ライフサイエンスコーパス: high-fat

1 2) a low fat diet containing n-3 PUFAs, 3) a high fat (41% kcal) diet rich in n-3 PUFAs, 4) a high fa

2 or 0.45 kg fresh strawberries) to a high-fat (50 g total fat) meal on postprandial vascular

3 C57BL/6 female mice were fed a chow (21%) or high-fat (60%) diet and further divided by housing in st

4 t (CD) and Western diet (WD), which contains high fat and carbohydrate, were used to feed wild type (

5 e Western world, obesogenic diets containing high fat and high sugar (HFHS) are commonly consumed dur

6 e Western world, obesogenic diets containing high fat and high sugar (HFHS) are commonly consumed dur

7 eventing metabolic endotoxemia in rats fed a high fat and sucrose diet.

8 ion, and increased apoptosis when a combined high-fat and high-glucose diet was given, seemingly due

9 ells by either a high-fat diet or a combined high-fat and high-glucose diet, but not by high-glucose

10 pocaloric low-fat and high-carbohydrate or a high-fat and low-carbohydrate diet for 10 wk.

11 5% CI: -0.20, 4.28 kg; P = 0.07) more on the high-fat and low-carbohydrate diet than on the low-fat a

12 Ci (P = 0.0146) for white bread than did the high-fat breakfast and a lower II value (P = 0.0285) tha

13 1) values than did the high-carbohydrate and high-fat breakfasts.

14 actility and relaxation when challenged with high fat buffer.

15 se effects when they are incorporated into a high-fat, calorie-dense meal.

16 Importantly, following a high-fat challenge, carriers in the Amish Complex Diseas

17 In addition, previous experience with high-fat consumption reduced dendritic spine density in

18 on of this technology for food products with high fat contents.

19 and cholesterol metabolism, compared to the high-fat control group.

20 Higher intake of high-fat dairy food and milk was not associated with all

21 the hypotheses that a greater consumption of high-fat dairy improves fertility or that a greater cons

22 ere was no clear association between low- or high-fat dairy intake and fecundability in either cohort

23 stern (high in red meat, refined grains, and high-fat dairy) and prudent (high in fruits, vegetables,

24 mine the effects of two different diets-very high fat diet (HFD) and moderately high fat plus cholest

25 search demonstrated that a brief (two weeks) high fat diet (HFD) caused insulin resistance in rat ske

26 Under conditions of high fat diet (HFD) consumption, glucose dyshomeostasis

27 reverse some of the negative consequences of high fat diet (HFD) consumption.

28 In this study we fed mice a high fat diet (HFD) for seven weeks followed by addition

29 We examined the effect of chronic high fat diet (HFD) on amyloid deposition and cognition

30 s of age, and to HF diabetic mice induced by high fat diet (HFD) plus streptozotocin (STZ) in C57BL/6

31 lysis of the effects after placing mice on a high fat diet (HFD) regimen demonstrated that running di

32 Atherosclerosis was induced by feeding high fat diet (HFD) to mice for 10 weeks, followed by fi

33 ows: (i) Control, (ii) Control + L/Zi, (iii) High Fat Diet (HFD), and (iv) HFD+ L/Z.

34 ceptible to the harmful cognitive effects of high fat diet (HFD)-induced IR due to apoE isoform-speci

35 BALB/c mice are known to be resistant to high fat diet (HFD)-induced obesity, however the genetic

36 We established a standardized mouse model of high fat diet (HFD)-induced steatosis followed by Omega3

37 ensitive, glucose-tolerant, and resistant to high fat diet (HFD)-induced toxicity.

38 sed fuel oxidation and oxidative damage upon high fat diet (HFD).

39 llular carcinogenesis using a streptozotocin-high fat diet (STZ-HFD) induced nonalcoholic steatohepat

40 Type 2 diabetes (T2D) was induced by high fat diet administration in M.sh.

41 reased body weight, which was exacerbated by high fat diet and driven by increased food intake.

