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1 tics of human osteosarcomas, including being highly metastatic.
2 e C-alpha (PKC-alpha), is both malignant and highly metastatic.
3 strogen unresponsive, fully tumorigenic, and highly metastatic.
4                     ARCaP is tumorigenic and highly metastatic.
5                   Colorectal cancer (CRC) is highly metastatic.
6 ancers arising from neuroendocrine cells are highly metastatic.
7  ablation, chemo- and radiotherapy, but also highly metastatic.
8             PKCdelta levels are increased in highly metastatic 13762NF mammary tumor cells (MTLn3) co
9                   In vitro incubation of the highly metastatic 253J B-V cells and the IFN-alpha-resis
10 able to suppress the metastatic phenotype in highly metastatic 4T1 and MDA-MB-231 SCP28 cells, as wel
11 tand the chemoresistance mechanism using the highly metastatic 4T1 breast cancer model, which emulate
12 is associated with doxorubicin resistance of highly metastatic 4T1 breast cancer.
13 py was tested against weakly immunogenic and highly metastatic 4T1 breast tumor using SU6668, an angi
14 y, cleaved caspase-3 decreased by 45% in the highly metastatic 4T1 cells after hypoxia.
15 e nonmetastatic 67NR cells and lowest in the highly metastatic 4T1 cells.
16 C expression by small interfering RNA in the highly metastatic 4T1 mammary tumor cell line expressing
17 erapy strategy in the weakly immunogenic and highly metastatic 4T1 murine mammary cancer model.
18                                       In the highly metastatic A375-M human melanoma cells, p190-RhoC
19                    Transfection of AP-2 into highly metastatic A375SM melanoma cells (AP-2-negative a
20         First, the PLD2 gene was silenced in highly metastatic, aggressive breast cancer cells (MDA-M
21                                          The highly metastatic amelanotic C8161 human melanoma line w
22 o a transition from androgen-dependence to a highly metastatic and androgen refractory (androgen depl
23  (TN) breast cancers (ER(-)PR(-)HER2(-)) are highly metastatic and associated with poor prognosis.
24          Pancreatic ductal adenocarcinoma is highly metastatic and current preoperative evaluation of
25                       Because the disease is highly metastatic and difficult to diagnosis until late
26 lectively knock down GnT-V expression in the highly metastatic and invasive human breast carcinoma ce
27                   Cells lacking vinculin are highly metastatic and motile.
28 xpressed pluripotency-associated genes, were highly metastatic and showed long-term in vivo tumorigen
29 cer cell lines and selected subpopulation of highly metastatic and tumorigenic cells (ALDH(high)) str
30 hotopic growth and spontaneous metastasis of highly metastatic, androgen-insensitive caveolin-1-secre
31 ane glycoprotein found on the surface of the highly metastatic ascites 13762 rat mammary adenocarcino
32  abundantly expressed on the cell surface of highly metastatic ascites 13762 rat mammary adenocarcino
33 viral Gag-like protein p58gag expressed in a highly metastatic ascites rat mammary adenocarcinoma has
34 f the Mr 85,000 standard form of CD44 in the highly metastatic AT3.1 rat prostatic cells greatly supp
35  containing this region was transferred into highly metastatic AT6.3 rat prostate cancer cells by mic
36 f B16 melanoma and at much reduced levels in highly metastatic B16 variants.
37  that it was substantially down-regulated in highly metastatic B16-F10 melanoma cells, which contribu
38                   In the poorly immunogenic, highly metastatic, B16/F10.9 tumor model a dramatic redu
39 d SENP7L levels lessens the dissemination of highly metastatic BCa cells to the lungs from primary im
40                                Here, using a highly metastatic breast cancer (4T1) model, we show tha
41  breast carcinoma and on the cell surface of highly metastatic breast cancer cell line MDA-MB-231.
42 re linked to MCT expression in MDA-MB-231, a highly metastatic breast cancer cell line.
43 e show that the loss of KiSS-1 expression in highly metastatic breast cancer cell lines correlates di
44 e 2, I-branching enzyme, is overexpressed in highly metastatic breast cancer cell lines of human and
45 type plasminogen activator, and cytokines in highly metastatic breast cancer cell lines.
