ライフサイエンスコーパス: histochemistry

1 PVH was confirmed with in situ hybridization histochemistry.

2 haracteristic of IBM, including abnormal tau histochemistry.

3 brain using RT-PCR and in situ hybridisation histochemistry.

4 points, and processed for nucleic acids and histochemistry.

5 s using nonradioactive in situ hybridization histochemistry.

6 de adenine dinucleotide phosphate-diaphorase histochemistry.

7 nd NTrs was determined by immunofluorescence histochemistry.

8 NCs using quantitative in situ hybridization histochemistry.

9 igated using quantitative cytochrome oxidase histochemistry.

10 Transplanted cells were localized by histochemistry.

11 ng confocal microscopy and immunofluorescent histochemistry.

12 on cells, as indicated by cytochrome oxidase histochemistry.

13 on biochemical assays, immunoreactivity, and histochemistry.

14 ions processed by using tetramethylbenzidine histochemistry.

15 sittacus undulatus), was examined using iron histochemistry.

16 , they are very useful tools in quantitative histochemistry.

17 ne dinucleotide phosphate (NADPH)-diaphorase histochemistry.

18 cells, indicated by cytochrome oxidase (CO) histochemistry.

19 dinucleotide phosphate-diaphorase (NADPH-d) histochemistry.

20 upported by enzyme assays, Western blot, and histochemistry.

21 t turtle retinas by using immunofluorescence histochemistry.

22 ta in the rat brain by in situ hybridization histochemistry.

23 ts was determined with in situ hybridization histochemistry.

24 ma model using both a quantitative ELISA and histochemistry.

25 adult murine brain by in situ hybridization histochemistry.

26 ype and RET-deficient mouse embryos by TUNEL histochemistry.

27 ght for Wisteria floribunda agglutinin (WFA) histochemistry.

28 n the same sections by in situ hybridization histochemistry.

29 fragmentation using in situ nick translation histochemistry.

30 nous gene, as shown by in situ hybridization histochemistry.

31 nfirmed by in situ reverse transcriptase PCR histochemistry.

32 onse to KGF as assessed by bromodeoxyuridine histochemistry.

33 A expression, dendritic structure and immuno-histochemistry.

34 itative analysis of conventional chromogenic histochemistry.

35 us (SCN) were assessed by cytochrome oxidase histochemistry.

36 land gorillas using immunohistochemistry and histochemistry.

37 identified based on cytochrome oxidase (CO) histochemistry.

38 ion (PCR) analysis and in situ hybridization histochemistry.

39 e tissue processed for in situ hybridization histochemistry.

40 es as evidenced by improved ERGs and retinal histochemistry.

41 cro-positron emission tomography imaging and histochemistry.

42 n situ hybridization, and immunofluorescence histochemistry.

43 nzymatic activity was demonstrated by enzyme histochemistry.

44 wisteria floribunda agglutinin (WFA) lectin histochemistry.

45 ochondrial marker Cytochrome C Oxidase (COX) histochemistry.

46 outing in the rat dentate gyrus using Timm's histochemistry: (1) repeated spaced ECS; (2) daily admin

47 sion of iron-related genes, (2) nonheme iron histochemistry, (3) immunohistochemistry for proteins of

48 lei were also visualized by NADPH-diaphorase histochemistry, a marker of nNOS activity.

49 e dinucleotide phosphate diaphorase (NADPHd) histochemistry, a marker of nNOS activity.

50 T) immunoreactivity and acetylcholinesterase histochemistry (AChE).

51 rains were compared using cytochrome oxidase histochemistry, an endogenous marker of regional metabol

52 e issues, we performed in situ hybridization histochemistry analyses and found that Sirt1 mRNA is hig

53 ortex was labeled by cytochrome oxidase (CO) histochemistry analysis or [(3)H]proline autoradiography

54 t2 mRNA were measured by using hybridization histochemistry and a semiquantitative reverse transcript

55 gree of demyelination assessed by Black-Gold histochemistry and activation of glial cells assessed by

56 diated biotin-dUTP nick end labeling (TUNEL) histochemistry and agarose gel electrophoresis.

57 ith classical and contemporary techniques in histochemistry and allows unambiguous in vivo detection

58 lized to Muller glial cells by hybridization histochemistry and by immunohistochemistry.