42 Both stress and high fat diet can alter the gut microbiota and contribut

43 Here the authors show that high fat diet causes calpain-1-dependent degradation of

44 ecent studies demonstrate that rodents fed a high fat diet exhibit retinal dysfunction concomitant wi

45 re assessed during cold stress and following high fat diet feeding.

46 We verified that high fat diet lipid accumulation is also capable of indu

47 Furthermore, the high fat diet lowered bone marrow B cell frequency accom

48 and attenuates the inflammatory impact of a high fat diet on glucose tolerance and insulin resistanc

49 atherosclerotic plaque in rats feeding with high fat diet plus balloon injury.

50 With this aim, rats were fed a high fat diet with 5% sucrose in the drinking water (HFS

51 urthermore, in wild-type C57BL/6J mice fed a high fat diet, 2-weeks supplementation with Lab4 probiot

52 vulnerable to the anxiogenic effects of the high fat diet, and obese male mice showed decreased loco

53 sorders, obesity was induced in mice using a high fat diet, and the mice were subsequently stressed u

54 When hyperlipidemic mice are given a high fat diet, CC appear in aortic sinus within 1 week.

55 In murine obesity induced by a high fat diet, ex vivo IgM and IgG were elevated with un

56 d peripheral macrophages and is required for high fat diet-dependent weight gain in mice.

57 Transplantation of AT-LSK sorted from high fat diet-fed (HFD) mice is sufficient to induce ATM

58 CAST overexpression did not influence high fat diet-induced body weight and fat mass gain thro

59 ity runner; LCR) displayed susceptibility to high fat diet-induced steatosis in association with redu

60 on of ER stress in mice completely abolishes high fat diet-induced upregulation of TRIP-Br2 in BAT.

61 (PDH) within a single day of feeding mice a high fat diet.

62 protective immunity, which can be altered by high fat diet.

63 ting the adverse metabolic consequences of a high fat diet.

64 is development, and insulin resistance under high fat diet.

65 Some mice were placed on an atherogenic high-fat diet (16% fat, 41% carbohydrate, and 1.25% chol

66 KEY POINTS: A high-fat diet (60% kcal from fat) is associated with mot

67 ic syndrome-accompanying features induced by high-fat diet (HF), such as dyslipidaemia, glucose intol

68 High-fat diet (HFD) accelerates these effects in apoE4-T

69 ential exposure to postnatal over nutrition, high-fat diet (HFD) after weaning, followed later by ova

70 onse of human skeletal muscle to 9 days of a high-fat diet (HFD) alone (Sed-HFD) or in combination wi

71 Maternal high-fat diet (HFD) alters hypothalamic developmental pr

72 of vitamin D-enriched mushrooms extracts on high-fat diet (HFD) animal model of non-alcoholic steato

73 and liver as distinct features of mice fed a high-fat diet (HFD) as well as obese patients.

74 atosis formation in transgenic CCDC3 mice on high-fat diet (HFD) by reducing the expression of hepati

75 Feeding a high-fat diet (HFD) coupled with sugar, mimicking a West

76 In addition, high-fat diet (HFD) feeding induced vascular miR-204 and

77 effects of hyperinsulinaemia associated with high-fat diet (HFD) feeding on the cardiac beta2 -adrene

78 During high-fat diet (HFD) feeding, macrophage-specific JAK2 kn

79 ecies and their changes induced by long-term high-fat diet (HFD) feeding.

80 rodents and causes excess weight gain during high-fat diet (HFD) feeding.

81 Mice fed a high-fat diet (HFD) for 10 weeks were divided into group

82 and p35IL-12(-/-) (p35(-/-)) mice were fed a high-fat diet (HFD) for 12 weeks.

