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1           Furthermore, CCNY abolishes LTP in hippocampal slices.
2 ged presynaptic [Ca(2+)] transients in mouse hippocampal slices.
3 Cajal-Retzius cells optogenetically in mouse hippocampal slices.
4 erent components of the CA3 network in mouse hippocampal slices.
5 ongoing epileptiform activity in mouse acute hippocampal slices.
6 d post-tetanic potentiation and LTP in mouse hippocampal slices.
7 ocampal neurons and CA1 pyramidal neurons in hippocampal slices.
8 mini of dentate gyrus neurons in adult mouse hippocampal slices.
9 on, but not long-term potentiation, in acute hippocampal slices.
10  dendrites of CA1 pyramidal neurons in mouse hippocampal slices.
11 -access two-photon microscopy in acute mouse hippocampal slices.
12 tional hemichannels in astrocytes from mouse hippocampal slices.
13 ed oligomer-mediated LTP impairment in mouse hippocampal slices.
14 ivation inhibited Thr-840 phosphorylation in hippocampal slices.
15 ed reduction of the sAHP observed ex vivo in hippocampal slices.
16 d long-term potentiation inhibition in mouse hippocampal slices.
17 ative electrophysiological systems using rat hippocampal slices.
18 pocampal networks in dissociated culture and hippocampal slices.
19 ate gyrus granule cells (DGGCs) in adult rat hippocampal slices.
20 lasticity at mossy fiber-CA3 synapses in rat hippocampal slices.
21 in and impaired synaptic plasticity in adult hippocampal slices.
22 ng SWRs that occurred spontaneously in mouse hippocampal slices.
23 locked long-term potentiation (LTP) in acute hippocampal slices.
24 artments in early and later-stage AD mice in hippocampal slices.
25 binant noradrenaline transporters and in rat hippocampal slices.
26 arbachol-induced gamma oscillations in mouse hippocampal slices.
27 y reduces epileptiform bursting in TSC2(+/-) hippocampal slices.
28  plasticity and synaptic transmission in the hippocampal slices.
29 fected cells, primary neuronal cultures, and hippocampal slices.
30 k activity or acute effects on plasticity in hippocampal slices.
31 ion in stratum radiatum of area CA1 in mouse hippocampal slices.
32 zed in cultured cortical neurons or in acute hippocampal slices.
33 ission in the CA1 area compared to wild-type hippocampal slices.
34 ion of inhibitory transmission (iLTD) in rat hippocampal slices.
35 ed NMDA-induced neurotoxicity in acute mouse hippocampal slices.
36  5-HT1A/5-HT7 agonist) reversed mGluR-LTD in hippocampal slices.
37 ein regulation were observed in cultured rat hippocampal slices.
38 ignaling kinases, at spine synapses in adult hippocampal slices.
39 he induction, of BDNF-dependent LTP in acute hippocampal slices.
40 n of gamma oscillations in kainate-activated hippocampal slices.
41 ently enhances cAMP in neuronal cultures and hippocampal slices.
42     We addressed these issues by using mouse hippocampal slices.
43 defective LTP in primary neuron cultures and hippocampal slices.
44 using electrophysiological recordings in rat hippocampal slices.
45 engthening lasting tens of minutes in rodent hippocampal slices.
46 ignalling in CA1 pyramidal cell dendrites in hippocampal slices.
47 and whole-cell recordings in wild-type mouse hippocampal slices.
48 ong-lasting synaptic potentiation (L-LTP) in hippocampal slices.
49 lls in the more physiological environment of hippocampal slices.
50 ry, but not inhibitory, neurotransmission in hippocampal slices.
51 of long-term depression, measured in ex vivo hippocampal slices.
52 cues presynaptic responses in culture and in hippocampal slices.
53  of CA1 pyramidal neurons in cultured murine hippocampal slices.
54  in an in vitro model of seizure activity in hippocampal slices.
55 ost-synaptic potentials (EPSPs) in adult rat hippocampal slices.
56 ing electrophysiological field recordings in hippocampal slices.
57 d pulse facilitation (PPF) in KO and control hippocampal slices.
58 cies, consistently with previous findings in hippocampal slices.
59  min) reduced FMRP levels in wild-type mouse hippocampal slices.
60 ated ERK1/2 are also elevated in Syngap(+/-) hippocampal slices.
61 onists on pyramidal neurons in mouse and rat hippocampal slices.
62 n patch clamp in whole cell configuration in hippocampal slices.
