ライフサイエンスコーパス: histocompatability

1 roglia and astrocytes, and included class II histocompatability antigens, the pro-inflammatory cytoki

2 strains but were ineffective across a major histocompatability barrier.

3 In this allograft across major histocompatability barriers, the transplanted heart unde

4 pression during the differentiation of major histocompatability class 1 (MHC I)-restricted thymocytes

5 the invariant chain (li, CD74) of the major histocompatability class II complex (MHCII) undergoes in

6 The major histocompatability class II heterodimer (class II) is ex

7 De novo expression of major histocompatability class II molecules was increased in b

8 Major histocompatability class II proteins are transmembrane a

9 to induce humoral immune tolerance to major histocompatability complex (MHC) antigens.

10 sion-induced alloimmunization to donor major histocompatability complex (MHC) antigens.

11 rived from these exogenous antigens on major histocompatability complex (MHC) class I molecules and i

12 controlled by inhibitory receptors for major histocompatability complex (MHC) class I, including the

13 We previously demonstrated major histocompatability complex (MHC) class I-deficient (beta

14 mpetence requires engagement of a self-major histocompatability complex (MHC) class I-specific inhibi

15 riptional activation of genes encoding major histocompatability complex (MHC) class II molecules and

16 ng cognate T cell-B cell interactions, major histocompatability complex (MHC) class II molecules tran

17 smaller peptides, and present them on major histocompatability complex (MHC) class II molecules with

18 naive CD8 T cells require self-peptide-major histocompatability complex (MHC) complexes for maintenan

19 bodies to CD11c and antigen-presenting major histocompatability complex (MHC) II products.

20 The Major Histocompatability Complex (MHC) is one of the best know

21 To address this problem, the peptide-major histocompatability complex (MHC) ligand for a given popu

22 PSORS1 is located within the major histocompatability complex (MHC) locus on 6p21.3.

23 natural processing and presentation of major histocompatability complex (MHC) molecules as peptide by

24 recognize antigenic peptides bound to major histocompatability complex (MHC) molecules through commo

25 ens in association with Class I and II major histocompatability complex (MHC) molecules.

26 mocytes even in the absence of class I major histocompatability complex (MHC) proteins, ligands that

27 Genetic analysis of a mouse model of major histocompatability complex (MHC)-associated autoimmune t

28 thally irradiated mice receiving fully major histocompatability complex (MHC)-mismatched bone marrow

29 t disease (GVHD) after the infusion of major histocompatability complex (MHC)-mismatched donor bone m

30 esponsiveness unless modulated by self-major histocompatability complex (MHC)-specific inhibitory rec

31 f the invariant chain (Ii) of class II major histocompatability complex (MHC-II) and aequorin was loc

32 enetic effects including those for the major histocompatability complex (MHC; particularly the HLA-DR

33 3, alpha-sarcomeric actin, CD3, CD11c, major histocompatability complex class I, hematoxylin-eosin).

34 nnexin 43, alpha-sarcomeric actin, and major histocompatability complex class I.

35 antigen presentation in the context of major histocompatability complex class I.

36 atitis through induction of hepatocyte major histocompatability complex class I.

37 cells; P<0.0001) and cells expressing major histocompatability complex class II (9 +/- 3 versus 40 +

38 Major histocompatability complex class II (MHCII) molecules ar

39 n of CD8alpha and IL-12 p40 as well as major histocompatability complex class II and other costimulat

40 This block was independent of major histocompatability complex class II expression and was a

41 t facilitates folding and transport of major histocompatability complex class II molecules; here it i

42 These DCs were immature (major histocompatability complex class II(low)) and expressed

43 presenting cells, measured as class II major histocompatability complex expressing cells, in lymph no

44 8 T cell responses in a cell contact-, major histocompatability complex I-, and influenza peptide-dep

45 tigen presentation, including class II major histocompatability complex molecules (HLA-DP, -DQ, and -

46 izes cells bearing Ld and Kbm3 class I major histocompatability complex molecules.

47 rately characterized the hypervariable major histocompatability complex region as well as demonstrati

48 Major histocompatability complex type II proteins (MHC II) are

49 ate considerably less than that of the major histocompatability complex, discovery of recent risk var

50 gainst CML cells but low reactivity to major histocompatability complex-matched normal bone marrow ce

51 cells in a non-antigen-dependent, non-major histocompatability complex-restricted fashion.

52 d as recipients of syngeneic, multiple minor histocompatability (H)-disparate, or major histocompatib

53 or and recipient differ at two or more major histocompatability loci.

54 Here, we show that Id3 is required for major histocompatability (MHC) class I- and class II-restricte