ライフサイエンスコーパス: hindlimb

1 entivectors in the skeletal muscle of murine hindlimb.

2 nd neovascularization in the murine ischemic hindlimb.

3 echanisms that change proportions within the hindlimb.

4 the recovered proprioceptive function of the hindlimb.

5 k as monitored by five muscle nerves in each hindlimb.

6 flow, and functional recovery in an ischemic hindlimb.

7 f motor recovery, especially in ipsilesional hindlimb.

8 r cells, leading to severe truncation of the hindlimb.

9 rea of primary motor cortex representing the hindlimb.

10 al and lateral IO affected both forelimb and hindlimb.

11 the trunk body wall not associated with the hindlimb.

12 ting acute and chronic regeneration in a rat hindlimb.

13 are differentially modified in forelimb and hindlimb.

14 of the flight muscles and enlargement of the hindlimbs.

15 , followed by stepping-like movements in the hindlimbs.

16 brate cats walking on a treadmill with their hindlimbs.

17 rve, and the dorsal motor nerve in fore- and hindlimbs.

18 nic proteins in ischemic AMPKalpha2(DeltaMC) hindlimbs.

19 tep-training as opposed to training only the hindlimbs.

20 absence of a variable distal portion of the hindlimbs.

21 Mice underwent hindlimb Achilles' tendon transection and dorsal burn in

22 aining the forelimbs in conjunction with the hindlimbs after a thoracic spinal cord hemisection in ad

23 e, anesthetized mice were suspended with the hindlimb allowed to move freely in space.

24 rons that project to L5 selectively disrupts hindlimb alternation allowing a continuum of walking to

25 These data illustrate that hindlimb alternation can be manipulated independently fr

26 Both hindlimb and dorsal muscles were studied at 7, 14 and 21

27 e consistently observed a protrusion between hindlimb and forelimb representation, which in rats corr

28 ly higher blood flow restoration in ischemic hindlimb and higher capillaries density at histological

29 athway regulates skeletal development of the hindlimb and lower jaw through discrete populations of c

30 EB, an enhancer of Tbx4, produces defects in hindlimbs and genitalia, establishing the importance of

31 d functional survival of AD-MSCs in ischemic hindlimbs and provoked a synergetic effect with AD-MSCs

32 echanism when the animal was standing on its hindlimbs and which was partially dependent on the endog

33 ifferentially expressed between forelimb and hindlimb, and across different stages.

34 rongly negatively allometric relative to the hindlimb, and patterns of vascular canal orientation pro

35 rneuronal networks linking the forelimbs and hindlimbs are amenable to a rehabilitation training para

36 rated (predictable) sensory signals from the hindlimbs are likely to occur.

37 eropods, the pelvic girdle is downsized, the hindlimbs are short, and all of the limb bones are solid

38 ctions of RFA, CFA, and the caudally located hindlimb area (CHA), which is a part of M1, were determi

39 described, besides few reports of a rostral hindlimb area.

40 me3) analyses on its developing forelimb and hindlimb autopods at sequential embryonic stages to deci

41 tly test this hypothesis, we applied passive hindlimb bike exercise after complete thoracic transecti

42 otein synthesis rates that match the reduced hindlimb blood flow and oxygen consumption rates in IUGR

43 muscle protein synthesis rates match reduced hindlimb blood flow and oxygen consumption rates in the

44 These adaptations resulted in hindlimb blood flow rates in IUGR that were similar to c

45 Absolute hindlimb blood flow was reduced in IUGR (IUGR: 32.9 +/-

46 gen and nutrient supply to the fetus affects hindlimb blood flow, substrate uptake and protein accret

47 a synergetic effect with AD-MSCs to restore hindlimb blood perfusion and limb functions.

48 ly in a discrete posterior domain of nascent hindlimb bud mesenchyme.

49 of the embryo, at the level of the posterior hindlimb buds and anterior tail.

50 in pythons, and HOXD gene expression in the hindlimb buds progresses to the distal phase, forming an

51 Python hindlimb buds then develop transitory pre-chondrogenic c

52 ranscription is weak and transient in python hindlimb buds, leading to early termination of a genetic

53 the ancestral enhancer drives expression in hindlimbs but not forelimbs, in locations that have been

54 by bilateral mechanical hypersensitivity of hindlimbs, but corpus callosotomy eliminated the analges

55 Unlike Grem1, Chrdl1 is expressed in the hindlimb by a small subset of ZPA cells and their descen

56 li (VF neurons) is a potent activator of the hindlimb central pattern generators (CPGs) in rodent spi

57 ficantly improved the locomotor deficits and hindlimb clasping phenotype, both in male and female mic

58 noticeable tremor, and most animals showed a hindlimb clasping phenotype.