63 ogy in dorsal root ganglia (DRG) neurons and hippocampal slices.
64 naptic plasticity and spine numbers in acute hippocampal slices 2-3 weeks later.
65                                           In hippocampal slices, 24(S)-HC enhances the ability of sub
66 napses is greatly enhanced in cultured mouse hippocampal slices after chronic (60 h) network-activity
67 sicle pool in CA3-CA1 synapses from an acute hippocampal slice and for the characterization of its an
68    Using direct measurements of LTP in acute hippocampal slices and an in vitro LTP model of stimulat
69  with electrophysiological analysis on acute hippocampal slices and ATP assays in purified cell cultu
70                           Here, we use acute hippocampal slices and combine two-photon excitation Ca(
71 ptor 2 (GluR2) subunit surface expression in hippocampal slices and cultured hippocampal neurons, an
72                                           In hippocampal slices and cultured neurons we also observed
73 ase probability inhibitory synapses in mouse hippocampal slices and fits well with the observation th
74 -clamp and current-clamp recordings in acute hippocampal slices and focal applications of irreversibl
75  from post-hoc-identified cells in acute rat hippocampal slices and identified a subpopulation of mol
76                 Despite extensive studies in hippocampal slices and incentive from computational theo
77                                           In hippocampal slices and intact hippocampal preparations f
78 specifically induced during LTP induction in hippocampal slices and its knockdown in the hippocampus
79     Electrophysiological recordings in acute hippocampal slices and primary hippocampal neuronal cult
80 l, a small-molecule Eg5 inhibitor, on LTP in hippocampal slices and synapse loss in neuronal cultures
81           NRG-1 acutely depotentiates LTP in hippocampal slices, and blocking ErbB kinase activity in
82 ical effect of THC was evaluated using acute hippocampal slices, and hippocampal cannabinoid receptor
83  recycling versus degradation for LTD in rat hippocampal slices, and their correlation with receptor
84 n imaging of FM1-43 vesicular release in rat hippocampal slices; and (ii) transgenic mice expressing
85  expression in glutamatergic terminals, when hippocampal slices are incubated with low concentration
86 ency, measured on pyramidal neurons in acute hippocampal slices at 270 DAT, was reduced in epileptic
87 re made with control stimulation in the same hippocampal slices at 5 minutes, 30 minutes, and 2 hours
88                                           In hippocampal slices, beta(2) signaling decreases cAMP lev
89 term potentiation (LTP) in the CA1 region of hippocampal slices but had no significant effect on LTP
90 d robust electrophysiological effects in rat hippocampal slices, but showed lower efficacy in striatu
91 ylation had no effect on glutamate uptake in hippocampal slices, but simultaneous inhibition of both
92 ileptiform activity has been demonstrated in hippocampal slices, but use in humans will require more
93 l activity, and synaptic plasticity in acute hippocampal slices by combining electrophysiological ext
94 cetylcholine was optogenetically released in hippocampal slices by expressing the excitatory optogene
95  address this issue, we induced SD in murine hippocampal slices by focal KCl microinjection and visua
96 d that facilitation of spontaneous SPW-Rs in hippocampal slices by increasing gap-junction coupling o
97 ane excitability of CA1 pyramidal neurons in hippocampal slices by lowering the spike threshold possi
98          Importantly, in primary neurons and hippocampal slices, CALHM1 activation facilitated the ph
99                          We demonstrate that hippocampal slices can be imaged through transparent gra
100                                Astrocytes in hippocampal slices can dynamically regulate synaptic tra
101 whole-cell patch-clamp recordings from acute hippocampal slices, (+)-CIQ, the active enantiomer of th
102 t in decreasing water content in OGD-exposed hippocampal slices, compared with mu, delta, and kappa s
103 any as approximately 50% of synapses in some hippocampal slice conditions.
104 n of CA3-CA1 synaptic input-output curves in hippocampal slices confirmed an age-related decrease in
105                      As such, experiments in hippocampal slices continue to progress our understandin
106 ockout mice express EtOH-sensitive mIPSCs in hippocampal slices, correlating with upregulated GABAAR
107                                       In rat hippocampal slices, cortical synaptoneurosomes, and cult
108 beta-amyloidosis by establishing a long-term hippocampal slice culture (HSC) model.
