ライフサイエンスコーパス: hilar

1 Bilateral hilar (18)F-FDG-avid foci are often noted on PET studies

2 y-one patients with the finding of bilateral hilar (18)F-FDG-avid foci underwent a staging-only PET s

3 Thirty-nine IPNBs were identified in hilar (23/144), intrahepatic (4/86), and distal (12/113)

4 , cerebrospinal fluid (CSF) pleocytosis, and hilar adenopathy more frequently than NMOSD (p < 0.05);

5 evidence of supraclavicular or contralateral hilar adenopathy.

6 s in hippocampal mossy fiber synapses of the hilar and CA3 regions.

7 Complex renal tumors, such as hilar and endophytic lesions, have also been performed r

8 ity, lobular ground-glass opacification, and hilar and intercostal systemic collaterals were more pre

9 ion is the primary modality of treatment for hilar and intrahepatic cholangiocarcinoma (HCCA-ICCA).

10 A than in DSA, whereas the visibility of the hilar and intrarenal vessels was significantly worse (P

11 measuring 4.7 cm as well as several enlarged hilar and ipsilateral mediastinal lymph nodes.

12 gnostic performance of MR imaging in staging hilar and mediastinal lymph nodes in NSCLC on both a per

13 eedle aspiration (EBUS-TBNA) biopsies of the hilar and mediastinal lymph nodes, but the feasibility a

14 ed our hospital for examination of bilateral hilar and mediastinal lymphadenopathy.

15 ck-propagation aNN can be trained to predict hilar and mediastinal nodal involvement with greater acc

16 monkeypox antigen was detected in the lung, hilar and submandibular lymph nodes, spleen, and colon.

17 reased biopsy success rates for pretracheal, hilar, and high pretracheal adenopathy.

18 group and lung parenchymal, airway, pleural, hilar, and mediastinal abnormalities systematically revi

19 ide Y-containing interneurons in the dentate hilar area play an important role in inhibiting the acti

20 ition is caused by cholinergic activation of hilar astrocytes, which provide glutamatergic excitation

21 neurons, as well as a small number of mature hilar astrocytes.

22 HEGCs were much more likely to have a hilar basal dendrite than normotopic granule cells.

23 s labeled by injecting RV after SE displayed hilar basal dendrites and ectopic migration, but not spr

24 SE revealed normally located DGCs exhibiting hilar basal dendrites and mossy fiber sprouting (MFS) wh

25 could explain the role of granule cells with hilar basal dendrites in contributing to hyperexcitabili

26 locarpine-epileptogenesis exhibited aberrant hilar basal dendrites.

27 Temoporfin-PDT can safely be delivered to hilar bile duct cancer patients and results in prolonged

28 n 71 [range 47-88] years) with nonresectable hilar bile duct cancer were treated with T-PDT (median 1

29 ves palliation and survival in nonresectable hilar bile duct cancer.

30 patients and results in prolonged patency of hilar bile ducts, a trend for longer survival time, and

31 the SE-experienced rat, excitatory drive to hilar border inhibitory interneurons is weakened through

32 produced a greater depression of GC input to hilar border interneurons in SE-experienced rats than in

33 s (GCs), perforant path, and CA3 inputs onto hilar border interneurons of the dentate gyrus were exam

34 yrus, often hundreds of micrometers from the hilar border, in the lucidum and radiatum layers.

35 GluR) modulation of glutamatergic input onto hilar-border interneurones and its regulation of feedbac

36 Many BrdU-labeled cells at the hilar/CA3 border also were double-labeled with a neurona

37 the population of granule-like cells at the hilar/CA3 border and CA3 bursts.

38 y double-labeled neurons were located at the hilar/CA3 border.

39 le cell layer and hilus combined, and 33% of hilar calretinin-positive cells.

