ライフサイエンスコーパス: hillock

1 er neurons, which project to the M-cell axon hillock.

2 at the presumptive domain boundary, the axon hillock.

3 ing were present in the vicinity of the axon hillock.

4 forward along the dendrite to the soma-axon hillock.

5 ated with the formation of well-defined etch hillocks.

6 he accumulation of the mRNA both at the axon hillock after the treatment with 5-HT and at the opposit

7 The cell body, axon hillock and basal dendritic compartments achieve unique

8 eins abolished both accumulation at the axon hillock and long-term facilitation.

9 in mRNA and protein accumulation at the axon hillock and number of syntaxin granules in the SN axon w

10 he accumulation of sensorin mRNA at the axon hillock and the stability of sensorin mRNA exported to d

11 onal cell bodies, primary dendrites and axon hillocks and function in neuronal protection and stabili

12 h is also accompanied with the formations of hillocks and granules due to the dewetting of Au films a

13 electrical activation occurred near the axon hillock, and that dendrites contributed little.

14 ion of syntaxin mRNA and protein at the axon hillock, and the increase in syntaxin granules in SN axo

15 and protein were targeted away from the axon hillock, and the number of syntaxin granules in the SN a

16 f atomically flat Si(100) terminated by etch hillocks bounded by {111}- and {110}-oriented microfacet

17 nsities, cells in serum-coated dishes formed hillocks, but cells in noncoated dishes did not.

18 The induction of hillocks by NAC was correlated with downregulation of al

19 Hillocks capped by different pentameric proteins spontan

20 oduces multifocal nodular structures, i.e., "hillocks," consisting of cells and extracellular matrix.

21 urons, syntaxin mRNA accumulates at the axon hillock during long-term facilitation of sensory-motor n

22 In summary, key steps in hillock formation are: (1) migration, (2) secretion of f

23 Antibodies to fibronectin blocked hillock formation by cells on serum-coated substrata and

24 Smooth muscle cell and mesangial cell hillock formation have been proposed as in vitro models

25 Hillock formation is associated with induction of a diff

26 r and how the cellular redox state modulates hillock formation.

27 cer of glutathione, dramatically facilitated hillock formation.

28 pericellular fibronectin-fibrils was key to hillock formation.

29 relocalization of syntaxin mRNA to the axon hillock from the opposite pole in the cell body of the s

30 action potentials initiate first in the axon hillock/initial segment (AH-IS) region because of a loca

31 The goldfish M-cell initial segment/axon hillock is surrounded by a composite axon cap consisting

32 were found surrounding cell bodies and axon hillocks most often in the buccal and abdominal ganglia.

33 xin mRNA and protein accumulated at the axon hillock of SNs during the initial phase of synapse forma

34 of high levels of sensorin mRNA in the axon hillock of the SN and in SN neurites contacting L7.

35 At the nodes of Ranvier and the axon hillocks of central neurons, VGSCs are associated with t

36 lse can be initiated in either the soma-axon hillock or in the distal primary dendrite, and that the

37 e at a fixed location, typically at the axon hillock or initial segment, although action potentials,

38 A high density of Na+ channels in the axon hillock, or initial segment, is believed to determine th

39 ial to SI >/= 1.0 to generate/develop growth hillocks, or other factors.

40 t the impulse always arises in the soma-axon hillock region, with back-propagation through excitable

41 ts were located near the nucleus or the axon hillock region.

42 generating spikes of depolarization at axon hillock regions and propagating the initial rising phase

43 Ca2+ release within the perinuclear and axon hillock regions provide a mechanism for preferential ini

44 ilayered ridges and nodules, termed hills or hillocks, separated by less populated areas termed valle

45 ron including an axon, initial segment, axon hillock, soma, and simplified dendritic tree was used to

46 on potential (AP) successfully from the axon hillock to a synaptic terminal.

47 e and menadione inhibited the development of hillocks triggered by either NAC, glutathione, or prolon

48 y distributed arrays of "tips" (tall conical hillocks) upon oxidation of palladium (Pd) thin films.