109      Like PSD-95, activity blockade in a rat hippocampal slice culture increases SynDIG1 palmitoylati
110 uced tau hyperphosphorylation in organotypic hippocampal slice cultures (OHSC) and applied marker-ind
111 g from ROS and RNS production in organotypic hippocampal slice cultures (OHSC), as well as its potent
112 ellular fluid collected from rat organotypic hippocampal slice cultures (OHSCs) by electroosmotic flo
113 We have tested this by examining organotypic hippocampal slice cultures (OHSCs) exposed to oxygen glu
114 oA in the extracellular space of organotypic hippocampal slice cultures (OHSCs).
115 tensiometric Ca(2+) indicator in organotypic hippocampal slice cultures (one- and two-photon) and the
116 (i.e. post-treatment protocol in organotypic hippocampal slice cultures and cortical neurons) can be
117                            Using organotypic hippocampal slice cultures and primary neuron hippocampa
118 function, we used sparse transfection of rat hippocampal slice cultures and whole-cell recordings in
119                 Expression of CRISPR/Cas9 in hippocampal slice cultures completely eliminated NMDA re
120 se deprivation-induced injury in organotypic hippocampal slice cultures confirmed that calpains were
121 in AMPAR ischaemic plasticity in organotypic hippocampal slice cultures exposed to oxygen glucose dep
122 egulated, in cortical neuron and organotypic hippocampal slice cultures from rat, either by the previ
123  PAK3, or inhibition of PAK3 function in rat hippocampal slice cultures interfere with activity-media
124 ltures of primary hippocampal neurons and in hippocampal slice cultures is similarly enhanced by PCB-
125                     To this end, we infected hippocampal slice cultures of different rat strains with
126 The effects were confirmed in vitro by using hippocampal slice cultures of female mice.
127                        We imaged organotypic hippocampal slice cultures of rat, in which astrocytes m
128 ntate granule cells in organotypic entorhino-hippocampal slice cultures of Thy1-GFP mice.
129                                 In entorhino-hippocampal slice cultures prepared from SP-deficient mi
130 By analyzing CA1 pyramidal neurons in mutant hippocampal slice cultures that are essentially devoid o
131 orded from CA3 pyramidal neuron pairs in rat hippocampal slice cultures to characterize synaptic and
132  biolistically transfected astrocytes in rat hippocampal slice cultures to facilitate fluorescent con
133 g activity of hundreds of neurons in cortico-hippocampal slice cultures using a high-density 512-elec
134 bolic responses to traumatic brain injury in hippocampal slice cultures, and observed marked upregula
135 microglia-neuron interactions in organotypic hippocampal slice cultures, i.e., postnatal cortical tis
136  via RyR- and miR132-dependent mechanisms in hippocampal slice cultures.
137 del of rMS using mouse organotypic entorhino-hippocampal slice cultures.
138  using rapid high-pressure freezing (HPF) of hippocampal slice cultures.
139 tive NMDAR antagonist, D-APV, in organotypic hippocampal slice cultures.
140 ere also produced in ex vivo optic nerve and hippocampal slice cultures.
141 on compared to single agents in immature rat hippocampal slice cultures.
142 nockdown of PYK2 blocks LTD, but not LTP, in hippocampal slice cultures.
143 beta aggregation and deposition in long-term hippocampal slice cultures.
144 y synapses over prolonged periods of time in hippocampal slice cultures.
145                                  In isolated hippocampal slices, decaying long-term potentiation can
146                                 We find that hippocampal slices, either prepared from rats following
147                                   Whole-cell hippocampal slice electrophysiological recordings and mo
148 ional assay (EC50, 36 nM) and carbachol in a hippocampal slice electrophysiology assay (EC50, 165 nM)
149 n of MS-DBB glutamatergic fiber terminals in hippocampal slices elicited weak postsynaptic responses
150 voflurane-induced preconditioning in the rat hippocampal slice enhances the hypoxic hyperpolarization
151  addition, acute treatment of an organotypic hippocampal slice epilepsy model with Sema4D reveals tha
152 inant or synthetic Aeta-alpha was applied on hippocampal slices ex vivo, long-term potentiation was l
153 naptic currents in both cultured neurons and hippocampal slices exposed to E2, while their frequency
154 strocytic lactate was also observed in acute hippocampal slices exposed to NH4(+) and in the somatose
155 y upregulates synaptic NMDAR currents in rat hippocampal slices, facilitating induction of long-term
156                  In biolistically transduced hippocampal slices, fluorescently tagged GLT-1 puncta ov
157                          In acutely prepared hippocampal slices, frequency and amplitude of mEPSCs an
158 ced dendritic spine expansion is impaired in hippocampal slices from 15- and 21-d-old synaptopodin-de
159 ayed for restoration of synaptic function in hippocampal slices from AD-transgenic mice, reversal of
160 U0409551 directly enhances NMDAR function in hippocampal slices from adult male SR-/- mice.
161 affer collateral/commissural fibers in acute hippocampal slices from adult mice transduced with the g
162 ptic responses of mature and immature GCs in hippocampal slices from adult mice.