40 e frequently in patients with lower lobe and hilar cancers combined compared with upper lobe cancers

41 posterior margin of the inferior vena cava (hilar-caval line) on lateral radiographs; this line corr

42 ted over (n = 66) or anterior to (n = 2) the hilar-caval line.

43 ts with operatively confirmed stage I and II hilar CCA in 1993.

44 n for patients with localized, node-negative hilar CCA.

45 h survival after resection for patients with hilar CCA.

46 was first used in patients with unresectable hilar CCAs, these methods have yet to reach broad applic

47 al, and molecular profile similar to that of hilar CCs (from mucin-producing cholangiocytes), whereas

48 nd molecular features of 85 resected CCs (14 hilar CCs [so-called Klatskin tumor], 71 intrahepatic CC

49 Immunoreactivity was similar in muc-ICCs and hilar CCs and in mixed-ICCs and CLCs.

50 and MUC1 were significantly up-regulated in hilar CCs and muc-ICCs compared with mixed-ICCs and CLCs

51 Clinicopathologically, muc-ICCs and hilar CCs showed a predominantly (peri-)hilar location,

52 Hilar cell assemblies, identified by simultaneous triple

53 promoters, and prevented kainic acid-induced hilar cell death.

54 -) mice showed a unique pattern of selective hilar cell loss (i.e., endfolium sclerosis), which so fa

55 planted animals produced similarly extensive hilar cell loss and immediate granule cell disinhibition

56 tide Y mRNA were compared with the degree of hilar cell loss determined by immunohistochemistry again

57 nterneurones occurs in proportion to overall hilar cell loss, and therefore the hypothesis of a selec

58 onsistently abolished after extensive (>85%) hilar cell loss.

59 Hilar cells are often among the first lost in hippocampa

60 layer, in the granule cell layer, and in the hilar cells of the dentate gyrus as well as in isolated

61 s and presynaptic burst discharges, allowing hilar cells to respond over a large dynamic range of inp

62 erneurones with respect to the non-GABAergic hilar cells, i.e. the mossy cells.

63 from the decrease in the total population of hilar cells, indicating no preferential survival of inte

64 Sixty-five patients with unresectable hilar cholangiocarcinoma (CCA) have undergone orthotopic

65 de (LN) assessment after liver resection for hilar cholangiocarcinoma (HC) is still controversial, an

66 ntrahepatic cholangiocarcinoma (IH; n = 23), hilar cholangiocarcinoma (Hilar; n = 54), gallbladder (G

67 of intrahepatic cholangiocarcinoma (ICC) and hilar cholangiocarcinoma (Klatskin tumors) is limited to

68 y resectable gallbladder cancer (n = 44) and hilar cholangiocarcinoma (n = 56) were prospectively eva

69 A total of 257 patients (144 hilar cholangiocarcinoma [HCCA] and 113 distal bile duct

70 umor extent, the proposed staging system for hilar cholangiocarcinoma accurately predicts resectabili

71 rround the issues of extent of resection for hilar cholangiocarcinoma and whether the histopathology

72 ts receiving endoscopic biliary drainage for hilar cholangiocarcinoma between September 1995 and Dece

73 The yield of laparoscopy is lower for hilar cholangiocarcinoma but can be improved by targetin

74 formed a review of the English literature on hilar cholangiocarcinoma from 1990 to 2007.

75 Between 1997 and 2004, 80 patients with hilar cholangiocarcinoma having surgery were reviewed.

76 ic resection in hepatocellular carcinoma and hilar cholangiocarcinoma improves survival.

