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1 alues of both positive and negative dP/dt in heterozygous mutants.
2 bosis response was severely reduced for both heterozygous mutants.
3 e dental epithelium and mesenchyme of double heterozygous mutants.
4 cient to obtain gene conversion in initially heterozygous mutants.
5 ignificantly reduced in Clock homozygous and heterozygous mutants.
6  that was no more severe than that of single heterozygous mutants.
7  of wild-type alpha1beta2gamma2 receptors or heterozygous mutant alpha1(A322D)beta2gamma2 receptors,
8 rafficking and/or accelerated endocytosis of heterozygous mutant alpha1beta2gamma2 receptors containi
9                                              Heterozygous mutants also exhibited delayed recovery of
10 cartilage from the knee joints of the double-heterozygous mutant and surgically treated mice were exa
11  copy numbers in ovulated oocytes from EndoG heterozygous mutant and wild-type mice are similar, sugg
12 e strain but decreasing catalase activity in heterozygous mutants and no detectable catalase in a hom
13                                      We used heterozygous mutants and targeted RNA interference-media
14 between those persons who were homozygous or heterozygous mutants and wildtype/pseudo-wildtype (p = 0
15 e 73.3% for p53 wild-type fetuses, 52.5% for heterozygous mutants, and 2.2% for p53-null mutants.
16 our onset, incidence and progression in Pten-heterozygous mutants, and leads to female sterility with
17                                           In heterozygous mutant animals, we observed a relative decr
18           We also show that Xpc Trp53 double heterozygous mutants are more predisposed to skin cancer
19                      Prior to seizure onset, heterozygous mutants are not defective in motor learning
20 enes expressed during wing development in 27 heterozygous mutants, as well as their co-isogenic wild
21                       Here we show that aged heterozygous mutant (Atp2a2(+/-)) mice develop squamous
22 ables instant germline modifications, making heterozygous mutants available within 18 wk.
23 res from E12.5 homozygous mutant (bcl-x-/-), heterozygous mutant (bcl-x+/-), and wild-type (bcl-x+/+)
24 one morphogenetic protein receptor-2 (BMPR2)-heterozygous, mutant (BMPR2(+/-)) mice have a genetic tr
25                           In contrast to all heterozygous mutants, both systolic and diastolic functi
26                                              Heterozygous mutant BRCA1 cell clones also showed a high
27                       We found that even the heterozygous mutant cell lines showed cell survival defe
28                            We show that Apex heterozygous mutant cells and animals are abnormally sen
29                                        While heterozygous mutant cells had diminished signaling in re
30                     When compared with Ptch1 heterozygous mutants, compound Ptch1/Hic1 heterozygotes
31 nctionally independent domains, we undertook heterozygous mutant crossing between Phr1(Delta8,9) and
32                       Interestingly, the MK2 heterozygous mutants display an intermediate level of pr
33                                Ventricles of heterozygous mutants displayed a 50% reduction in mlc-2v
34                              In mouse, Hnf1b heterozygous mutants do not exhibit any phenotype, where
35  Myocardial hypoplasia in GATA4/GATA6 double heterozygous mutant embryos is associated with reduced p
36  finding of exencephaly in about 15% of rybp heterozygous mutant embryos, and by Rybp's distinct neur
37  misexpressed in engrailed cells in hedgehog heterozygous mutant embryos, larvae show a dominant phen
38                                    Using p53 heterozygous mutant epithelial cells from Li-Fraumeni sy
39                                 Although the heterozygous mutant ES cells were able to generate low c
40                                              Heterozygous mutants exhibited cardiac function comparab
41                                Finally, BIR1 heterozygous mutants exhibited grossly altered cell morp
42                       Compound Cdc42, tinman heterozygous mutant flies exhibited impaired cardiac out
43 phenotypic characterization of mice that are heterozygous mutants for the Apex gene (Apex+/-).
44                                              Heterozygous mutants (Foxc1(+/-) and NC-Foxc1(+/-)) exhi
45 s knockout (SR-/-) and glycine transporter 1 heterozygous mutant (GlyT1-/+).
