ライフサイエンスコーパス: high-frequency stimulation

1 ng less than half the energy of conventional high frequency stimulation.

2 and these inputs can be potentiated through high frequency stimulation.

3 current, reducing neuronal ability to follow high frequency stimulation.

4 nt of Ad-LacZ infected cells with periods of high frequency stimulation.

5 facilitated, after strong depolarizations or high frequency stimulation.

6 dergo long-term potentiation (LTP) following high frequency stimulation.

7 long-term increase in latency caused by the high frequency stimulation.

8 ed before and immediately after cessation of high frequency stimulation.

9 the time period of 30 s after switching off high frequency stimulation.

10 onic movements where it was suppressed after high frequency stimulation.

11 NMDA receptor-independent form of LTP during high frequency stimulation.

12 ascade is accurate and fast enough to follow high frequency stimulation.

13 t in location toward the AZ periphery during high-frequency stimulation.

14 viding constant SV size during long-lasting, high-frequency stimulation.

15 of long-term potentiation by short bursts of high-frequency stimulation.

16 ery near highly active exocytic sites during high-frequency stimulation.

17 fusing fully with the plasma membrane during high-frequency stimulation.

18 e more quickly than wild-type in response to high-frequency stimulation.

19 tral synapses rapidly depress in response to high-frequency stimulation.

20 es of baroreceptor neurons is reduced during high-frequency stimulation.

21 GLUT1 recycles more slowly during prolonged, high-frequency stimulation.

22 charge transfer and current potentiation by high-frequency stimulation.

23 NMDAR-mediated currents induced by bursts of high-frequency stimulation.

24 vous system are transiently depressed during high-frequency stimulation.

25 current and failure of the neurons to follow high-frequency stimulation.

26 release at 15%-20% of the normal rate during high-frequency stimulation.

27 ontent and facilitation of EPP amplitudes at high-frequency stimulation.

28 tentials and NMDA receptor activation during high-frequency stimulation.

29 of synaptic NMDAR currents during bursts of high-frequency stimulation.

30 neurotransmission over prolonged periods of high-frequency stimulation.

31 A3 induced by either brief or long trains of high-frequency stimulation.

32 ired the maintenance of LTP induced by brief high-frequency stimulation.

33 ntiation of neuromuscular transmission after high-frequency stimulation.

34 magnesium-free bathing medium, or following high-frequency stimulation.

35 role comes into play primarily during brief high-frequency stimulation.

36 synaptic depression during brief periods of high-frequency stimulation.

37 he stronger activation that occurs following high-frequency stimulation.

38 tiation was lower than 1 hour after the last high-frequency stimulation.

39 , and a reduction in their ability to follow high-frequency stimulation.

40 se excitatory synaptic currents triggered by high-frequency stimulation.

41 reas LTD required low intracellular cAMP and high-frequency stimulation.

42 plasticity lasting for tens of seconds after high-frequency stimulation.

43 had been previously potentiated by repeated high-frequency stimulation.

44 to an increase in Nav1.1 availability during high-frequency stimulation.

45 prevents excessive glutamate release during high-frequency stimulation.

46 of LTP that is BDNF independent, induced by high-frequency stimulation.

47 Loss of FMRP led to enhanced responses to high-frequency stimulation.

48 postsynaptic calcium, and can be reversed by high-frequency stimulation.

49 intensify postsynaptic Ca(2+) signals during high-frequency stimulation.

50 it regulates excitability during periods of high-frequency stimulation.

51 equency stimulations and for 76 (50%) of the high-frequency stimulations.

52 High frequency stimulation (100 or 40 Hz) in layer IV in

53 cilitation, and response to a brief train of high-frequency stimulation (100 Hz, 40 pulses) that exha

54 eceptor-independent LTP: LTP induced by very high-frequency stimulation (200 Hz-LTP), LTP induced by

55 However, when high frequency stimulation (30 Hz for 10 s) preceded imm

56 lation (depression) of EPSP amplitude during high-frequency stimulation (-39 +/- 4% and -53 +/- 4%, u

57 rminals and increased synaptic fatigue under high-frequency stimulation, accompanied by the developme

58 ning receptors preferentially desensitize at high-frequency stimulation, accounting for the inability

59 prevented this decrease indicating that the high frequency stimulation activated the NMDA receptor a

60 An additional bout of high-frequency stimulation administered under urethane c

61 fact, if LY294002 was withdrawn 5 min after high-frequency stimulation, an LTP of fEPSP was seen.

