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1 tics of human osteosarcomas, including being highly metastatic.
2 e C-alpha (PKC-alpha), is both malignant and highly metastatic.
3 strogen unresponsive, fully tumorigenic, and highly metastatic.
4 ARCaP is tumorigenic and highly metastatic.
5 Colorectal cancer (CRC) is highly metastatic.
6 ancers arising from neuroendocrine cells are highly metastatic.
7 ablation, chemo- and radiotherapy, but also highly metastatic.
10 able to suppress the metastatic phenotype in highly metastatic 4T1 and MDA-MB-231 SCP28 cells, as wel
11 tand the chemoresistance mechanism using the highly metastatic 4T1 breast cancer model, which emulate
13 py was tested against weakly immunogenic and highly metastatic 4T1 breast tumor using SU6668, an angi
16 C expression by small interfering RNA in the highly metastatic 4T1 mammary tumor cell line expressing
22 o a transition from androgen-dependence to a highly metastatic and androgen refractory (androgen depl
23 (TN) breast cancers (ER(-)PR(-)HER2(-)) are highly metastatic and associated with poor prognosis.
26 lectively knock down GnT-V expression in the highly metastatic and invasive human breast carcinoma ce
28 xpressed pluripotency-associated genes, were highly metastatic and showed long-term in vivo tumorigen
29 cer cell lines and selected subpopulation of highly metastatic and tumorigenic cells (ALDH(high)) str
30 hotopic growth and spontaneous metastasis of highly metastatic, androgen-insensitive caveolin-1-secre
31 ane glycoprotein found on the surface of the highly metastatic ascites 13762 rat mammary adenocarcino
32 abundantly expressed on the cell surface of highly metastatic ascites 13762 rat mammary adenocarcino
33 viral Gag-like protein p58gag expressed in a highly metastatic ascites rat mammary adenocarcinoma has
34 f the Mr 85,000 standard form of CD44 in the highly metastatic AT3.1 rat prostatic cells greatly supp
35 containing this region was transferred into highly metastatic AT6.3 rat prostate cancer cells by mic
37 that it was substantially down-regulated in highly metastatic B16-F10 melanoma cells, which contribu
39 d SENP7L levels lessens the dissemination of highly metastatic BCa cells to the lungs from primary im
41 breast carcinoma and on the cell surface of highly metastatic breast cancer cell line MDA-MB-231.
43 e show that the loss of KiSS-1 expression in highly metastatic breast cancer cell lines correlates di
44 e 2, I-branching enzyme, is overexpressed in highly metastatic breast cancer cell lines of human and
47 onditioning of naive mice with exosomes from highly metastatic breast cancer cells revealed the accum
51 we found that stable SDPR overexpression in highly metastatic breast cancer model cell lines inhibit
52 on of hyaluronan synthase 2 (HAS2) occurs in highly metastatic breast cancer stem-like cells (CSC) de
54 is greater than that in normal tissue, with highly metastatic breast epithelial cells expressing the
55 Furthermore, blockade of activated Stat3 in highly metastatic C4 cells significantly suppressed the
56 e expression of a dominant-negative Stat3 in highly metastatic C4 tumor cells inhibited the MMP-2 pro
57 K-1735 melanoma system, we demonstrated that highly metastatic C4, M2, and X21 tumor cells express el
58 l that Trp consumption and Kyn production by highly metastatic cancer cells (HT29) were significantly
59 and invasion and that MTA1 overexpression in highly metastatic cancer cells drives cell migration and
61 cells from hepatocellular carcinoma (HCC), a highly metastatic cancer, undergo epithelial to amoeboid
63 y a poorly metastatic cell line to that by a highly metastatic cell line 24 h after injection in the
64 -2 mRNA stabilization in MDA-MB-231 cells, a highly metastatic cell line derived from a human mammary
65 ential reversal of oncogenic properties of a highly metastatic cell line with the introduction of non
66 ially expressed (greater than 2-fold) in all highly metastatic cell lines relative to their reference