83 rats of both sexes that were maintained on a high-fat diet (HFD) for 6 weeks prior to DRG inflammatio

84 Here, we report that mice fed a high-fat diet (HFD) for as little as 1-3 days show incre

85 In C57BL/6 (B6) mice, obesity induced by a high-fat diet (HFD) has major effects on visceral epidid

86 mmation pathways in the obesity induced by a high-fat diet (HFD) in rodents.

87 range with significant weight reduction in a high-fat diet (HFD) induced diabetic mouse model and a g

88 ABSTRACT: The consumption of a high-fat diet (HFD) is associated with myenteric neurode

89 this study was to understand the impacts of high-fat diet (HFD) on the insulin-adrenergic receptor s

90 d replacement mice were fed either a control high-fat diet (HFD) or an HFD supplemented with 3% n-3 P

91 ene expression in C57BL/6NTac mice fed a 60% high-fat diet (HFD) or control diet for up to 16 weeks.

92 f multi-tissues from outbred mice fed with a high-fat diet (HFD) or regular chow at weeks 1, 9, and 1

93 Consumption of a high-fat diet (HFD) results in suppression of ATP citrat

94 /6 J mice were fed a control diet (CON) or a high-fat diet (HFD) with or without 0.2% (w/w) RES durin

95 modification accumulation in mice exposed to high-fat diet (HFD), injected with streptozotocin, or bo

96 Despite becoming obese on a high-fat diet (HFD), mice lacking FcgammaRIIB globally o

97 fed mice either a chow diet (CD), a 16 week high-fat diet (HFD), or a CR diet to compare and contras

98 When fed a high-fat diet (HFD), the transgenic mice displayed a sig

99 s after ischemia reperfusion injury (IRI) in high-fat diet (HFD)-fed mice.

100 In a mouse model, high-fat diet (HFD)-fed offspring have cognitive and exe

101 in Ern1(f/f); Lyz2-Cre mice largely reversed high-fat diet (HFD)-induced M1-M2 imbalance in white adi

102 ry immune responses and markedly exacerbated high-fat diet (HFD)-induced NAFLD pathogenesis.

103 eas AEG-1(DeltaHEP) mice were protected from high-fat diet (HFD)-induced NASH.

104 transcripts and metabolites in response to a high-fat diet (HFD)-induced obesity and/or exercise.

105 High-fat diet (HFD)-induced obesity is accompanied by in

106 e relevance of adipose tissue ERalpha during high-fat diet (HFD)-induced obesity using female aP2-Cre

107 it is protected against a cold challenge and high-fat diet (HFD)-induced obesity with associated insu

108 ive pancreatic islet dysfunction with age or high-fat diet (HFD).

109 downregulated during weight gain induced by high-fat diet (HFD).

110 ased RNA-seq analysis in liver from mice fed high-fat diet +/- Fenretinide.

111 A high-fat diet accelerated stroke incidence.

112 Adding a high-salt, high-fat diet accelerates endothelial senescence and ins

113 n a mouse model of sporadic AAD induced by a high-fat diet and angiotensin II infusion, ADAMTS-4 defi

114 icient ovariectomized female mice were fed a high-fat diet and concomitantly administered with vehicl

115 to show an increase in RMR in response to a high-fat diet and deoxycorticosterone acetate-salt (DOCA

116 nant GDF15 induces weight loss in mice fed a high-fat diet and in nonhuman primates with spontaneous

117 bserved in hearts from rats fed a lard-based high-fat diet and in rodent and human cardiomyocytes upo

118 ture adipocytes in multiple tissues during a high-fat diet and in skin during hair follicle growth.

119 al proteins in human skeletal muscle after a high-fat diet and resistance exercise.

120 eration tissues were fed a low-fat diet or a high-fat diet and treated with vehicle or dasatinib.

121 brogates their abnormal phenotype, even upon high-fat diet challenge.

122 ion, and were more insulin resistant after a high-fat diet challenge.