62 can account for the complex kinetics during high frequency stimulation and cause stimulus-history-de

63 onic features are the quickest to respond to high frequency stimulation and may thus directly relate

64 ated in area CA1 in response to LTP-inducing high frequency stimulation and that this activation requ

65 apid changes in synaptic efficacy induced by high-frequency stimulation and BDNF at central excitator

66 eadily releasable pool of vesicles evoked by high-frequency stimulation and by increasing the fractio

67 ndent protein kinase C (PKC) is activated by high-frequency stimulation and mediates post-tetanic pot

68 napsins boost the release probability during high-frequency stimulation and suggest that this effect

69 stimulus is recruited within milliseconds by high-frequency stimulation and support an ultrafast reco

70 ORNs to better synchronize their output with high-frequency stimulation and to perceive brief stimuli

71 yfish opener muscle is elicited by prolonged high frequency stimulation, and arises from an increase

72 mined depletion at individual synapses using high frequency stimulation, and estimated the size of th

73 long-term potentiation of EPSPs triggered by high frequency stimulation, and prevented the EPSP-spike

74 stioned because endogenous activation during high-frequency stimulation appears to have little impact

75 voked field EPSPs was induced selectively by high-frequency stimulation applied to the outer third of

76 demand and greater specificity than current high frequency stimulation approaches, and may lower the

77 We used high-frequency stimulation as a known activator of Ca(2+

78 The effects of high frequency stimulation at GP sites can be prevented

79 lly designed amplifier allowing simultaneous high frequency stimulation at therapeutic parameter sett

80 Upon high-frequency stimulation at 100 Hz, successive became

81 ction of presynaptic miniature release after high-frequency stimulation at Drosophila neuromuscular j

82 with both binomial statistical analysis and high-frequency stimulation at the amphibian neuromuscula

83 d electrically evoked synaptic responses for high-frequency stimulation at three synapses in the mous

84 ition of firing by 5-CT is diminished during high-frequency stimulation, because relief from synaptic

85 and theoretically, that the neurons support high-frequency stimulation beyond the inverse membrane t

86 Burst high-frequency stimulation (BHFS) (10 stimuli with 100 H

87 Interestingly, after chronic high-frequency stimulation, both the short- and long-ter

88 In response to high-frequency stimulation, both wild-type and mutant ne

89 itation (PPF) was enhanced; (2) responses to high frequency stimulation bursts were distorted; (3) lo

90 ed enhanced LTP in the hippocampal CA1 after high-frequency stimulation, but Ng-/- mice were affected

91 and induction of long-term potentiation with high-frequency stimulation, but not theta burst stimulat

92 o achieve results similar to STN lesions and high-frequency stimulation, but with fewer side effects.

93 ression reduced long-term potentiation after high-frequency stimulation compared with WT littermates

94 nced synaptic fatigue at CA1 synapses during high-frequency stimulation, compared with wild-type anim

95 indow limited to the first few minutes after high-frequency stimulation delivery.

96 h stellate cells and Purkinje cells, whereas high-frequency stimulation depresses stellate cell synap

97 onses of mitral and external tufted cells to high frequency stimulation did not originate presyaptica

98 High-frequency stimulation did not increase the overlap

99 maximized, and p alone is decreased, another high-frequency stimulation elicits additional LTP.

100 riggered synaptic transmission; furthermore, high-frequency stimulation elicits synaptic facilitation

101 calcium influx in presynaptic neurons during high-frequency stimulation, enhanced synaptic vesicle re

102 Brief, high-frequency stimulation evoked repetitive postsynapti

103 nhanced synaptic depression during prolonged high-frequency stimulation followed by delayed recovery.

104 During trains of high-frequency stimulation GABAA and GABAB receptors wor

105 It has been hypothesized that during high frequency stimulation glutamate a major neurotransm

106 charge transferred across the synapse during high frequency stimulations (>5 Hz).