46 coding PMCA1 caused embryolethality, whereas heterozygous mutants had no overt disease phenotype.
47                                          The heterozygous mutant has no overt phenotype, and its leaf
48                                              Heterozygous mutants have 67% of the enzyme activity of
49                                              Heterozygous mutants have axonal defects in the adult ey
50 hus, heterozygous and homozygous or compound heterozygous mutants have very different clinical phenot
51 t of WT KRAS with a mutant allele sensitized heterozygous mutant HCT116 cells to treatment.
52 CA2 mRNA was reduced by approximately 45% in heterozygous mutant hearts and that SERCA2 protein and m
53 crog/L; 24 had homozygous mutant or compound heterozygous mutant HFE genotypes.
54 can affect muscle function, we have analyzed heterozygous mutants in depth.
55 f chondrogenesis and osteogenesis, while the heterozygous mutant is dwarfed.
56 mal gene can be disrupted, and the resulting heterozygous mutant is unlikely to display a phenotype.
57 s, the phenotype of the lymph gland of Zfpr8 heterozygous mutants is dominantly enhanced by the l(1)d
58  ciliary ganglion and we find that in Phox2b heterozygous mutants it is highly atrophic.
59                       By contrast, kinesin-8 heterozygous mutant (KIP3/kip3) spindles exhibited the s
60                                              Heterozygous mutant Kit(V558Delta)/+ mice that develop s
61                                              Heterozygous mutant KitV558Delta/+ mice develop symptoms
62  comparison with wild-type alphaA protein of heterozygous mutant lenses.
63                                          The heterozygous mutant (LPAT2/lpat2) had minimal altered ve
64                                              Heterozygous mutant (luxoid.Zbtb16(LU)/J) mice deficient
65                                          The heterozygous mutant mice (Acc1(+/-)) had normal fertilit
66 e phenotypes in growth plate chondrocytes of heterozygous mutant mice (Gnas(+/E2-)).
67                                              Heterozygous mutant mice (ngf+/-) have reduced levels of
68                                          The heterozygous mutant mice also developed similar prostati
69 in the mammalian heart, we examined compound heterozygous mutant mice and found conduction system and
70                 The abnormal EEG patterns in heterozygous mutant mice and the influence of genetic ba
71                                        Snf2h heterozygous mutant mice are born at the expected freque
72             In addition, both homozygous and heterozygous mutant mice are highly resistant to the sei
73 oints was dramatically reduced in the double-heterozygous mutant mice compared with that in the type
74 nally, we propose that soluble KITL in Sld/+ heterozygous mutant mice complements a dosage effect of
75                          We report here that heterozygous mutant mice develop into adulthood without
76                          We report here that heterozygous mutant mice develop into adulthood without
77                                          The heterozygous mutant mice display insulin hypersensitivit
78                                          The heterozygous mutant mice do not develop seizures.
79                                     The Chd2 heterozygous mutant mice exhibit increased extramedullar
80 nterestingly, we found that cells from EndoG heterozygous mutant mice exhibit increased resistance to
81 ectrophysiological studies demonstrated that heterozygous mutant mice have a specific defect in hippo
82 opment, we generated, through breeding, IRF4 heterozygous mutant mice in the NZB background (NZB IRF4
83 taneous cell death of spermatogonia in EndoG heterozygous mutant mice is significantly reduced compar
84 ed an F2 intercross between insulin receptor heterozygous mutant mice on B6 and 129/Sv backgrounds (B
85                         However, breeding of heterozygous mutant mice on the 129 background generated
86                         Closer inspection of heterozygous mutant mice revealed a range of defects wit
87                                              Heterozygous mutant mice show a phenotype similar to hum
88                 Fibroblasts taken from adult heterozygous mutant mice show an apparent alteration in
89 scular endothelial cells isolated from Foxc2 heterozygous mutant mice showed a marked reduction in It
90                                              Heterozygous mutant mice showed postnatal growth delay w
91                       The resulting compound heterozygous mutant mice showed significantly accelerate
92                The progression in the double-heterozygous mutant mice was delayed by approximately 6
93 , microvessel outgrowth from aortas of Foxc2 heterozygous mutant mice was reduced.