123 High-fat diet equally worsened glucose tolerance in AgRP

124 In high-fat diet fed and genetically obese ob/ob mice, P300

125 nalysis of intestinal and renal tissues from high-fat diet fed mice, critical for maintaining metabol

126 While aging impairs the osteogenic lineage, high-fat diet feeding activates expansion of the adipoge

127 privation and excessive storage of energy by high-fat diet feeding dampen the suppressive effect of l

128 le to the negative metabolic consequences of high-fat diet feeding than male mice, for reasons that a

129 ood intake and weight gain in lean mice upon high-fat diet feeding, and this injection paradigm reduc

130 proved insulin sensitivity despite prolonged high-fat diet feeding.

131 e) mice, but this effect was attenuated with high-fat diet feeding.

132 ignal transducer protein of IL-6, were fed a high-fat diet for 12 weeks.

133 actor receptor 2 knockout mice, either fed a high-fat diet for 12-14 weeks, or age-matched lean contr

134 rendered obese and pre-diabetic by feeding a high-fat diet for 15 weeks and then treated with LH-21 o

135 m-graecum), C57BL/6J mice were fed a low- or high-fat diet for 16 weeks with or without 2% (w/w) fenu

136 KEY POINTS: Maternal high-fat diet impairs brown adipocyte function and corre

137 with control littermates after 10 weeks of a high-fat diet in male mice.

138 High-fat diet in sedentary mice led to endoplasmic retic

139 rnal transcriptome is not recapitulated by a high-fat diet in young adult mice, it is significantly p

140 A high-fat diet increases bacterial lipopolysaccharide (LP

141 In high-fat diet induced fatty-liver mice, AM580 reduced Ap

142 DNA microbiome sequencing, which showed that high-fat diet induces reduction of Parasutterella sp. in

143 othesis, behavioral tests including chow and high-fat diet intake, meal patterns, conditioned place p

144 Female mice fed a high-fat diet maintain CX3CL1-CX3CR1 levels while male m

145 The high-fat diet not only accelerated Src-induced prostate

146 egates the detrimental effects of a maternal high-fat diet on glucose tolerance and hepatocyte glucos

147 ophagy was induced in beta cells by either a high-fat diet or a combined high-fat and high-glucose di

148 tively by caloric deficit and not altered by high-fat diet or stress.

149 Our data showed that rats fed a high-fat diet or that were centrally administered thapsi

150 We found that a high-fat diet promotes myofibroblast differentiation by

151 Consumption of a high-fat diet protects mice from ventilator-induced lung

152 It is currently not fully understood how a high-fat diet reprograms adipose-derived stem cells into

153 tively) were fed regular chow (control) or a high-fat diet supplemented with 30% d-fructose in drinki

154 lation exposure of mice fed normal chow or a high-fat diet to airborne fine particulate matters (PM2.

155 esterol mediates the metastatic effects of a high-fat diet via its oxysterol metabolite, 27-hydroxych

156 Steatotic liver induced by high-fat diet was more vulnerable to IRI.

157 Induction of Cyp2e1 protein by high-fat diet was suppressed in Gsta4(-/-) and dKO group

158 oprotein receptor-null (Ldlr(-/-)) mice on a high-fat diet were orally administered with vehicle cont

159 On a high-fat diet, although no differences in body weight an

160 ts against hepatic steatosis in mice fed the high-fat diet, as a novel agonist of these receptors.

161 Under a high-fat diet, deletion of PKD1 in beta-cells worsened h

162 e (IKK) occurs rapidly upon consumption of a high-fat diet, even prior to significant weight gain.

163 On high-fat diet, JAK2L mice had hepatic steatosis and insu

164 When challenged with a high-fat diet, mice carrying one copy of KD-mTOR mutant

165 When challenged with a high-fat diet, Ocy-PPARgamma(-/-) mice retain glycemic c

166 ith a smaller increases in body weight under high-fat diet, smaller fat deposits, increased beta-oxid

167 When placed on the atherogenic high-fat diet, the KO mice developed features of nonalco

168 In response to the high-fat diet, the T allele was associated with a higher

169 When fed a high-fat diet, these mice exhibited normal food intake b

170 eased Src kinase activity is associated with high-fat diet-accelerated progression of prostate tumors

171 e is essential to facilitate Src-induced and high-fat diet-accelerated tumor progression.

