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1 protective immunity, which can be altered by high fat diet.
2 ting the adverse metabolic consequences of a high fat diet.
3 is development, and insulin resistance under high fat diet.
4 (PDH) within a single day of feeding mice a high fat diet.
5 function of these mice fed a control chow or high-fat diet.
6 and glucose intolerance in mice exposed to a high-fat diet.
7 , and hepatic steatosis when challenged with high-fat diet.
8 mber or size distribution on either a low or high-fat diet.
9 olic deficits caused by the consumption of a high-fat diet.
10 potently decreasing weight gain in mice on a high-fat diet.
11 is did not occur when Ad-FLD mice were fed a high-fat diet.
12 s of irisin were observed in mice fed with a high-fat diet.
13 ity to metabolic disease in the context of a high-fat diet.
14 hat were maintained on either a regular or a high-fat diet.
15 bolic deficits displayed by mice consuming a high-fat diet.
16 sing hZnT8 WT or hZnT8 R325W fed a normal or high-fat diet.
17 red with littermate controls) after eating a high-fat diet.
18 ke and greater diet-induced obesity when fed high-fat diet.
19 o the same extent as control mice when fed a high-fat diet.
20 db/db, streptozotocin-induced and mice fed a high-fat diet.
21 ion of adipose tissue when challenged with a high-fat diet.
22 I)) and wild-type mice (gsk3(WT)) were fed a high-fat diet.
23 fatty liver disease in mice maintained on a high-fat diet.
24 d decreases vascular inflammation induced by high-fat diet.
25 on of these mice fed chow or after 6 wk of a high-fat diet.
26 3 in mice fed either a normal chow diet or a high-fat diet.
27 imbalance, and reduced animal longevity on a high-fat diet.
28 e to reduce obesity even when mice are fed a high-fat diet.
29 ght gain and protected from hyperglycemia on high-fat diet.
30 and fibrosis prevented, on both low-fat and high-fat diets.
31 turbed in Gpr119(betacell-/-) mice on RC and high-fat diets.
33 urthermore, in wild-type C57BL/6J mice fed a high fat diet, 2-weeks supplementation with Lab4 probiot
34 % fat, 15% protein, and 60-65% carbohydrate; high-fat diet: 40-45% fat, 15% protein, and 40-45% carbo
38 eased Src kinase activity is associated with high-fat diet-accelerated progression of prostate tumors
41 2+/-)Rosa(tdT+/-) mouse model subjected to a high-fat diet, adipocytes of atrial EAT derived from a s
47 n a mouse model of sporadic AAD induced by a high-fat diet and angiotensin II infusion, ADAMTS-4 defi
48 ice exhibit reduced effortful responding for high-fat diet and compulsive grooming, whereas group-hou
49 icient ovariectomized female mice were fed a high-fat diet and concomitantly administered with vehicl
50 to show an increase in RMR in response to a high-fat diet and deoxycorticosterone acetate-salt (DOCA
51 nant GDF15 induces weight loss in mice fed a high-fat diet and in nonhuman primates with spontaneous
52 bserved in hearts from rats fed a lard-based high-fat diet and in rodent and human cardiomyocytes upo
53 ture adipocytes in multiple tissues during a high-fat diet and in skin during hair follicle growth.
55 retinal disease, we weaned mice to chow or a high-fat diet and tested the hypothesis that diet-induce
56 eration tissues were fed a low-fat diet or a high-fat diet and treated with vehicle or dasatinib.
57 vulnerable to the anxiogenic effects of the high fat diet, and obese male mice showed decreased loco
58 sorders, obesity was induced in mice using a high fat diet, and the mice were subsequently stressed u
59 ue size at the aortic root and the aorta for high-fat diet animals as compared with Ldlr(-/-) control
60 ts against hepatic steatosis in mice fed the high-fat diet, as a novel agonist of these receptors.
62 olipoprotein E (ApoE) knockout mice fed with high-fat diet, BM nestin(+) cells regulate the egress of
63 id lesions were induced in FVB mice fed on a high-fat diet by streptozotocin injection followed by li
74 e (IKK) occurs rapidly upon consumption of a high-fat diet, even prior to significant weight gain.