163 o mGlu5 receptor activation were abnormal in hippocampal slices from AS mice compared with wild-type
164 etanic stimulation (TS) in the CA1 region of hippocampal slices from both nulliparous female and male
165                              In contrast, in hippocampal slices from calpain-1 knock-out (KO) mice, a
166 spine resolution in pyramidal neurons in rat hippocampal slices from either sex.
167                  Cells transcribing P2rx7 in hippocampal slices from epileptic mice displayed enhance
168               To test those possibilities in hippocampal slices from epileptic pilocarpine-treated ra
169                           Moreover, treating hippocampal slices from fmr1 KO mice with Kv4 channel bl
170                 We examined this issue using hippocampal slices from guinea pigs and mice.
171 ts, we recorded whole-cell currents in acute hippocampal slices from hAPP mice with and without tau.
172 ced oscillatory activity generated in CA3 of hippocampal slices from heterozygous GAD67-GFP (Deltaneo
173                                              Hippocampal slices from juvenile and adult GluN3A KO mic
174                                  Using acute hippocampal slices from mice of either sex with genetic
175                        Here we show in acute hippocampal slices from mice that endogenous NPY, releas
176  on glutamatergic neurons in neocortical and hippocampal slices from neonatal mouse pups in vitro, bu
177 d local field potentials and single cells in hippocampal slices from normal rats.
178 n during whole-cell patch clamp recording in hippocampal slices from pilocarpine-treated rats.
179 function and calcium homeostasis using acute hippocampal slices from PS conditional knockout mice and
180  absence of NMDAR activation, we examined in hippocampal slices from rats and mice, an NMDAR-independ
181                      Ex vivo preparations of hippocampal slices from rats that have been subjected to
182                                           In hippocampal slices from rats treated with nicotine for 1
183 alyzed NMDA-dependent synaptic plasticity in hippocampal slices from Tg(CJD) mice, which model a gene
184 erm potentiation (LTP), in the CA1 region of hippocampal slices from this mouse, is impared at Tg2576
185 ostsynaptic potentials (IPSPs) between acute hippocampal slices from Ts65Dn mice and diploid (2N) wil
186  peptide, reduces glutamate release in acute hippocampal slices from wild-type but not APP deficient
187  induced by theta burst stimulation (TBS) in hippocampal slices from wild-type mice was associated wi
188                      In cornu ammonis area 1 hippocampal slices from wild-type mice, TBB impairs neur
189  associated with phosphorylated PAK in adult hippocampal slices from wild-type, but not Fmr1-KO, mice
190  minority of O/A interneurons in MLA-treated hippocampal slices from WT animals and alpha7(-/-) mice,
191                                  Here, using hippocampal slices from young adult male rats and mice,
192 -clamp recording and biochemical analyses in hippocampal slices from young adult rats, we show that E
193 itors cycloheximide and emetine to acute rat hippocampal slices have no effect on total KCC2 protein
194                                           In hippocampal slices, IGF-II promotes IGF-II receptor-depe
195 ictal-like activity induced by incubation of hippocampal slices in an Mg(2+)-free solution led to a f
196 that GluN2C expression is increased in acute hippocampal slices in response to ischemia.
197 l and anatomical techniques applied to mouse hippocampal slices in vitro to directly address this que
198 he dentate gyrus (DG) or CA1 region of mouse hippocampal slices in vitro.
199 pplied these neuropeptides directly to human hippocampal slices in vitro.
200 d paired patch-clamp recordings in acute rat hippocampal slices, in combination with post hoc identif
201 hanol locally increases 3alpha,5alpha-THP in hippocampal slices, in the absence of adrenal influence.
202 ry/inhibitory imbalance observed in SynII(-) hippocampal slices indirectly, not by correcting the def
203 e, we report that neuronal depolarization in hippocampal slices induces a calcium and protein phospha
204 easing O-GlcNAcylation in Sprague Dawley rat hippocampal slices induces an NMDA receptor and protein
205  potentiation (LTP) in the CA1 region of the hippocampal slice is not altered in Dyt1 DeltaGAG hetero
206        Electrophysiology studies showed that hippocampal slices isolated from these mice are more sus
207 ed short-term synaptic plasticity in a mouse hippocampal slice model can be reduced by LEI105.
