ライフサイエンスコーパス: hind paw

1 n the mechanical sensitivity of the affected hind paw.

2 d- and mechano-allodynia) in the ipsilateral hind paw.

3 n (50muL, 10%) in the plantar surface of the hind paw.

4 heat stimuli applied to their tumor-bearing hind paw.

5 ing task performed by the previously injured hind paw.

6 timuli applied to the plantar surface of the hind paw.

7 of 1% lambda-carrageenan into the mouse left hind paw.

8 Lewis rats by injecting carrageenan into the hind paw.

9 mechanical and hypotonic stimulation of the hind paw.

10 nts were acquired from sections of arthritic hind paws.

11 nization induced an erosive arthritis of the hind paws.

12 etermined in histology sections of arthritic hind paws.

13 nded to the proximal joints of the front and hind paws.

14 ts following noxious heat stimulation of the hind paws.

15 and (4) rats treated with formalin into the hind paw 30 min after subcutaneous morphine injection (m

16 sly, (2) rats treated with FORMALIN into the hind paw 30 min after subcutaneous normal saline injecti

17 ts develop hyperalgesia and allodynia in the hind paw after L5 spinal nerve ligation.

18 Having established facial and hind-paw allodynia as a useful animal surrogate of heada

19 Facial and hind-paw allodynia associated with dural stimulation is

20 IM elicited robust facial and hind-paw allodynia, which peaked within 3 hours.

21 nical or thermal stimulus to the ipsilateral hind paw and lower limb of the rat.

22 noxious thermal stimulus to the ipsilateral hind paw and lower limb.

23 pplied to different areas of the ipsilateral hind paw and lower limb.

24 he response to mechanical stimulation of the hind paws and face.

25 ia and mechanical allodynia occurred in both hind paws and forepaws by 7 d postlesion and were mainta

26 fied by measurement of ankle swelling in the hind paws and histologic examination.

27 liter) into the plantar surface of the right hind paw, and 24 rats were injected with 50 microliter s

28 vented the development of hypoalgesia in the hind paw, and reduced superoxide and nitrotyrosine level

29 Unlike the traditional hind paw assay, the time-course of nociceptive behavior

30 igation, but maintained in the contralateral hind paw at control levels.

31 onse to graded mechanical stimulation of the hind paws (brush, pressure, and pinch).

32 ntibody delayed the onset of swelling of the hind paws but, more importantly, inhibited the developme

33 n the tail were similar to those seen in the hind paw, but were limited to licking.

34 Noxious stimulation of the hind paw by subcutaneous injection of 0.5% formalin into

35 obes and received noxious stimulation to the hind paw by subcutaneous injection of 0.5% formalin solu

36 d as periarticular erythema and edema in the hind paws by days 24-26 after the first injection, with

37 n of TLQP-21-stimulated macrophages into rat hind paw caused mechanical hypersensitivity.

38 pain in which carrageenan injection into the hind paw causes hypersensitivity to heat stimuli, TNF-al

39 Histologic analyses were done on hind paws collected on day 32 following the pristane inj

40 the lymph nodes, liver, kidneys, spleen, and hind paw containing the injection site were removed and

41 ry function and on density of innervation in hind paws contralaterally as well as ipsilaterally to th

42 s of different bending forces to the plantar hind paw, developed in the untrained group 3 weeks after

43 the effects of spinal adrenal transplants on hind paw edema and the anterograde transport of substanc

44 cell line) and using the carrageenan-induced hind paw edema model on rats.

45 Hind paw edema, inflammatory cell infiltration, and oste

46 d morphine resulted in significantly reduced hind paw flinching compared with morphine alone in the f

47 Robust formalin-evoked edema, as well as hind paw flinching, was observed in striated muscle cont

48 including the rota-rod, open-field tests and hind-paw footprint analysis.

49 Relative to hind paws, forepaws performed ~4 times more steps, they

50 rats under resting conditions and following hind paw formalin injection.

51 Using a rodent pain assay that combines the hind-paw formalin model with the place-conditioning para

52 domly assigned to five groups; each had both hind paws immersed in water at different temperatures (n

53 increased withdrawal latency of the inflamed hind paws in the sham-operated rats but not in those wit

54 Hind paw incision was used in separate groups of animals

55 Rats that experience hind-paw incision injury at 3 days of age, display an in

56 The injection of carrageenin into the rat hind paw induced a decrease in the mechanical nociceptiv

57 Injection of formalin into the hind paw induces a biphasic pain response; the first pha

58 taneous injection of formalin into the rat's hind paw induces microglial activation in the spinal cor

59 We established a S. aureus hind paw infection in diabetic db/db and nondiabetic Lep

60 Nondiabetic +/+ mice resolved the S. aureus hind paw infection within 10 days, whereas db/db mice wi

61 ted an antinociceptive effect in rats with a hind paw inflammation, without exhibiting characteristic

62 cal- and thermal-pain hypersensitivity after hind-paw inflammation compared with wild-type littermate

63 The hind paw inflammatory score showed a decrease in the int

64 stemically injected into rats prior or after hind paw injection of formalin.

