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4 ed in a small but significant improvement in hip bone density, did not significantly reduce hip fract
7 iation between dietary patterns, measures of hip bone geometry, and subsequent fracture risk are scar
8 men, PAD was associated with higher rates of hip bone loss and increased risk of nonspine fractures.
9 one mineral density at the hip and the heel, hip bone loss over 2 years, and fractures during 3.5 yea
11 intake during childhood and adolescence and hip bone mineral content and bone mineral density (P < 0
12 scence was associated with a 3% reduction in hip bone mineral content and bone mineral density (P < 0
13 oints were changes in lumbar spine and total hip bone mineral densities (BMDs); secondary endpoints w
14 3.72%, 95% CI 1.54 to 5.89; p=0.26), nor did hip bone mineral density (2.09%, 95% CI -1.45 to 5.63 vs
15 yunsaturated fatty acid and fish intakes and hip bone mineral density (BMD) at baseline (1988-1989; n
16 mary endpoint was percentage change in total hip bone mineral density (BMD) from baseline to week 48
22 percentage changes in lumbar spine and total hip bone mineral density at week 48, assessed by dual en
23 howed a smaller decrease in lumbar spine and hip bone mineral density but greater accumulation of lim
25 ne mineral density secondary outcomes, total hip bone mineral density increased more in the teriparat
28 c effect of weight change on change in total hip bone mineral density was evaluated over 4 years (199
30 th differences in percentage change in total hip bone mineral density were 0.79 percentage point (95%
31 n (serum type I collagen C-telopeptide), low hip bone mineral density, absence of urticaria pigmentos
33 els adjusted for age, body mass index (BMI), hip bone mineral density, knee surgery or pain, and phys
34 cs sex, serum type I collagen C-telopeptide, hip bone mineral density, urticaria pigmentosa, and alco
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