107 itors that inhibited LTP when present during high-frequency stimulation had no effect on potentiated

108 blocked the stabilization of LTP induced by high frequency stimulation (HFS) in hippocampal slices.

109 everal laboratories have observed that brief high frequency stimulation (HFS) of cortical afferents i

110 tients, 31 sides) and then separately during high frequency stimulation (HFS) of the STN (16 patients

111 High frequency stimulation (HFS) of the STN improved tre

112 f inhibition of LTPs that were induced using high frequency stimulation (HFS), versus theta burst sti

113 N55212-2 and anandamide, but not cOA blocked high frequency stimulation (HFS)-LTP.

114 and 20 minutes following exposure to visual high-frequency stimulation (HFS) ( approximately 8.8 Hz,

115 erm changes in synaptic efficacy produced by high-frequency stimulation (HFS) of glutamatergic affere

116 High-frequency stimulation (HFS) of neocortical afferent

117 (LTP) of synaptic strength that results from high-frequency stimulation (HFS) of the afferent pathway

118 e activation of postsynaptic ASIC-1as during high-frequency stimulation (HFS) of the presynaptic nerv

119 glutamate is released within the STN during high-frequency stimulation (HFS) of the STN.

120 Our previous study showed that high-frequency stimulation (HFS) of the subthalamic nucl

121 long-term potentiation (LTP) after a single high-frequency stimulation (HFS) or two spaced HFSs at 1

122 Surprisingly, the same high-frequency stimulation (HFS) protocol induces presyn

123 tentiation (LTP) induced by the same type of high-frequency stimulation (HFS) that induces LTP at MF

124 mGluR7 activation during high-frequency stimulation (HFS) triggers presynaptic LT

125 /y) and Shank3B(-/-) lines, and administered high-frequency stimulation (HFS) via implanted electrode

126 degrees C (control group) and, 90 min later, high-frequency stimulation (HFS) was applied to the medi

127 synapses exhibited deficits in responses to high-frequency stimulation (HFS), as well as in long-ter

128 undergo long-term depression (LTD) following high-frequency stimulation (HFS), in contrast to MF-pyra

129 e clamping the postsynaptic cell soma during high-frequency stimulation (HFS), intracellular injectio

130 activity induced by K+ depolarization or by high-frequency stimulation (HFS), known to induce synaps

131 ion (LTP) induced in hippocampal area CA1 by high-frequency stimulation (HFS).

132 enhancement of synaptic transmission during high-frequency stimulation (HFS).

133 tenuation of the synaptic fatigue induced by high-frequency stimulation (HFS).

134 est the Ca2+ dependence of the VOCC, we used high-frequency stimulation (HFS).

135 FPL-LTD occludes LTD induced by high-frequency stimulation (HFS-LTD) and requires elevat

136 atrial fibrillation, endocardial continuous high frequency stimulation identified GP sites producing

137 paroxysmal atrial fibrillation, synchronized high frequency stimulation identified sites initiating p

138 CAP area declined more rapidly during high frequency stimulation if monocarboxylate transport

139 ronounced short-term depression (STD) during high-frequency stimulation; (ii) a conversion of that ST

140 der to potentiate with a burst of relatively high frequency stimulation in MeA-trained birds: the cha

141 f PFC responses resulting from vHipp and BLA high-frequency stimulation in adult rats that received r

142 of synaptic vesicle (SV) endocytosis during high-frequency stimulation in central nerve terminals.

143 Furthermore, high-frequency stimulation in Uva simultaneously suppres

144 However, high-frequency stimulation in Uva suppressed auditory-ev

145 Acetylcholine synapses are depressed by high-frequency stimulation, in part due to desensitizati

146 in a decrease in transmitter release during high-frequency stimulation, indicating an inhibition of

147 afts contracted when stimulated ex vivo, and high frequency stimulation induced tetanus.

148 In addition, high-frequency stimulation induced a consistent long-ter

149 Prolonged high-frequency stimulation induced NMDA receptor-depende

150 GRF1 can also mediate high frequency stimulation-induced LTP (HFS-LTP) in mice

151 cellular cleavage of proBDNF greatly reduced high-frequency stimulation-induced extracellular mBDNF.

152 High-frequency stimulation-induced long-term depression,

153 to a priming effect that enhanced subsequent high-frequency stimulation-induced LTP.

