ライフサイエンスコーパス: high BP

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1 ere periodontitis, hypertension history, and high BP.

2 tension, poorly controlled hypertension, and high BP levels are associated with a decline in pCBF.

3 with increased events rates at both low and high BP values, both unadjusted and after adjustment for

4 important in reducing adulthood obesity and high-BP risk.

5 se-response effect for the length of time at high BP levels.

6 ge from 1999-2000 to 2011-2012 in borderline high BP (7.6% [95% CI, 5.8-9.8] vs 9.4% [95% CI, 7.2-11.

7 11.9]; P = .90) or either high or borderline high BP (10.6% [8.4-13.1] vs 11.0% [95% CI, 8.8-13.4]; P

8 and 2011-2012, but either high or borderline high BP remained stable.

9 The presence of both high BP and diabetes was associated with fourfold odds o

10 tabolic syndrome and its two key components, high BP and diabetes were associated with age-related ca

11 he individual metabolic syndrome components, high BP was associated with all three cataract types; di

12 In contrast, high BP is not associated with higher risk of mortality

13 In either case, overperfusion during high BP may cause oxidative injury to the outer retina,

14 .42-2.40) for diabetes, 1.92 (1.47-2.52) for high BP, and 1.27 (1.04-1.55) for metabolic syndrome.

15 We constructed a genetic risk score for high BP by using 314 published BP loci in 277 005 indivi

16 ffspring with high BP whose parents also had high BP showed an unexpected rise in plasma epinephrine

17 n in offspring with low BP whose parents had high BP or in offspring with high BP whose parents had l

18 Irrespective of family history, high BP is associated with increased body weight and hyp

19 Although the groups presented with identical high BP, endothelial-specific Epas1 gene deletion accent

20 There was a decrease in high BP between 1999-2000 (3.0% [95% CI, 2.0-4.3]) and 2

21 ation of the optimally rapid brain kinetics, high BP and brain uptake, and favorable metabolic profil

22 r and metabolic diseases, including obesity, high BP, diabetes, CKD, myocardial infarction, and strok

23 was to determine whether this attenuation of high BP is associated with prevention of other pathophys

24 ense (AT1R-AS) attenuates the development of high BP in the spontaneously hypertensive (SH) rat model

25 e responsible for the increased frequency of high BP and kidney disease in African Americans, with pa

26 These results document the importance of high BP as a modifiable risk factor for ESRD in China.

27 -based recommendations for the management of high BP and should meet the clinical needs of most patie

28 novel SNPs in MDM4 and HRH1 with sequelae of high BP including coronary artery disease (CAD), left ve

29 AD is associated with central obesity and/or high BP.

30 e than 1 in 10 had either borderline high or high BP.

31 patient's private physician for persistently high BP.

32 he predominant causes of ESRD are reportedly high BP and diabetes mellitus.

33 In the high-BP group, BF(ONH) had no significant change during

34 ages of disclosing genetic predisposition to high BP for risk stratification needs careful evaluation

35 e role of these factors in predisposition to high BP, we studied 100 young adults with high or low BP

36 personal BP and a familial predisposition to high BP.

37 pared with those without high triglycerides, high BP, and MetS after adjusting for potential confound

38 Untreated high BP, or hypertension (HTN), is associated with incre

39 Severe periodontitis was associated with high BP, with OR of 2.93 (95% CI: 1.25 to 6.84), after a

40 Offspring with high BP whose parents also had high BP showed an unexpec

41 ose parents had high BP or in offspring with high BP whose parents had low BP.

42 Subjects with high BP, irrespective of parental BP, were heavier (P=.0

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