77 urvival in patients with surgically resected hilar cholangiocarcinoma in a single institution over th

78 Hilar cholangiocarcinoma is a rare tumor with a poor pro

79 Hilar cholangiocarcinoma is an uncommon tumor with a poo

80 Endoscopic biliary drainage of hilar cholangiocarcinoma is controversial with respect t

81 SEMS insertion for the palliation of hilar cholangiocarcinoma offers higher technical and cli

82 s the majority of patients with unresectable hilar cholangiocarcinoma or gallbladder carcinoma, there

83 r the study period was compared with that of hilar cholangiocarcinoma patients (HCCA) seen during the

84 From 1985 to 2006, all patients with hilar cholangiocarcinoma referred to a tertiary surgical

85 Hilar cholangiocarcinoma remains a difficult challenge f

86 y gallbladder cancer and patients with T2/T3 hilar cholangiocarcinoma should undergo staging laparosc

87 ompares outcome after resection of papillary hilar cholangiocarcinoma to that of the more common nodu

88 uated endoscopic palliation in patients with hilar cholangiocarcinoma using self-expandable metallic

89 urvival was most favorable when resection of hilar cholangiocarcinoma was accomplished with margin-ne

90 ogic, and survival data on all patients with hilar cholangiocarcinoma were analyzed.

91 For patients with early stage hilar cholangiocarcinoma which is unresectable or arisin

92 and bile-duct resection can be performed for hilar cholangiocarcinoma with acceptable mortality, thou

93 In patients with hilar cholangiocarcinoma, concomitant hepatic resection

94 In patients with hilar cholangiocarcinoma, long-term survival depends cri

95 ant therapy followed by liver transplant for hilar cholangiocarcinoma, placed in context with the mos

96 locally advanced but potentially resectable hilar cholangiocarcinoma, the yield of laparoscopy was g

97 ve and only potentially curative therapy for hilar cholangiocarcinoma.

98 section remains the mainstay of treatment of hilar cholangiocarcinoma.

99 herapy followed by liver transplantation for hilar cholangiocarcinoma.

100 ates after aggressive surgical resections of hilar cholangiocarcinoma.

101 t determinant of survival after resection of hilar cholangiocarcinoma.

102 between patients with gallbladder cancer and hilar cholangiocarcinoma.

103 lecystectomy), but only 25% in patients with hilar cholangiocarcinoma.

104 This bidirectional plasticity enables hilar circuitry to regulate the flow of information thro

105 experimental murine PGD models were studied: hilar clamp and orthotopic lung transplantation after pr

106 NETs were increased after either hilar clamp or OLT-PCI compared with surgical control su

107 However, hilar clamping also renders the entire kidney ischemic,

108 tial nephrectomies performed with or without hilar clamping for clinical stage T1b tumors.

109 The reviewed data show that foregoing hilar clamping for T1b tumors is not only feasible, but

110 ist between cholecystokinin (CCK)-expressing hilar commissural associational path (HICAP) cells and a

111 d intraoperative blood loss when compared to hilar controlled procedures.

112 m can be an effective alternative to classic hilar dissection in cases when the latter is difficult o

113 dated videoscopic guidance and a traditional hilar dissection without rib spreading.

114 Our results demonstrate that hilar ectopic DGCs preferentially synapse onto adult-bor

115 seizure frequency and (1) the percentage of hilar ectopic DGCs, (2) the amount of mossy fiber sprout

116 ic times before or after SE decreased MFS or hilar ectopic DGCs, supporting the RV labeling results.

117 Hilar ectopic GCs (EGCs) can potentially provide insight

118 Hilar ectopic granule cells (HEGCs) are hyperexcitable a

119 Together, these data indicate that hilar ectopic granule cells are postsynaptic to mossy fi

120 th recent physiological results showing that hilar ectopic granule cells from epileptic rats are more

121 Electron microscopy was used to study hilar ectopic granule cells that were located 20-40 micr

122 Stereological methods were used to measure hilar ectopic granule cells, mossy cells, mossy fiber sp

123 nificantly correlated with the generation of hilar ectopic granule cells, the number of mossy cells,

124 es with the somata and proximal dendrites of hilar ectopic granule cells.

125 In the pilocarpine mTLE model, hilar-ectopic DGCs arise as a result of an aberrant chai

126 ws abnormal dispersion and the appearance of hilar-ectopic DGCs.

