ライフサイエンスコーパス: hippocampal

1 In adult mice, attenuation of hippocampal ACSS2 expression impairs long-term spatial m

2 We found that greater hippocampal activation at baseline was associated with i

3 05], 6 months [F3,20 = 5.74, p = .007]) with hippocampal activation predicting PTSD symptoms at 3 and

4 iven trial was correlated significantly with hippocampal activation such that more fixations were ass

5 more fixations were associated with stronger hippocampal activation.

6 We then recorded hippocampal activity as disoriented mice performed a cla

7 model, we examined the relationship between hippocampal activity during a memory task using fMRI and

8 oencephalographic (iEEG) work has shown that hippocampal activity during encoding predicts subsequent

9 emory encoding [1, 2], little is known about hippocampal activity during spontaneous memory retrieval

10 ring navigation of the city, right posterior hippocampal activity indexes the change in the number of

11 ailable for future travel and right anterior hippocampal activity reflects global properties of the s

12 The impact of dentate mossy cells on hippocampal activity remained uncertain despite a long h

13 ound-biased AC patterns predicted subsequent hippocampal activity.

14 Epileptic seizures potently modulate hippocampal adult neurogenesis, and adult-born dentate g

15 formance in aged rats, prevented many of the hippocampal age-related gene expression changes.

16 ent with GZ667161 slowed the accumulation of hippocampal aggregates of alpha-synuclein, ubiquitin, an

17 e EtOH intoxication and CIE, upregulation of hippocampal alpha4betagamma2 subtypes, as well as increa

18 -AG-mediated synaptic suppression at ventral hippocampal-amygdala glutamatergic synapses and amygdala

19 tified dendritic morphology in CA1 pyramidal hippocampal and adjacent somatosensory pyramidal cortica

20 Persistent activity of hippocampal and amygdala neurons was stimulus-specific,

21 Many studies report smaller hippocampal and amygdala volumes in posttraumatic stress

22 We show that betaIII spectrin in hippocampal and cortical neurons from rodent embryos of

23 he cellular effects of TDP-43 dysfunction in hippocampal and cortical neurons.

24 dually or in combinations, in cultured mouse hippocampal and cortical neurons.

25 e similarity, and possible homology, between hippocampal and DD/DL circuitry.

26 Here we tested this idea by recording from hippocampal and entorhinal neurons during a task that re

27 sequences during spontaneous ictal events in hippocampal and neocortical circuits in experimental mod

28 SIGNIFICANCE STATEMENT: The hippocampal and oculomotor networks have each been studi

29 These results help shape models of how hippocampal and prefrontal regions support navigation, p

30 n the posterior cingulate, lateral parietal, hippocampal, and parahippocampal cortices and amygdala,

31 We discovered that place fields in hippocampal area CA1 are produced by a synaptic potentia

32 Activity in hippocampal area CA1 is essential for consolidating epis

33 Using chronic two-photon Ca(2+) imaging in hippocampal area CA1 of wild-type and Df(16)A(+/-) mice,

34 tin D1 (NPD1), which is decreased in the CA1 hippocampal area of patients with early-stage Alzheimer'

35 ween interhemispheric homotopic cortical and hippocampal areas, as well as in cortico-striatal pathwa

36 ack loop and connecting functionally diverse hippocampal areas.

37 BI compared with vehicle, and a reduction in hippocampal astrogliosis.

38 ed that increased rates of TDP-43-associated hippocampal atrophy might occur at least 10 years before

39 We measured hippocampal atrophy morphometrically, and all brains wer

40 ression measuring cognition and function and hippocampal atrophy were observed.

41 hese deficits were accompanied by pronounced hippocampal atrophy, including subfields cornu ammonis 2

42 tective, rescued memory deficits and reduced hippocampal atrophy.

43 Moreover, a significant increase in hippocampal BDNF was detected in APPSWE /PS1dE9 mice exp

44 duced a reversal of age-dependent effects on hippocampal brain-derived neurotrophic factor (BDNF) BDN

45 Higher hippocampal, but not prefrontal GABA, predicted stronger

46 rectly impacted region and the contralateral hippocampal CA1 area and was accompanied by increased lo

47 1, but not ID mutants, rescued spine loss in hippocampal CA1 neurons in Grasp1 knockout mice.

48 ishes GABA-mediated synaptic transmission in hippocampal CA1 neurons without affecting their excitato

49 es and near dendritic spines of mouse dorsal hippocampal CA1 neurons.