172 Vsig4 (-/-) mice are susceptible to high-fat diet-caused obesity and murine hepatitis virus

173 c clamps and ex vivo in isolated islets from high-fat diet-fed betaPKD1KO mice without changes in isl

174 ance and motivation in female offspring from high-fat diet-fed dams.

175 in obese adipose tissue and have shown that high-fat diet-induced (HFD-induced) insulin resistance i

176 terozygous for p32 are resistant to age- and high-fat diet-induced ailments, including obesity, hyper

177 ssed highly in the liver, protects mice from high-fat diet-induced and aging-induced obesity and hepa

178 -/- mice with Arhgef1-deficient BM prevented high-fat diet-induced atherosclerosis, while reconstitut

179 eptor (LDLR) and Arhgef1 were protected from high-fat diet-induced atherosclerosis.

180 ansporter ZIP14 during pharmacologically and high-fat diet-induced ER stress using Zip14(-/-) (KO) mi

181 e, Nox2-deficient mice are protected against high-fat diet-induced hepatic steatosis and insulin resi

182 led that SLC13A5 deletion protects mice from high-fat diet-induced hepatic steatosis and that mutatio

183 he counteractive effect of PM2.5 exposure on high-fat diet-induced hepatic steatosis was mediated thr

184 is in the mouse liver but protects mice from high-fat diet-induced hyperglycemia.

185 A-L exacerbates or alleviates, respectively, high-fat diet-induced mitochondrial dysfunction, hepatos

186 Here we report that in the NTS of high-fat diet-induced obese (DIO) rats, the apoA-IV mRNA

187 normalised elevated levels of FGF21 in both high-fat diet-induced obese mice and in genetically obes

188 PM2.5 exposure relieved hepatic steatosis in high-fat diet-induced obese mice.

189 primary bone marrow-derived macrophages and high-fat diet-induced obese mice.

190 rove hepatic glucose and lipid metabolism in high-fat diet-induced obese mice.

191 cells, zebrafish and in live tissues from a high-fat diet-induced obese mouse model.

192 adipose tissues were resistant to developing high-fat diet-induced obesity and had significantly redu

193 Finally, moderate alcohol prevented high-fat diet-induced obesity and metabolic dysfunction.

194 rexpression of Id1 causes age-associated and high-fat diet-induced obesity in mice.

195 High-fat diet-induced obesity is a major risk factor for

196 acute fasting) and excessive energy storage (high-fat diet-induced obesity) blunt the effect of lepti

197 trol intervention protects offspring against high-fat diet-induced obesity.

198 with type 2 diabetes, and C57BL/6J mice with high-fat diet-induced obesity.

199 w the metabolism of myristic acid stimulates high-fat diet-mediated prostate tumor progression.

200 ght the potential of probiotics to attenuate high-fat diet-related metabolic disorder.

201 In high-fat diet-treated mice, knockout of the endothelial

202 e to reduce obesity even when mice are fed a high-fat diet.

203 ght gain and protected from hyperglycemia on high-fat diet.

204 and glucose intolerance in mice exposed to a high-fat diet.

205 , and hepatic steatosis when challenged with high-fat diet.

206 function of these mice fed a control chow or high-fat diet.

207 mber or size distribution on either a low or high-fat diet.

208 potently decreasing weight gain in mice on a high-fat diet.

209 is did not occur when Ad-FLD mice were fed a high-fat diet.

210 s of irisin were observed in mice fed with a high-fat diet.

211 olic deficits caused by the consumption of a high-fat diet.

212 ity to metabolic disease in the context of a high-fat diet.

213 hat were maintained on either a regular or a high-fat diet.

214 bolic deficits displayed by mice consuming a high-fat diet.