59 ormal, were more active and developed severe hindlimb clasping phenotypes.

60 eficient mice developed forelimb stereotypy, hindlimb clasping, excessive grooming and hypo-activity

61 ycle duration relationships differed in both hindlimbs concurrently during split-belt walking.

62 sured the cardiovascular responses to static hindlimb contraction or tendon stretch before and after

63 cally transformed mice and the disruption of hindlimb coordination following ablation of descending L

64 en verified that neurons in the deafferented hindlimb cortex increased their responsiveness to tactil

65 forepaw sensory cortex into the deafferented hindlimb cortex, associated with sprouting of corticospi

66 erwent massive changes after injury and that hindlimb cortical areas were recruited to control the fo

67 reover, the tissue perfusion in the ischemic hindlimb could be quantitatively measured by the dynamic

68 Fore-hindlimb coupling favored a more stable diagonal couplin

69 ns serve as a major link between SCA and the hindlimb CPGs and that the ability of SCA to induce step

70 deletion of the SAM domain induces a complex hindlimb defect associated with down-regulation of Trp63

71 Here we report that hindlimb development arrests in python embryos as a resu

72 nheritance; implicating 13 genes involved in hindlimb development in bilateral cases and 11 in unilat

73 d SMARCA4, known to play a role in embryonic hindlimb development.

74 howed that Has2 conditional mutant (Has2cko) hindlimbs display digit-specific patterning defects with

75 raising the possibility that re-emergence of hindlimbs during snake evolution did not require de novo

76 limb enhancer played a role in reduction of hindlimbs during snake evolution.

77 ence for a successive loss of forelimbs then hindlimbs during snake evolution.

78 relimbs were engaged simultaneously with the hindlimbs during treadmill step-training as opposed to t

79 kb1-mutant nerves and muscle atrophy lead to hindlimb dysfunction and peripheral neuropathy.

80 unctuated evolution of allometric scaling of hindlimb elements during the radiation of Dipodoidea.

81 demonstrate that the HLEB of snakes has lost hindlimb enhancer function while retaining genital activ

82 ng scratching and behave like neurons in the hindlimb enlargement.

83 ratching project descending axons toward the hindlimb enlargement.

84 contralateral L5, which contains many of the hindlimb extensor MNs activated by the LVST.

85 nd tracing the pathway transynaptically from hindlimb extensor muscles using rabies virus (RV).

86 ats, these results show that the encoding of hindlimb features in motor cortex dynamics is comparable

87 of interest in the ischemic and nonischemic hindlimbs for quantification of regional changes in CT-d

88 hat actively engaging the forelimbs improves hindlimb function and that one likely mechanism underlyi

89 therapy have the clear potential to protect hindlimb function from such adverse influence.

90 lectrophysiological analyses showed improved hindlimb function when the forelimbs were engaged simult

91 Hindlimb glucose uptake and lactate output rates were si

92 Net hindlimb glucose uptake and lactate output rates were si

93 mg/kg resulted in significant improvement in hindlimb grip strength and a 30% decrease in inflammatio

94 less of a decline in normalized forelimb and hindlimb grip strength and declines in in vitro EDL forc

95 placental insufficiency that develop to slow hindlimb growth and muscle protein accretion.

96 Metabolic adaptations to slow hindlimb growth are probably hormonally-mediated by mech

97 The loss of V2b INs results in hindlimb hyperextension and a delay in the transition fr

98 ation of limb morphology and forelimb versus hindlimb identity.

99 ty electrical stimulation of the ipsilateral hindlimb in awake rats evoked field potentials in the C1

100 of blood vasculature in the mouse brain and hindlimb in the NIR-IIb window with short exposure time

101 cing muscle damage and recovery in the lower hindlimbs in mice.

102 oral and pelvic fins in fishes and fore- and hindlimbs in tetrapods.

103 q palmitoylation in ischemic and nonischemic hindlimbs in vivo In summary, we demonstrate that NAC ac

104 ythmic pattern is highly correlated with the hindlimb ipsilateral flexor activities.

