ライフサイエンスコーパス: hind leg

1 ond to tactile stimulation or movements of a hind leg.

2 l melanoma cells after s.c. injection in the hind leg.

3 uropil regions of the ipsilateral middle and hind legs.

4 ke approximately fourfold and also increased hind leg 1-MX metabolism by 50%, suggesting increased ex

5 cts in chimeric mice, characterized by short hind legs, aberrant limb features, split lumbar vertebra

6 stimulus, orienting behavior (rearing on the hind legs), and food cup behavior (placing the head insi

7 dominant, causes progressive weakness of the hind legs, and there is severe demyelination in the peri

8 lts demonstrate that insulin increases total hind leg blood flow and metabolism of 1-MX, suggesting a

9 cterized by a distinctive dysfunction of the hind legs, causing uncoordinated movements.

10 radiation-induced (single dose, 35 or 45 Gy) hind leg contraction in C3H/Hen mice.

11 umping was generated by slow contractions of hind leg depressor muscles and then stored by bending sp

12 e's center of mass, pollen is carried on the hind legs, farther from the center of mass.

13 this study, behaviors (open field, grooming, hind-leg gait, water maze, and acoustic startle reflex)

14 vels, muscle interstitial oxygen saturation, hind leg glucose extraction, and muscle insulin clearanc

15 Insulin treatment increased hind leg glucose uptake approximately fourfold and also

16 ously, orienting behavior (rearing up on the hind legs) habituated across trials in normo-active cont

17 Spinalized rats given shock whenever 1 hind leg is extended learn to maintain that leg in a fle

18 In addition, hind leg kick force, produced by stimulating the extenso

19 ogical and life history traits: body weight, hind leg length, parasite burden, horn length, horn grow

20 nents of shape: body width, body height, and hind-leg length.

21 Blood flow distribution within the hind leg muscles was assessed by measuring the metabolis

22 Metabolism of intact hind-leg muscles from young rats was studied in vitro un

23 a recruitment of capillary blood flow in rat hind leg not mimicked by epinephrine.

24 tumors transplanted into the subcutis of the hind leg of Nembutal-anesthetized (50 mg/kg) Fischer 344

25 .OH-induced structural changes in DNA of the hind leg of the BALB/c mouse.

26 n two identified motoneurons innervating the hind leg of the locust: the FETi-FlTi synapse (fast exte

27 cells were intramuscularly injected into the hind legs of 18 nude mice.

28 MCA, injected into the hind legs of mice, produced a variety of significant str

29 with Poly:ICLC plus OVA protein in the neck, hind leg, or foreleg for drainage into the cervical, ing

30 ese mice exhibit motor defects which lead to hind leg paralysis and tremor.

31 sin A(-/-) mice developed slowly progressive hind leg paralysis with clinical onset at approximately

32 Hind-leg paralysis preceded the death of the mice.

33 njection of 0.75% bupivacaine into the right hind leg prior to CIP was used for peripheral nerve bloc

34 ard oil (100%) to the lateral surface of the hind leg produced a facilitation of the tail-flick refle

35 Brief electrical stimulation of a single hind leg proprioceptor, the lump receptor (LR), led to p

36 ergy and its quick release to accelerate the hind legs rapidly.

37 dition, immunohistochemistry of mouse embryo hind legs showed that Sox9 phosphorylated at serine 211

38 es that exhibit more moderate enlargement of hind legs, such as mantids and cockroaches.

39 ic dystrophic mice a progressive lameness of hind legs, suggestive of a nerve defect.

40 raptorial front legs, and the two propulsive hind legs to produce a controlled jump with a precise la

41 Dytiscus marginalis simultaneously uses its hind legs to propel itself through the water.

42 ) and femoral blood flow (80%) and decreased hind leg vascular resistance (31%).

43 but increased femoral blood flow and lowered hind leg vascular resistance to a similar extent as insu

44 od pressure, heart rate, femoral blood flow, hind leg vascular resistance, and glucose uptake were me

45 If one hind leg was cocked then the spikes only occurred in mot

46 sults suggest that diversification of insect hind legs was influenced by changes in both the spatial

47 roduced normally, they developed progressive hind leg weakness and decline in motor coordination at 1

48 mice with subcutaneous PC3 xenografts in the hind leg were treated with 2ME2 (75 mg/kg) p.o. for 5 da

49 ther than the middle legs, and also that the hind legs were able to generate a larger angular velocit

50 Second, we concluded that the hind legs were able to propel the beetle farther than th

51 First, both hind legs were moved into a cocked position by high-freq

52 Mice bearing the sarcoma FSA in the hind legs were treated daily for 10 days with SC-'236 (6

53 as shown that spinal rats given shock to the hind leg when it is in an extended position (contingent

54 nditioned orienting behavior (rearing on the hind legs) when a visual stimulus was paired with food.

55 Forty-two BDIX rats were inoculated in each hind leg with a DHD/K12/TRb tumor cell line.

56 ssive mouse mutation, exhibits ataxia of the hind legs with a slight side-to-side wobble while walkin

57 individuals regularly touching others on the hind legs within populations that have become concentrat