ライフサイエンスコーパス: histomorphometry

1 the width of the wound by computer-assisted histomorphometry.

2 in the wound boundaries by computer-assisted histomorphometry.

3 was not significantly different from that at histomorphometry.

4 the right tibiae were removed on day 28 for histomorphometry.

5 IH volumes correlated strongly with ex vivo histomorphometry.

6 e assessed with biochemical markers and bone histomorphometry.

7 outgrowth after 4 weeks was quantified using histomorphometry.

8 als were given fluorochrome bone markers for histomorphometry.

9 using quantitative coronary angiography and histomorphometry.

10 ound, invasive hemodynamic measurements, and histomorphometry.

11 calcified histology and cellular and dynamic histomorphometry.

12 aluated mineral and bone disorders with bone histomorphometry.

13 ation parameters were assessed using dynamic histomorphometry.

14 sessed by micro-computed tomography (CT) and histomorphometry.

15 emoved and atrial thrombi were quantified by histomorphometry.

16 joints as demonstrated by histology and bone histomorphometry.

17 lar inflammatory infiltrate and quantitative histomorphometry.

18 o the cell-free zone using computer assisted histomorphometry.

19 was scored using a qualitative scale and by histomorphometry.

20 , and 9 was analyzed using computer-assisted histomorphometry.

21 d 12 mo as measured by bone densitometry and histomorphometry.

22 ures by micro computed tomography (muCT) and histomorphometry.

23 , bone mineral density measurement, and bone histomorphometry.

24 Bone structure in the femur was assessed by histomorphometry.

25 4) was accomplished using computer-assisted histomorphometry.

26 ), were accomplished using computer-assisted histomorphometry.

27 in the optic nerve were compared by detailed histomorphometry.

28 biochemical markers of bone resorption, and histomorphometry.

29 true infarction size as measured by means of histomorphometry (36% +/- 3, r = 0.90).

30 ced after TbetaRI-I treatment as detected by histomorphometry analysis compared with the placebo cont

31 ecreased osteoblast number, as quantified by histomorphometry and 3D-microtomography.

32 Furthermore, histomorphometry and biochemical assays demonstrated a s

33 ects hMMC biology using quantitative (immuno)histomorphometry and electron microscopy.

34 Herein, we combined histomorphometry and IHC on human iliac biopsy specimens

35 Both histomorphometry and IVUS showed that IH was concentric

36 post-surgery and analyzed through histology, histomorphometry and micro-computed tomography (muCT).

37 Histomorphometry and mineralization density analysis of

38 stingly, osteoblast activity, as measured by histomorphometry and osteocalcin expression, is strongly

39 ems pathology analysis program that includes histomorphometry and quantitative multiplex biomarker as

40 Quantitation was achieved by histomorphometry and real-time RT-PCR for human osteocal

41 Clinical outcomes were determined, and renal histomorphometry and sequencing of Mendelian nephrotic s

42 e evaluated using micro-computed tomography, histomorphometry and targeted gene profiling.

43 ic and SD-OCT volumes (8 microm, range 4-19, histomorphometry, and 10 microm, range 4-26, SD-OCT) and

44 examined by a combination of micro-CT, bone histomorphometry, and biomechanical testing and compared

45 al quantitative computed tomography, by bone histomorphometry, and by measurements of bone cell apopt

46 ensity using micro-computed tomography, bone histomorphometry, and characteristics of primary bone ma

47 s were examined blindly by light microscopy, histomorphometry, and color computer image analysis.

48 osteonectin in bone, we used contact x-ray, histomorphometry, and Northern blot analysis to characte

49 cans were correlated with plain radiographs, histomorphometry, and soft-tissue measurements.

50 yzed using dual energy x-ray absorptiometry, histomorphometry, and vertebral compression testing.

51 d fluorescent labeling with tetracycline and histomorphometry are described.

52 Digital radiology and histomorphometry are described.

53 s quantified by angiography, ultrasound, and histomorphometry at 30 days.

54 d perimeter, there was no difference in bone histomorphometry between the two groups.

55 Dynamic histomorphometry confirmed a significant decrease in tra

56 Histomorphometry confirmed increased cortical bone thick

57 By histomorphometry, cross-sectional area narrowing by plaq

58 Dynamic histomorphometry demonstrated an associated decrease in

59 Quantitative (immuno)histomorphometry demonstrated that Tfam(EKO) mice showed

60 In contrast, quantitative islet histomorphometry demonstrates that the total islet numbe

61 stration were in good agreement with ex vivo histomorphometry (Elastica van Gieson stain) and gadolin

62 -computed tomography, and dynamic and static histomorphometry end points were assessed.

63 rillhole model using micro-CT, histology and histomorphometry evaluation.

