ライフサイエンスコーパス: hippocalcin

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1 HP channels is the diffusible calcium sensor hippocalcin.

2 binding by neuronal calcium sensors such as hippocalcin.

3 unis et al. now show that calcium binding to hippocalcin, a member of the NCS family, is one of the n

4 We obtained the first crystal structure of hippocalcin and alignment with other NCS proteins showed

5 Finally we show that hippocalcin and neurocalcin delta both increase the calc

6 We propose that hippocalcin binds to the SH3 region of PSD-95 under basa

7 These considerations question whether hippocalcin can be the sole calcium sensor for the slow

8 e dystonia-causing mutations strongly affect hippocalcin cellular functions which suggest a central r

9 Mutations T71N and A190T in hippocalcin did not affect stability, calcium-binding af

10 [Ca2+] was examined in HeLa cells expressing hippocalcin-enhanced yellow fluorescent protein (EYFP) t

11 a2+] ramp and revealed that translocation of hippocalcin-EYFP initiated at around 180 nM and was half

12 tamine induced a reversible translocation of hippocalcin-EYFP.

13 n and overexpression strategies to show that hippocalcin functions as a calcium sensor for the slow a

14 these mutations on the physiological role of hippocalcin has not yet been elucidated.

15 Introduction of hippocalcin in cultured hippocampal neurons leads to a p

16 Translocation of hippocalcin in response to increased cytosolic [Ca2+] wa

17 Hippocalcin is a neuronal calcium sensor protein that po

18 The data show that hippocalcin is a sensitive Ca2+ sensor capable of respon

19 3 is essential for maintaining the sAHP when hippocalcin is ablated, a condition that likely impairs

20 urrent in the cerebral cortex, an area where hippocalcin is expressed at much lower levels than in hi

21 However, while hippocalcin is very strongly expressed in the hippocampu

22 that the decay of the sAHP was prolonged in Hippocalcin knockout mice, and that the decay was sensit

23 al neuron sAHP current in both wild-type and Hippocalcin knockout mice, indicating that the gating of

24 wild-type mice do not elicit the I(sAHP) in hippocalcin knockout mice.

25 those of serine/threonine kinase 16 (STK16), hippocalcin-like 1 (HPCAL1) as well as neurocalcin-delta

26 ing that the neuronal calcium sensor protein hippocalcin may allow for these dual signaling processes

27 Functional studies suggest that hippocalcin might play a role in regulating voltage-depe

28 in KCl-depolarised cells expressing mutated hippocalcin, mostly driven by N-type voltage-gated calci

29 This implies that hippocalcin must bind to the plasma membrane to mediate

30 onounced I(sAHP), while neurons expressing a hippocalcin mutant lacking N-terminal myristoylation exh

31 idisciplinary approach, we demonstrated that hippocalcin oligomerises in a calcium-dependent manner a

32 between the neuronal calcium sensor protein hippocalcin, the clathrin adaptor molecule AP2, the post

33 kinetics in wild-type mice and mice lacking Hippocalcin, the putative sAHP calcium sensor.

34 Ca2+-free hippocalcin was freely diffusible, as shown by photoblea

35 ecently, the neuronal calcium sensor protein hippocalcin was identified as a calcium sensor for the s

36 d A190T in the neuronal calcium sensor (NCS) hippocalcin were identified as the genetic cause of prim

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