46                The restoration of miR-203 in highly metastatic breast cancer cells inhibited tumor ce
47 onditioning of naive mice with exosomes from highly metastatic breast cancer cells revealed the accum
48               These genes are upregulated in highly metastatic breast cancer cells, and their increas
49 verexpression of protein kinase Calpha), and highly metastatic breast cancer cells.
50  miR-203 was significantly down-regulated in highly metastatic breast cancer cells.
51  we found that stable SDPR overexpression in highly metastatic breast cancer model cell lines inhibit
52 on of hyaluronan synthase 2 (HAS2) occurs in highly metastatic breast cancer stem-like cells (CSC) de
53             Overexpression of AP-2alpha into highly metastatic breast cell lines did not alter KiSS-1
54  is greater than that in normal tissue, with highly metastatic breast epithelial cells expressing the
55  Furthermore, blockade of activated Stat3 in highly metastatic C4 cells significantly suppressed the
56 e expression of a dominant-negative Stat3 in highly metastatic C4 tumor cells inhibited the MMP-2 pro
57 K-1735 melanoma system, we demonstrated that highly metastatic C4, M2, and X21 tumor cells express el
58 l that Trp consumption and Kyn production by highly metastatic cancer cells (HT29) were significantly
59 and invasion and that MTA1 overexpression in highly metastatic cancer cells drives cell migration and
60                                          For highly metastatic cancer cells, the pH measured at the s
61 cells from hepatocellular carcinoma (HCC), a highly metastatic cancer, undergo epithelial to amoeboid
62                           In conclusion, the highly metastatic capability of a unique TS subpopulatio
63 y a poorly metastatic cell line to that by a highly metastatic cell line 24 h after injection in the
64 -2 mRNA stabilization in MDA-MB-231 cells, a highly metastatic cell line derived from a human mammary
65 ential reversal of oncogenic properties of a highly metastatic cell line with the introduction of non
66 ially expressed (greater than 2-fold) in all highly metastatic cell lines relative to their reference
67                        The undifferentiated, highly metastatic cell lines with high metastatic potent
68               We found that in vivo selected highly metastatic cell populations showed little genetic
69                                              Highly metastatic cells (MDA-MB-435) expressing high lev
70              Within minutes of adhesion, the highly metastatic cells acquire the ability of enhanced
71 ere we show that loss of c-KIT expression in highly metastatic cells correlates with loss of expressi
72 at up-regulation of MCAM/MUC18 expression in highly metastatic cells correlates with loss of expressi
73 ration of miR-10b antagomirs to mice bearing highly metastatic cells does not reduce primary mammary
74                                              Highly metastatic cells evade this tumor-suppressive pat
75 ompared with poorly metastatic cancer cells, highly metastatic cells expressed increased levels of th
76                                              Highly metastatic cells growing in culture constitutivel
77                                              Highly metastatic cells growing in the prostate expresse
78 usly shown that enforced c-KIT expression in highly metastatic cells inhibited tumor growth and metas
79                 Silencing of IL-13Ralpha2 in highly metastatic cells led to a decrease in adhesion ca
80            Does it arise from rare, variant, highly metastatic cells or does a primary tumor progress
81                      Introduction of Psap in highly metastatic cells significantly reduced the occurr
82                               Loss of CC3 in highly metastatic cells such as SCLC might render them r
83                                              Highly metastatic cells survived to produce numerous lun
84                                     When the highly metastatic cells were compared with their low met
85                    The pH at the surfaces of highly metastatic cells within tumors was found to be ab
86  kinase expression was first demonstrated in highly metastatic cells, whilst re-expression of the pro
87 elta levels were relatively increased in the highly metastatic cells.
88  transcripts displaying reduced stability in highly metastatic cells.
89 gainst poorly metastatic cells compared with highly metastatic cells.
90 ignificantly impairs the bone progression of highly metastatic cells.
91 s phosphoinositide 3-kinase, AKT, and SRC in highly metastatic cells.
92 n upon miR-200 overexpression toward that of highly metastatic cells.