59 ressing cell bodies by in situ hybridization histochemistry and by labeling beta-galactosidase driven

60 dinucleotide phosphate diaphorase (NADPH-d) histochemistry and conventional microelectrode technique

61 eral adult trigeminal relay nuclei by NADPHd histochemistry and demonstrate that fibers from DR conta

62 ls were investigated by acetylcholinesterase histochemistry and dual immunolocalization.

63 15- to 18-month-old Gsalpha transgenic mice, histochemistry and electron microscopy illustrated the e

64 The evolution of research utilizing histochemistry and electron microscopy, along with disci

65 We employed histochemistry and fluorophore-assisted carbohydrate ele

66 en from the double null mice, as assessed by histochemistry and gas chromatography-mass spectrometry.

67 rcs) is demonstrated by acetylcholinesterase histochemistry and gene expression for class III beta-tu

68 Using in situ hybridization (ISH) histochemistry and gene-specific riboprobes, the current

69 Using in situ hybridization histochemistry and immunoblot analyses, we further exami

70 In situ hybridization histochemistry and immunocytochemistry revealed TIP39-co

71 lacZ, in combination with fluorescent X-gal histochemistry and immunocytochemistry to assess levels

72 In situ hybridization histochemistry and immunocytochemistry were used to exam

73 In situ hybridization histochemistry and immunocytochemistry were used to map

74 e prominent secretory granules identified by histochemistry and immunodetection for the MC-specific g

75 le from affected patients was evaluated with histochemistry and immunohistochemical stains for dystro

76 Cell phenotypes were analyzed by histochemistry and immunohistochemistry and by reverse t

77 In this study, we used in situ hybridization histochemistry and immunohistochemistry to map the distr

78 Combined in situ hybridization histochemistry and immunohistochemistry with the neurona

79 Using in situ hybridization histochemistry and immunohistochemistry, we confirmed th

80 media to neointima by in situ hybridization histochemistry and immunohistochemistry.

81 the rat neostriatum by in situ hybridization histochemistry and immunohistochemistry.

82 ydrolysis deficiency, as observed in situ by histochemistry and in primary smooth muscle cell culture

83 ne dinucleotide phosphate (NADPH)-diaphorase histochemistry and in situ hybridization.

84 trapezoid body (MNTB) with NADPH-diaphorase histochemistry and in situ hybridization.

85 , light and electron microscopy, specialized histochemistry and indirect immunofluorescence microscop

86 ) were examined with cytochrome oxidase (CO) histochemistry and neurofilament protein (NF) immunoreac

87 dinucleotide phosphate-diaphorase (NADPH-d) histochemistry and NOS immunocytochemistry-to demonstrat

88 n's disease (PD) using in situ hybridization histochemistry and oligodeoxynucleotide (single-stranded

89 ere investigated using in situ hybridization histochemistry and oligodeoxynucleotide (single-stranded

90 oridia were identified in stool specimens by histochemistry and PCR of 30 (18.9%) of 159 HIV-infected

91 Histochemistry and polymerase chain reaction demonstrate

92 oscopy (SEM), immunofluorescence microscopy, histochemistry and quantitative RT-PCR methods.

93 We used in situ hybridization histochemistry and reverse transcriptase-PCR amplificati

94 ections were prepared for beta-galactosidase histochemistry and rhodopsin, TfR, or GFAP immunocytoche

95 ne dinucleotide phosphate-diaphorase (NADPH) histochemistry and SMI-32 immunocytochemistry to label p

96 assessments, spinal cords were collected and histochemistry and stereology were conducted to evaluate

97 Sections underwent immunocytochemistry or histochemistry and the overlap of microvascular and func

98 ter than adjacent divisions with CO and AChE histochemistry and was moderately stained with calbindin

99 Myocardial histochemistry and Western blot analysis revealed a sign

100 But, based on histochemistry and Western blot analysis, Pd is found al

101 specimens were evaluated by immunoperoxidase histochemistry and Western blot analysis.

102 of which was studied by means of PCR, X-gal histochemistry, and beta-galactosidase immunocytochemist

103 ccinate dehydrogenase and cytochrome oxidase histochemistry, and electron microscopy correlated with

104 additional methods in rats (immunoblotting, histochemistry, and electron microscopy).

105 Liver function, morphology, histochemistry, and fibrotic parameters were examined.

106 oxidase (CO) and acetylcholinesterase (AChE) histochemistry, and immunocytochemistry for calbindin.

107 rse transcription-PCR, in situ hybridization histochemistry, and immunocytochemistry.