94                                              Heterozygous mutant mice were viable, but embryos exhibi
95                                        Adult heterozygous mutant mice with a targeted disruption for
96                                              Heterozygous mutant mice with deletion of hypoxia-respon
97                                    Mating of heterozygous mutant mice yielded wild-type and heterozyg
98 f osteoclasts were not altered in the double heterozygous mutant mice, indicating that the defect lie
99 chanism of MHE, both Ext1(+/-) and Ext2(+/-) heterozygous mutant mice, which mimic the genetic status
100 ciency also accelerated tumorigenesis in p53 heterozygous mutant mice.
101 ter behavioral recovery than in wild-type or heterozygous mutant mice.
102 f the Steel locus increase TGCT incidence in heterozygous mutant mice.
103 omal breaks as well as aneuploidy in primary heterozygous mutant mouse embryonic fibroblasts.
104                                Analysis of a heterozygous mutant mouse in which the NF-kappaB-depende
105 which manifests as visual dysfunction in the heterozygous mutant mouse, B6;C3-Opa1(Q285STOP).
106 , a finding relevant to our understanding of heterozygous mutant myosins found in disease states like
107 ablated in Schwann cells in the context of a heterozygous mutant (Nf1+/-) microenvironment.
108 ablated in Schwann cells in the context of a heterozygous mutant (Nf1+/-) microenvironment.
109 trocytes from NKCC1 wild-type (NKCC1+/+) and heterozygous mutant (NKCC1+/-) mice.
110          When the mutations were examined in heterozygous mutant/nonmutant genotypes, more than half
111 the activity of the wild-type transposase in heterozygous mutant/nonmutant genotypes.
112 ary phase survival was prolonged in a double heterozygous mutant of the metabolic enzyme non-quiescen
113 ductive tracts of Hoxa-10 and Hoxa-11 single heterozygous mutants of both sexes were primarily unaffe
114 rease in deposition of AA when compared with heterozygous mutant or wild-type animals.
115 ic mice bearing both the v-Ha-ras gene and a heterozygous mutant p53 allele tend to retain the Ink4a
116 cted with an apparent dominant ZSD in whom a heterozygous mutant PEX6 allele (c.2578C>T [p.Arg860Trp]
117                                              Heterozygous mutant plants showed no visible leaf phenot
118                                 Selfing of a heterozygous mutant produced normal-sized and shrunken s
119                            We found that pnr heterozygous mutants result in defective cardiac perform
120 ient for the Bloom's syndrome helicase, such heterozygous mutants segregate homozygous daughter cells
121 e extreme fertility observed in the compound heterozygous mutant sheep.
122                                              Heterozygous mutants show a slow onset of degeneration i
123                                 In contrast, heterozygous mutants show an exaggerated escape response
124                             We conclude that heterozygous mutants show neither a molecular nor a phys
125                                          The heterozygous mutants showed slower growth kinetics and e
126 ice had no detectable CuZn-SOD activity, and heterozygous mutants (Sod1 +/-) showed a 50% decrease co
127  further suggestive of selection against the heterozygous mutant state as tumors progress.
128 We obtained homozygous cells from 40% of the heterozygous mutants tested.
129                          Moreover, in double heterozygous mutants, the lower incisors were consistent
130 00(DeltaKASH), or in klar and msp-300 double heterozygous mutants, the MSP-300 nuclear ring and the a
131                                              Heterozygous mutant thymocytes had a severe defect in p5
132 nctionally complemented lpat2 in transformed heterozygous mutants to produce the lpat2/lpat2 genotype
133                         We have grown > 6000 heterozygous mutants together and exposed them to a star
134                      By flow cytometry, some heterozygous mutants were polyploid accumulating >4 N DN
135                               Notch2(Q2319X) heterozygous mutants were smaller and had shorter femurs
136 ty could be rescued by transformation of the heterozygous mutant with a 35S:LPAAT1 construct.
137                                 In ephrin-B1 heterozygous mutants, X inactivation generates ephrin-B1
138                                  Breeding of heterozygous mutants yielded a normal Mendelian ratio am

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