76 ecent studies demonstrate that rodents fed a high fat diet exhibit retinal dysfunction concomitant wi
79 us (EDL) fibre bundles obtained from chronic high-fat diet fed mice had lower basal oxygen consumptio
80 nalysis of intestinal and renal tissues from high-fat diet fed mice, critical for maintaining metabol
82 c clamps and ex vivo in isolated islets from high-fat diet-fed betaPKD1KO mice without changes in isl
87 While aging impairs the osteogenic lineage, high-fat diet feeding activates expansion of the adipoge
88 privation and excessive storage of energy by high-fat diet feeding dampen the suppressive effect of l
89 dy site-specific myeloid cell behavior after high-fat diet feeding or tumor necrosis factor stimulati
90 le to the negative metabolic consequences of high-fat diet feeding than male mice, for reasons that a
91 ood intake and weight gain in lean mice upon high-fat diet feeding, and this injection paradigm reduc
97 actor receptor 2 knockout mice, either fed a high-fat diet for 12-14 weeks, or age-matched lean contr
98 rendered obese and pre-diabetic by feeding a high-fat diet for 15 weeks and then treated with LH-21 o
99 m-graecum), C57BL/6J mice were fed a low- or high-fat diet for 16 weeks with or without 2% (w/w) fenu
100 rofiling of aortas from Apoe(-/-) mice fed a high-fat diet for 4 weeks, 8 weeks, or 4 months revealed
101 e 32 weeks and after 20 weeks of standard or high-fat diet, Gucy1a3(-/-)/Ldlr(-/-) mice exhibited a s
102 ic syndrome-accompanying features induced by high-fat diet (HF), such as dyslipidaemia, glucose intol
103 mine the effects of two different diets-very high fat diet (HFD) and moderately high fat plus cholest
105 search demonstrated that a brief (two weeks) high fat diet (HFD) caused insulin resistance in rat ske
111 s of age, and to HF diabetic mice induced by high fat diet (HFD) plus streptozotocin (STZ) in C57BL/6
112 lysis of the effects after placing mice on a high fat diet (HFD) regimen demonstrated that running di
115 ceptible to the harmful cognitive effects of high fat diet (HFD)-induced IR due to apoE isoform-speci
116 0mvarphiKD) which resists the development of high fat diet (HFD)-induced metabolic diseases due to en
117 BALB/c mice are known to be resistant to high fat diet (HFD)-induced obesity, however the genetic
119 We established a standardized mouse model of high fat diet (HFD)-induced steatosis followed by Omega3
124 mitic acid (PA)- or oleic acid (OA)-enriched high-fat diet (HFD) (20% of calories from FA) or a norma
126 ential exposure to postnatal over nutrition, high-fat diet (HFD) after weaning, followed later by ova
127 onse of human skeletal muscle to 9 days of a high-fat diet (HFD) alone (Sed-HFD) or in combination wi
130 of vitamin D-enriched mushrooms extracts on high-fat diet (HFD) animal model of non-alcoholic steato
132 atosis formation in transgenic CCDC3 mice on high-fat diet (HFD) by reducing the expression of hepati
134 rozygous mice (S2HET) were challenged with a high-fat diet (HFD) containing 45% of kilocalories from
137 effects of hyperinsulinaemia associated with high-fat diet (HFD) feeding on the cardiac beta2 -adrene
144 rats of both sexes that were maintained on a high-fat diet (HFD) for 6 weeks prior to DRG inflammatio
146 In C57BL/6 (B6) mice, obesity induced by a high-fat diet (HFD) has major effects on visceral epidid
148 range with significant weight reduction in a high-fat diet (HFD) induced diabetic mouse model and a g
149 ss (CC) mouse genetic resource population to high-fat diet (HFD) induced T2D-like disease to evaluate
151 this study was to understand the impacts of high-fat diet (HFD) on the insulin-adrenergic receptor s
152 (ACE2KO) and wild-type (WT) mice were fed a high-fat diet (HFD) or a control diet and studied at 6 m
153 d replacement mice were fed either a control high-fat diet (HFD) or an HFD supplemented with 3% n-3 P
154 ene expression in C57BL/6NTac mice fed a 60% high-fat diet (HFD) or control diet for up to 16 weeks.
155 f multi-tissues from outbred mice fed with a high-fat diet (HFD) or regular chow at weeks 1, 9, and 1
158 Experiments using parabiotic mice fed a high-fat diet (HFD) showed differential trafficking of A
159 /6 J mice were fed a control diet (CON) or a high-fat diet (HFD) with or without 0.2% (w/w) RES durin
162 modification accumulation in mice exposed to high-fat diet (HFD), injected with streptozotocin, or bo
165 fed mice either a chow diet (CD), a 16 week high-fat diet (HFD), or a CR diet to compare and contras
172 in Ern1(f/f); Lyz2-Cre mice largely reversed high-fat diet (HFD)-induced M1-M2 imbalance in white adi
173 lose), fiber markedly protected mice against high-fat diet (HFD)-induced metabolic syndrome, the effe
177 transcripts and metabolites in response to a high-fat diet (HFD)-induced obesity and/or exercise.
178 y polypeptide (GIP) receptor (GIPR) prevents high-fat diet (HFD)-induced obesity in mice due to speci
181 e relevance of adipose tissue ERalpha during high-fat diet (HFD)-induced obesity using female aP2-Cre
182 it is protected against a cold challenge and high-fat diet (HFD)-induced obesity with associated insu
183 kout (GPRKO) female mice were protected from high-fat diet (HFD)-induced obesity, blood glucose intol
186 mine the contributions of macrophage Nrp1 in high-fat diet (HFD)-instigated insulin resistance in viv
199 rnal transcriptome is not recapitulated by a high-fat diet in young adult mice, it is significantly p
200 nduced reduction of plasma TGs in mice fed a high-fat diet, in postprandial clearance studies, and wh
205 RNA activator (SRA) (SRAKO) are resistant to high fat diet-induced obesity with a phenotype that incl
206 ity runner; LCR) displayed susceptibility to high fat diet-induced steatosis in association with redu
207 on of ER stress in mice completely abolishes high fat diet-induced upregulation of TRIP-Br2 in BAT.