105 In a mouse hindlimb ischaemia model, we examine the effects of hMSC

106 ike CD31(+)/c-Kit(+) cells in mice following hindlimb ischaemia.

107 show impaired functional recovery following hindlimb ischaemia.

108 In mice with unilateral hindlimb ischemia (40%-50% reduction in flow), ultrasoun

109 the susceptibility of the peripheral limb to hindlimb ischemia (HLI).

110 These animals developed advanced hindlimb ischemia and digital autoamputation, secondary

111 njection, and iii) vascular flow recovery to hindlimb ischemia as indicated by laser Doppler and alph

112 ypic outcomes that follow surgically induced hindlimb ischemia between inbred mouse strains to identi

113 that miR-93 enhances perfusion recovery from hindlimb ischemia by modulation of multiple genes that c

114 These events can reverse hindlimb ischemia in mice for >24 hours and increase mus

115 essential for arteriogenesis in response to hindlimb ischemia in mice.

116 We induced hindlimb ischemia in wild-type and Dhh(-/-) mice.

117 on in rat tibialis anterior (TA) muscle in a hindlimb ischemia model (i.e., ligation of the iliac art

118 d flow recovery and neovessel formation in a hindlimb ischemia model compared with nondiabetic mice.

119 In a mouse hindlimb ischemia model of peripheral arterial disease,

120 ved the recovery of blood flow in the murine hindlimb ischemia model of peripheral artery disease.

121 c catalase transgenic mice (Cat-Tg mice) and hindlimb ischemia model to address the role of endogenou

122 In a murine hindlimb ischemia model, TUDCA-treated MSC transplantati

123 blood vessel perfusion function in a murine hindlimb ischemia model.

124 In this study, In vivo experiments with hindlimb ischemia models revealed that XBP1 deficiency i

125 ge numbers of newly formed arterioles in the hindlimb ischemia mouse model.

126 reperfusion after occlusion was removed) or hindlimb ischemia reperfusion injury (left leg tournique

127 Unilateral hindlimb ischemia was induced by ligating femoral artery

128 apillary density and perfusion recovery from hindlimb ischemia, and antagomirs to miR-93 attenuated p

129 ng with larger extent of tissue damage after hindlimb ischemia, as compared to wild-type (WT) litterm

130 in their extent of perfusion recovery after hindlimb ischemia, we found that the mouse strain with h

131 mps in diabetes using a novel in vivo murine hindlimb ischemia-amputation model.

132 the murine femoral artery ligation model of hindlimb ischemia.

133 d miR-155 as a downregulated microRNA during hindlimb ischemia.

134 ndent vasodilator pathways, and in mice with hindlimb ischemia.

135 od flow recovery and capillary density after hindlimb ischemia.

136 ponse to ischemic injury in animal models of hindlimb ischemia.

137 crucial for capillary angiogenesis in rodent hindlimb ischemia.

138 erformed in vivo studies in animals model of hindlimb ischemia.

139 pressing MYOCD and TERT in a murine model of hindlimb ischemia.

140 icroRNAs involved in perfusion recovery from hindlimb ischemia.

141 was prepared from C57BL/6-mice subjected to hindlimb ischemia.

142 d in vivo angiogenic properties in mice with hindlimb ischemia.

143 d pigs were subjected to RIPC (4x5/5 minutes hindlimb ischemia/reperfusion) or placebo (PLA) before 6

144 In the hindlimb, Isl1-lineages broadly contributed to the mesen

145 Rats dosed with CK-2066260 showed increased hindlimb isometric and isokinetic force in response to s

146 hat control antagonistic muscles at a single hindlimb joint.

147 ulatory movements to bilaterally synchronous hindlimb kick propulsion in the adult.

148 on responses in the hindlimb motor cortex to hindlimb kinematics and hindlimb muscle synergies across

149 cy, associated with altered EMG patterns and hindlimb kinematics during gait.

150 Hindlimb kinematics, SO fascicle and muscle-tendon unit

151 Siberia with small scales around the distal hindlimb, larger imbricated scales around the tail, mono

152 Morphological comparison showed that lower hindlimb length in the introduced populations tended to

153 Slower hindlimb linear growth and muscle protein synthesis rate

154 We demonstrate slower hindlimb linear growth and muscle protein synthesis rate

155 nical coupling, to partly recover unassisted hindlimb locomotion after complete spinal cord injury.

