribute to heterosis.
33 allelic and/or isoform differences linked to heterosis.
34 fic proteins correlate with higher levels of heterosis.
35 at consistently exhibited either low or high heterosis across a variety of environments were examined
41 lluscs, related phenomena, marker-associated heterosis and distortion of marker segregation ratios, h
43 ting (over)dominance as the genetic cause of heterosis and estimating the (over)dominance coefficient
45 ad strongly supports the dominance theory of heterosis and inbreeding depression and establishes the
47 ed to explore the molecular genetic basis of heterosis and outbreeding depression, and how their magn
49 or maintenance of genetic diversity - social heterosis and social genomes - can similarly explain the
51 at an inhibitor of photosynthesis eliminated heterosis and that higher light intensities enhanced bot
55 among populations and compared mean fitness, heterosis, and inbreeding depression for eight large and
56 wer and repeatability of allozyme-associated heterosis, and that the allozyme-associated heterosis de
58 put forward to explain the genetic basis of heterosis are the general dominance and the local overdo
59 mic and epigenetic perspectives suggest that heterosis arises from allelic interactions between paren
60 reproductive yield as cumulative outcomes of heterosis at different levels, tissues, and times of dev
61 isease with an earlier onset indicating that heterosis at Esr2 plays a significant role in regulating
65 he molecular mechanisms of hybrids vigor (or heterosis) between Dura, Pisifera and their hybrid proge
66 ene regulation, quantitative trait loci, and heterosis, but one that is not easily applied to sexuall
67 herefore, the (over)dominance hypothesis for heterosis can be tested by estimating h, under either do
69 ing a robust hybridization platform in wheat.Heterosis can rapidly boost yield in crop species but de
70 cepticism that surrounds allozyme-associated heterosis comes from inconsistent and unreliable detecti
72 heterosis, and that the allozyme-associated heterosis detected in this study was the result of gener
73 Different mechanisms have been proposed for heterosis: dominance, overdominance, epistasis, epigenet
74 tern and epigenetic mechanisms contribute to heterosis during early flower development in allopolyplo
83 herited in autosomal fashion; the absence of heterosis for male fertility among the MA lines was ther
87 nt an example of discovery and validation of heterosis generated by a combination of repulsion linkag
91 However, previous theoretical studies on heterosis have been based on bi-parental segregating pop
93 distance between hybridizing parents affects heterosis; however, the mechanisms for this remain uncle
94 ple, the genetic and physiological causes of heterosis (hybrid vigor) have remained elusive for nearl
95 re consistent with the well-known effects of heterosis (hybrid vigour) described when outcrossing ani
97 Large increases in biomass and yield in high-heterosis hybrids suggest that alterations in bioenerget
99 es between natural populations can result in heterosis if recessive or nearly recessive deleterious m
100 ther, our data suggest that the magnitude of heterosis in A. suecica is environmentally regulated, ar
101 tal lines can directly or indirectly trigger heterosis in Arabidopsis hybrids independent of genetic
102 pite the importance and wide exploitation of heterosis in commercial crop breeding, the molecular mec
103 e of favorable genes to explain the observed heterosis in grain yield and other traits, although epis
105 s been shown to affect metabolic and biomass heterosis in interspecific hybrids or allotetraploids.
106 echanisms that may cause allozyme-associated heterosis in natural populations has proven difficult.
109 eveloping hybrids with greater expression of heterosis in productivity and concentrations of provitam
113 aving repulsion linkage between two inbreds, heterosis in the hybrid can appear as a single locus wit
114 e gene-expression patterns underlying growth heterosis in the Pacific oyster (Crassostrea gigas) in t
116 It may be that this example of molecular heterosis in vitro provides the basis for maintenance of
117 Arabidopsis thaliana shows hybrid vigor (heterosis) in progeny of crosses between Columbia-0 and
120 Various models have been posited to explain heterosis, including dominance, overdominance, and pseud
123 this study suggest that a major component of heterosis is a mechanism that is modulated by dosage-sen
132 effect was an increase in apparent allozyme heterosis later in ontogeny coinciding with a series of
133 ds included in this study exhibit a range of heterosis levels; however, we did not observe difference
135 gulation, indicating that expression GxE and heterosis may result from the evolution of transcription
136 ctions corresponding to the evolutionary and heterosis mechanisms, asking whether any effects of gene
138 nalysis of the congenic lines argues against heterosis of outbred backgrounds contributing to Egfrtm1
139 crosatellite markers and immediate recovery (heterosis) of egg viability and flight metabolic rate in
141 ng depression and the converse phenomenon of heterosis or hybrid vigor remain poorly understood despi
144 ynthesis, we describe a case of single-locus heterosis, or overdominance, where the heterozygote disp
145 es identifying a specific genetic example of heterosis, our research indicated that integrated molecu
146 addition to genetic factors contributing to heterosis, our results strongly suggest that epigenetic
147 enotype, revealing effects of strain dosage, heterosis, parent of origin, epistasis, and sex-specific
155 hybrids differed in the level of high-parent heterosis relative to the derived triploid inbreds.
157 lating level of heterozygosity and degree of heterosis should take into account this nonuniform distr
158 e correlations are viewed as a phenomenon of heterosis, so that it cannot possibly occur under within
159 As such, modes of inheritance that drive heterosis, such as dominance or overdominance, may be co
160 approximately 350 candidate genes for growth heterosis that exhibit concordant nonadditive expression
161 nd Hardy-Weinberg analysis suggested partial heterosis, that is, an increased risk for heterozygotes,
163 inance vs overdominance as an explanation of heterosis; the classical vs balance hypothesis for genet
164 nes directly contribute to, or merely mimic, heterosis, they may aid generation of more vigorous and
165 :1) additive loci, dominant contributions to heterosis to outnumber overdominant, and extensive pleio
166 ing the early developmental manifestation of heterosis under fluctuating environmental conditions in
167 ssed the possible epigenetic contribution to heterosis using epigenetic inbred lines (epiRILs) with v
168 effect; 2) the ranking of factors affecting heterosis was dominance > dominance-by-dominance > over-
169 mplementation hypothesis in a direct manner, heterosis was examined in diploid inbreds and reciprocal
170 fitness was 68% lower in small populations; heterosis was significantly greater for small (mean = 70
171 ensive model to explain the phenomenology of heterosis, we provide the details of what needs to be ex
172 lant size and cell number are reminiscent of heterosis, which also increases plant size primarily thr
173 ci showed a significant increase in apparent heterosis with ontogeny, while one locus showed a signif
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