208 in obese adipose tissue and have shown that high-fat diet-induced (HFD-induced) insulin resistance i
209 terozygous for p32 are resistant to age- and high-fat diet-induced ailments, including obesity, hyper
210 ssed highly in the liver, protects mice from high-fat diet-induced and aging-induced obesity and hepa
211 -/- mice with Arhgef1-deficient BM prevented high-fat diet-induced atherosclerosis, while reconstitut
213 weeks of VWR exercise in obese mice rescued high-fat diet-induced decreased muscle GLUT4 protein and
215 ansporter ZIP14 during pharmacologically and high-fat diet-induced ER stress using Zip14(-/-) (KO) mi
216 e, Nox2-deficient mice are protected against high-fat diet-induced hepatic steatosis and insulin resi
217 led that SLC13A5 deletion protects mice from high-fat diet-induced hepatic steatosis and that mutatio
218 ific gp130 knockout mice were protected from high-fat diet-induced hepatic steatosis as well as from
219 he counteractive effect of PM2.5 exposure on high-fat diet-induced hepatic steatosis was mediated thr
223 A-L exacerbates or alleviates, respectively, high-fat diet-induced mitochondrial dysfunction, hepatos
226 normalised elevated levels of FGF21 in both high-fat diet-induced obese mice and in genetically obes
231 adipose tissues were resistant to developing high-fat diet-induced obesity and had significantly redu
235 acute fasting) and excessive energy storage (high-fat diet-induced obesity) blunt the effect of lepti
239 virus-mediated depletion of COP1 ameliorates high-fat diet-induced steatosis in mouse liver and impro
240 ed thermogenic EE, which protected mice from high-fat diet-induced weight gain at ambient temperature
241 ponse, which requires domains that influence high-fat-diet-induced chronic inflammation and alter cel
242 mption of stevioside prevents development of high-fat-diet-induced diabetic hyperglycaemia in wild-ty
243 stine-specific disruption of Hif2a, in which high-fat-diet-induced hepatic steatosis and obesity were
244 roves mitochondrial function and ameliorates high-fat-diet-induced hepatic steatosis, glucose toleran
245 rgeted activation of Hh signaling suppresses high-fat-diet-induced obesity and improves whole-body gl
246 e used mouse models of a regular diet and of high-fat-diet-induced obesity to investigate the role of
249 sts increased insulin secretion and reversed high-fat-diet-induced weight gain and insulin resistance
250 DNA microbiome sequencing, which showed that high-fat diet induces reduction of Parasutterella sp. in
251 othesis, behavioral tests including chow and high-fat diet intake, meal patterns, conditioned place p
253 ation to a ketogenic low carbohydrate (CHO), high fat diet (LCHF) during 3 weeks of intensified train
258 more susceptible to the adverse effects of a high-fat diet, manifested in increased body weight and a
260 ice, consistent with the hypothesis that the high-fat diet-mediated reduction of hippocampal SIRT1 co
264 nt-associated osteomyelitis in normal versus high-fat-diet obese/T2D mice and found that S. aureus in
266 and attenuates the inflammatory impact of a high fat diet on glucose tolerance and insulin resistanc
267 egates the detrimental effects of a maternal high-fat diet on glucose tolerance and hepatocyte glucos
268 ed at two different doses to C57BL/6 mice on high fat diet or standard diet for a period of 17weeks.
269 ophagy was induced in beta cells by either a high-fat diet or a combined high-fat and high-glucose di
272 nning to obtain mechanistic insight into how high-fat diets perturb stem cell function and cause canc
276 female mice display increased weight gain on high-fat diet, reduced behavioral despair, and increased
278 re elevated in adipose depots in response to high-fat diet regimens and during the aging process; how
280 It is currently not fully understood how a high-fat diet reprograms adipose-derived stem cells into
281 ith a smaller increases in body weight under high-fat diet, smaller fat deposits, increased beta-oxid
282 th Il6 gene (controls), were fed a chow or a high-fat diet; some mice were given injections of recomb
283 Furthermore, dusp6-deficient mice have a high-fat-diet-specific transcriptomic response to revers
284 llular carcinogenesis using a streptozotocin-high fat diet (STZ-HFD) induced nonalcoholic steatohepat
285 tively) were fed regular chow (control) or a high-fat diet supplemented with 30% d-fructose in drinki
287 s, insulin secretion and autoimmunity with a high-fat diet supports a shared mechanism for type 1 (T1
291 lation exposure of mice fed normal chow or a high-fat diet to airborne fine particulate matters (PM2.
295 esterol mediates the metastatic effects of a high-fat diet via its oxysterol metabolite, 27-hydroxych
298 oprotein receptor-null (Ldlr(-/-)) mice on a high-fat diet were orally administered with vehicle cont
299 ced weight gain in mice that had access to a high-fat diet, while not altering general locomotor acti
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