215 sing hZnT8 WT or hZnT8 R325W fed a normal or high-fat diet.

216 3 in mice fed either a normal chow diet or a high-fat diet.

217 imbalance, and reduced animal longevity on a high-fat diet.

218 % fat, 15% protein, and 60-65% carbohydrate; high-fat diet: 40-45% fat, 15% protein, and 40-45% carbo

219 The role of A2AARs in regulating high-fat-diet (HFD)-induced metabolic derangements is un

220 nt-associated osteomyelitis in normal versus high-fat-diet obese/T2D mice and found that S. aureus in

221 nhibition in a mouse model of T2DM (i.e., in high-fat-diet-fed animals).

222 mption of stevioside prevents development of high-fat-diet-induced diabetic hyperglycaemia in wild-ty

223 stine-specific disruption of Hif2a, in which high-fat-diet-induced hepatic steatosis and obesity were

224 roves mitochondrial function and ameliorates high-fat-diet-induced hepatic steatosis, glucose toleran

225 rgeted activation of Hh signaling suppresses high-fat-diet-induced obesity and improves whole-body gl

226 e used mouse models of a regular diet and of high-fat-diet-induced obesity to investigate the role of

227 receptors control pancreatic dysfunction in high-fat-diet-induced obesity.

228 ned with increased oxygen consumption during high-fat-diet-induced obesity.

229 sts increased insulin secretion and reversed high-fat-diet-induced weight gain and insulin resistance

230 Overconsumption of high-fat diets (HFDs) can critically affect synaptic and

231 turbed in Gpr119(betacell-/-) mice on RC and high-fat diets.

232 and fibrosis prevented, on both low-fat and high-fat diets.

233 dult beta cell improved glucose tolerance in high fat-fed animals.

234 an effect not present in the offspring from high fat-fed dams that had trained.

235 protein to low-density lipoprotein ratios in high fat-fed mice without affecting circulating total ch

236 t-binding protein 1/CD36 in hepatocytes from high fat-fed mice.

237 did transiently improve glucose tolerance in high fat-fed mice.

238 High fat-fed sedentary dams produced female offspring wi

239 Offspring from high fat-fed trained dams had normalized glucose toleran

240 High fat feeding alters both the number of ILC2 and thei

241 High fat feeding impairs skeletal muscle metabolic flexi

242 le in limiting obesity, how ILC2s respond to high fat feeding is poorly understood, and their direct

243 eliorates some of the deleterious effects of high fat feeding, we investigated the transcriptional an

244 njury to steatohepatitis in NASH produced by high-fat feeding during development but appear less impo

245 the beta-cell adaptive secretory response to high-fat feeding in mice.

246 reverse the detrimental effects of maternal high-fat feeding on offspring metabolism of female mice.

247 After high-fat feeding, Gpr119(-/-) mice exhibited reduced fat

248 -cell PKD1 under basal conditions and during high-fat feeding.

249 l patterns, conditioned place preference for high-fat food, cue-induced reinstatement of sucrose-seek

250 ipoprotein E knock-out (apoE(-/-)) mice on a high fat (HF) diet as an atherosclerotic obesity model,

251 ts for 6 weeks before they received a 6-week high fat (HF) diet under isocaloric conditions.

252 were lower in TRPC1 KO mice that were fed a high-fat (HF) (45% fat) diet and exercised as compared w

253 iver extracellular matrix (ECM) expands with high-fat (HF) feeding.

254 od) was used to derive an energy-dense (ED), high-fat (HF), low-fiber density (LFD) dietary pattern w

255 either a low-fat (10% kcal) or one of three high-fat (HF, 60% kcal) diets rich in saturated fatty ac

256 Mice fed high fat, high cholesterol, cholate (HFHCC) diet for thr

257 oaded Huh7 cells and in livers of rats fed a high-fat, high-fructose diet.

258 senescence (n=15) were fed normal chow or a high-fat, high-salt diet (WD).