208                                 Although the hippocampal slice model may not represent the neurons di
209                               Using in vitro hippocampal slice models from rats and mice, we performe
210 ntiseizure activity in two acute ex vivo rat hippocampal slice models of epileptiform activity.
211 deleting Pten from neurons in an organotypic hippocampal slice network.
212                                       In rat hippocampal slices, neuronal activity rapidly decreased
213 -4 weeks) dramatically increased the sAHP in hippocampal slice neurons from young-adult rats.
214                                     In mouse hippocampal slices, NMDAR EPSCs in a singly activated CA
215                                              Hippocampal slices obtained 4 d after the induction of i
216 s were absent on GFAP-positive astrocytes in hippocampal slices obtained from GFAP-A7KO offspring fro
217 fer collateral stimulation, were detected in hippocampal slices obtained from Pin1(-/-) mice compared
218 nction and rescues long-term potentiation in hippocampal slices obtained from SR-/- mice.
219 eceptor antagonist (SR144528) or absent from hippocampal slices of CB2 receptor knock-out mice.
220 nd inhibitory synaptic transmission in acute hippocampal slices of Cntnap2(-/-) mice.
221 uced inhibition of long-term potentiation in hippocampal slices of cognitively normal mice.
222  function and recovery upon reoxygenation in hippocampal slices of mice lacking APP (APP(-/-)) or sel
223 kingly, electrophysiological recordings from hippocampal slices of mice lacking PRMT8 reveal multiple
224 rial and synaptic dysfunction in organotypic hippocampal slices of rats.
225 pines inhibited these forms of plasticity in hippocampal slices of rodents.
226  glutamate (mGlu5) receptors was enhanced in hippocampal slices of Ube3A(m-/p+) mice, which model AS.
227                                           In hippocampal slices, oligomeric Abeta induces eNMDAR-medi
228 or oxygen-glucose deprivation in organotypic hippocampal slices or neurons, p75(NTR) is rapidly induc
229                          We demonstrate that hippocampal slices overexpressing PKMzeta show enhanced
230 tual fear memory and to show enhanced LTP in hippocampal slices overexpressing PKMzeta.
231                                           In hippocampal slices, photolysis of MNI-D-aspartate (4-met
232 nd intracellular recording techniques in the hippocampal slice preparation of immature rats.
233 eptor and subsequent CREB phosphorylation in hippocampal slice preparations and its administration im
234                          We used organotypic hippocampal slices prepared from 6-d-old Thy1-YFP mice a
235                                  Using acute hippocampal slices prepared from adult mice, we report t
236 ion (mGluR-LTD) at CA3-CA1 synapses in acute hippocampal slices prepared from CGG KI mice relative to
237 ntiation (LTP) in experiments using standard hippocampal slice protocols.
238 that early expression of NR2A in organotypic hippocampal slices reduces the number of synapses and th
239 ition of PDE-4 by pharmacologic treatment in hippocampal slices rescues the enhanced mGluR-dependent
240 elective OXTR agonist [Thr(4),Gly(7)]-OXT to hippocampal slices resulted in an acute and lasting pote
241     Electrophysiological recordings of acute hippocampal slices revealed that genetic inactivation of
242 Specifically, our experimental data in mouse hippocampal slices show that acetylcholine biases STDP t
243 tions; and (3) multineuron Ca(2+) imaging in hippocampal slices showed increased spontaneous neuronal
244 AO) in rats and hMCT2 transgenic mice and of hippocampal slices subjected to ASH was assessed, as wel
245 ein levels and in the functional recovery of hippocampal slices subjected to ASH.
246 cation to the postsynaptic fraction in acute hippocampal slices subjected to long-term potentiation.
247 ong-term potentiation in Vav-deficient mouse hippocampal slices, suggesting that Vav-dependent regula
248                                              Hippocampal slices taken from AT mice exhibited decrease
249 ophysiological study of synaptic function in hippocampal slices taken from Clcn3(-/-) mice before the
250 air recordings were performed in organotypic hippocampal slices taken from genetically modified mice
251 pression (LTD) were significantly reduced in hippocampal slices taken from inflamed animals.