50 questions, we recorded intracellularly from hippocampal CA1 of awake, behaving male mice to examine

51 induction of long-term potentiation (LTP) in hippocampal CA1 pyramidal cells and hippocampal-dependen

52 (EC) and Schaffer collateral (SC) inputs to hippocampal CA1 pyramidal neurons (PNs) produces a long-

53 Following global cerebral ischemia, hippocampal CA1 pyramidal neurons are more vulnerable to

54 Using dynamic clamp in hippocampal CA1 pyramidal neurons in brain slices, we sh

55 f total and mushroom dendritic spines in the hippocampal CA1 region.

56 presynaptic organization, especially in the hippocampal CA1 stratum radiatum, and also diminishes GA

57 In the hippocampal CA1 stratum radiatum, the values of the CB1

58 eceptors (OXTRs) are abundantly expressed in hippocampal CA2 and CA3 regions, but there are little kn

59 he Teevra cells, selectively innervating the hippocampal CA3 area bypassing CA1, CA2, and the dentate

60 bility, in the absence of synaptic input, in hippocampal CA3 neurons, a classical focus for epileptog

61 g RNA sequencing of ribosome-bound mRNA from hippocampal CA3 neurons, we found remarkable sex differe

62 The hippocampal CA3 region is classically viewed as a homoge

63 vel form of long-term synaptic depression at hippocampal CA3-CA1 synapses (O-GlcNAc LTD).

64 del of Ca2+, CaM, and seven highly-expressed hippocampal CaM binding proteins, we find that competiti

65 valuated using acute hippocampal slices, and hippocampal cannabinoid receptor type 1 and brain-derive

66 t in stimulating cAMP signaling in the mouse hippocampal cell line HT-22 leading to an increase in CR

67 Following this training period, hippocampal cerebral blood flow (CBF) was measured by fu

68 initial insult during which maladaptation of hippocampal circuitries takes place.

69 hodopsin for the optogenetic manipulation of hippocampal circuitry with light.

70 Hippocampal cis-meQTL-CpGs are enriched in flanking regi

71 PFC (mPFC) activity on spatial learning and hippocampal coding in a plus maze task that requires bot

72 ampal high-frequency band activity in single hippocampal contacts.

73 including DLPFC, thus supporting a specific hippocampal contribution to contextually mediated memory

74 arborization within the apical dendrites of hippocampal cornu ammonis 1 and granule cell neurons, ef

75 that during sleep there is a rapid cortical-hippocampal-cortical loop of information flow around the

76 ail, supporting functional specialization of hippocampal-cortical networks.

77 t prefrontal GABA, predicted stronger fronto-hippocampal coupling during suppression, suggesting that

78 Our data from mouse primary hippocampal cultures demonstrate that menin and its calp

79 In mouse and rat primary hippocampal cultures, synaptic activity caused an up-reg

80 c exposure in these animals are seizures and hippocampal damage, and they have been proposed as a nat

81 entiation of neural progenitors in the adult hippocampal dentate gyrus (DG), one of the select region

82 pathophysiological mechanisms involving the hippocampal dentate gyrus have been proposed.

83 (LTP) in hippocampal CA1 pyramidal cells and hippocampal-dependent cognitive function.

84 conditional knock-out mice exhibit impaired hippocampal-dependent fear learning.

85 the hippocampus of middle-aged mice enhances hippocampal-dependent learning and memory and restores i

86 ejuvenates hippocampal function and improves hippocampal-dependent learning.

87 pocampus, as transducing OCN's regulation of hippocampal-dependent memory in part through inositol 1,

88 pheral metabolism and cognition, deficits in hippocampal-dependent spatial learning and memory were e

89 t aerobic fitness levels are associated with hippocampal DG-related memory, which is consistent with

90 he novel object recognition task, suggesting hippocampal dysfunction.

91 for targeting ageing- or disease-associated hippocampal dysfunction.

92 ve decline in neural connectivity, affecting hippocampal efferent pathways documented by magnetic res

93 ht to provide a teaching signal that enables hippocampal encoding of memories, but its role during re

94 map-like knowledge structure directly from a hippocampal-entorhinal functional magnetic resonance ima

95 uggest that common circuit mechanisms in the hippocampal-entorhinal system are used to represent dive

96 Here, we show that the human hippocampal-entorhinal system can represent relationship

97 The hippocampal-entorhinal system encodes a map of space tha

98 nriched with epilepsy-eQTLs than with normal hippocampal eQTLs from two larger independent published

99 schizophrenia) are more enriched with normal hippocampal eQTLs than with epilepsy-eQTLs.