65 Unilateral hind paw injections of complete Freund's adjuvant produc

66 hind-paw injury at Postnatal Day (PD) 2, (b) hind-paw injury at PD 5, (c) anesthesia exposure only on

67 igned to 1 of 4 experimental conditions: (a) hind-paw injury at Postnatal Day (PD) 2, (b) hind-paw in

68 Subjects receiving a unilateral neonatal hind-paw injury showed decreased mechanical threshold (h

69 Immune challenge took place in the hind paw ipsilateral or contralateral to an injured scia

70 Sensitivity was lost in the hind paw ipsilateral to spinal nerve ligation, but maint

71 late test (52 degrees C): the mean (+/-S.D.) hind paw lick latency of rats in the high anti-NGF titer

72 ured before injection of formalin or CFA and hind paw licking/biting timed during the late-phase of t

73 Contralesional hind paws lost 54% of innervation in tibial-innervated e

74 e, reversible, dose-dependent attenuation of hind paw mechanical allodynia for up to 1h after adminis

75 orepaw steps were classified as exploratory, hind paw movement as locomotive.

76 ) was implanted subcutaneously on the dorsal hind paw of C57 mice while the tumor-free contralateral

77 neously into the plantar portion of the left hind paw of male Holtzman-strain Sprague-Dawley rats.

78 sponse when injected subcutaneously into the hind paw of mice.

79 thermal hyperalgesia when injected into the hind paw of mice.

80 te Freund's adjuvant subcutaneously into one hind paw of rats with dorsolateral funiculus lesions and

81 Arthritis was induced in the right hind paw of six rats; the left hind paw served as an int

82 on of Complete Freund's Adjuvant in the left hind paw of Sprague-Dawley rat.

83 was injected into the plantar surface of one hind paw of the rat to induce hyperalgesia in the inject

84 une response was observed bilaterally in the hind paws of animals subjected to unilateral mononeuropa

85 Swelling was also observed in the hind paws of hamsters infused with only Mphi-FBb or only

86 chemokine mRNA transcripts obtained from the hind paws of immunized mice, whereas FcgammaRI(-/-) mice

87 ng complete Freund's adjuvant (CFA) into the hind paws of rats.

88 ng complete Freund's adjuvant (CFA) into the hind paws of rats.

89 V1-lineage afferents in the epidermis of the hind paws of the reporter mice showed that EGTA and MDL2

90 that challenging the skin of the calf of the hind paw or the cheek of previously sensitized mice with

91 uitry, we used an animal model of persistent hind paw peripheral inflammation.

92 ntly reduced pain-related behavior following hind paw plantar formalin injection in rats.

93 were significantly correlated with the RVS (hind paws R = -0.94, front paws R = -0.81, combined R =

94 Periorbital and hind paw sensory thresholds were measured to detect cuta

95 in the right hind paw of six rats; the left hind paw served as an internal control.

96 responses were compared and were related to hind-paw skin temperatures measured during stimulation o

97 yographic responses to graded suprathreshold hind-paw stimuli in the 4 weeks following adult incision

98 cement on unmyelinated fiber function in the hind paw, sural nerve C-fiber morphometry, sciatic nerve

99 to IL-17 partially inhibited the significant hind paw swelling and histopathological changes observed

100 Inflammation (hind paw swelling) was quantified throughout the clinica

101 g gait, reluctance or inability to move, and hind paw swelling.

102 carrageenan (2%) into genital or nongenital (hind paw, tail, cheek) regions.

103 old (hyperalgesia) on the previously injured hind paw throughout development.

104 alysis, and immunohistochemistry; plasma and hind paw tissue levels of cytokines and chemokines (incl

105 Exposure of the hind paw to 1,500 J m(-2) UVB radiation caused an increa

106 The sensitivity of the hind paws to noxious heat (Hargreaves test), innocuous t

107 itial development of neuropathic pain in the hind paw upon injury to the sciatic nerve, but the abnor

108 Severe swelling of the hind paws was detected 8 days after infection and increa

109 Swelling of the hind paws was detected in 100, 100, and 50% of hamsters

110 Swelling of the hind paws was detected within 8 h of infection, increase

111 Inflammation in the hind paws was evaluated by assessing cytokine and chemok

112 overy from anesthesia, motor function of the hind paws was scored as follows: 0, no evidence of defic

113 njection of carrageenan into the ipsilateral hind paw, was investigated.

114 y subcutaneous inoculation of QHGAD67 in the hind paws, was substantially increased compared to anima

115 ng complete Freund's adjuvant (CFA) into one hind paw, we systematically evaluated the anti-hyperalge

116 r 4 weeks of arthritis, mice were killed and hind paws were assessed histologically for joint damage.

117 Results demonstrated that inflamed hind paws were edematous, and the withdrawal thresholds

118 Hind paws were evaluated for histologic evidence of infl

119 nd the withdrawal thresholds of the inflamed hind paws were significantly lower after formalin or CFA

120 ar junctions of the lumbrical muscles of the hind-paw were vulnerable in both SMA and ALS, with a los

121 ulas, and sensory thresholds of the face and hind-paws were characterized.

122 lymphocytes developed severe swelling of the hind paws when infected with B. burgdorferi.

123 erkeratotic calluses on Krt16(-/-) front and hind paws, which severely compromise the animals' abilit

124 or rat glabrous skin blood perfusion in both hind paws, while a simultaneous heart rate (HR) and DRRs

125 ts were injected subcutaneously in the right hind paw with (99m)Tc-SPIONs (25-50 MBq, approximately 0

126 bar (L5)-DRG induced hyperalgesia in the rat hind paw with a profile similar to that of intraplantar

127 inal cord, rats displayed markedly decreased hind paw withdrawal thresholds, indicative of below-leve

128 esic responses to thermal stimulation of the hind paw without alterations in rearing behavior or body