252 of long-term potentiation (LTP) in adult rat hippocampal slices that can account for one temporal seg
253                   We show in rat organotypic hippocampal slices that prolonged network silencing indu
254 ssion and also caused synaptic depression in hippocampal slices that was consistent with AMPAR downre
255  we show, using whole-cell recordings in rat hippocampal slices, that group I metabotropic glutamate
256      Herein, we investigated whether in mice hippocampal slices these distinct forms of LTP are speci
257                                           In hippocampal slices, this increase in cleavage products w
258  we report that brief exposure of adult male hippocampal slices to 1 nM E2 increases the percentage o
259 tage-sensitive dye imaging (VSDi) in ventral hippocampal slices to determine the effects of sazetidin
260 gether with electrophysiology in acute mouse hippocampal slices to dissect the roles of P/Q- and N-ty
261  of tonic GABAA receptor currents in ex vivo hippocampal slices to examine GAT-1 operation under vary
262 geted optogenetic stimulation in acute mouse hippocampal slices to examine how one class of interneur
263 re used chained microRNAs in rat organotypic hippocampal slices to generate a reduced background of e
264  electrophysiologically tagged NMDARs in rat hippocampal slices to identify the molecular determinant
265 e, we used extracellular field recordings in hippocampal slices to investigate adaptations in synapti
266 he CaMKIIalpha promoter in mice and prepared hippocampal slices to produce a model of intrinsic CA1 g
267  oxygenase 1, and 4) immunohistochemistry of hippocampal slices to quantify vital neurons.
268                                  Using mouse hippocampal slices to study acute oxygen glucose depriva
269 that in rat dentate granule cells in ex vivo hippocampal slices, tonic currents are predominantly gen
270                                 In wild-type hippocampal slices treated with exogenous amyloid beta p
271 fore, a peptidomimetic furoxan was tested in hippocampal slices treated with oligomeric amyloid-beta
272                                           In hippocampal slices, treatment with the GABA(B) receptor
273            Application of VU0463271 to mouse hippocampal slices under low-Mg(2+) conditions induced u
274 reased alpha2beta1gamma1 GABAAR pentamers in hippocampal slices using cell-surface cross-linking, fol
275  quantified its buffering capacity in murine hippocampal slices using confocal calcium imaging and th
276 that immunostaining in the CA1 region of rat hippocampal slices was confined to pyramidal neurons.
277 lation of prohibitin in neuronal cultures or hippocampal slices was markedly neuroprotective, whereas
278 3 pyramidal neurons on postnatal day 0-6 rat hippocampal slices, we detected robust GlyR activity as
279 tive dye to image electrical activity in rat hippocampal slices, we explored how long-term potentiati
280                           In organotypic rat hippocampal slices, we find that a nonphosphorylatable P
281 -cell confocal microscopy of rat organotypic hippocampal slices, we find that enhancing neuronal acti
282                                           In hippocampal slices, we found that the GluN2B-selective a
283 using paired whole-cell recordings in rodent hippocampal slices, we report that presynaptic protein s
284 s in young 3xTg-AD and nontransgenic (NonTg) hippocampal slices, we show that increased RyR-evoked ca
285               Using paired recordings in rat hippocampal slices, we show that inhibition in the DG is
286                Here, using mouse organotypic hippocampal slices, we show that the extracellular AMPAR
287 ectrophysiological recordings from acute rat hippocampal slices, we tested the critical BCM predictio
288 ngth of excitatory inputs on PV-INs in acute hippocampal slices were also dependent on Narp and Narp-
289                                     When rat hippocampal slices were chronically treated with neuregu
290                                              Hippocampal slices were cultured and treated with microm
291           For the in situ model of ischemia, hippocampal slices were exposed to oxygen glucose depriv
292 atal day 1 to 7 or milk only (Controls), and hippocampal slices were prepared from Control- and CNN-t
293                                In adulthood, hippocampal slices were prepared.
294 how that when organotypic cultures of rodent hippocampal slices were treated with a CB2 receptor agon
295 ular field recordings from the CA1 region of hippocampal slices, whereas behavioral techniques were u
296 on and LTP was increased in CaMKIIalpha-HM3D hippocampal slices, whereas slices from CaMKIIalpha-HM4D
297 ork activation (GNA) in the developing mouse hippocampal slices, which is measured macroscopically by
298  Here, we report that treatment of adult rat hippocampal slices with BDNF or with tetraethylammonium
299          Here we report that pretreatment of hippocampal slices with GLP-1(9-36)(amide) prevented imp
300 dress this issue, we probed NA-treated mouse hippocampal slices with pharmacological inhibitors targe

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