108 ells, as shown by single-cell RT-PCR, enzyme histochemistry, and immunofluorescence.

109 Our proteomics, histochemistry, and immunohistochemistry data also revea

110 real-time polymerase chain reaction, (immuno)histochemistry, and light and electron microscopy.

111 had mild exercise intolerance, normal muscle histochemistry, and normal respiratory chain activity in

112 d using immunofluorescence or lectin binding histochemistry, and percentages of single and double-lab

113 Developmental analysis by histology, histochemistry, and SEM revealed a significant reduction

114 Histopathology and histochemistry are providing a better understanding of t

115 nsitometry, microphotometry, and video-based histochemistry, are effective in quantitative and detail

116 amide adenine diphosphate (NADPH)-diaphorase histochemistry as an indicator of nitric oxide synthase

117 ntrols using quantitative cytochrome oxidase histochemistry as an intracellular measure of oxidative

118 tivity using quantitative cytochrome oxidase histochemistry as in our previous study using congenital

119 Lectin histochemistry as well as tissue binding patterns of TCo

120 re identified by acetylcholinesterase (AChE) histochemistry, as well as immunostaining for calbindin-

121 The senescence-associated beta-galactosidase histochemistry assay, adapted for use in the primate pos

122 sured retrospectively using a novel affinity histochemistry assay.

123 r, and were alkaline phosphatase positive by histochemistry but showed no other evidence of bone line

124 sera corroborated the results of the NADPH-d histochemistry by staining the same two cells in the cer

125 ional roles in the retina using fluorescence histochemistry, confocal microscopy, immuno-electron mic

126 romatography (HPLC), cytochrome oxidase (CO) histochemistry, cresyl violet, or demonstration of TCAs

127 Dihydroxyphenylalanine (DOPA) histochemistry demonstrated an increase in melanization

128 tibialis anterior (TA) muscle morphology and histochemistry demonstrated an increase in the cross sec

129 beta-Galactosidase histochemistry demonstrated gene expression throughout t

130 Affinity histochemistry demonstrated that hyaluronan accumulates

131 Electron microscopic histochemistry disclosed a complex array of corneal PGs

132 Brain histochemistry disclosed abnormal aggregates of ferritin

133 Histochemistry documented successful implantation of myo

134 s for these mutations was analyzed by enzyme histochemistry during embryogenesis, postnatal developme

135 of H5N1 virus receptors, by virus and lectin histochemistry, during highly pathogenic avian influenza

136 pathological tests with regard to studies of histochemistry examination.

137 lateral symmetry of cytochrome oxidase (COX) histochemistry following unilateral eye enucleation was

138 hicle for the adjuvant by using fluorescence histochemistry for catecholamines, with morphometric ana

139 e healing pattern was assessed by histology, histochemistry for collagen deposition, and immunohistoc

140 ere analyzed utilizing in situ hybridization histochemistry for enkephalin (ENK) mRNA in the ventral

141 on microscopy, we performed immunoperoxidase histochemistry for ICAM-5 in mouse visual cortex at post

142 transcriptase (RT)-PCR, immunostaining, and histochemistry for key proteins underlying ATP secretion

143 ial cells) and GFAP (astrocytes), and enzyme histochemistry for menadione-dependent a-glycerophosphat

144 such injury, we performed immunofluorescence histochemistry for metabotropic glutamate receptor 1alph

145 Myeloperoxidase histochemistry for neutrophils was performed in jejunal

146 Myeloperoxidase histochemistry for neutrophils was performed in jejunal

147 studied with myosin immunohistochemistry and histochemistry for the mitochondrial enzyme, nicotinamid

148 ation was confirmed by in situ hybridization histochemistry for two strongly down-regulated genes, my

149 By quantitative histochemistry, hepatic fibrosis in treated animals was

150 Liver tissues were analyzed by histochemistry, hydroxyproline determination, reverse-tr

151 In situ hybridization histochemistry identified strong mRNA labeling for BMP-4

152 We analyzed structural changes in muscle by histochemistry, immunocytochemistry, and electron micros

153 enucleated, and retinal sections studied by histochemistry, immunofluorescence labeling, and confoca

154 Histochemistry, immunohistochemistry, and electron micro

155 Using histochemistry, immunohistochemistry, and image analysis

156 uction profiles were evaluated by histology, histochemistry, immunohistochemistry, and quantitative a

157 MS cases together with complex IV/complex II histochemistry, immunohistochemistry, laser dissection m

158 MS cases together with complex IV/complex II histochemistry, immunohistochemistry, laser dissection m

159 hology were evaluated using biochemistry and histochemistry in 62 subjects with a premortem diagnosis

160 f NADPHd activity was demonstrated by NADPHd histochemistry in both central and peripheral nervous sy

161 ha regulation, we used in situ hybridization histochemistry in C57Bl/6J mice and Sprague-Dawley rats.