127 ntate gyrus and increased Dab1 expression in hilar-ectopic neuroblasts.

128 Consistently, hilar EGC and GC layer GCs had similar dendritic lengths

129 ensional analysis revealed that, remarkably, hilar EGC dendrites often extended along the longitudina

130 However, hilar EGC dendrites were topologically more complex, wit

131 efferent output, could help explain why the hilar EGC population could impair DG function.

132 Axonal reconstruction demonstrated that hilar EGCs contributed to mossy fiber sprouting.

133 tively analyzed the structural morphology of hilar EGCs from adult male rats several months after pil

134 Additionally, hilar EGCs that develop after SE may contribute to epile

135 Hilar EGCs were physiologically identified in slices, in

136 A chest x-ray showed bilateral hilar enlargement, thickening of the right paratracheal

137 n within the hilus was effective in reducing hilar excitation of granule cells, although this release

138 study of patients with cancer with bilateral hilar foci on 1 or 2 sequential (18)F-FDG PET studies be

139 ntial and EPSC frequencies were increased in hilar GABA neurons from slices ipsilateral to the injury

140 excitatory synaptic activity was detected in hilar GABA neurons ipsilateral to the injury after gluta

141 s suggest that excitatory drive to surviving hilar GABA neurons is enhanced by convergent input from

142 short period of time prevented age-dependent hilar GABAergic interneuron decline and cognitive defici

143 with PB for 6 weeks prevented age-dependent hilar GABAergic interneuron decline and learning and mem

144 c mice; eliminating endogenous Tau prevented hilar GABAergic interneuron loss and the learning and me

145 n neurotoxic apoE4 fragment transgenic mice, hilar GABAergic interneuron loss was even more pronounce

146 n (KI) mice had an age-dependent decrease in hilar GABAergic interneurons that correlated with the ex

147 poE4-KI mice had an age-dependent decline in hilar GABAergic interneurons that correlated with the ex

148 in (KI) mice had an age-dependent decline in hilar GABAergic interneurons that correlated with the ex

149 causes age- and Tau-dependent impairment of hilar GABAergic interneurons, leading to learning and me

150 group, a trend towards increased numbers of hilar GAD cells was observed, even over and above that o

151 ed to test for monosynaptic connections from hilar GPHNs to granule cells.

152 ules did not arise directly from preexisting hilar IHBDs.

153 s, which provide glutamatergic excitation of hilar inhibitory interneurons.

154 This rewiring of circuitry regulating hilar inhibitory neurons may reflect an important compen

155 sionally reconstructed 216-microm-long spiny hilar interneuron axon segment in the outer third of the

156 The numbers of hilar interneurones expressing somatostatin mRNA and neu

157 The percentage decrease in the number of hilar interneurones labelled with either GAD67 or parval

158 ntrast, SOMIs with axon in the hilus, termed hilar interneurons (HILs), provide perisomatic inhibitio

159 ells and excitability of surviving GABAergic hilar interneurons 8-13 weeks after cortical contusion b

160 nerability of a particular subset of dentate hilar interneurons after prolonged SE.

161 ta-estradiol in ovariectomized rats protects hilar interneurons against seizure-induced injury, inclu

162 Hilar interneurons also show synapse specificity and pre

163 We also observed that hilar interneurons and associated NG2(+) cells are simil

164 utamatergic and monosynaptically excite both hilar interneurons and mossy cells.

165 Although many somatostatin-positive hilar interneurons effectively transported FG from the d

166 erefore, in the outer molecular layer, spiny hilar interneurons form synaptic contacts that appear to

167 eneration and tauopathy occurred earliest in hilar interneurons in apoE4 fragment transgenic mice; el

168 of the transplanted neurons with endogenous hilar interneurons in mice without TLE.