156 Four adult cats that recovered stable hindlimb locomotion after spinal transection were implan

157 the spinal cord that control left and right hindlimb locomotion can differentially and simultaneousl

158 Hindlimb locomotion was restored by reestablished integr

159 cats and primates, cortical contribution to hindlimb locomotor movements is not critical in rats.

160 ence that, despite its broad contribution to hindlimb mesenchyme and facial epithelium, the Isl1-beta

161 s that give rise to Shh-expressing posterior hindlimb mesenchyme and Fgf8-expressing mandibular epith

162 g 26 days revealed a significant increase in hindlimb microvascular density in response to experiment

163 ls differentially to contralateral trunk and hindlimb MNs in the mammalian spinal cord.

164 emia reperfusion and injured leg muscle in a hindlimb model of ischemia reperfusion.

165 In a murine ischaemic hindlimb model, MRTF-A or Tss4 promotes neovascularizati

166 sly enhanced the rhythmic performance of the hindlimbs more robustly than ES or IS alone.

167 onized with the spatiotemporal activation of hindlimb motoneurons.

168 aling in retinocollicular development and in hindlimb motor axon guidance, suggesting that chick and

169 in rats suggests that cortical engagement in hindlimb motor control may depend on the behavioral cont

170 temporally scaled activity occurs in the rat hindlimb motor cortex in the absence of motor output and

171 targeted deletion of FGFR1 and FGFR2 in the hindlimb motor cortex limits the formation of new synaps

172 whole-body kinematics, muscle synergies, and hindlimb motor cortex modulation in freely moving rats p

173 We found that the activation of hindlimb motor cortex preceded gait initiation.

174 ped the neuronal population responses in the hindlimb motor cortex to hindlimb kinematics and hindlim

175 ubsets of lumbar motor neurons: HGF supports hindlimb motor neurons through c-Met; CNTF supports subs

176 neuronal activity, were used to replace the hindlimb movement as a trigger for reward in real-time (

177 saphenous nerve block before, but not after, hindlimb movement blocked movement-induced BTP.

178 ouse (lamb1t) exhibits intermittent dystonic hindlimb movements and postures when awake, and hyperext

179 In reduced preparations, hindlimb movements can be generated by a minimal network

180 ted axons in the implants and improvement in hindlimb movements.

181 We further show that, in ischaemic rabbit hindlimbs, MRTF-A as well as Tss4 induce functional neov

182 ion were implanted with electrodes to record hindlimb muscle activity chronically and to stimulate th

183 ith higher levels of microRNA-93 (miR-93) in hindlimb muscle before ischemia and the greater ability

184 ce mean arterial pressure (MAP) and increase hindlimb muscle blood flow in the exercising rat.

185 logical analyses revealed that Stac3-deleted hindlimb muscle contained more slow type-like fibers tha

186 o frequent electrostimulation, Stac3-deleted hindlimb muscle contracted but the maximal tension gener

187 sponses to electrically-induced intermittent hindlimb muscle contractions.

188 nal discharges reliably evoke contra-lateral hindlimb muscle contractions.

189 Arimoclomol significantly improved hindlimb muscle force and contractile characteristics, r

190 Rodents dosed with CK-2066260 show increased hindlimb muscle force and power in response to submaxima

191 ted against muscle fatigue and increased mdx hindlimb muscle force by 40%, a value comparable to curr

192 antibody (GSK577548) significantly improves hindlimb muscle innervation at 90 days, a late symptomat

193 limb motor cortex to hindlimb kinematics and hindlimb muscle synergies across a spectrum of natural l

194 adherin/CD31+/CD45-) isolated from uninjured hindlimb muscle tissue undergo in vivo EndMT when transp

195 unctions in skeletal muscle, at least in the hindlimb muscle.

196 etermined the contractility of Stac3-deleted hindlimb muscle.

197 0 in fibres of the entire posterior group of hindlimb muscles (gastrocnemius, soleus, and plantaris)

198 d vascular conductance for the 28 individual hindlimb muscles and muscle parts correlated positively

199 Likewise, the hindlimb muscles of rats with ligated femoral arteries s

200 Likewise in five rats whose hindlimb muscles were freely perfused, the pressor and c

201 Primary myoblasts were isolated from hindlimb muscles, and after 3 days in growth media (20%

202 rological impairment, delayed denervation of hindlimb muscles, and prolonged survival of spinal motor

203 s area control movements involving trunk and hindlimb muscles, those in the intermediate part control

204 on in coordinating the activity of trunk and hindlimb muscles.

