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   1  had a lower incidence of palpable tumor and high-grade prostatic intraepithelial neoplasia.         
     2 in the first year following the diagnosis of high-grade prostatic intraepithelial neoplasia.         
     3 se AR expression as early as at the stage of high-grade prostatic intraepithelial neoplasia.         
     4 gland and gradually facilitates the onset of high-grade prostatic intraepithelial neoplasia.         
     5  5% of benign prostatic hyperplasias, 20% of high-grade prostatic intraepithelial neoplasias, 6% of T
     6 ade prostatic intraepithelial neoplasias and high-grade prostatic intraepithelial neoplasias among th
     7 mens examined by in situ analysis, including high-grade prostatic intraepithelial neoplasia and andro
     8 ficant increase in cathepsin H expression in high-grade prostatic intraepithelial neoplasia and carci
     9 s are capable of cell division, all cases of high-grade prostatic intraepithelial neoplasia and invas
    10 inversely correlated with Skp2 expression in high-grade prostatic intraepithelial neoplasia and PCa. 
    11 f positive nuclei decreased significantly in high-grade prostatic intraepithelial neoplasia and PCa. 
    12 s frequently increased in malignant cells of high-grade prostatic intraepithelial neoplasia and prost
    13 static epithelium, its expression is lost in high-grade prostatic intraepithelial neoplasia and prost
  
    15 ironment proteins may promote progression of high-grade prostatic intraepithelial neoplasia and/or pr
    16 a tissue microarray comprising 158 tumor, 18 high-grade prostatic intraepithelial neoplasia, and 91 n
    17 ession of EphA2 in prostatic adenocarcinoma, high-grade prostatic intraepithelial neoplasia, and adja
    18 xpressed by adenocarcinomas of the prostate, high-grade prostatic intraepithelial neoplasia, and four
    19 n of COX-2 in established prostate cancer or high-grade prostatic intraepithelial neoplasia, as compa
    20 PSA (HR = 3.90 for > 10 v 2.5 to 3.9 ng/mL), high-grade prostatic intraepithelial neoplasia/atypical 
    21 f treatment for "worried well" patients with high-grade prostatic intraepithelial neoplasia combined 
    22 arcinomas and the presumed precursor lesion (high-grade prostatic intraepithelial neoplasia) consiste
    23 ently, beginning at approximately 10 months, high-grade prostatic intraepithelial neoplasias develope
    24 he 12T-10 large probasin promoter-Tag mouse, high-grade prostatic intraepithelial neoplasia develops 
    25 undantly and ubiquitously expressed in human high-grade prostatic intraepithelial neoplasia, Gleason 
  
  
    28 mg in PCa prevention among men with isolated high-grade prostatic intraepithelial neoplasia (HGPIN) o
    29 motes marked acceleration and progression of high-grade prostatic intraepithelial neoplasia (HGPIN) t
    30 genic mouse models of Pca that progress from high-grade prostatic intraepithelial neoplasia (HGPIN) t
    31 cinogenesis, present in approximately 70% of high-grade prostatic intraepithelial neoplasia (high-gra
    32 in in histologically normal prostate glands, high-grade prostatic intraepithelial neoplasia, invasive
    33             Cell lines derived from low- and high-grade prostatic intraepithelial neoplasia, invasive
    34 n, 12%) was significantly lower than that in high-grade prostatic intraepithelial neoplasia (mean, 67
    35 w-grade prostatic intraepithelial neoplasia, high-grade prostatic intraepithelial neoplasia, microinv
  
    37  The risk of cancer following a diagnosis of high-grade prostatic intraepithelial neoplasia on needle
  
  
    40 an in benign prostatic tissue (P < 0.001) or high-grade prostatic intraepithelial neoplasia (P < 0.00
    41 clonal ERG rearrangements were found both in high grade prostatic intraepithelial neoplasia (PIN) and
    42 pared from tissue sample sections containing high grade prostatic intraepithelial neoplasia (PIN), bu
    43 ithelial neoplasia strongly resembling human high-grade prostatic intraepithelial neoplasia (PIN), a 
    44 ELISA of tissue extracts of normal prostate, high-grade prostatic intraepithelial neoplasia (PIN), an
  
    46 of 13 MCRs in the putative precursor lesion, high-grade prostatic intraepithelial neoplasia (PIN), sh
  
    48 ostate tissue, benign prostatic hyperplasia, high-grade prostatic intraepithelial neoplasia, primary 
    49 late with Gleason score, their occurrence in high-grade prostatic intraepithelial neoplasia, suggests
    50 t mice expressing LT, tumors progressed from high-grade prostatic intraepithelial neoplasia to poorly
  
    52 of Pten loss, with early neoplastic lesions (high-grade prostatic intraepithelial neoplasia) with str
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