169 tions and synaptic connections made by spiny hilar interneurons labeled with biocytin in gerbils in v

170 Some of hilar interneurons of the dentate gyrus express neuropep

171 , in all groups, we observed a population of hilar interneurons that were CHT-immunoreactive.

172 connectivity may be mechanisms for surviving hilar interneurons to inhibit more granule cells and com

173 gnaling events that render somatostatinergic hilar interneurons vulnerable to SE-induced cell death.

174 s of NPY expression in the cortex, CA1, CA3, hilar interneurons, and granule cells of the dentate gyr

175 erneurons and suggest that the loss of spiny hilar interneurons, as occurs in temporal lobe epilepsy,

176 ilar mossy cells, but not peptide-containing hilar interneurons, indicating that mossy cells are the

177 t-born granule cells establish synapses with hilar interneurons, mossy cells and CA3 pyramidal cells

178 n this region, glutamatergic mossy cells and hilar interneurons, or the organization of local circuit

179 populations of dentate gyrus mossy cells and hilar interneurons.

180 of their intralamellar connections are onto hilar interneurons.

181 nd in vulnerable somatostatin-immunoreactive hilar interneurons.

182 estradiol enhances NPY expression within the hilar interneurons.

183 ve cohort of patients with bile duct cancer (hilar, intrahepatic, or distal) was reviewed, and those

184 Hilar IPSPs have low failure rates, are blocked by the G

185 The dominant positive polarity of hilar LFPs was only produced by the synchronous activati

186 An in vivo murine hilar ligation model of IR injury was used, in which lef

187 and hilar CCs showed a predominantly (peri-)hilar location, smaller tumor size, and more lymphatic a

188 on, and proliferative responses of pulmonary hilar lymph node CD4 T cells to autologous lung extracts

189 of 120 days or less (HR, 2.6; P = 0.01) and hilar lymph node invasion (HR = 2.2; P = 0.03), but not

190 tments (bronchoalveolar lavage, lung tissue, hilar lymph node) at AAD and LIT; systemic compartments

191 ed from T lymphocytes in blood, airways, and hilar lymph node, with responses predominantly localized

192 ells in the bronchoalveolar lavage (BAL) and hilar lymph nodes (HLN) at the resolution stage of this

193 t been characterized in cells from pulmonary hilar lymph nodes (PHLN).

194 ed enlarged hyperattenuating mediastinal and hilar lymph nodes and edema of mediastinal fat.

195 ge II disease, that is, metastatic spread to hilar lymph nodes.

196 ulmonary nodules (31.4%), mediastinal and/or hilar lymphadenopathy (23%), mass-like consolidation (17

197 ess with incipient pneumonia, pleuritis, and hilar lymphadenopathy.

198 cic tuberculosis would be based primarily on hilar lymphadenopathy.

199 n-Hodgkin's lymphoma (NHL), to differentiate hilar lymphoma (HL) from HU of benign etiology.

200 dule (n = 1), multiple nodules (n = 1), or a hilar mass (n = 1).

201 raphy (CT) angiogram, there is a large right hilar mass and enlarged mediastinal lymph nodes but no p

202 n a marked decrease in the size of the right hilar mass and mediastinal lymph nodes.

203 tients underwent 155 liver transplants using hilar mass clamping.

204 nction; an ultrasound examination revealed a hilar mass, with hydronephrosis in five and stenosis of

205 symptomatic, and imaging typically reveals a hilar mass.

206 diagnosed in 5 of 77 patients (6.5%), while hilar/mediastinal lymphadenopathy was found in 25 of 76

207 N1 hilar metastases were diagnosed in 8 patients (12.9%) be

208 Excitatory hilar mossy cells (MCs) in the dentate gyrus receive inp

209 both p11 and SMARCA3 are highly enriched in hilar mossy cells and basket cells.

