ライフサイエンスコーパス: high-grade prostate cancer

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1 otential risks (including the possibility of high-grade prostate cancer).

2 men with systemic progression and death from high grade prostate cancer.

3 or SAPC as a potential prognostic marker for high grade prostate cancer.

4 ovel gene whose expression is inactivated in high grade prostate cancer.

5 gnosis of low-risk and the underdiagnosis of high-grade prostate cancer.

6 3.0% and 73.6%, respectively, for those with high-grade prostate cancer.

7 also associated with increased detection of high-grade prostate cancer.

8 n-7, could be involved in the development of high-grade prostate cancer.

9 hived specimens from untreated patients with high-grade prostate cancer.

10 There was no increase in high-grade prostate cancer.

11 enic, and eicosapentaenoic acids and risk of high-grade prostate cancer.

12 pendently associated with risk of lethal and high-grade prostate cancer.

13 d with decreased risk of prostate cancer and high-grade prostate cancer.

14 frequent allelic loss and downregulation in high-grade prostate cancer.

15 exual side effects and the increased risk of high-grade prostate cancer.

16 with an increased risk of advanced-stage and high-grade prostate cancer.

17 c alterations of KLF6 occur in a minority of high-grade prostate cancers.

18 expression is dramatically down-regulated in high grade prostate cancers (4/4) but is unaltered in lo

19 e 270, 148, and 88 cases of total, low-, and high-grade prostate cancers among African American men a

20 ification of both alternative treatments for high-grade prostate cancers and new biomarkers to predic

21 rmethylation is enriched in intermediate- to high-grade prostate cancers and not detectable in benign

22 ty is associated with an increase in risk of high-grade prostate cancer but with a decrease in risk o

23 proanthocyanidins (P for trend = 0.04) with high-grade prostate cancer, but not with advanced prosta

24 Rather, there was improved detection of high-grade prostate cancer due to decreased prostate vol

25 g; these findings were highly suspicious for high-grade prostate cancer (Fig 1).

26 hould be weighed with the potential risks of high-grade prostate cancer from 5-ARIs (although the maj

27 stry analysis revealed elevation of PROS1 in high grade prostate cancer (Gleason grade >/= 8), and fu

28 ade (HR, 1.14; 95% CI, 1.01 to 1.29) but not high-grade prostate cancer (HR, 0.83; 95% CI, 0.64 to 1.

29 index was positively associated with risk of high-grade prostate cancer in both non-Hispanic white me

30 n the BCPT) or apparent risk of finasteride (high-grade prostate cancer in the PCPT).

31 ncidence (HR, 1.02; 95% CI, 0.96 to 1.08) or high-grade prostate cancer incidence (HR, 0.91; 95% CI,

32 nearly and inversely associated with risk of high-grade prostate cancer (quartile 4 vs. 1: TFA 18:1,

33 ds, flavan-3-ols, and proanthocyanidins with high-grade prostate cancer risk varied by follow-up time

34 and vitamin D intake and total, advanced, or high-grade prostate cancer risk, whether for total intak

35 vidually both for high stage (RR = 2.23) and high grade prostate cancer (RR = 1.89).

36 rved its significant (p < 0.05) elevation in high grade prostate cancer seminal plasma samples.

37 ARA70 expression is also increased in high-grade prostate cancer tissues as well as the hormon

38 may be associated with an increased risk of high-grade prostate cancer tumors that would ultimately

39 Hsp27 and Twist expression each elevated in high-grade prostate cancer tumors.

40 High-grade prostate cancer was more common in the finast

41 oreactivity was significantly upregulated in high-grade prostate cancer when compared to benign prost

42 ferentiating low-grade from intermediate- or high-grade prostate cancer with diffusion-weighted MR im

43 Outcomes are also disparate among men with high-grade prostate cancer, with upwards of 65% having d

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