64 Histomorphometry for collagen and alpha-smooth muscle ac

65 ing of periodontal bone level (PBL) loss and histomorphometry for inflammatory cell infiltration and

66 ed, which can include descriptive histology, histomorphometry, immunostaining, 3D bone imaging, elect

67 BM composition was assessed by histomorphometry, immunostaining, and flow cytometry.

68 MC numbers and density were determined by histomorphometry in healthy and arthritic synovia.

69 Bone densitometry and histomorphometry in lymphoma-bearing mice revealed signi

70 hod is likely to eventually replace invasive histomorphometry in that it obviates the need to sacrifi

71 In addition, bone histomorphometry in young adults shows longer periods of

72 We evaluate the bone histomorphometry, in vitro proliferation, and alkaline p

73 ough sex-specific differences were observed, histomorphometry measurements revealed that both bone re

74 In addition to static histomorphometry, micro-computed tomography (muCT) and r

75 luated; alveolar bone loss was determined by histomorphometry, morphometry, and microcomputed tomogra

76 bone loss in periodontitis models, including histomorphometry, morphometry, and radiography.

77 2(-/-) and Fgf2(+/+) mice were determined by histomorphometry, nanoindentation, and quantitative reve

78 sessed by bone morphometric measurements and histomorphometry of block sections.

79 old stimuli and correlated with quantitative histomorphometry of myocardial architecture and connexin

80 The histology and histomorphometry of of the bone in cAF and AVF was signi

81 Histomorphometry of plaque cross-sectional area in the p

82 Histomorphometry of plaque cross-sectional area in the p

83 Histomorphometry of the aorta demonstrated larger athero

84 We performed immunohistochemistry and histomorphometry of the BM to assess microvascular densi

85 ing and osteoclast surface, as determined by histomorphometry of the femur; increased urinary deoxypy

86 Histomorphometry of the injured arteries showed striking

87 mice based on gross tissue observations and histomorphometry of tissue sections.

88 4), or 6 months (n=24) and were analyzed for histomorphometry or scanning electron microscopy.

89 When measured using histomorphometry, peritumoral BIO administration improve

90 crest, was examined by stereomicroscopy and histomorphometry, respectively.

91 elation and agreement between MR imaging and histomorphometry, respectively.

92 Similarly, histomorphometry revealed a greater bone formation rate

93 Cancellous bone histomorphometry revealed an increased number of osteocl

94 Histomorphometry revealed atRA-mediated reductions in ne

95 Histomorphometry revealed delayed dermal cell migration

96 Histomorphometry revealed increased bone formation in Ms

97 Histomorphometry revealed increased neovascularization i

98 Histomorphometry revealed limited evidence of osteogenes

99 Dynamic histomorphometry revealed reduced bone formation rate (-

100 Histomorphometry revealed that Bzb significantly increas

101 Histomorphometry revealed that half-dose gene of Fgf-9 m

102 Cancellous bone histomorphometry revealed that the increased bone mass w

103 ostsurgery were measured using densitometry, histomorphometry, scanning electron microscopy (SEM), an

104 Histomorphometry, scanning electron microscopy, and micr

105 muCT and histomorphometry showed consistently reduced trabecular

106 Histomorphometry showed expansile remodeling of Absorb-i

107 In addition, quantitative histomorphometry showed increased area fractions of coll

108 st number, and inflammation were assessed by histomorphometry, tartrate-resistant acid phosphatase hi

109 and neointima formation by computer-assisted histomorphometry techniques.

110 on cancellous bone structure from iliac bone histomorphometry that led to the demonstration that arch

111 roperties of the specimens, and we used bone histomorphometry to assess parameters of bone microstruc

112 n, proximal aortic lesion size quantified by histomorphometry was 5-fold-reduced in chow-fed ApoE+/-/

113 n, proximal aortic lesion size quantified by histomorphometry was 9-fold greater in chow-fed mice ino

114 Histomorphometry was also used to evaluate the area of n

115 Quantitative histomorphometry was performed at day 14, proliferating

116 d 7 PVOD) or surgery (10 PVOD), quantitative histomorphometry was performed in all analyzable arterie

117 Histomorphometry was performed on coronary stents from 1

118 Histomorphometry was performed using digitized photograp

119 The abnormal skeletal histomorphometry was reversed by BMP-7 therapy to normal

120 as assessed by biochemical markers and bone histomorphometry, was markedly decreased at both ages.

121 Following culture, cell viability and tissue histomorphometry were assessed with quantification of ma

122 ed calcium, osteoclast recruitment, and bone histomorphometry were evaluated.

123 Angiography and histomorphometry were performed at 6 months.

124 a from qualitative analysis and computerized histomorphometry were statistically processed at a signi