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1 f total and mushroom dendritic spines in the hippocampal CA1 region.
2 -801 on LTD and NMDAR signaling in the mouse hippocampal CA1 region.
3 naptic plasticity in slice recordings of the hippocampal CA1 region.
4 the medial entorhinal cortex (MECIII) to the hippocampal CA1 region.
5 nes) profiles in the stratum radiatum in the hippocampal CA1 region.
6 es and microglial cells were observed in the hippocampal CA1 region.
7 ces spontaneous epileptiform activity in the hippocampal CA1 region.
8 ning and long-term potentiation (LTP) in the hippocampal CA1 region.
9 ined if nicotine modulates DP and LTD in the hippocampal CA1 region.
10 ction of long-term potentiation (LTP) in the hippocampal CA1 region.
11 pulations and functional capabilities of the hippocampal CA1 region.
12 voked synaptic potential recorded in the rat hippocampal CA1 region.
13 eurite processes of remaining neurons of the hippocampal CA1 region.
14 the brain such as the pyramidal cells of the hippocampal CA1 region.
15 nd Archaerhodopsin in pyramidal cells in the hippocampal CA1 region, achieving high quality recording
16 a feedforward inhibitory microcircuit in the hippocampal CA1 region, an increased excitability of pyr
17 l impairments and increased apoptosis in the hippocampal CA1 region and cortex.
18 e most concentrated in stratum oriens of the hippocampal CA1 region and dentate inner molecular layer
19 own histone acetyltransferases (HATs) in the hippocampal CA1 region and find that K-acetyltransferase
20 th altered in vivo network activities in the hippocampal CA1 region and impaired synaptic function.
21 nd high-frequency ripple oscillations in the hippocampal CA1 region and is linked to experience, but
22 ng RTN3 will develop RIDNs, initially in the hippocampal CA1 region, and later in other hippocampal a
23                               Neurons in the hippocampal CA1 region are particularly sensitive to oxi
24 mation and impaired synaptic function in the hippocampal CA1 region as the result of epigenetic-depen
25 fect on GluR1 phosphorylation at T840 in the hippocampal CA1 region, bath application of NMDA induced
26 ts of the study show that ERalpha in the rat hippocampal CA1 region but not the uterus undergoes enha
27 xpressing cells in the stratum oriens of the hippocampal CA1 region confirmed that these cells were i
28 n analysis, was significantly reduced in the hippocampal CA1 region, cortex, striatum, and thalamus i
29 he anterior cingulate, basolateral amygdala, hippocampal CA1 region, dentate gyrus, nucleus accumbens
30 theless, knockdown of synaptotagmin-1 in the hippocampal CA1 region did not impede acquisition of rec
31 sed with lentiviral shRNA-HCN1 in the dorsal hippocampal CA1 region displayed antidepressant- and anx
32 tions of glutamate were not increased in the hippocampal CA1 region during a 2-h postmortem interval,
33 stigation of transcriptome plasticity in the hippocampal CA1 region in aging and AD models and sugges
34 e show that, in the pyramidal neurons of the hippocampal CA1 region in mice, blocking postsynaptic ex
35 s associated with learning and memory in the hippocampal CA1 region in rats.
36 nd the density of axospinous synapses in the hippocampal CA1 region in young rats, yet this is attenu
37                                          The hippocampal CA1 region is crucial for converting new mem
38 ts indicate that the induction of LTD in the hippocampal CA1 region is dependent on ionotropic, rathe
39                                          The hippocampal CA1 region is highly vulnerable to ischaemic
40 ther knockdown of HCN1 protein in the dorsal hippocampal CA1 region is sufficient to produce antidepr
41                                       In the hippocampal CA1 region, low-frequency stimulation (LFS;
42 elicited long-term potentiation (LTP) in the hippocampal CA1 region of 28-day-old control animals.
43               Inhibitory synapses in the rat hippocampal CA1 region of acute slices were studied usin
44 ls were trained on two memory tasks, and the hippocampal CA1 region of each was analyzed on an indivi
45  pyramidal neurons freshly isolated from the hippocampal CA1 region of immature (2- to 10-day-old) an
46  characterized events accompanying SD in the hippocampal CA1 region of murine brain slices, using who
47 not theta burst stimulation, is perturbed in hippocampal CA1 region of old but not young htau mice.
48 erties of basal synaptic transmission in the hippocampal CA1 region of PSD-93 and PSD-95 mutant mice
49 rays of electrodes in the dorsal and ventral hippocampal CA1 region of rats and recorded the neuronal
50 -phase long-term potentiation (L-LTP) in the hippocampal CA1 region of rodents.
51 of interneurons-theta-driving neurons in the hippocampal CA1 region of the mouse-to characterize the
52 inous synaptic density and plasticity in the hippocampal CA1 region of young female rats but fails to
53 reases the axospinous synapse density in the hippocampal CA1 region of young female rats but fails to
54 1 mice, a subpopulation of astrocytes in the hippocampal CA1 region prominently expressed nAChRbeta4
55 that nicotine reverses stabilized LTP in the hippocampal CA1 region, providing the first evidence tha
56 actic viral expression of Cre-recombinase in hippocampal CA1 region pyramidal neurons at postnatal da
57       Electrophysiological recordings in the hippocampal CA1 region revealed that short-term and long
58 taGABA(A)R) expression in the dentate gyrus, hippocampal CA1 region, thalamus, and striatum but not i
59                         Specifically, in the hippocampal CA1 region, the NL3(R451C) mutation caused a
60 aptic plasticity of pyramidal neurons in the hippocampal CA1 region to maintain an appropriate homeos
61  showed abnormal long-term plasticity in the hippocampal CA1 region together with deficits in learnin
62 lation-mediated synaptic potentiation in the hippocampal CA1 region was significantly reduced compare
63  pyramidal cells and field recordings in the hippocampal CA1 region, we investigated the specific con
64 ever, reduction of amyloid deposition in the hippocampal CA1 region, where RIDNs predominantly formed
65 d induction of long-term potentiation in the hippocampal CA1 region without affecting basal synaptic

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