71 ere we show that loss of c-KIT expression in highly metastatic cells correlates with loss of expressi
72 at up-regulation of MCAM/MUC18 expression in highly metastatic cells correlates with loss of expressi
73 ration of miR-10b antagomirs to mice bearing highly metastatic cells does not reduce primary mammary
75 ompared with poorly metastatic cancer cells, highly metastatic cells expressed increased levels of th
78 usly shown that enforced c-KIT expression in highly metastatic cells inhibited tumor growth and metas
86 kinase expression was first demonstrated in highly metastatic cells, whilst re-expression of the pro
93 etastatic variants, suggest that not only do highly-metastatic cells display constitutively elevated
104 e type II TGF-beta receptor (PyMT(mgko)) are highly metastatic compared with control PyMT-induced car
105 noma (NPC), an EBV-associated malignancy, is highly metastatic compared with other head and neck tumo
114 s evaluated, all were demonstrated to effect highly metastatic disease involving multiple organs, alt
115 pe of a murine model of pancreatic cancer, a highly metastatic disease that frequently displays p53 m
119 eomycin resistance gene, was introduced into highly metastatic Dunning AT6.1 prostate cancer cells by
120 xpression of a dominant-negative CDK5 in the highly metastatic Dunning AT6.3 prostate cancer cell lin
121 results in metastasis suppression in certain highly metastatic Dunning R-3327 rat prostatic cancer su
122 er to introduce human chromosome 16 into the highly metastatic Dunning rat prostatic cancer cell line
123 inally, EGFR-MET signaling was enhanced in a highly metastatic EGFR-mutant cell subpopulation, compar
124 minyltransferase (LARGE) gene in a cohort of highly metastatic epithelial cell lines derived from bre
126 53(R172H) mutation were undifferentiated and highly metastatic, exhibited minimal TP53 transactivatio
129 criptional regulation of the MMP-9 gene in a highly metastatic H-ras and v-myc transformed rat embryo
132 xpression of TIMP2 open reading frame in the highly metastatic HCC cell line, MHCC-97L, significantly
133 votal role in regulating RUNX2 expression in highly metastatic HNSCC cells, where it was downregulate
134 ricle injection into nude mice to identify a highly metastatic human breast cancer cell line (MDA-MET
135 oRNA-146b (miR-146a/b) when expressed in the highly metastatic human breast cancer cell line MDA-MB-2
136 functionally relevant betaAR subtype in the highly metastatic human breast cancer cell line MDA-MB-2
137 timigratory and antiinvasive effects against highly metastatic human breast cancer MDA-MB-231 cells v
138 in 3F (SEMA3F) was markedly downregulated in highly metastatic human cell lines in vitro and in vivo,
140 nst proteins preferentially expressed by the highly metastatic human epidermoid carcinoma cell line,
141 eLa (human cervical cancer), HCI-H460-LNM35 (highly metastatic human lung cancer) and B16 (murine mel
142 tisense cathepsin B-expressing clones of the highly metastatic human melanoma A375M and prostate carc
143 ected the c-KIT gene into the c-KIT negative highly metastatic human melanoma cell line A375SM and su
144 al human melanocyte cell line and weakly and highly metastatic human melanoma cell lines, we presentl
146 i-amino-C1-C3-alkane-sulfonic acid), against highly metastatic human pancreatic carcinoma cells injec
149 nvasive, murine colon cancer cells that were highly metastatic in an immunocompetent mouse model to i
151 nscript stability measurements in poorly and highly metastatic isogenic human breast cancer lines.
152 Evidence is now provided, using weakly and highly metastatic isogenic melanoma variants, that mda-9
156 cally defective liver stem cells (LSCs) into highly metastatic liver cancer cells in premalignant liv
160 e expression of CXCR4 in murine 4T1 cells, a highly metastatic mammary cancer cell line that is a mod
166 icantly, the HMG-Y protein isolated from the highly metastatic MCF-7/PKC-alpha cells possesses a uniq
167 tion of methylation reduces migration of the highly metastatic MDA-MB-231 breast cancer cell line.