162 Dual-labeling histochemistry in caudoputamen demonstrated that densely

163 We used cytochrome oxidase (CO) histochemistry in conjunction with other histological me

164 s by beta-galactosidase expression and X-gal histochemistry in gastrointestinal epithelia.

165 ed by semiquantitative in situ hybridization histochemistry in melanized neurons of human substantia

166 ession was examined by in situ hybridization histochemistry in rats injected intraperitoneally (i.p.)

167 studied by using NADPH-diaphorase (NADPH-d) histochemistry in the CNS and peripheral organs.

168 disease were processed using cholinesterase histochemistry in the presence or absence of rivastigmin

169 cells, as reflected by GAD67 mRNA expression histochemistry, in the rat substantia nigra pars reticul

170 Using histochemistry, in vivo Ca(2+) imaging, and behavioral a

171 leukocytic phytohemagglutinin (LPHA) lectin-histochemistry] in 119 archival specimens of human melan

172 rther characterized by in situ hybridization histochemistry, including pituitary adenylate cyclase ac

173 Hybridization histochemistry indicated robust KiSS-1 and GPR54 mRNA ex

174 ne serum and characterized using morphology, histochemistry, indirect immunofluorescence microscopy,

175 cultures together with in situ hybridization histochemistry (ISHH) in sections of adult rat tissue.

176 the present study used in situ hybridization histochemistry (ISHH) to map the temporal and sexually d

177 spinal cord, by using in situ hybridization histochemistry (ISHH) with a novel cRNA probe.

178 by using radioisotopic in situ hybridization histochemistry (ISHH) with a novel sensitive cRNA probe.

179 he CNS, as assessed by in situ hybridization histochemistry (ISHH), has been described previously in

180 In rat and monkey amygdala, WFA histochemistry labeled perineuronal nets, but not glial

181 ption polymerase chain reaction, immunoblot, histochemistry, laser-capture microscopy, and terminal d

182 Subcellular studies and histochemistry localized UGD protein to the perinuclear

183 (R(2)>0.80, p<1E-20) between MRI and immuno-histochemistry measurements with 95% lower bound of the

184 tagged" and imaged using conventional immuno-histochemistry methods.

185 Using NADPH-diaphorase histochemistry, neuronal nitric oxide synthase (nNOS) an

186 g overall normal tissue morphology and brain histochemistry, normal blood and urine chemistries, norm

187 vity were validated by in situ hybridization histochemistry of COX-2 mRNA and Western blot analysis.

188 and D(2) receptors and in situ hybridisation histochemistry of D(1) and D(2) mRNA were performed.

189 In this study, neo-Timm's histochemistry of MFs and immunocytochemistry of GluR1 w

190 Immune histochemistry of murine and human poststroke autoptic b

191 Horseradish peroxidase (HRP) histochemistry of OB following a 10-day recovery period

192 es used RT-PCR and beta-galactosidase (LacZ) histochemistry of retinas from transgenic mice heterozyg

193 Enzyme histochemistry of the adult rat brain for ectonucleotida

194 Histochemistry of the graft's muscularis externa showed

195 ol or injured liver and also by quantitative histochemistry of tissue sections.

196 Histochemistry of tissues from CNC patients is indicativ

197 ther, the integration of OR-PAM with (immuno)histochemistry offers a simple and versatile technique w

198 antitative confocal analysis of isolectin B4 histochemistry on days 7 and 14.

199 ing microtubule-associated protein-2 (MAP-2) histochemistry on P28 while the animals were hypoxic (n=

200 Their axons were visualized by X-gal histochemistry or anti-beta-galactosidase immunofluoresc

201 In normal rats, synapses labeled by Timm histochemistry or dynorphin immunohistochemistry were ra

202 ned for CTb immunocytochemistry or for CytOx histochemistry or for Nissl.

203 e dinucleotide phosphate diaphorase (NADPHd) histochemistry or immunocytochemistry using an antibody

204 in production at a cellular level by in situ histochemistry or immunocytochemistry.