93 etastatic variants, suggest that not only do highly-metastatic cells display constitutively elevated
94 g ligand activation of the EGFR, but only in highly-metastatic cells.
95 ecause they preferentially activate c-Src in highly-metastatic cells.
96 ces neoplastic transformation and promotes a highly metastatic cellular phenotype.
97 ch, in turn, are likely to contribute to the highly metastatic character of NPC.
98                Overexpression of NOS II in a highly metastatic clone by transfection with NOS II gene
99                                            A highly metastatic clone of K1735 cells, SW1-C, and its s
100                 Furthermore, both poorly and highly metastatic clones contained an identical p53 muta
101                                              Highly metastatic clones exhibited higher levels of NOS
102 rays to analyze the secretomes of poorly and highly metastatic colorectal cancer cells.
103            Suppression of Akt2 expression in highly metastatic colorectal carcinoma cells inhibits th
104 e type II TGF-beta receptor (PyMT(mgko)) are highly metastatic compared with control PyMT-induced car
105 noma (NPC), an EBV-associated malignancy, is highly metastatic compared with other head and neck tumo
106 d nitrogen radicals that kill weakly but not highly metastatic CRC cells.
107                                       In the highly metastatic CT26 murine colon cancer cell line, wh
108                          Nearly 10-fold more highly metastatic CX-1 cells survive within the livers o
109 lone A and MIP-101 cells at 24 h but <15% of highly metastatic CX-1 cells.
110 ne A and MIP-101 CRC cells die, whereas many highly metastatic CX-1 CRC cells survive.
111 ndent cells but was significantly reduced in highly metastatic derivative clones.
112 an prostate carcinoma cell line PC-3 and its highly metastatic derivative PC-3M.
113                         Ovarian cancer is an highly metastatic disease characterized by ascites forma
114 s evaluated, all were demonstrated to effect highly metastatic disease involving multiple organs, alt
115 pe of a murine model of pancreatic cancer, a highly metastatic disease that frequently displays p53 m
116                     Ovarian cancer (OC) is a highly metastatic disease, but no effective strategies t
117                          Ovarian cancer is a highly metastatic disease.
118                                              Highly metastatic Dunning AT3.1 rat prostate cancer cell
119 eomycin resistance gene, was introduced into highly metastatic Dunning AT6.1 prostate cancer cells by
120 xpression of a dominant-negative CDK5 in the highly metastatic Dunning AT6.3 prostate cancer cell lin
121 results in metastasis suppression in certain highly metastatic Dunning R-3327 rat prostatic cancer su
122 er to introduce human chromosome 16 into the highly metastatic Dunning rat prostatic cancer cell line
123 inally, EGFR-MET signaling was enhanced in a highly metastatic EGFR-mutant cell subpopulation, compar
124 minyltransferase (LARGE) gene in a cohort of highly metastatic epithelial cell lines derived from bre
125               This resulted in generation of highly metastatic epithelial-to-mesenchymal transition-l
126 53(R172H) mutation were undifferentiated and highly metastatic, exhibited minimal TP53 transactivatio
127 e and to an in vivo-derived subline that was highly metastatic for growth in the lungs.
128                            Osteosarcoma is a highly metastatic form of bone cancer in adolescents and
129 criptional regulation of the MMP-9 gene in a highly metastatic H-ras and v-myc transformed rat embryo