210 In addition to the dentate hilar mossy cells and CA3 pyramidal cells shown previous

211 Whereas hilar mossy cells and CA3 pyramidal cells were FG-labele

212 nt vulnerability of dendritically projecting hilar mossy cells and interneurons and the relative resi

213 lutamate receptor subunit 2 (GluR2)-positive hilar mossy cells and peptide-containing interneurons in

214 pothesis in light of reports that excitatory hilar mossy cells are not consistently vulnerable and in

215 Longitudinal projections of excitatory hilar mossy cells could be viewed as antithetical to lam

216 However, whether it is the disappearance of hilar mossy cells from the dentate gyrus circuitry after

217 reduces evoked IPSC amplitude recorded from hilar mossy cells in the rat dentate gyrus through a pre

218 eedback inhibition of GABAergic afferents to hilar mossy cells is governed by a complex relationship

219 The ADP observed in hilar mossy cells is produced by the opening of a Na(+)

220 We show that hilar mossy cells provide initial glutamatergic synapses

221 cells converge on the hilus, and excitatory hilar mossy cells redistribute these signals back to gra

222 ncy of miniature IPSCs (mIPSCs)recorded from hilar mossy cells without altering event amplitude, area

223 ipal cells, including dentate granule cells, hilar mossy cells, and hippocampal pyramidal cells, were

224 6 mice destroyed most GluR2-positive dentate hilar mossy cells, but not peptide-containing hilar inte

225 Hilar mossy cells, in particular, are thought to show dr

226 re, after epileptogenic treatments that kill hilar mossy cells, mossy fiber sprouting does not simply

227 min, and calretinin interneurons, as well as hilar mossy cells, new adult-born neurons, and recently

228 n null mice and an increase in the number of hilar mossy cells.

229 kappa opioid peptide dynorphin is present in hilar mossy fiber collaterals.

230 noma (IH; n = 23), hilar cholangiocarcinoma (Hilar; n = 54), gallbladder (GB; n = 32), and distal bil

231 ely to modulate neuronal excitability in the hilar network, and further reveal a mechanism that could

232 le cell discharges always produced extensive hilar neuron degeneration and immediate granule cell dis

233 ings are consistent with the hypotheses that hilar neuron loss and ectopic granule cells might contri

234 The degree of hilar neuron loss and mossy fiber sprouting correlated s

235 llar inhibition was intact in rats with <40% hilar neuron loss but was consistently abolished after e

236 ificantly affect granule cell proliferation, hilar neuron loss, or generation of ectopic granule cell

237 r of neuronal nuclear antigen-immunoreactive hilar neurones.

238 es revealed substantial loss of GFP-positive hilar neurons (GPHNs) but increased GFP-positive axon le

239 ptic barrages triggered persistent firing in hilar neurons (hilar up-states).

240 ons were among the many retrogradely labeled hilar neurons 2.5-4.5 mm longitudinally.

241 Population responses recorded in hilar neurons accurately encoded stimulus identity.

242 Patterns of synaptic input onto dentate hilar neurons predicted which of four pathways were stim

243 ry, including an extensive bilateral loss of hilar neurons throughout the hippocampal longitudinal ax

244 ause the total number of NeuN-immunoreactive hilar neurons was unaffected, the decline observed with

245 In addition, surviving NPY-immunoreactive hilar neurons were distributed preferentially in the sup

246 By contrast, other types of hilar neurons were less strongly depolarized by bath app

247 in SGCs and synaptic barrages in downstream hilar neurons without blocking fast synaptic transmissio

248 activation on the intrinsic excitability of hilar neurons.

249 lls evoke EPSPs with high failure rates onto hilar neurons.

250 rticular prominence is the loss of GABAergic hilar neurons.

251 ivation was repressed in STEP-immunoreactive hilar neurons.

252 of the hippocampal CA3 pyramidal and dentate hilar neurons.

253 dentate gyrus despite loss of immunopositive hilar neurons.