205 NADPH-diaphorase (NADPHd) histochemistry or NOS-immunostaining was combined with s

206 status of mutant mice, indicated by a lectin histochemistry pattern similar to that of wild-type mice

207 odologies: cytoarchitecture (cresyl violet), histochemistry (peanut agglutinin), immunocytochemistry

208 evelopment by means of in situ hybridization histochemistry, quantitative RT-PCR, and immunocytochemi

209 e specific lectin, and subsequent GNA lectin histochemistry refined the localization of N-glyans cont

210 mitochondria using immunohistochemistry and histochemistry, respectively, in chronic active and inac

211 In human amygdala, WFA lectin histochemistry resulted in labeling of perineuronal nets

212 Traditional microscopies in combination with histochemistry reveal glycogen accumulation within glia

213 In situ hybridization histochemistry revealed expression of Kir6.1/SUR2B mRNAs

214 In the Melibe brain, NADPH-d histochemistry revealed only a single pair of bilaterall

215 nses were improved (P<0.05) with Tempol, and histochemistry revealed oxidative stress in KW animals,

216 Quantitative in situ hybridization histochemistry revealed that baclofen (2.5 mg/kg, i.p.)

217 Histochemistry revealed that LNA-92a increased capillary

218 Immuno histochemistry revealed that the inclusions were immunor

219 Double-labeling immunofluorescence histochemistry revealed that the nNOS-like immunoreactiv

220 cks of tissue used for in situ hybridization histochemistry, revealed a marked reduction in the relat

221 Furthermore, histochemistry reveals markedly increased NEDD4-1 immuno

222 d the absence of NTPDase2, and ATPase enzyme histochemistry reveals no reaction product in taste buds

223 We used quantitative histochemistry, reverse transcription-PCR (RT-PCR), and

224 es followed by their localization with DPPIV histochemistry showed 3- to 5-fold increases in the numb

225 weeks after infection, in situ hybridization histochemistry showed a pattern of chronic overexpressio

226 Muscle histochemistry showed mitochondrial proliferation, and b

227 Immunocytochemistry/histochemistry showed that a TN-C-rich ECM surrounds Prx

228 Protein histochemistry showed that the tissue binding specificit

229 erminal sequences, and in situ hybridization histochemistry showed that these glypican-1 ligands are

230 In situ hybridization histochemistry shows that melanopsin expression is restr

231 In situ hybridization histochemistry shows that NAALADase-related mRNAs have a

232 We examined normal vessels and plaques by histochemistry, Southern blotting, and fluorescence in s

233 Using enzymatic histochemistry staining, we also demonstrated functional

234 Histochemistry study and real-time PCR further confirmed

235 s from typical confounding factors common to histochemistry, such as variation in reagent penetration

236 the site of transection combined with TUNEL histochemistry suggested that neuronal death, including

237 s early lineage progenitors, undetectable by histochemistry, that leave the bone marrow to enter the

238 tional protein immunolocalization and lignin histochemistry, these results suggest that the distinct

239 Here we used cytochrome oxidase (CO) histochemistry to demonstrate that GAP-43 heterozygous (

240 resent report, we used in situ hybridization histochemistry to demonstrate that the 5-HT(3B) subunit

241 t's visual cortex using alkaline phosphatase histochemistry to demonstrate the capillary endothelial

242 In this study, we used in situ hybridization histochemistry to determine the change in the levels of

243 used single and double in situ hybridization histochemistry to examine the distribution and coexisten

244 as analyzed using Western blotting and Golgi histochemistry to examine the hypothesized outcomes.

245 We used in situ hybridization histochemistry to examine the patterns of expression of

246 S-1 cortex by using postembedding immunogold histochemistry to examine the subcellular distribution o

247 ne dinucleotide phosphate (NADPH)-diaphorase histochemistry to identify populations of neurons contai

248 dinucleotide phosphate diaphorase (NADPH-d) histochemistry to identify the source of nitrergic inner

249 ne dinucleotide phosphate (NADPH)-diaphorase histochemistry to investigate nitric oxide as a possible

250 , we used double label in situ hybridization histochemistry to investigate the potential direct actio

251 Using serotonin transporter (5HT-T) histochemistry to label thalamocortical afferents (TCAs)

252 Here, we use X-gal histochemistry to map CRE-mediated gene transcription in

253 We used histochemistry to map differentially expressed proteins

254 boprobes, we performed in situ hybridization histochemistry to map the distribution of orexin recepto