130                                              Highly metastatic H7 murine pancreatic adenocarcinoma ce
131                 Another cell line, UMRC3, is highly metastatic, having lost TBR3 and TBR2 expression.
132 xpression of TIMP2 open reading frame in the highly metastatic HCC cell line, MHCC-97L, significantly
133 votal role in regulating RUNX2 expression in highly metastatic HNSCC cells, where it was downregulate
134 ricle injection into nude mice to identify a highly metastatic human breast cancer cell line (MDA-MET
135 oRNA-146b (miR-146a/b) when expressed in the highly metastatic human breast cancer cell line MDA-MB-2
136  functionally relevant betaAR subtype in the highly metastatic human breast cancer cell line MDA-MB-2
137 timigratory and antiinvasive effects against highly metastatic human breast cancer MDA-MB-231 cells v
138 in 3F (SEMA3F) was markedly downregulated in highly metastatic human cell lines in vitro and in vivo,
139              MUC2 levels were manipulated in highly metastatic human colon cancer cells using eukaryo
140 nst proteins preferentially expressed by the highly metastatic human epidermoid carcinoma cell line,
141 eLa (human cervical cancer), HCI-H460-LNM35 (highly metastatic human lung cancer) and B16 (murine mel
142 tisense cathepsin B-expressing clones of the highly metastatic human melanoma A375M and prostate carc
143 ected the c-KIT gene into the c-KIT negative highly metastatic human melanoma cell line A375SM and su
144 al human melanocyte cell line and weakly and highly metastatic human melanoma cell lines, we presentl
145                      Medium conditioned by a highly metastatic human pancreatic cancer cell line BxPC
146 i-amino-C1-C3-alkane-sulfonic acid), against highly metastatic human pancreatic carcinoma cells injec
147                                              Highly metastatic human pancreatic carcinoma L3.6pl cell
148                   The highly tumorigenic and highly metastatic human transitional cell carcinoma (TCC
149 nvasive, murine colon cancer cells that were highly metastatic in an immunocompetent mouse model to i
150          The primary neoplasm generated is a highly metastatic islet cell carcinoma of the pancreas.
151 nscript stability measurements in poorly and highly metastatic isogenic human breast cancer lines.
152   Evidence is now provided, using weakly and highly metastatic isogenic melanoma variants, that mda-9
153                                  CD44 on the highly metastatic KM12L4 clone is more heavily substitut
154                                 In addition, highly metastatic L3.6pl cells growing in the pancreas e
155 th transformation of SB2 melanoma cells to a highly metastatic line.
156 cally defective liver stem cells (LSCs) into highly metastatic liver cancer cells in premalignant liv
157 xA also reduced motility and invasiveness of highly metastatic LOX melanoma cells.
158                          Mucin purified from highly metastatic LS-Lim6 human colon cancer cells bound
159 ated with the Epstein-Barr virus (EBV), is a highly metastatic malignant tumor.
160 e expression of CXCR4 in murine 4T1 cells, a highly metastatic mammary cancer cell line that is a mod
161           Suppression of Twist expression in highly metastatic mammary carcinoma cells specifically i
162 early completely inhibits lung metastasis of highly metastatic mammary carcinoma cells.
163       We define ECM signatures of poorly and highly metastatic mammary carcinomas and these signature
164                                      Using a highly metastatic mammary tumor cell line that expresses
165             In the mouse, the development of highly metastatic mammary tumors is associated with an a
166 icantly, the HMG-Y protein isolated from the highly metastatic MCF-7/PKC-alpha cells possesses a uniq
167 tion of methylation reduces migration of the highly metastatic MDA-MB-231 breast cancer cell line.
168 gnificantly increased the penetration of the highly metastatic MDA-MB-231 breast cancer cells across
169  20S proteasome and 26S proteasome in intact highly metastatic MDA-MB-231 breast cancer cells, result
170 urthermore, forced expression of KLF4 in the highly metastatic MDA-MB-231 breast tumor cell line was
171 oduced a normal human chromosome 11 into the highly metastatic MDA-MB-435 breast carcinoma cell line
172 ry matrix supported motility and invasion in highly metastatic MDA-MB-435 cells, but not in cells wit
173 s found to suppress motility and invasion in highly metastatic MDA-MB-435 cells, whereas involution m
174 nes, nor was a difference observed between a highly metastatic melanoma cell line (A375SM) or its par
175  progression, we conducted a microarray on a highly metastatic melanoma cell line in which NFAT1 expr
176 ated receptor-1 (PAR-1)] is overexpressed in highly metastatic melanoma cell lines and in patients wi
177 wth (P < 0.01) and metastasis (P < 0.001) of highly metastatic melanoma cell lines in vivo.
178  showed that inducible expression of CD82 in highly metastatic melanoma cells significantly increased
179    Forced expression of TRAF2DeltaN in HHMSX highly metastatic melanoma cells that lack Fas expressio
180 on of the invasive and migratory behavior in highly metastatic melanoma cells, similar to the overexp
181 we use an in vivo selection scheme to select highly metastatic melanoma cells.