205 ages to the pool of satellite cells in adult hindlimb muscles.

206 n- and cartilage-like tissues from the lower hindlimb of Early Cretaceous Confuciusornis.

207 Unilateral exposure of the proximal hindlimb of mice (with or without ischemia produced by i

208 MSCs transplanted to the ischemic hindlimb of nude mice showed significantly higher BLI an

209 The hindlimb of theropod dinosaurs changed appreciably in th

210 ogenesis when transplanted into the ischemic hindlimbs of apolipoprotein E-null mice.

211 tested this hypothesis in the left and right hindlimbs of four intact and two chronic spinal-transect

212 of praying mantids (Mantidae), the elongated hindlimbs of grasshoppers (Orthoptera: Caelifera), and t

213 0% decrease in inflammation in the fore- and hindlimbs of mdx mice.

214 into the anterior tibial compartment of the hindlimbs of NOD-Rag1(null) IL2rgamma(null) immunodefici

215 mechanical hypersensitivity develops in the hindlimbs of rats in parallel with a reduction in all co

216 genitalia develop directly from the budding hindlimbs, or the remnants thereof, whereas in mice the

217 encephalomyelitis mouse model with complete hindlimb paralysis and death by 30 d after induction of

218 06 in mice results in progressive ataxia and hindlimb paralysis associated with motor neuron degenera

219 their ablation using PLP-CreERT resulted in hindlimb paralysis with immobility at approximately 30 d

220 nn cells displayed tremor that progressed to hindlimb paralysis, which correlated with diminished num

221 extended median survival by 50% and delayed hindlimb paralysis, with animals remaining ambulatory un

222 e decline of motor function and the onset of hindlimb paralysis.

223 disease severity, resulting in recovery from hindlimb paralysis.

224 imbs, leading to the accelerated recovery of hindlimb perfusion and superior muscle regeneration.

225 Hindlimb perfusion recovery was attenuated in Shc condit

226 Confuciusornis may indicate a more crouched hindlimb posture.

227 fusion, between-limb proportions, and within-hindlimb proportions all evolved independently of one an

228 ed the crocodylian-like ankle morphology and hindlimb proportions of stem archosaurs and early pseudo

229 In contrast, the hindlimbs provide relatively weak thrust in all simulati

230 dual specimens, showing that the forelimb-to-hindlimb ratio changed rapidly during the first or secon

231 y, that terminate as climbing fibers in the "hindlimb-receiving" parts of the C1 and C3 zones in the

232 ug of GsMTx4 into the arterial supply of the hindlimb reduced the peak pressor (control: 24 +/- 2, Gs

233 ug of GsMTx4 into the arterial supply of the hindlimb reduced the peak pressor (control: 24 +/- 5, Gs

234 ive DIs interfere with normal mouse ischemic hindlimb regeneration and suggest that their use could b

235 RFA, caudal forelimb area (CFA), and caudal hindlimb region were determined for comparison.

236 elopmental mechanisms, and elongation of the hindlimb relative to the forelimb is not simply due to g

237 6 mice were subjected to S/R with or without hindlimb RIC.

238 viable molecular mechanisms for diversity in hindlimb scale and feather distribution.

239 segments are functionally integrated in the hindlimb scratch network.

240 urons in mid-thoracic segments contribute to hindlimb scratching and may be part of a distributed mot

241 To address this issue, we use turtle hindlimb scratching as a model for fine motor control, s

242 -line, information encoded by neurons in the hindlimb sensorimotor cortex was assessed.

243 ed in the left hindlimb, such that the right hindlimb serves as an internal control.

244 Contrast ultrasound perfusion imaging of hindlimb skeletal muscle and femoral artery diameter mea

245 Total exercising hindlimb skeletal muscle blood flow (control: 108 +/- 8,

246 SMTC reduced total hindlimb skeletal muscle blood flow (control: 241 +/- 23

247 Subsequently, hindlimb skeletal muscle blood flow (radiolabelled micro

248 hyl-l-thiocitrulline (SMTC) would reduce rat hindlimb skeletal muscle blood flow and vascular conduct

249 MAP and hindlimb skeletal muscle blood flow and vascular conduct

250 ribute to the hyperaemic response within rat hindlimb skeletal muscle during low-speed treadmill runn

251 well as reduced parasite burden in heart and hindlimb skeletal muscle.

252 ke acutely influences signal transduction in hindlimb skeletal muscle.