100 stimulation leads to a reduction in ongoing hippocampal-evoked MSNs responses through the combined r

101 egular oscillations, coincident with highest hippocampal excitability.

102 ory deficit induced by FAE, and reversed the hippocampal expression changes in the thyroid hormone-in

103 Moreover, we found that hippocampal expression of a mutated version of TRIP8b fu

104 the same animals identified 2342 genes with hippocampal expression that was upregulated/downregulate

105 dal cells in CA1 appear to sprout across the hippocampal fissure to preferentially synapse onto early

106 Here, we tested whether hippocampal FKBP1b overexpression also counteracts aging

107 grid, border and head direction cells in the hippocampal formation and related structures.

108 The hippocampal formation is a brain structure integrally in

109 terminalis, mesocortical structures and the hippocampal formation.

110 d male mice using a viral vector rejuvenates hippocampal function and improves hippocampal-dependent

111 novel targets of intervention for improving hippocampal function in Rett syndrome individuals.

112 map has long been the dominant metaphor for hippocampal function, embracing the idea that place cell

113 previously unknown roles for TIMP2 in normal hippocampal function.

114 n adult and aged mice demonstrating improved hippocampal function.

115 How hippocampal GABA contributes to stopping unwanted though

116 eta-analyses have, however, reported reduced hippocampal grey matter volume in MDD and reduced white

117 replay of waking neocortical activity under hippocampal guidance leads to memory consolidation.

118 milarity analysis on the temporal pattern of hippocampal high-frequency band activity in single hippo

119 erexpression of CBSH3- or CBSH3+ in cultured hippocampal (HP) neurons.

120 g intrusive thoughts is also associated with hippocampal hyperactivity, arising from dysfunctional GA

121 ring, which may support sparse coding during hippocampal information processing.

122 ort a general molecular mechanism underlying hippocampal inhibitory synapse development.

123 g longitudinal multi-modal MRI, we monitored hippocampal injury and tissue reorganization during epil

124 he first evidence in humans linking neonatal hippocampal injury to adult dopamine dysfunction, and pr

125 ed the effect of maternal CB intake on mouse hippocampal interneurons largely focusing on cholecystok

126 This included viral transfection of hippocampal interneurons with channelrhodopsin for the o

127 ystems consolidation dynamics by maintaining hippocampal involvement in the memory.

128 ical mechanism for the expression of LTP and hippocampal learning.

129 Perinatal brain injuries, including hippocampal lesions, cause lasting changes in dopamine f

130 rments during scene discrimination following hippocampal lesions.

131 in the 6- to 10-Hz range dominate the rodent hippocampal local field potential during translational m

132 SSRIs selectively inhibited hippocampal long-term depression.

133 r PTHS mouse models demonstrated exaggerated hippocampal long-term potentiation (LTP), consistent wit

134 nsgenic mice and concomitantly rescues their hippocampal long-term potentiation deficit.

135 e were now far more bioactive: they impaired hippocampal LTP, decreased neuronal levels of beta2-adre

136 ate the mnemonic contributions of two direct hippocampal-medial prefrontal cortex (mPFC) pathways, on

137 cell layer of the dentate gyrus and rescues hippocampal memory defects in a mouse model of Kabuki sy

138 e prefrontal engram cells, with support from hippocampal memory engram cells, became functionally mat

139 y active lifestyle has beneficial effects on hippocampal memory function.

140 wing positive effect of physical exercise on hippocampal memory.

141 ortant role in novelty related modulation of hippocampal memory.

142 Our comprehensive maps of cis-acting hippocampal meQTLs and eQTLs provide a link between dise

143 Cis-acting SNPs of hippocampal meQTLs and eQTLs significantly overlap schiz

144 egulation of HMGB1 messenger RNA in enriched hippocampal microglia, an effect that persists for up to

145 CA4/dentate gyrus (DG), as well as impaired hippocampal microstructural integrity.

146 Fear Algorithm), a physiologically realistic hippocampal model positing that acquisition and retrieva

147 Thus, enhancing hippocampal muscarinic signaling using M1 mAChR PAMs res

148 tment also prevented BCCAO-induced damage to hippocampal myelin sheaths and oligodendrocytes, enhance

149 ssful memory consolidation requires coherent hippocampal-neocortical communication mediated by PV(+)