182 anocytes, we observed variably pigmented and highly metastatic melanoma with 100% penetrance.
183                                Exosomes from highly metastatic melanomas increased the metastatic beh
184  using retroviral vectors in EpRas cells and highly metastatic mesenchymal mouse colon carcinoma cell
185                                              Highly metastatic MeWo human melanoma cells were transfe
186                                        It is highly metastatic, migrating through lymph nodes to dist
187             To explore this possibility, the highly metastatic MMTV-PyMT mice were crossed with 25 AK
188                                 Furthermore, highly metastatic MNNG-HOS cells have increased levels o
189                                      Using a highly metastatic model of mammary cancer, we identified
190                       In vitro analysis of a highly metastatic mouse mammary tumor cell line ectopica
191                         Cells derived from a highly metastatic mouse model of SCCHN were used to conf
192             The two cell lines used were the highly metastatic MTLn3 cells and nonmetastatic MTC cell
193 erinuclear and at the leading edge), whereas highly metastatic MTLn3 cells have only a perinuclear di
194 beta 2m protein and RNA are not expressed in highly metastatic, multidrug-resistant MCF-7/Adr cells w
195  fluorescently labeled exosomes derived from highly metastatic murine breast cancer cells distributed
196 ting angiogenesis and lymphangiogenesis in a highly metastatic murine model of Burkitt's lymphoma (E
197                                    PANC02-H7 highly metastatic murine pancreatic adenocarcinoma cells
198  vivo gene therapy modalities to counter the highly metastatic nature of human melanoma.
199 any potential link between the virus and the highly metastatic nature of MCC.
200 k between MCPyV T antigen expression and the highly metastatic nature of MCC.
201 nduce apoptosis of human melanomas including highly metastatic ones despite their low surface Fas lev
202 in the majority of osteosarcoma cases and in highly metastatic osteosarcoma cell lines.
203  metastasis, poorly metastatic Panc02-H0 and highly metastatic Panc02-H7 cells were injected into the
204 B with an oncogenic Kras allele, we observed highly metastatic pancreatic adenocarcinomas.
205 ciency cooperates with Kras(G12D) to promote highly metastatic pancreatic cancer.
206                    We then demonstrated in a highly metastatic pancreatic mouse tumor model (Panc-02)
207 lencing MUC4 expression in an aggressive and highly metastatic pancreatic tumor cell line CD18/HPAF t
208 t case, the invasive ascents of the Tepui by highly metastatic PC-3 and noninvasive LNCaP prostate ca
209 nt LNCaP, hormone-independent DU145, PC-3 to highly metastatic PC-3M cancer cell lines.
210                                              Highly metastatic PC-3M human prostate cancer cells were
211                                              Highly metastatic PC-3M human prostate cancer cells were
212                                              Highly metastatic PC3 and PC3M-LN4 cells were found to a
213 poptosis in poorly metastatic PC3 M-Pro4 and highly metastatic PC3 M-LN4 subclones demonstrated that
214                                       In the highly metastatic PC3-MM2 cells, expression of a non-pho
215 sitively regulates E-cadherin and suppresses highly metastatic PCA cell invasion by modulating Rho pa
216 that miR-25 can act as a tumor suppressor in highly metastatic PCSCs by direct functional interaction
217          In a mouse model of osteosarcoma, a highly metastatic pediatric cancer, we found ezrin to be
218 ggest a possible molecular mechanism for the highly metastatic phenotype associated with MCC.
219 beta4 integrin was sufficient to revert this highly metastatic phenotype in the MNNG-HOS model withou
220 wth factor (EGF) receptor (EGFR) indicates a highly metastatic phenotype of breast cancer.
221 vels of GLI1 are likely to contribute to the highly metastatic phenotype of PDAC.
222 ogressively lost as the cells take on a more highly metastatic phenotype.
223 onsive, estrogen receptor (ER) negative, and highly metastatic phenotype.
224 s of proteases and angiogenic factors, and a highly metastatic phenotype.
225 opically to regulate an undifferentiated and highly metastatic phenotype.