255 henotype, we have used in situ hybridization histochemistry to map the IKAP mRNA in sections of whole

256 By using Fluoro-jade histochemistry to mark neurodegeneration and dual immuno

257 ogical techniques with in situ hybridization histochemistry to produce both 2D and 3D images and to v

258 odies) and fluorescent in situ hybridization histochemistry to search for Y chromosome-positive cells

259 In this study we used in situ hybridization histochemistry to show that TCF7L2 has a unique expressi

260 tion-PCR (RT-PCR), and in situ hybridization histochemistry to study cholinesterase expression during

261 We used Wisteria Floribunda agglutinin histochemistry to visualize PNNs to investigate whether

262 in conjunction with cytochrome oxidase (CO) histochemistry, to investigate the distribution of thala

263 gment was performed after beta-galactosidase histochemistry using 0.1% to 1% potassium permanganate i

264 5-HTT in mouse brain, in situ hybridization histochemistry using 35S-labeled riboprobes was performe

265 The complementary technique of Southwestern histochemistry using a labeled Smad-binding element demo

266 In situ hybridization histochemistry using an antisense probe to this novel re

267 culture was studied by in situ hybridization histochemistry using an intron-specific VP heteronuclear

268 Immunofluorescence histochemistry using antibodies to three domains of the

269 s ('shape modules') were located by electron histochemistry using Cupromeronic blue methodology.

270 alamus and amygdala by in situ hybridization histochemistry using monkey-specific cRNA probes.

271 pecimens were assessed by immunofluorescence histochemistry using polyclonal antibodies specific for

272 lla and rat vestibular brainstem; diaphorase histochemistry was done in the chinchilla periphery.

273 Immunofluorescent histochemistry was employed to detect changes in peptidi

274 In situ hybridization histochemistry was performed for such "claustrum-enriche

275 dase (COX) and succinate dehydrogenase (SDH) histochemistry was performed on 46 EOM samples to determ

276 Double-label fluorescent histochemistry was used for IT and AVT (by using antibod

277 ative and double-label in situ hybridization histochemistry was used selectively to confirm a number

278 First, isotopic in situ hybridization histochemistry was used to examine mRNA expression of pa

279 r immunohistochemistry or cytochrome oxydase histochemistry was used to reveal thalamic afferent patt

280 tion, immunoblotting, and immunofluorescence histochemistry, we demonstrate the following.

281 Using histochemistry, we demonstrated accumulation of mucus an

282 Using enzymatic histochemistry, we detected ecto-AMPase activity in dent

283 Finally, using Timm histochemistry, we detected progressive sprouting of mos

284 Using Fos histochemistry, we found changes in the activation of se

285 By using double in situ hybridization histochemistry, we found co-expression of the functional

286 In conclusion, using in situ hybridization histochemistry, we have shown that mRNA for both the exc

287 in situ hybridization and immunofluorescence histochemistry, we show that hippocampal D4R mRNA and pr

288 l c-fos mRNA probe for in situ hybridization histochemistry, we systematically analyzed and identifie

289 dinucleotide phosphate diaphorase (NADPH-d) histochemistry were used to explore the existence of sex

290 Culture, 16S ribosomal DNA sequencing, and histochemistry were used to guide subsequent FISH.

291 n Pde6b(H620Q) homozygotes was documented by histochemistry, whereas PDE6beta expression and activity

292 g Holtzman rat pups using cytochrome oxidase histochemistry, which reflects long-term changes in brai

293 s against NOS and NADPH-diaphorase (NADPH-d) histochemistry, which, with the exception of the primary

294 cts were processed for in situ hybridization histochemistry with (35)S-oligonucleotide probes for GAT

295 ion were determined by in situ hybridization histochemistry with a digoxigenin (DIG)-labeled antisens

296 We have shown by Western blotting and immuno-histochemistry with a polyclonal antibody to a specific

297 uleus were measured by in situ hybridization histochemistry with autoradiographic analysis.

298 Using double labeling fluorescent histochemistry with confocal microscopy we found TRalpha

299 In situ hybridization histochemistry with digoxigenin-labeled oligonucleotide

300 In situ hybridization histochemistry with SPACRCAN riboprobes indicates that h

301 Conventional histochemistry yields rich morphological data from tissu