150 rstanding representations of neural codes of hippocampal-neocortical networks during sleep would reve

151 ctor, grid and place cells in the entorhinal-hippocampal network form the brain's navigational system

152 at repeats occurred independently of classic hippocampal network states like theta oscillations or sh

153 ly long patterns of repeated activity in the hippocampal network that occur nonstochastically, are tr

154 fferentiation of doublecortin-positive adult hippocampal neural progenitors into functionally mature

155 Replay of hippocampal neural representations during sleep is thoug

156 Hippocampal neural stem cells (NSCs) integrate inputs fr

157 for the first time that the extent of early hippocampal neurodegeneration and progressive microstruc

158 s expressed in astrocytes could affect adult hippocampal neurogenesis and contribute to aspects of ps

159 tant DISC1) in astrocytes would affect adult hippocampal neurogenesis and hippocampus-dependent behav

160 hology and neuronal stem cells suggests that hippocampal neurogenesis impairment is an important fact

161 Many antidepressants stimulate adult hippocampal neurogenesis, but the mechanisms by which th

162 identify Sema7A as a key regulator of adult hippocampal neurogenesis, providing an example of how di

163 rbofuran exposure has detrimental effects on hippocampal neurogenesis, the generation of new neurons

164 a therapeutic effect that has been linked to hippocampal neurogenesis.

165 ession, which, importantly, impacted several hippocampal neuron plasticity processes.

166 Compound 23 protected hippocampal neuronal cells from the excitotoxic insult,

167 n of GIRK channel function and expression in hippocampal neuronal injury during prolonged epileptic s

168 In adult hippocampal neuronal precursor cells, RIT1 controls an A

169 genetic conditional ablation of Hdac1 in CA1 hippocampal neurons (i.e., Camk2a-cre;Hdac1(fl/fl)), we

170 Silencing CYLD in hippocampal neurons abolishes NMDA-induced chemical long

171 promote the production of Ephexin5 in mature hippocampal neurons and in mice expressing human amyloid

172 nti-NL1 antibodies reduced AbetaO binding to hippocampal neurons and prevented AbetaO-induced neurona

173 ic calcium signals along dendritic arbors of hippocampal neurons and relate this to measures of synap

174 nsufficient nuclear calcium signaling in CA1 hippocampal neurons and, consequently, reduced expressio

175 high-cholesterol diet, cholesterol levels in hippocampal neurons are increased.

176 endocrine and neuropeptidergic signaling in hippocampal neurons as a novel substrate of importance i

177 Following CCI injury, hippocampal neurons downregulated d-serine levels, while

178 e scrutinized cryo-electron tomograms of rat hippocampal neurons for the occurrence and spatial distr

179 ine, structural remodeling of prefrontal and hippocampal neurons has been proposed as critical.

180 from the cytosol/dendrites to the nucleus of hippocampal neurons in the mouse brain.

181 n and is able to restore their deficiency in hippocampal neurons obtained from PS1-M146V-KI AD mouse

182 Short-term exposure of cultured rat hippocampal neurons or ex vivo human cortical slices to

183 t-mediated repulsion events in primary mouse hippocampal neurons over 9 min at 2 s temporal resolutio

184 er to deliver precise amounts of reagents to hippocampal neurons to elicit time- and dose-precise res

185 erived endocrine signals act on receptors in hippocampal neurons to reduce (leptin, glucagon-like pep

186 echanisms to determine theta-phase timing of hippocampal neurons to support memory and spatial naviga

187 GD/ischemia contributes to neuronal death in hippocampal neurons via diverse effects on NADPH oxidase

188 impact of Cav-1 on structural plasticity of hippocampal neurons with age.

189 vs. AE3(-/-) mice show that AE3 (present in hippocampal neurons, not astrocytes; mediates HCO3(-) ef

190 sed in a cell line derived from PrP knockout hippocampal neurons, NpL2.

191 ulates in the somatodendritic compartment of hippocampal neurons, where it forms immobile complexes o

192 s reshaping includes the addition of newborn hippocampal neurons.

193 N1 controls the plasticity of cultured mouse hippocampal neurons.

194 ical player in Rac1 regulation during OGD in hippocampal neurons.

195 stributes in clusters along the dendrites of hippocampal neurons.

196 go in real time within axons or dendrites in hippocampal neurons.

197 down occludes OGD-induced Rac1 activation in hippocampal neurons.

198 against the deleterious impact of AbetaOs on hippocampal neurons.

