ライフサイエンスコーパス: highly metastatic cell

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1 n upon miR-200 overexpression toward that of highly metastatic cells.

2 elta levels were relatively increased in the highly metastatic cells.

3 transcripts displaying reduced stability in highly metastatic cells.

4 gainst poorly metastatic cells compared with highly metastatic cells.

5 ignificantly impairs the bone progression of highly metastatic cells.

6 s phosphoinositide 3-kinase, AKT, and SRC in highly metastatic cells.

7 g ligand activation of the EGFR, but only in highly-metastatic cells.

8 ecause they preferentially activate c-Src in highly-metastatic cells.

9 Within minutes of adhesion, the highly metastatic cells acquire the ability of enhanced

10 ere we show that loss of c-KIT expression in highly metastatic cells correlates with loss of expressi

11 at up-regulation of MCAM/MUC18 expression in highly metastatic cells correlates with loss of expressi

12 etastatic variants, suggest that not only do highly-metastatic cells display constitutively elevated

13 ration of miR-10b antagomirs to mice bearing highly metastatic cells does not reduce primary mammary

14 Highly metastatic cells evade this tumor-suppressive pat

15 ompared with poorly metastatic cancer cells, highly metastatic cells expressed increased levels of th

16 Highly metastatic cells growing in culture constitutivel

17 Highly metastatic cells growing in the prostate expresse

18 usly shown that enforced c-KIT expression in highly metastatic cells inhibited tumor growth and metas

19 Silencing of IL-13Ralpha2 in highly metastatic cells led to a decrease in adhesion ca

20 y a poorly metastatic cell line to that by a highly metastatic cell line 24 h after injection in the

21 -2 mRNA stabilization in MDA-MB-231 cells, a highly metastatic cell line derived from a human mammary

22 ential reversal of oncogenic properties of a highly metastatic cell line with the introduction of non

23 ially expressed (greater than 2-fold) in all highly metastatic cell lines relative to their reference

24 The undifferentiated, highly metastatic cell lines with high metastatic potent

25 Highly metastatic cells (MDA-MB-435) expressing high lev

26 Does it arise from rare, variant, highly metastatic cells or does a primary tumor progress

27 We found that in vivo selected highly metastatic cell populations showed little genetic

28 Introduction of Psap in highly metastatic cells significantly reduced the occurr

29 Loss of CC3 in highly metastatic cells such as SCLC might render them r

30 Highly metastatic cells survived to produce numerous lun

31 When the highly metastatic cells were compared with their low met

32 kinase expression was first demonstrated in highly metastatic cells, whilst re-expression of the pro

33 The pH at the surfaces of highly metastatic cells within tumors was found to be ab

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