259 tently, beta-cell expansion in response to a high-fat, high-sucrose (HFHS) diet was significantly imp

260 In nonhuman primates, a 2-year high-fat, high-sucrose diet increased hepatic mIndy expr

261 tis (NASH) when challenged with a lipogenic, high-fat, high-sucrose diet.

262 bacteria in mice undergoing switches between high-fat, high-sugar (HFHSD) and low-fat, plant-polysacc

263 ese mice, even as they continue to consume a high-fat, high-sugar diet.

264 le C57Bl6J mice were fed a control (CD) or a high fat/high sucrose (HF/HS) diet for 4-10 weeks, and t

265 resent study was to evaluate the impact of a high fat/high sucrose diet on retinal insulin signaling

266 Addition of excess cholesterol to a high-fat/high-sucrose diet produced greater steatosis in

267 ncreases susceptibility to acute and chronic high-fat/high-sucrose diet-induced steatosis, without ob

268 in iWAT, and develop glucose intolerance and high fat-induced hepatic steatosis.

269 % CI: 1.02, 1.34), while risk was higher for high fat intake during both adolescence and midlife.

270 feeding, and this injection paradigm reduced high-fat intake and obesity in diet-induced obese (DIO)

271 Several studies associated high-fat intakes with a high incidence of age-related ma

272 Adaptation to a ketogenic low carbohydrate, high fat (LCHF) diet markedly increases rates of whole-b

273 At high fat level a small increase of aroma release was obs

274 ence >98 cm were randomly assigned to a very high-fat, low-carbohydrate (VHFLC; 73% of energy fat and

275 -matched, overweight males consumed 9 d of a high-fat, low-carbohydrate diet during which time they e

276 High-fat, low-carbohydrate diets, known as ketogenic die

277 High-fat, low-carbohydrate ketogenic diets (KDs) have sh

278 on prior to and after five-weeks on control, high-fat, low-fat (18%, 40% and 10% energy from fat, res

279 survival after infection, with those on the high fat/low protein diet showing 30% survival at 8 days

280 A high fat mass index was associated with a higher Rint (Z

281 RNA was observed to be up-regulated by acute high-fat meal ingestion in both rodents and humans.

282 Therefore, administering cannabinoids with a high-fat meal or in lipid-based formulations has the pot

283 disorder (MDD) on inflammatory responses to high-fat meals.

284 ments and within each group (low, medium and high fat milk) using the three methods.

285 PUFAs, 4) a high fat n-6 PUFA diet, or 5) a high fat monounsaturated diet.

286 fat (41% kcal) diet rich in n-3 PUFAs, 4) a high fat n-6 PUFA diet, or 5) a high fat monounsaturated

287 Rats self-administered 60% high fat or chow 45 mg pellets and were then tested for

288 Three inbred ILSXISS strains were fed high-fat or chow diets and subjected to metabolic phenot

289 ic breakfast meals (i.e., high carbohydrate, high fat, or high protein) on separate days in a random

290 iets-very high fat diet (HFD) and moderately high fat plus cholesterol diet (HFC)-on wildtype (WT) an

291 ue inflammatory secretion profile induced by high-fat regimens.

292 Retinas of mice fed a high fat/sucrose diet also exhibited elevated levels of

293 Mice fed a high fat/sucrose diet exhibited attenuated Akt phosphory

294 axis as a key molecular mechanism whereby a high fat/sucrose diet impairs insulin action in retina.

295 We have recently validated a high fat/sugar water-induced animal (an isogenic strain

296 -VTA oleate blunted the rewarding effects of high-fat/sugar food in an operant task and inhibited DA

297 ates in the diet because feeding mice a very high-fat, very low-carbohydrate diet did not affect cell

298 Finally, incubation of craving for chow and high fat was accompanied by an increase in calcium-perme

299 High fat was chosen for comparison to determine whether

300 sponding for cues previously associated with high fat was greater than chow at both 1 and 30 days.