226 dermal growth factor receptor)-positive, and highly metastatic phenotype.
227 oma (PDAC), a cancer type characterized by a highly metastatic phenotype.
228                             p27 knockdown in highly metastatic PI3K-activated cells reduces STAT3 bin
229                  Rapid, specific adhesion of highly metastatic prostate adenocarcinoma cells (PC3M-LN
230 n at both the mRNA and protein levels in the highly metastatic prostate cancer cell line PC3.
231 st completely inhibited lung colonization of highly metastatic prostate cancer cells without affectin
232 tional regulator, is abnormally expressed in highly metastatic prostate cancer cells.
233                                 In contrast, highly metastatic prostate carcinoma cells were resistan
234  pluripotency in embryonic stem cells and in highly metastatic prostate tumors.
235 ersely, recombinant expression of Cav-1 in a highly metastatic PyMT mammary carcinoma-derived cell li
236 were transfected and stably expressed in the highly metastatic rat Dunning MAT-LyLu prostate cancer c
237 d with lycopene against proliferation of the highly metastatic rat prostate adenocarcinoma MAT-LyLu c
238  SPARC selectively supports the migration of highly metastatic relative to less metastatic prostate c
239                       We used J774 tumors (a highly metastatic reticulum cell sarcoma line) and PDT w
240 r to introduce normal human chromosomes into highly metastatic rodent prostatic cancer cells to map t
241 mplete inhibition on invasion (P < 0.001) of highly metastatic SiHa cells via reduced transcriptional
242 6.1, without metastasis suppression in other highly metastatic sublines, such as AT3.1.
243 in-7 expression in poorly differentiated and highly metastatic SW620 colon cancer cells induced epith
244 ong epithelial gene program were enriched in highly metastatic TICs, while a second subpopulation wit
245  for these cells revealed that CSCs that are highly metastatic to bone and brain expressed significan
246 -MB cells, a breast cancer cell line that is highly metastatic to bone.
247                  A murine melanoma cell line highly metastatic to lymph nodes (B16F10) was implanted
248 , FAK expression is increased in a number of highly metastatic tumor cell lines.
249 ate that secretion of inhibitory TFPI-2 by a highly metastatic tumor cell markedly inhibits its growt
250  within extracellular vesicles secreted from highly metastatic tumor cells can be internalized by wea
251        In vivo tumor modeling studies with a highly metastatic tumor line, PC-3ML cells, revealed tha
252 titumor efficacy and overall survival in two highly metastatic tumor models.
253 rate that several proteins characteristic of highly metastatic tumors (LTBP3, SNED1, EGLN1, and S100A
254 ant organs, whereas inhibition of miR-105 in highly metastatic tumors alleviates these effects.
255 n, which can be defeated in the evolution of highly metastatic tumors by combined loss of both p66(Sh
256 sing a mouse model of metastasis reveal that highly metastatic tumors express proteolyzed cyclin E an
257                  Instead, these mice develop highly metastatic tumors of neuroendocrine, not epitheli
258 the control clones produced rapidly growing, highly metastatic tumors within 2 wk of inoculation on c
259 oss in the mouse lung to promote aggressive, highly metastatic tumors, that are initially sensitive t
260  we found that 4.1B expression is reduced in highly metastatic tumors.
261 or discrimination between non-metastatic and highly metastatic tumors.
262 e human-human somatic cell fusions between a highly metastatic, undifferentiated, ER-negative line of
263 rom the compound mutant prostates, which was highly metastatic upon orthotopic transplantation.
264 o search for metastasis-related mutations in highly metastatic uveal melanomas of the eye.
265 an prostate cancer cell line LNCaP, with its highly metastatic variant LNCaP-LN3, by two-dimensional
266 man breast carcinoma MDA-MB-435-F-L cells, a highly metastatic variant of human breast cancer MDA-MB-
267 c GI101A human breast cancer cell line and a highly metastatic variant, GILM2.
268 y be essential for this process, we isolated highly metastatic variants from a poorly metastatic huma
269 ic vs. immunosuppressed newborn rat-selected highly metastatic variants.
270                          However, HCT116 was highly metastatic with 68% metastasis observed in liver

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