168 gnificantly increased the penetration of the highly metastatic MDA-MB-231 breast cancer cells across
169 20S proteasome and 26S proteasome in intact highly metastatic MDA-MB-231 breast cancer cells, result
170 urthermore, forced expression of KLF4 in the highly metastatic MDA-MB-231 breast tumor cell line was
171 oduced a normal human chromosome 11 into the highly metastatic MDA-MB-435 breast carcinoma cell line
172 ry matrix supported motility and invasion in highly metastatic MDA-MB-435 cells, but not in cells wit
173 s found to suppress motility and invasion in highly metastatic MDA-MB-435 cells, whereas involution m
174 nes, nor was a difference observed between a highly metastatic melanoma cell line (A375SM) or its par
175 progression, we conducted a microarray on a highly metastatic melanoma cell line in which NFAT1 expr
176 ated receptor-1 (PAR-1)] is overexpressed in highly metastatic melanoma cell lines and in patients wi
178 showed that inducible expression of CD82 in highly metastatic melanoma cells significantly increased
179 Forced expression of TRAF2DeltaN in HHMSX highly metastatic melanoma cells that lack Fas expressio
180 on of the invasive and migratory behavior in highly metastatic melanoma cells, similar to the overexp
184 using retroviral vectors in EpRas cells and highly metastatic mesenchymal mouse colon carcinoma cell
193 erinuclear and at the leading edge), whereas highly metastatic MTLn3 cells have only a perinuclear di
194 beta 2m protein and RNA are not expressed in highly metastatic, multidrug-resistant MCF-7/Adr cells w
195 fluorescently labeled exosomes derived from highly metastatic murine breast cancer cells distributed
196 ting angiogenesis and lymphangiogenesis in a highly metastatic murine model of Burkitt's lymphoma (E
201 nduce apoptosis of human melanomas including highly metastatic ones despite their low surface Fas lev
203 metastasis, poorly metastatic Panc02-H0 and highly metastatic Panc02-H7 cells were injected into the
207 lencing MUC4 expression in an aggressive and highly metastatic pancreatic tumor cell line CD18/HPAF t
208 t case, the invasive ascents of the Tepui by highly metastatic PC-3 and noninvasive LNCaP prostate ca
213 poptosis in poorly metastatic PC3 M-Pro4 and highly metastatic PC3 M-LN4 subclones demonstrated that
215 sitively regulates E-cadherin and suppresses highly metastatic PCA cell invasion by modulating Rho pa
216 that miR-25 can act as a tumor suppressor in highly metastatic PCSCs by direct functional interaction
219 beta4 integrin was sufficient to revert this highly metastatic phenotype in the MNNG-HOS model withou
231 st completely inhibited lung colonization of highly metastatic prostate cancer cells without affectin
235 ersely, recombinant expression of Cav-1 in a highly metastatic PyMT mammary carcinoma-derived cell li
236 were transfected and stably expressed in the highly metastatic rat Dunning MAT-LyLu prostate cancer c
237 d with lycopene against proliferation of the highly metastatic rat prostate adenocarcinoma MAT-LyLu c
238 SPARC selectively supports the migration of highly metastatic relative to less metastatic prostate c
240 r to introduce normal human chromosomes into highly metastatic rodent prostatic cancer cells to map t
241 mplete inhibition on invasion (P < 0.001) of highly metastatic SiHa cells via reduced transcriptional
243 in-7 expression in poorly differentiated and highly metastatic SW620 colon cancer cells induced epith
244 ong epithelial gene program were enriched in highly metastatic TICs, while a second subpopulation wit
245 for these cells revealed that CSCs that are highly metastatic to bone and brain expressed significan
249 ate that secretion of inhibitory TFPI-2 by a highly metastatic tumor cell markedly inhibits its growt
250 within extracellular vesicles secreted from highly metastatic tumor cells can be internalized by wea
253 rate that several proteins characteristic of highly metastatic tumors (LTBP3, SNED1, EGLN1, and S100A
255 n, which can be defeated in the evolution of highly metastatic tumors by combined loss of both p66(Sh
256 sing a mouse model of metastasis reveal that highly metastatic tumors express proteolyzed cyclin E an
258 the control clones produced rapidly growing, highly metastatic tumors within 2 wk of inoculation on c
259 oss in the mouse lung to promote aggressive, highly metastatic tumors, that are initially sensitive t
262 e human-human somatic cell fusions between a highly metastatic, undifferentiated, ER-negative line of
265 an prostate cancer cell line LNCaP, with its highly metastatic variant LNCaP-LN3, by two-dimensional
266 man breast carcinoma MDA-MB-435-F-L cells, a highly metastatic variant of human breast cancer MDA-MB-
268 y be essential for this process, we isolated highly metastatic variants from a poorly metastatic huma
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