199 r protein complex, in the development of rat hippocampal neurons.

200 physiological properties of mitochondria in hippocampal neurons.

201 tor (GABAAR)-mediated transmission in mature hippocampal neurons.

202 an OGD-induced increase in Rac1 activity in hippocampal neurons; however, the identity of an antagon

203 Adult hippocampal neuroprogenitors give rise to both neurons a

204 heterozygosity and found that they exhibited hippocampal NMDA receptor hyperfunction, which likely dr

205 TgF344-AD rats displayed progressive loss of hippocampal norepinephrine levels and locus coeruleus fi

206 o, few studies have tested the importance of hippocampal-OFC interactions.

207 nel activity, some of which were not seen in hippocampal or cortical neurons, and resulting in neuron

208 n addition, ICV, dorsal hippocampal, ventral hippocampal, or ACC infusions immediately 'before' testi

209 With hippocampal output suppressed, rats were slower to adapt

210 temporal, occipital, anterior, and posterior hippocampal, parahippocampal, and fusiform regions, as w

211 of two other brain diseases associated with hippocampal pathology (Alzheimer's disease and schizophr

212 In contrast, hippocampal patients were unable to grasp the higher-ord

213 ever, it is not fully understood how and why hippocampal patterns become separated.

214 affected in a PKA-dependent manner, whereas hippocampal-PFC projections involved in SWM were not aff

215 Further, we extended the scope of hippocampal phase precession to include object informati

216 Reactivation of hippocampal place cell sequences during behavioral immob

217 atial cues, consistent with prior studies of hippocampal place cells and providing a rich representat

218 the role of this neuron-specific protein in hippocampal plasticity and cognitive function.

219 l kinetics of AMPARs, necessary for synaptic hippocampal plasticity, and memory recall.

220 3CR1 is involved in synaptic development and hippocampal plasticity.

221 We also observed an expected increase in hippocampal pO2 (15 +/- 4% from baseline) in response to

222 The results suggest that hippocampal pre-activation of expected stimuli is a faci

223 complicated by multiple direct and indirect hippocampal-prefrontal connections.

224 cate that Abeta has a differential effect on hippocampal proliferative cells by inhibiting neuronal p

225 and lack of extinction-dependent changes in hippocampal PSD CaMKII expression and S831 GluA1 phospho

226 -dependent protein kinase II (CaMKII) to the hippocampal PSD.

227 Tests in CA1 hippocampal pyramidal cells reveal that a slow AHP is re

228 GIRK2a and GIRK2c) expressed individually in hippocampal pyramidal neurons lacking GIRK2.

229 Here, in hippocampal pyramidal neurons of both primary cultures a

230 moval via RNAi-mediated knockdown in the CA1 hippocampal region in immature animals causes rapid and

231 d for levels of GABA or Glx in right or left hippocampal region.

232 al prefrontal cortex (l-DLPFC) and bilateral hippocampal regions of 19 older adults (age +/- SD: 66.1

233 During a self-paced spatial task, awake hippocampal replay occurring either immediately before m

234 Hippocampal replays have been demonstrated to play a cru

235 rocessing of memory during sleep affects the hippocampal representation itself, is unclear.

236 nsolidation processes during sleep shape the hippocampal representation itself.

237 , presumably by neural memory replay, shapes hippocampal representations and enhances computations of

238 ignaled by the mPFC become incorporated into hippocampal representations and support prospective codi

239 To resolve this debate, we characterized hippocampal representations during reorientation.

240 (eliminating spatial and visual similarity), hippocampal representations paradoxically became relativ

241 They find that tau deletion impairs hippocampal response to insulin through IRS-1 and PTEN d

242 found that tau deletion leads to an impaired hippocampal response to insulin, caused by altered IRS-1

243 (NREM) sleep-the thalamo-cortical spindles, hippocampal ripples, and the cortical slow oscillations-

244 ssociative cognitive processes, suggesting a hippocampal role in making consistent decisions.

245 peer-rearing, PR) in archived rhesus macaque hippocampal samples (male, n = 13).

246 Hippocampal samples harvested from these mice following

247 vity-dependent long-term depression (LTD) at hippocampal Schaffer collateral (SC)-CA1 synapses.

248 ecreased levels of long-term potentiation at hippocampal Schaffer collateral-CA1 synapses.