253 The hindlimb skeletal muscles were exposed to ultrasound fro

254 Isl1-expressing cells caused agenesis of the hindlimb skeleton and absence of the lower jaw (agnathia

255 nt in all skin tissues, localized kPhd2KO in hindlimb skin tissues did not have similar effects, excl

256 igin and therefore evoked by transmission in hindlimb SOCPs.

257 s sequentially from visual to barrel then to hindlimb somatosensory; the second principle is correlat

258 regulatory changes in the genes encoding the hindlimb-specific transcription factor Pitx1 and forelim

259 Rats showed significant improvement in hindlimb stepping ability, quadrupedal weight support, a

260 surface of the spinal cord at L3-L7 induced hindlimb stepping-like movements on a moving treadmill b

261 he sensory signal, some animals received the hindlimb stimulation only during phase 2.

262 h injury is specifically induced in the left hindlimb, such that the right hindlimb serves as an inte

263 models of ischemia in the heart, brain, and hindlimb that only in the brain does NADPH oxidase 4 (NO

264 bited shifts in investment from forelimbs to hindlimbs that were qualitatively similar to anatomical

265 Moreover, in the hindlimb there are only two muscle absence/presence diff

266 f altered regulation of CaV2.1 channels, the hindlimb tibialis anterior muscle in IM-AA mice exhibite

267 ETHODS AND When injected into mouse ischemic hindlimb tissue, CD34Exo, but not the CD34Exo-depleted c

268 ns result from a partial transformation from hindlimb to forelimb identity mediated by cis-regulatory

269 of preservation solutions in a syngenic rat hindlimb transplant model.

270 both a sciatic nerve cut-and-repair and rat hindlimb transplant model.

271 of nerve regeneration after nerve injury and hindlimb transplantation.

272 Similarly, rats undergoing allogeneic hindlimb transplants treated with local injection of MSC

273 LEW) (n = 4) and allogeneic (BN-LEW) (n = 4) hindlimb transplants were performed and assessed for neu

274 oved nerve regeneration following allogeneic hindlimb transplants.

275 is rat recipients of mismatched Brown Norway hindlimb transplants.

276 ts received full-mismatched Brown Norway rat hindlimb transplants.

277 he tibialis anterior muscle and the onset of hindlimb tremor.

278 obot impedance control at the pelvis allowed hindlimb, trunk, and forelimb mechanical interactions.

279 r-Brown Norway rats at 10 months of age were hindlimb unloaded for a period of 2 weeks.

280 rmine if absence of MAT reduced bone loss in hindlimb-unloaded (HU) mice.

281 s, toxin injuries, ischemia-reperfusion, and hindlimb unloading and reloading.

282 IR-II) fluorescence imaging, to image murine hindlimb vasculature and blood flow in an experimental m

283 nto the body, we demonstrated that the mouse hindlimb vasculature could be imaged with higher spatial

284 wild-type littermates resulting in impaired hindlimb vasodilatation to the ACE substrate, bradykinin

285 rgonomic pain models (eccentric exercise and hindlimb vibration) and in a model of endometriosis.

286 The soleus of the immobilized-reambulated hindlimb was found to have a greater amount of muscle da

287 entation provide evidence that growth of the hindlimb was particularly rapid during the middle part o

288 These mice display hindlimb weakness and impaired axonal conduction in scia

289 spinalized adult rats can recover unassisted hindlimb weight support and locomotion without explicit

290 bot rehabilitation that promotes recovery of hindlimb weight support functions on trunk motor cortex

291 1) ; P < 0.005), although flow normalized to hindlimb weight was similar between groups.

292 Hindlimb weight, linear growth rate, muscle protein accr

293 , P < 0.0001) was negatively associated with hindlimb weight.

294 orepinephrine was negatively associated with hindlimb weight.

295 Divided tibial bones in one hindlimb were gradually lengthened at 0.7 mm per day usi

296 cond order intramuscular arterioles from rat hindlimb were isolated, cut longitudinally, fixed, and i

297 restingly, CD34Exo, when treated to ischemic hindlimbs, were most efficiently internalized by endothe

298 menter-induced movement of the tumor-bearing hindlimb with a context produces conditioned place avoid

299 nglion neurons, reversed hypersensitivity of hindlimb withdrawal reflexes, and reduced ongoing pain a

300 ion (T10), cats recovered stepping with both hindlimbs within 3 weeks.