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1 pid levels, and a neuromuscular abnormality (hind-limb clasping).
2  They also exhibited reduced body weight and hind limb clasping.
3 e absence of striatal pathology, begins with hind-limb clasping during tail suspension and tail stiff
4 vere neurobehavioral abnormalities including hind limb clasping, overt seizures, motor impairment and
5 ge of phenotypes including ataxia, front and hind limb clasping, reduced brain size, and smaller neur
6 ral deficits on the accelerating rotorod and hind limb clasping tests in transgenic HD mice.
7 tionally, Kcne3(-/-) mice exhibited abnormal hind-limb clasping upon tail suspension (63% of Kcne3(-/

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