254 excitability, and control aspirations of the hilar neuropil or of interneurons in the presence of GAB

255 tion, we performed patch-clamp recordings on hilar NG2(+) cells and interneurons between 3 and 21 pos

256 We first observed that hilar NG2(+) cells exhibit spontaneous glutamatergic EPS

257 Hilar nodes and parenchymal lung tissue were processed a

258 way from the staple line, and in ipsilateral hilar nodes.

259 flammation and Th2 cytokines in restimulated hilar nodes.

260 her, we show that beta-estradiol upregulates hilar NPY and that this leads to enhancement in dentate

261 Hilar NPY neurons showed modest spike frequency adaptati

262 ting lymph node metastases in lower lobe and hilar NSCLC compared with upper lobe NSCLC.

263 ry stenting (HR 3.274, P=0.019), distal (non-hilar) obstruction of the bile ducts (HR 3.711, P=0.008)

264 r and more complex tumors including those in hilar or central locations, in kidneys with multiple mas

265 However, hilar path activity could be seen after LTP, with or wit

266 U69,593 also inhibited hilar path induced long-term potentiation (LTP) of granu

267 The hilar pathology was often associated with hepatic artery

268 ells and among somatostatin (SOM)-containing hilar perforant path-associated (HIPP) interneurons.

269 Hilar-perforant-path-associated interneurons (HIPP cells

270 gh excitatory mossy cells of the hippocampal hilar region are known to project both to dentate granul

271 study dissected the fascia dentata (FD) and hilar region from the CA and subicular cell fields of th

272 It is suggested that the dentate gyrus and hilar region in the hippocampus perform memory selection

273 so was observed in large bile ducts from the hilar region of primary sclerosing cholangitis patients.

274 microglial cells and their processes in the hilar region of the dentate gyrus of adult Sprague-Dawle

275 ed in the subventricular zone but not in the hilar region of the dentate gyrus.

276 tostatin mRNA and neuropeptide Y mRNA in the hilar region of the hippocampal formation of patients wi

277 ived significant synaptic input from the CA3/hilar region, especially under conditions of experimenta

278 e found to be exclusively located within the hilar region.

279 The CMV promoter produced expression in hilar regions and pyramidal neurons, with minimal expres

280 of symmetric and mild uptake limited to the hilar regions that are stable on 2 sequential PET studie

281 In AA rats, sympathetic nerve density in the hilar regions, where NA nerves enter the spleen, was inc

282 mprove behavioral performance fully restored hilar SOM expression in aged, memory-impaired rats.

283 ether, these findings suggest that surviving hilar somatostatin interneurons enlarge, extend dendrite

284 ble among animals, but was as high as 96% of hilar somatostatin-positive interneurons, 84% of parvalb

285 In area CA3, repetitive hilar stimuli frequently evoked multiple population spik

286 Classic dissection of the hilar structures during the hepatectomy portion of the l

287 In such cases, clamping of the hepatic hilar structures en mass can be an effective alternative

288 rm the hepatectomy with mass clamping of the hilar structures was an intraoperative judgment made whe

289 surface of the kidney, as well as posterior hilar structures.

290 Intralamellar hilar synapses appear to function primarily by integrati

291 ially inhibit mossy cells; 81% of inhibitory hilar synapses are onto mossy cells.

292 n the lung adjacent to a central mediastinal/hilar thoracic mass were excluded (range,.59 to.91).

293 Thus, restricted hilar transplantation of inhibitory interneurons restore

294 Overlap in the pathogenesis of GB and Hilar tumors was suggested.

295 1b tumors and more complex tumors, including hilar tumors, central tumors or tumors in solitary kidne

296 Hilar up-states triggered functional inhibition in granu

297 riggered persistent firing in hilar neurons (hilar up-states).

298 prevalence and characterize the patterns of hilar uptake (HU) on 67Ga-citrate imaging after cyclopho

299 scarring of the hilum (n = 137) or extensive hilar varices (n = 18) were encountered.

300 The expression profiles of GB and Hilar were more similar to each other than either was to