154 The facilitatory effects of the high-frequency stimulation-induced priming event itself

155 ease evoked by RGC activation and found that high-frequency stimulation induces a long-lasting subthr

156 High-frequency stimulation induces a transient calcium e

157 ibited long-term potentiation in response to high-frequency stimulation instead of the expected depre

158 High frequency stimulation is used to identify GP sites

159 We show that actin polymerization induced by high-frequency stimulation is blocked by local inhibitio

160 long-term potentiation (LTP) triggered by a high-frequency stimulation is facilitated in hypocretin

161 Chronic high-frequency stimulation is safe and highly effective

162 High-frequency stimulation leads to a transient increase

163 High-frequency stimulation leads to post-tetanic potenti

164 ABSTRACT: High-frequency stimulation leads to post-tetanic potenti

165 High frequency stimulation led to a significant reductio

166 targeted regional approach through localized high-frequency stimulation (LHFS) using low-amplitude el

167 Our findings suggest that high frequency stimulation may suppress pathologically e

168 ies have investigated how activity evoked by high-frequency stimulation may interact with the existin

169 that chronic ventromedial prefrontal cortex high-frequency stimulation may serve as a novel effectiv

170 Subsequently, the high frequency stimulation-mediated synaptic potentiatio

171 imal LG-S EPSP amplitude and the response to high-frequency stimulation (modulation) recorded in untr

172 High-frequency stimulation, multivesicular release (MVR)

173 ibited neither reliable phase-locking during high-frequency stimulation nor sensitivity to stimulus d

174 High frequency stimulation of mouse hippocampal slices r

175 High frequency stimulation of the afferents in vivo lead

176 High frequency stimulation of the cZI results in greater

177 d by action potential-evoked release (during high frequency stimulation of the mossy fibres), tonic i

178 synapses that are inactive, normally during high-frequency stimulation of a converging synaptic inpu

179 We examined the effect of high-frequency stimulation of a crucial component of the

180 We found that high-frequency stimulation of afferent fibers elicits sy

181 h to preserve stable AP waveforms, even upon high-frequency stimulation of chromaffin cells during st

182 of potentiation induced in vivo by repeated high-frequency stimulation of cortical association fiber

183 duced by either glutamate uptake blockade or high-frequency stimulation of corticostriatal tracts.

184 s pharmacologically prevented LTP induced by high-frequency stimulation of glutamatergic axons, or by

185 ause its activation usually requires strong, high-frequency stimulation of inputs.

186 High-frequency stimulation of mossy fibers induced long-

187 Here we demonstrate that high-frequency stimulation of mossy fibres in rat hippoc

188 ngs from dentate granule cells revealed that high-frequency stimulation of perforant path afferents i

189 iated synaptic depression, induced either by high-frequency stimulation of PF or mGluR1 agonist DHPG,

190 slices, we now demonstrate that conditioning high-frequency stimulation of primary afferents activate

191 -CA1 EPSPs, ADX71743 reversed the ability of high-frequency stimulation of SC afferents to reduce IPS

192 a manner that can be reversed by subsequent high-frequency stimulation of SC inputs.

193 on of long-term potentiation (LTP) evoked by high-frequency stimulation of Schaffer collaterals, and

194 High-frequency stimulation of thalamic inputs led to a l

195 nced the magnitude of dentate LTP induced by high-frequency stimulation of the angular bundle.

196 classes of neural progenitors, we found that high-frequency stimulation of the ATN increases symmetri

197 l timed postsynaptic action potential during high-frequency stimulation of the auditory nerve.

198 ious research has demonstrated that repeated high-frequency stimulation of the granule cell layer of

199 uces long-term potentiation (LTP) induced by high-frequency stimulation of the intracortical axons in

200 We also found that sustained, high-frequency stimulation of the locus coeruleus at fre

201 LTP was induced by high-frequency stimulation of the medial perforant path

202 Short bursts of high-frequency stimulation of the ML elicited Ca2+ trans

203 resent study aims to delineate the effect of high-frequency stimulation of the MLR (MLR-HFS) on gait

204 High-frequency stimulation of the perforant path project

205 on of their resting membrane potential after high-frequency stimulation of the perforant path.