249 e dentate gyrus translate to the severity of hippocampal sclerosis and seizure burden in chronic epil

250 -resistant focal epilepsy requiring surgery, hippocampal sclerosis was the most common histopathologi

251 The most common categories were hippocampal sclerosis, found in 36.4% of the patients (8

252 during consolidation.SIGNIFICANCE STATEMENT Hippocampal sharp-wave ripples (SWRs) occur both in the

253 e in mood-related brain regions, and reduced hippocampal signaling proteins, such as brain-derived ne

254 Despite extensive studies in hippocampal slices and incentive from computational theo

255 ency, measured on pyramidal neurons in acute hippocampal slices at 270 DAT, was reduced in epileptic

256 Using acute hippocampal slices from mice of either sex with genetic

257 alyzed NMDA-dependent synaptic plasticity in hippocampal slices from Tg(CJD) mice, which model a gene

258 kingly, electrophysiological recordings from hippocampal slices of mice lacking PRMT8 reveal multiple

259 Herein, we investigated whether in mice hippocampal slices these distinct forms of LTP are speci

260 reased alpha2beta1gamma1 GABAAR pentamers in hippocampal slices using cell-surface cross-linking, fol

261 ical effect of THC was evaluated using acute hippocampal slices, and hippocampal cannabinoid receptor

262 Previous work indicates that hippocampal-specific alterations in CREB signaling and s

263 attributed to obesity-induced alterations in hippocampal structure and function in some patients.

264 To identify the genetic underpinnings of hippocampal structure here we perform a genome-wide asso

265 We also demonstrated that hippocampal subfield volume reduction was associated wit

266 eval of episodic memory for which individual hippocampal subfields and entorhinal cortex layers contr

267 ed the functional contribution of individual hippocampal subfields during a perceptual discrimination

268 ty was not merely an indirect consequence of hippocampal suppression; rather, both the hippocampus an

269 either 12 or 16 months of age fully reverses hippocampal synapse loss and completely rescues preexist

270 nts reveal a novel type of plasticity at CA1 hippocampal synapses that is expressed by the activation

271 ately 27 nm localization precision at single hippocampal synapses under physiological conditions.

272 during NMDA-receptor-dependent LTP at mature hippocampal synapses.

273 ling and modulate betaAR-mediated effects on hippocampal synaptic plasticity and cognitive function.

274 ociated reductions in NMDA receptor-mediated hippocampal synaptic plasticity.

275 Hippocampal tail volume is proposed as a potentially use

276 controlling for age and total brain volume, hippocampal tail volume was larger in the MDD cohort com

277 is pivotal for long-term depression at adult hippocampal temporoammonic-CA1 synapses.

278 ACA neurons increased just after the peak of hippocampal theta wave.

279 epilepsy-eQTL analysis is superior to normal hippocampal tissue eQTL analyses for identifying the var

280 es correlated with the volume of compromised hippocampal tissue.

281 Together, our findings indicate that ventral hippocampal trkB is essential to goal-directed action se

282 In addition, ICV, dorsal hippocampal, ventral hippocampal, or ACC infusions immed

283 he authors investigated group differences in hippocampal volume (both including and excluding amyloid

284 omposite AD signature cortical thickness and hippocampal volume (HVa), and underwent (11) C-Pittsburg

285 e amyloid scans, and the association between hippocampal volume and cortical amyloid uptake.

286 stronger negative association between right hippocampal volume and HbA1c was found in patients with

287 Structural abnormalities in hippocampal volume and shape are found in several common

288 rather than mitosis, have an impact on adult hippocampal volume and that these genes represent import

289 as positively associated with left and right hippocampal volume and with delayed recall.

290 t genetic variants associated with decreased hippocampal volume are also associated with increased ri

291 ospective study was to examine whether lower hippocampal volume in late-life depression is associated

292 Hippocampal volume loss is a hallmark of clinical depres

293 genotype, with a 0.032-mL decrease in right hippocampal volume per 14% increase in HbA1c (P = 0.0007

294 Lower hippocampal volume was not related to amyloid pathology

295 s on demographics, comorbidities, cognition, hippocampal volume, and small-vessel disease but not on

296 dependent loci significantly associated with hippocampal volume, four of them novel.

297 se events was inversely associated with left hippocampal volume.

298 al, occipital and frontal cortex and reduced hippocampal volume.

299 severity were associated with smaller right hippocampal volume.

300 went magnetic resonance imaging to determine hippocampal volumes and cognitive assessment to evaluate

301 s, AQP4 knock-down in mice not only impaired hippocampal volumetric plasticity after exposure to enri