206 the fidelity of synaptic transmission during high-frequency stimulation of the presynaptic cell.

207 ty (LTP-IE) of jcBNST neurons in response to high-frequency stimulation of the stria terminalis.

208 cal evidence of the therapeutic potential of high-frequency stimulation of the subthalamic nucleus (S

209 ery, primarily in the form of the bilateral, high-frequency stimulation of the subthalamic nucleus, i

210 performed a multicenter trial of unilateral high-frequency stimulation of the ventral intermedius nu

211 t, we report here that repeated low-current, high-frequency stimulation of the VTA provoked afterdisc

212 re involved in the mechanism of DBS, and how high-frequency stimulation of these cells may lead to al

213 High-frequency stimulation of these glutamatergic inputs

214 icostriatal long-term depression (LTD) after high-frequency stimulation of tissue from AC5KO mice.

215 In a rodent model, DBS-like high-frequency stimulation of VS can either enhance or i

216 ort, we describe the facilitatory effects of high-frequency stimulation on the induction of homosynap

217 on the elimination of vagal reflex evoked by high frequency stimulation or an extensive approach at a

218 (LTP) induction by either single or repeated high frequency stimulation or theta burst stimulation in

219 had no significant effect on LTP induced by high-frequency stimulation or long-term depression induc

220 nhibitors suppressed the induction of LTP by high-frequency stimulation or low-frequency stimulation

221 ctor, a neurotrophin that is released during high-frequency stimulation patterns used to elicit LTP.

222 In contrast, high-frequency stimulation preferentially increased extr

223 High-frequency stimulation produced sustained potentiati

224 al for long-term potentiation induced with a high frequency stimulation protocol (HFS-LTP) in the dor

225 Furthermore, high-frequency stimulation resulted in increased facilit

226 However, high-frequency stimulation revealed greatly elevated pos

227 , and enhanced use-dependent inactivation on high-frequency stimulation; S246L is likely a benign pol

228 t on potentiated synapses when applied after high-frequency stimulation, suggesting that PI3-kinase i

229 These findings suggest that high frequency stimulation suppresses epileptiform activ

230 However, when challenged with sustained high frequency stimulation, synapses in old mice exhibit

231 ced at both sites with either theta burst or high-frequency stimulation (TBS or HFS).

232 ess refractoriness, and improved response to high-frequency stimulation than dysmyelinated counterpar

233 spines, which can be reversed by subsequent high-frequency stimulation that induces LTP.

234 stration was so robust that after repetitive high-frequency stimulation the Ca content of responsive

235 During high-frequency stimulation, the greatest firing rate and

236 During high-frequency stimulation, the synapse undergoes short-

237 In response to high-frequency stimulation, the synaptic-vesicle pool ca

238 tion of late-phase LTP expression induced by high-frequency stimulation; the early phase of LTP is un

239 Furthermore, analysis of high-frequency stimulation trains suggests an impairment

240 nmol) into area CA3 15 min before delivering high-frequency stimulation (two times at 100 Hz, 1 sec).

241 that chronic ventromedial prefrontal cortex high-frequency stimulation upregulated neurogenesis-asso

242 es were achieved by daily amygdala kindling, high frequency stimulation was delivered to the SNr bila

243 Suppression of spontaneous activity by high frequency stimulation was found to be frequency (<

244 LTP was blocked when high-frequency stimulation was applied in the presence o

245 LTP of NMDA and AMPA EPSCs after high-frequency stimulation was reduced by prior inhibiti

246 lation that immediately follows LTP-inducing high-frequency stimulation, we wondered whether NMDAR-in

247 lobe, 111 (27%) low-frequency and 176 (55%) high-frequency stimulations were associated with a clini

248 quency facilitation tests and LTP induced by high-frequency stimulation when compared with untreated

249 itical for long-term potentiation induced by high-frequency stimulation, whereas proBDNF facilitate l

250 High-frequency stimulation, which evoked robust long-ter

251 stered the specific GR agonist RU 28362 that high-frequency stimulation, which normally produces LTP,

252 tached recordings, mitral cells responded to high frequency stimulation with sustained responses, whe

253 uch adaptation allows a neuron to respond to high-frequency stimulation with lower-frequency firing t